H. Yamasaki

N. Dumaz, A. Ouhtit, H. Yamasaki, 1997, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

Okadaic acid and phorbol esters: comparative effects of these tumor promoters on cell transformation, intercellular communication and differentiation in vitro.

T. Sugimura, H. Yamasaki, D. Fitzgerald, 1990, Japanese journal of cancer research : Gann.

Aberrant control of connexin expression and functions in multistage rat and human hepatocarcinogenesis

V. Krutovskikh, H. Yamasaki, Y. Omori, 1995 .

Lack of tumor-promoting effects of flavonoids: studies on rat liver preneoplastic foci and on in vivo and in vitro gap junctional intercellular communication.

V. Krutovskikh, H. Yamasaki, C. Chaumontet, 1996, Nutrition and cancer.

Transgeneration transmission of carcinogenic risk.

S. Narod, H. Yamasaki, S. Narod, 1992, Carcinogenesis.

An update on minding the gap in cancer.

C. Naus, H. Yamasaki, A. Tabernero, 2018, Biochimica et biophysica acta. Biomembranes.

Further evidence for the involvement of gap-junctional intercellular communication in induction and maintenance of transformed foci in BALB/c 3T3 cells.

H. Yamasaki, F. Katoh, 1988, Cancer research.

Long-term connexin-mediated bystander effect in highly tumorigenic human cells in vivo in herpes simplex virus thymidine kinase/ganciclovir gene therapy

H. Yamasaki, A. Rolland, A. Duflot-Dancer, 1998, Gene Therapy.

Increased susceptibility to urethane-induced lung tumors in mice with decreased expression of connexin43.

H. Yamasaki, M. Mesnil, T. C. da Silva, 2004, Carcinogenesis.

Delayed liver regeneration and increased susceptibility to chemical hepatocarcinogenesis in transgenic mice expressing a dominant-negative mutant of connexin32 only in the liver.

V. Krutovskikh, H. Yamasaki, Y. Omori, 2003, Carcinogenesis.

Co‐ordinated expression of connexins 26 and 32 in human endometrial glandular epithelium during the reproductive cycle and the influence of hormone replacement therapy

R. Kudo, H. Yamasaki, M. Mori, 1997, International journal of cancer.

The inhibitory effect of connexin 32 gene on metastasis in renal cell carcinoma

H. Yamasaki, Y. Nagashima, T. Yano, 2008, Molecular carcinogenesis.

G→A mutations in p53 and Ha‐ras genes in esophageal papillomas induced by N‐nitrosomethylbenzylamine in two strains of rats

H. Yamasaki, H. Nakazawa, J. Lozano, 1994, Molecular carcinogenesis.

Hypermethylation of the CpG island of connexin 32, a candiate tumor suppressor gene in renal cell carcinomas from hemodialysis patients.

H. Yamasaki, T. Yano, F. Ito, 2004, Cancer letters.

Use of the Syrian hamster embryo (SHE) cell transformation assay for predicting the carcinogenic potential of chemicals. Introduction.

H. Yamasaki, 1996, Mutation research.

Growth suppression of transformed cells by a human placental extract not related to transforming growth factor Β

H. Yamasaki, J. Tayot, J. Klein, 2005, Journal of Cancer Research and Clinical Oncology.

Human hemangiosarcomas have a common polymorphism but no mutations in the connexin37 gene

K. Ishak, Tsuyoshi Saito, V. Krutovskikh, 2000, International journal of cancer.

Possible role of activated ras genes in human esophageal carcinogenesis

S. Hirohashi, A. Fusco, N. Martel, 1993, International journal of cancer.

Relationship between chemically induced ha‐ras mutation and transformation of BALB/c 3T3 cells: Evidence for chemical‐specific activation and cell type—specific recruitment of oncogene in transformation

H. Yamasaki, H. Nakazawa, A. Aguelon, 2006, Molecular carcinogenesis.

Aberrant expression, function and localization of connexins in human esophageal carcinoma cell lines with different degrees of tumorigenicity

A. Fusco, H. Yamasaki, M. Oyamada, 2005, Journal of Cancer Research and Clinical Oncology.

Gap-junctional communication alterations at various regulatory levels of connexin expression and function during animal and human carcinogenesis

