N. Baptiste

发表

PML is induced by oncogenic ras and promotes premature senescence.

S. Lowe, E. Stanchina, C. Prives, 2000, Genes & development.

Potentiation of the antitumor activity of 5-fluorouracil in colon carcinoma cells by the combination of interferon and deoxyribonucleosides results from complementary effects on thymidine phosphorylase.

N. Baptiste, S. Wadler, E. Schwartz, 1994, Cancer research.

Regulation of expression of thymidine phosphorylase/platelet-derived endothelial cell growth factor in human colon carcinoma cells.

N. Baptiste, E. Schwartz, E. Wan, 1998, Cancer research.

5-Ethoxy-2'-deoxyuridine, a novel substrate for thymidine phosphorylase, potentiates the antitumor activity of 5-fluorouracil when used in combination with interferon, an inducer of thymidine phosphorylase expression.

N. Baptiste, S. Wadler, E. Schwartz, 1995, Cancer research.

p53 in the Cytoplasm A Question of Overkill?

C. Prives, N. Baptiste, 2004, Cell.

The proline-rich domain of p53 is required for cooperation with anti-neoplastic agents to promote apoptosis of tumor cells

C. Prives, P. Friedlander, Xinbin Chen, 2002, Oncogene.

Developmental regulation of the 3-methylcholanthrene- and dioxin-inducible CYP1A5 gene in chick embryo liver in vivo.

N. Baptiste, C. Bentivegna, M. Ihnat, 1998, Toxicology and applied pharmacology.

Dissection of the sequence-specific DNA binding and exonuclease activities reveals a superactive yet apoptotically impaired mutant p53 protein

C. Prives, K. Mckinney, A. Zupnick, 2009, Cell cycle.

Thymidine Phosphorylase Mediates the Sensitivity of Human Colon Carcinoma Cells to 5-Fluorouracil (*)

N. Baptiste, S. Wadler, E. Schwartz, 1995, The Journal of Biological Chemistry.