K. Will

发表

Specific Mismatch Recognition in Heteroduplex Intermediates by p53 Suggests a Role in Fidelity Control of Homologous Recombination

L. Wiesmüller, W. Deppert, Christine Dudenhöffer, 1998, Molecular and Cellular Biology.

P53‐Dependent UVB Responsiveness of Human Keratinocytes Can Be Altered by Cultivation on Cell Cycle‐arrested Dermal Fibroblasts

K. Will, M. Neben, T. Schmidt-Rose, 2000 .

P53-Dependent UVB Responsiveness of Human Keratinocytes Can Be Altered by Cultivation on Cell Cycle-arrested Dermal Fibroblasts

K. Will, M. Neben, T. Schmidt-Rose, 2000, Photochemistry and photobiology.

Analysis of UV-B-induced DNA damage and its repair in heat-shocked skin cells.

K. Will, T. Schmidt-Rose, K. Wittern, 1999, Journal of photochemistry and photobiology. B, Biology.

Species- and tissue-specific expression of the C-terminal alternatively spliced form of the tumor suppressor p53.

W. Deppert, G. Warnecke, K. Will, 1995, Nucleic acids research.

Missense variation of the CFTR gene codon 507

K. Will, M. Stuhrmann, H. Ellemunter, 1992, Human mutation.

A Novel Donor Splice Site in Intron I I of the CFTR Gene, Created by Mutation

X. Estivill, K. Will, T. Casals, 2007 .

Alpha-glucosylrutin : an innovative antioxidant in skin protection

K. Will, K. Wittern, F. Rippke, 2001 .

Transcript analysis of CFTR nonsense mutations in lymphocytes and nasal epithelial cells from cystic fibrosis patients

T. Dörk, K. Will, B. Tümmler, 1995, Human mutation.

A novel donor splice site in intron 11 of the CFTR gene, created by mutation 1811+1.6kbA-->G, produces a new exon: high frequency in Spanish cystic fibrosis chromosomes and association with severe phenotype.

X. Estivill, T. Dörk, K. Will, 1995, American journal of human genetics.

Analysis of mutant p 53 for MAR-DNA binding : determining the dominant-oncogenic function of mutant p 53

W. Deppert, K. Will, 2001 .

Specific interaction of mutant p53 with regions of matrix attachment region DNA elements (MARs) with a high potential for base-unpairing.

L. Wiesmüller, W. Deppert, G. Warnecke, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Alternative splicing in the first nucleotide binding fold of CFTR.

K. Will, J. Schmidtke, M. Stuhrmann, 1993, Human molecular genetics.

Present state of DNA-diagnosis for cystic fibrosis and attitudes concerning a heterozygote screening in the GDR.

V. Steinbicker, K. Will, C. Coutelle, 1991, Advances in experimental medicine and biology.

A novel exon in the cystic fibrosis transmembrane conductance regulator gene activated by the nonsense mutation E92X in airway epithelial cells of patients with cystic fibrosis.

T. Meitinger, T. Dörk, K. Will, 1994, The Journal of clinical investigation.

High affinity MAR‐DNA binding is a common property of murine and human mutant p53

W. Deppert, G. Warnecke, N. Albrechtsen, 1998, Journal of cellular biochemistry.

A 32‐bp deletion (2991del32) in the cystic fibrosis gene associated with CFTR mRNA reduction

T. Dörk, M. Krawczak, K. Will, 1994, Human mutation.

CFTR transcripts are undetectable in lymphocytes and respiratory epithelial cells of a CF patient homozygous for the nonsense mutation R553X.

K. Will, J. Reiss, J. Schmidtke, 1993, Journal of medical genetics.

Lpphoq tes and Nasal Epithelial Cells From Cystic Fibrosis Patients

K. Will, M. Stuhrmann, H. Ellemunter, 2005 .

A donor splice mutation (405 + 1 G-->A) in cystic fibrosis associated with exon skipping in epithelial CFTR mRNA.

T. Dörk, K. Will, B. Tümmler, 1993, Human molecular genetics.