V. Krutovskikh, W. Jongen, H. Yamasaki, 1993 .

Lack of Correlation between the Gap Junctional Communication Capacity of Human Colon Cancer Cell Lines and Expression of the DCC Gene, a Homologue of a Cell Adhesion Molecule (N‐CAM)

H. Yamasaki, M. Mesnil, C. Piccoli, 1993, Japanese journal of cancer research : Gann.

Induction of a bystander effect in HeLa cells by using a bigenic vector carrying viral thymidine kinase and connexin32 genes

H. Yamasaki, T. Tanaka, M. Mesnil, 2001, Molecular carcinogenesis.

CHROMOSOMAL DAMAGE AND GAP JUNCTIONAL INTERCELLULAR COMMUNICATION IN MESOTHELIOMA CELL LINES AND CULTURED HUMAN PRIMARY MESOTHELIAL CELLS TREATED WITH MMMF

H. Yamasaki, T. Tuomi, K. Linnainmaa, 1991 .

Long‐term remission in an elderly patient with mantle cell leukemia treated with low‐dose cyclophosphamide

H. Mitsuya, H. Yamasaki, Motomi Osato, 2000, American journal of hematology.

Multistage carcinogenesis: implications for risk estimation

H. Yamasaki, 1988, Cancer and Metastasis Reviews.

Gap junction proteins connexin32 and connexin43 partially acquire growth-suppressive function in HeLa cells by deletion of their C-terminal tails.

H. Yamasaki, Y. Omori, 1999, Carcinogenesis.

Growth control of 3T3 fibroblast cell lines established from connexin 43–deficient mice

H. Yamasaki, Y. Omori, K. Yamakage, 1998, Molecular carcinogenesis.

Connexin 32 mutations from X-linked Charcot-Marie-Tooth disease patients: functional defects and dominant negative effects.

H. Yamasaki, Y. Omori, M. Mesnil, 1996, Molecular biology of the cell.

Role of blocked gap junctional intercellular communication in non-genotoxic carcinogenesis.

V. Krutovskikh, H. Yamasaki, Y. Omori, 1995, Toxicology letters.

Cell‐cell communication and growth control of normal and cancer cells: Evidence and hypothesis

A. Balmain, J. C. Barrett, H. Moses, 1993 .

Induction of cell transformation by mutated 16K vacuolar H+-atpase (ductin) is accompanied by down-regulation of gap junctional intercellular communication and translocation of connexin 43 in NIH3T3 cells

Tsuyoshi Saito, H. Yamasaki, R. Schlegel, 1998, Oncogene.

Involvement of gap junctions in tumor suppression: analysis of genetically-manipulated mice.

H. Yamasaki, Y. Omori, K. Yamakage, 2001, Mutation research.

Inhibition of rat liver gap junction intercellular communication by tumor-promoting agents in vivo. Association with aberrant localization of connexin proteins.

V. Krutovskikh, H. Yamasaki, G. Mazzoleni, 1995, Laboratory investigation; a journal of technical methods and pathology.

Changes in gap junction protein (connexin 32) gene expression during rat liver carcinogenesis

H. Yamasaki, D. Fitzgerald, H. Tsuda, 1989, Journal of cellular biochemistry.

Phenobarbital specifically reduces gap junction protein mRNA level in rat liver

H. Yamasaki, D. Fitzgerald, M. Mesnil, 1988, Molecular carcinogenesis.

An improved long-term culture of rat hepatocytes to detect liver tumour-promoting agents: results with phenobarbital.

H. Yamasaki, M. Mesnil, C. Piccoli, 1993, European journal of pharmacology.

High frequency of ki‐ras amplification and p53 gene mutations in adenocarcinomas of the human esophagus

H. Yamasaki, H. Nakazawa, J. Lozano, 1995, Molecular carcinogenesis.

Patched (ptch)-associated preferential expression of smoothened (smoh) in human basal cell carcinoma of the skin.

I. Vořechovský, M. Kallassy, H. Yamasaki, 1997, Cancer research.

Inhibitory effect of tumor promoting phorbol esters and mezerein on human mixed lymphocyte reaction.

N. Martel, H. Yamasaki, 1981, Cancer letters.

Nuclear localization of β‐catenin in normal and carcinogenic endometrium

Tsuyoshi Saito, R. Kudo, H. Yamasaki, 1999, Molecular carcinogenesis.

A derivative of oleamide potently inhibits the spontaneous metastasis of mouse melanoma BL6 cells.

