P. Morris

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p53 Suppresses the Activation of the Bcl-2 Promoter by the Brn-3a POU Family Transcription Factor*

D. Latchman, C. Midgley, V. Budhram-Mahadeo, 1999, The Journal of Biological Chemistry.

Brn-3b enhances the pro-apoptotic effects of p53 but not its induction of cell cycle arrest by cooperating in trans-activation of bax expression

D. Latchman, V. Budhram-Mahadeo, P. Morris, 2006, Nucleic acids research.

Brn-3 b enhances the pro-apoptotic effects of p 53 but not its induction of cell cycle arrest by cooperating in transactivation of bax expression

D. Latchman, V. Budhram-Mahadeo, P. Morris, 2006 .

Functional role of position 22 in the homeodomain of Brn‐3 transcription factors

D. Latchman, P. Morris, R. Palmer, 1998, Neuroreport.

Inhibition of DNA binding of the NF-Y transcription factor by the pyrrolobenzodiazepine-polyamide conjugate GWL-78

R. Mantovani, P. Morris, D. Hochhauser, 2008, Molecular Cancer Therapeutics.

Modulation of topoisomerase IIα expression by a DNA sequence-specific polyamide

R. Mantovani, P. Morris, D. Hochhauser, 2007, Molecular Cancer Therapeutics.

The Brn-3a transcription factor inhibits the pro-apoptotic effect of p53 and enhances cell cycle arrest by differentially regulating the activity of the p53 target genes encoding Bax and p21CIP1/Waf1

D. Latchman, P. Morris, Vishwanie Budram-Mahadeo, 2002, Oncogene.

The HPV-activating cellular transcription factor Brn-3a is overexpressed in CIN3 cervical lesions.

A. Singer, D. Latchman, P. Morris, 1998, The Journal of clinical investigation.

Regulation of Neurite Outgrowth and SNAP-25 Gene Expression by the Brn-3a Transcription Factor (*)

T. Möröy, D. Latchman, P. Morris, 1995, The Journal of Biological Chemistry.

Differential regulation of genes encoding synaptic proteins by members of the Brn-3 subfamily of POU transcription factors.

D. Latchman, P. Morris, N. Lakin, 1996, Brain research. Molecular brain research.

A Single Amino Acid Change Converts an Inhibitory Transcription Factor into an Activator (*)

D. Latchman, P. Morris, S. Dawson, 1996, The Journal of Biological Chemistry.

The opposite and antagonistic effects of the closely related POU family transcription factors Brn-3a and Brn-3b on the activity of a target promoter are dependent on differences in the POU domain

D. Latchman, P. Morris, K. Lillycrop, 1994, Molecular and cellular biology.

A single residue within the homeodomain of the Brn‐3 POU family transcription factors determines whether they activate or repress the SNAP‐25 promoter

D. Latchman, P. Morris, Michael C. Wilson, 1997, Neuroreport.

Cytokines and Peripheral Tolerance to Alloantigen

M. Dallman, P. Morris, M. Wood, 1993, Immunological reviews.

Activation of the α-Internexin Promoter by the Brn-3a Transcription Factor Is Dependent on the N-terminal Region of the Protein (*)

T. Möröy, D. Latchman, R. Liem, 1995, The Journal of Biological Chemistry.

The Different Activities of the Two Activation Domains of the Brn-3a Transcription Factor Are Dependent on the Context of the Binding Site (*)

D. Latchman, V. Budhram-Mahadeo, P. Morris, 1996, The Journal of Biological Chemistry.

D S Latchman, D. Latchman, P. Sassone-Corsi, 2001, The international journal of biochemistry & cell biology.

Brn-3a transcription factor blocks p53-mediated activation of proapoptotic target genes Noxa and Bax in vitro and in vivo to determine cell fate.

D. Latchman, V. Budhram-Mahadeo, P. Morris, 2005, The Journal of Biological Chemistry.

Brn-3a transcription factor blocks p53-mediated activation of proapoptotic target genes Noxa and Bax in vitro and in vivo to determine cell fate.

D. Latchman, V. Budhram-Mahadeo, P. Morris, 2015, The Journal of Biological Chemistry.

Coordinate Induction of the Three Neurofilament Genes by the Brn-3a Transcription Factor*

M. Schwartz, D. Latchman, P. Morris, 1997, The Journal of Biological Chemistry.

The DNA target site for the Brn-3 POU family transcription factors can confer responsiveness to cyclic AMP and removal of serum in neuronal cells.

D. Latchman, V. Budhram-Mahadeo, P. Morris, 1994, Nucleic acids research.

Brn-3a Transcription Factor Blocks p53-mediated Activation of Proapoptotic Target Genes Noxa and Bax in Vitro and in Vivo to Determine Cell Fate*

D. Latchman, V. Budhram-Mahadeo, P. Morris, 2005, Journal of Biological Chemistry.

The octamer binding site in the HPV16 regulatory region produces opposite effects on gene expression in cervical and non-cervical cells.

D. Latchman, P. Morris, C. Ring, 1993, Nucleic acids research.

The Brn-3a POU family transcription factor stimulates p53 gene expression in human and mouse tumour cells

D. Latchman, V. Budhram-Mahadeo, P. Morris, 2002, Neuroscience Letters.

11-Hydroxy-β-steroid dehydrogenase gene expression in canine adipose tissue and adipocytes: stimulation by lipopolysaccharide and tumor necrosis factor α.

P. Morris, P. Trayhurn, A. German, 2011, Domestic animal endocrinology.

The HPV activating transcription factor Brn-3a is over expressed in CIN3 lesions

A. Singer, D. Latchman, P. Morris, 1998 .

The Brn‐3c transcription factor contains a neuronal‐specific activation domain

D. Latchman, P. Morris, M. D. Smith, 1998, Neuroreport.

Immediate upstream promoter regions required for neurospecific expression of SNAP-25

D. Latchman, P. Morris, Michael C. Wilson, 1995, Journal of Molecular Neuroscience.

Transactivation of the human papilloma virus 16 octamer motif by the octamer binding protein Oct-2 requires both the N and C terminal activation domains.

P. Morris, K. Lillycrop, D. Latchman, 1993, Nucleic Acids Research.