E. Ensor

发表

Brn-3b enhances the pro-apoptotic effects of p53 but not its induction of cell cycle arrest by cooperating in trans-activation of bax expression

D. Latchman, V. Budhram-Mahadeo, P. Morris, 2006, Nucleic acids research.

Brn-3 b enhances the pro-apoptotic effects of p 53 but not its induction of cell cycle arrest by cooperating in transactivation of bax expression

D. Latchman, V. Budhram-Mahadeo, P. Morris, 2006 .

A Minimal CGRP Gene Promoter is Inducible by Nerve Growth Factor in Adult Rat Dorsal Root Ganglion Neurons But Not in PC12 Phaeochromocytoma Cells

D. Latchman, E. Ensor, J. Winter, 1995, The European journal of neuroscience.

Induction of the Oct-2 transcription factor in primary sensory neurons during inflammation is nerve growth factor-dependent

D. Latchman, C. Woolf, E. Ensor, 1996, Neuroscience Letters.

Sensory neurons from mice lacking the Brn-3b POU family transcription factor are resistant to death-inducing stimuli both in vitro and in vivo.

D. Latchman, E. Ensor, Martin D Payne Smith, 2003, Brain research. Molecular brain research.

Brn-3a Activates the Expression of Bcl-xL and Promotes Neuronal Survival in Vivo as Well as in Vitro

D. Latchman, Martin D. Smith, E. Ensor, 2001, Molecular and Cellular Neuroscience.

Bcl-2 Transcription from the Proximal P2 Promoter Is Activated in Neuronal Cells by the Brn-3a POU Family Transcription Factor*

D. Latchman, L. Boxer, E. Ensor, 1998, The Journal of Biological Chemistry.

Doppel expression is regulated by the Brn-3a and Brn-3b transcription factors

D. Latchman, D. R. Brown, E. Ensor, 2004, Neuroreport.

Activation of the gene encoding decay accelerating factor following nerve growth factor treatment of sensory neurons is mediated by promoter sequences within 206 bases of the transcriptional start site

D. Latchman, E. Ensor, D. Lublin, 1996, Journal of neuroscience research.

Cold-sensitive, menthol-insensitive neurons in the murine sympathetic nervous system

M. Koltzenburg, E. Ensor, Martin Smith, 2004, Neuroreport.

Regulation of NGFI-A (Egr-1) gene expression by the POU domain transcription factor Brn-3a.

D. Latchman, E. Ensor, M. Smith, 1999, Brain research. Molecular brain research.

Distinct domains of Brn-3a regulate apoptosis and neurite outgrowth in vivo

D. Latchman, E. Ensor, D. Faulkes, 2004, Neuroreport.

The BRN-3A Transcription Factor Protects Sensory but Not Sympathetic Neurons from Programmed Cell Death/Apoptosis*

D. Latchman, E. Ensor, M. D. Smith, 2001, The Journal of Biological Chemistry.

Nerve growth factor induces expression of immediate-early genes NGFI-A (Egr-1) and NGFI-B (nur 77) in adult rat dorsal root ganglion neurons.

D. Latchman, E. Ensor, J. Winter, 1994, Brain research. Molecular brain research.

Nerve growth factor induces the Oct‐2 transcription factor in sensory neurons with the kinetics of an immediate‐early gene

D. Latchman, E. Ensor, J. Wood, 1995, Journal of neuroscience research.

Nerve growth factor treatment of sensory neuron primary cultures causes elevated levels of the mRNA encoding the ATP synthase beta-subunit as detected by a novel PCR-based differential cloning method.

D. Latchman, E. Ensor, G. Kendall, 1996, European journal of biochemistry.

The POU domain transcription factor Brn-3a protects cortical neurons from apoptosis

D. Latchman, E. Ensor, R. Kinloch, 2001, Neuroreport.