Y. Kitamura, H. Nojima, H. Yamasaki, 2004, Carcinogenesis.

Genomic instability in oral squamous cell carcinoma: relationship to betel-quid chewing.

S. Zienolddiny, H. Yamasaki, G. Thomas, 2004, Oral oncology.

Effects of sodium L-ascorbate, uracil, butylated hydroxyanisole and extracellular pH on junctional intercellular communication of BALB/c 3T3 cells.

S. Fukushima, H. Yamasaki, N. Ito, 1988, Carcinogenesis.

Role of activator protein‐1 and methylation function in 12‐o‐tetradecanoylphorbol‐13‐acetate—mediated inhibition of differentiation of friend erythroleukemia cells

H. Yamasaki, H. Nakazawa, A. Sergeant, 1992, Molecular carcinogenesis.

High degree of myeloid differentiation and granulocytosis is associated with t(8;21) smoldering leukemia.

K. Yamaguchi, E. Matutes, H. Yamasaki, 1995, Leukemia.

p53 gene mutation and loss of heterozygosity are associated with increased risk of disease progression in adult T cell leukemia.

H. Yamasaki, Motomi Osato, K. Takatsuki, 1995, Leukemia.

Low-dose cytarabine and aclarubicin in combination with granulocyte colony-stimulating factor for elderly patients with previously untreated acute myeloid leukemia.

H. Mitsuya, H. Yamasaki, H. Tsuda, 2010, Leukemia research.

L-myc allele polymorphism and prognosis for metastases in Russian gastric cancer patients.

H. Yamasaki, N. Mironov, A. Aguelon, 1994, European journal of cancer.

Stimulation of intercellular communication of poor‐communicating cells by gap‐junction‐competent cells enhances the HSV‐TK/GCV bystander effect in vitro

H. Yamasaki, T. Tanaka, M. Mesnil, 2001, International journal of cancer.

Identification and quantification of a carcinogen-induced molecular initiation event in cell transformation.

H. Yamasaki, H. Nakazawa, A. Aguelon, 1992, Oncogene.

Inhibition of cell communication between Balb/c 3t3 cells by tumor promoters and protection by cAMP

N. Martel, H. Yamasaki, Y. Kanno, 1984, Journal of cellular physiology.

Protective effect of cAMP on tumor promoter-mediated inhibition of cell-cell communication.

H. Yamasaki, Y. Kanno, T. Enomoto, 1984, Experimental cell research.

Increased genetic stability of HeLa cells after connexin 43 gene transfection.

W. Zhu, H. Yamasaki, N. Mironov, 1997, Cancer research.

Role of the Cytoplasmic Loop Domain of Cx43 in Its Intracellular Localization and Function: Possible Interaction with Cadherin

H. Yamasaki, Yumi Kawasaki, Chika Nambara, 2007, Journal of Membrane Biology.

Induction of skin papillomas, carcinomas, and sarcomas in mice in which the connexin 43 gene is heterologously deleted.

H. Yamasaki, Y. Omori, K. Yamakage, 2000, The Journal of investigative dermatology.

Gene Associated With Myeloblastic Leukemias AML1/PEBP2?B Biallelic and Heterozygous Point Mutations in the Runt Domain of the

H. Yamasaki, Yoshiaki Ito, Motomi Osato, 2013 .

NBT-II carcinoma behaviour is not dependent on cell-cell communication through gap junctions.

J. Thiery, J. Jouanneau, H. Yamasaki, 2002, Biochemical and biophysical research communications.

Cellular and molecular methods to study the role of gap junctional intercellular communication in toxicology.

H. Yamasaki, 1997, Toxicology in vitro : an international journal published in association with BIBRA.

Tumor Promoter-Mediated Modulation of Cell Differentiation and Communication: The Phorbol Ester — Oncogene Connection

M. Hollstein, H. Yamasaki, 1987 .

Specific viral oncogenes cause differential effects on cell‐to‐cell communication, relevant to the suppression of the transformed phenotype by normal cells

M. Bignami, H. Yamasaki, S. Rosa, 1988, Molecular carcinogenesis.

Tissue‐specific activating mutations of Ha‐ and Ki‐ras oncogenes in skin, lung, and liver tumors induced in mice following transplacental exposure to DMBA

M. Hollstein, N. Martel, H. Yamasaki, 1990, Molecular carcinogenesis.

Regulation of cellular invasion and matrix metalloproteinase activity in HepG2 cell by connexin 26 transfection

H. Yamasaki, T. Yano, 2001, Molecular carcinogenesis.

Selective lack of intercellular communication between transformed and nontransformed cells as a common property of chemical and oncogene transformation of BALB/c 3T3 cells.

M. Hollstein, N. Martel, H. Yamasaki, 1987, Cancer research.

Reduced gap junctional intercellular communication and altered biological effects in mouse osteoblast and rat liver oval cell lines transfected with dominant‐negative connexin 43

J. Trosko, Chia‐cheng Chang, V. Krutovskikh, 2003, Molecular carcinogenesis.

Connexins in tumour suppression and cancer therapy.

W. Zhu, V. Krutovskikh, H. Yamasaki, 1999, Novartis Foundation symposium.

Inhibition of junctional intercellular communication as a possible short-term test to detect tumor-promoting agents: results with nine chemicals tested by dye transfer assay in Chinese hamster V79 cells.

H. Yamasaki, M. Zeilmaker, 1986, Cancer research.

Modulation of cellular phorbol ester binding and/or protein kinase C activity by human placental fractions.

H. Yamasaki, J. Tayot, E. Hamel, 1988, Biochimica et biophysica acta.

Cell-Cell Interaction and Carcinogenesis

H. Yamasaki, 1986, Toxicologic pathology.

Inhibition of gap junctional intercellular communication in Syrian hamster embryo cells by TPA, retinoic acid and DDT.

H. Yamasaki, T. Sanner, E. Rivedal, 1994, Carcinogenesis.

Performance of tests for cell transformation and gap-junction intercellular communication for detecting nongenotoxic carcinogenic activity.

H. Yamasaki, S. Swierenga, 1992, IARC scientific publications.

Role of cell-cell communication in tumor suppression.

H. Yamasaki, 1990, Immunology series.

Transforming Growth Factor ÃŸas a Potent Promoter in Two-Stage BALB/c 3T3 Cell Transformation1

W. Birchmeier, H. Yamasaki, Élisabeth, 2006 .

A human T-cell lymphotropic virus type-I carrier with chronic renal failure, aplastic anemia, myelopathy, uveitis, Sjögren's syndrome and panniculitis.

H. Yamasaki, Motomi Osato, M. Kawakita, 1996, Internal medicine.

Possible molecular mechanism of loss of homologous and heterologous gap junctional intercellular communication in rat liver epithelial cell lines.

H. Yamasaki, M. Mesnil, M. Asamoto, 1994, Cell adhesion and communication.

Dominant-negative abrogation of connexin-mediated cell growth control by mutant connexin genes

H. Yamasaki, A. Duflot-Dancer, M. Mesnil, 1997, Oncogene.

Epigenetic inactivation of connexin 32 in renal cell carcinoma from hemodialytic patients.

H. Yamasaki, T. Yano, F. Ito, 2004, Kidney international.

Gap junctional communication and carcinogenesis

H. Yamasaki, 1990 .

Membrane interaction and modulation of gene expression by tumor promoters.

H. Yamasaki, Y. Kanno, T. Enomoto, 1983, Princess Takamatsu symposia.

Cytotoxic effect of the Her-2/Her-1 inhibitor PKI-166 on renal cancer cells expressing the connexin 32 gene.

H. Yamasaki, T. Yano, K. Ueno, 2005, Journal of pharmacological sciences.

Cell Transformation Assays as Predictors of Human Carcinogenicity

E Sabbioni, M Balls, N. Fusenig, 1999, Alternatives to laboratory animals : ATLA.

To explore the program of cell differentiation in Friend murine eryth roleukemia (MEL) cells, we used three clonal variants: phorbol 12-my

H. Yamasaki, A. Vaughan, A. Constantinou, 2006 .

Evidence for UV-associated activation of telomerase in human skin.

A. Ouhtit, H. Yamasaki, H. Nakazawa, 1997, Cancer research.

UV and skin cancer: specific p53 gene mutation in normal skin as a biologically relevant exposure measurement.

D. English, N. Martel, H. Yamasaki, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Regulation of connexin 43-mediated gap junctional intercellular communication by Ca2+ in mouse epidermal cells is controlled by E- cadherin

T. Slaga, D. Gros, M. Takeichi, 1991, The Journal of cell biology.

Long-term Effect of a Tumor Promoter , 12-O-Tetradecanoylphorbol-IS-Acetate , on Induced Differentiation of Friend Leukemia Cells 1

N. Martel, H. Yamasaki, L. Saint-Vincent, 2006 .

Tumour promoter-mediated reversible inhibition of cell-cell communication (electrical coupling). Relationship with phorbol ester binding and de novo macromolecule synthesis.

N. Martel, H. Yamasaki, Y. Kanno, 1983, Experimental cell research.

Bystander effect in herpes simplex virus-thymidine kinase/ganciclovir cancer gene therapy: role of gap-junctional intercellular communication.

H. Yamasaki, M. Mesnil, 2000, Cancer research.

Bystander killing of cancer cells by herpes simplex virus thymidine kinase gene is mediated by connexins.

K. Willecke, H. Yamasaki, G. Tiraby, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Aberrant expression of gap junction gene in primary human hepatocellular carcinomas: Increased expression of cardiac‐type gap junction gene connexin 43

V. Krutovskikh, H. Yamasaki, M. Mesnil, 1990, Molecular carcinogenesis.

Intercellular communication of transformed and non-transformed rat liver epithelial cells. Modulation by TPA.

R. Montesano, H. Yamasaki, M. Mesnil, 1986, Experimental cell research.

Cell contact but not junctional communication (dye coupling) with biliary epithelial cells is required for hepatocytes to maintain differentiated functions.

H. Yamasaki, C. Guguen-Guillouzo, M. Mesnil, 1987, Experimental cell research.

Genetic control of the regulation of cell susceptibility to carcinogenic polycyclic hydrocarbons by cyclic AMP

L. Sachs, H. Yamasaki, E. Huberman, 1974, International journal of cancer.

Biallelic and heterozygous point mutations in the runt domain of the AML1/PEBP2alphaB gene associated with myeloblastic leukemias.

H. Yamasaki, Motomi Osato, K. Shigesada, 1999, Blood.

Allele‐specific PCR analysis of p53 codon 249 AGT transversion in liver tissues from patients with viral hepatitis

G. Batist, H. Yamasaki, H. Nakazawa, 1996, International journal of cancer.

In vitro and in vivo effects of retrovirus-mediated transfer of the connexin 43 gene in malignant gliomas: consequences for HSVtk/GCV anticancer gene therapy

G. Finocchiaro, H. Yamasaki, S. Benedetti, 1998, Gene Therapy.

A tumor suppressor gene, Cx26, also mediates the bystander effect in HeLa cells.

H. Yamasaki, M. Mesnil, C. Piccoli, 1997, Cancer research.

Inhibition of intrinsic gap‐junction intercellular communication and enhancement of tumorigenicity of the rat bladder carcinoma cell line BC31 by a dominant‐negative connexin 43 mutant

V. Krutovskikh, H. Yamasaki, H. Tsuda, 1998, Molecular carcinogenesis.

Understanding Multi-Stage Carcinogenesis at the Molecular Level: Notes on Recent Progress

M. Hollstein, H. Yamasaki, 1989 .

Genetic and epigenetic changes of intercellular communication genes during multistage carcinogenesis.

V. Krutovskikh, H. Yamasaki, Y. Omori, 1999, Cancer detection and prevention.

The expression of the tumor suppressor gene connexin 26 is not mediated by methylation in human esophageal cancer cells

H. Yamasaki, J. Loncarek, M. Mesnil, 2003, Molecular carcinogenesis.

Growth of only highly tumorigenic cell lines is inhibited by EAP, a human placental fraction.

C. Chiodino, H. Yamasaki, J. Klein, 1993, Cancer letters.

Role of disrupted gap junctional intercellular communication in detection and characterization of carcinogens.

H. Yamasaki, 1996, Mutation research.

Altered homologous and heterologous gap‐junctional intercellular communication in primary human liver tumors associated with aberrant protein localization but not gene mutation of connexin 32

V. Krutovskikh, H. Yamasaki, F. Berger, 2007, International journal of cancer.

Aberrant expression and function of gap junctions during carcinogenesis.

H. Yamasaki, 1991, Environmental health perspectives.

Molecular mechanisms of TPA‐mediated inhibition of Gap‐junctional intercellular communication: Evidence for action on the assembly or function but not the expression of connexin 43 in rat liver epithelial cells

D. Gros, H. Yamasaki, M. Asamoto, 1991, Molecular carcinogenesis.

Decreased connexin 32 expression is associated with cellular proliferation and progression of hepatocarcinogenesis in the rat

K. Ozaki, H. Yamasaki, H. Tsuda, 1995 .

Tumor promotion--mechanisms and implication to risk estimation.

H. Yamasaki, 2009, Acta pharmacologica et toxicologica.

ROLE OF ONCOGENES AND INTERCELLULAR COMMUNICATION

H. Yamasaki, 1990 .

Molecular and Cellular Mechanisms of Multistage Carcinogenesis; Role of Oncogenes and Intercellular Communication

H. Yamasaki, 1990 .

Novel method for selective killing of transformed rodent cells through intercellular communication, with possible therapeutic applications.

H. Yamasaki, F. Katoh, 1988, Cancer research.

Dose and frequency effect in mouse skin tumor promotion.

N. Day, H. Becher, H. Yamasaki, 1989, Cancer research.

Continuous suppression of globin gene expression and differentiation of Friend erythroleukemia cells by phorbol 12-myristate 13-acetate (PMA) despite the loss of PMA binding sites by down regulation.

A. Fusco, N. Martel, H. Yamasaki, 1984, Proceedings of the National Academy of Sciences of the United States of America.

Characterization of a human placental factor which inhibits specific binding of phorbol esters to cultured cells.

N. Martel, H. Yamasaki, J. Tayot, 1984, Carcinogenesis.

Tumor promoters cause a rapid and reversible inhibition of the formation and maintenance of electrical cell coupling in culture.

H. Yamasaki, Y. Kanno, T. Enomoto, 1981, Proceedings of the National Academy of Sciences of the United States of America.

Long-term effect of a tumor promoter, 12-O-tetradecanoylphorbol-13-acetate, on induced differentiation of Friend leukemia cells.

N. Martel, H. Yamasaki, L. Saint-Vincent, 1980, Cancer research.

Malignant transformation of simian virus 40–immortalized human milk epithelial cells by chemical carcinogenesis accompanied by loss of heterozygosity on chromosome 1 but not microsatellite instability

H. Yamasaki, H. Lahm, K. Sasaki, 1998, Molecular carcinogenesis.

Connexin 32 potentiates vinblastine‐induced cytotoxicity in renal cell carcinoma cells

H. Yamasaki, T. Yano, K. Ueno, 2007, Molecular carcinogenesis.

Intercellular communication and carcinogenesis.

V. Krutovskikh, H. Yamasaki, Y. Omori, 1995, Mutation research.

A novel sensitive method to detect frameshift mutations in exonic repeat sequences of cancer-related genes.

W. Zhu, H. Yamasaki, L. Jansen, 1999, Carcinogenesis.

Gap junctional intercellular communication and connexin expression in normal and SV 40-transformed human liver cells in vitro.

H. Yamasaki, N. Marceau, D. Fitzgerald, 1993, Cancer letters.

Implementation of EPA Revised Cancer Assessment Guidelines: Incorporation of Mechanistic and Pharmacokinetic Data.

H J Clewell, M E Andersen, C B Frederick, 1997, Fundamental and applied toxicology : official journal of the Society of Toxicology.

Overexpression of estrogen receptor-α gene suppresses gap junctional intercellular communication in endometrial carcinoma cells

Tsuyoshi Saito, R. Kudo, H. Yamasaki, 2004, Oncogene.

Suppressed gap junctional intercellular communication in carcinogenesis of endometrium

Tsuyoshi Saito, R. Kudo, H. Yamasaki, 2001, International journal of cancer.

Transformation assay of established cell lines: mechanisms and application. Proceedings of a workshop organized by IARC in collaboration with the US National Cancer Institute and the US Environmental Protection Agency. Lyon, 15-17 February 1984.

H. Yamasaki, T. Kakunaga, 1985, IARC scientific publications.

Role of connexin (gap junction) genes in cell growth control and carcinogenesis.

V. Krutovskikh, H. Yamasaki, Y. Omori, 1999, Comptes rendus de l'Academie des sciences. Serie III, Sciences de la vie.

Negative growth control of HeLa cells by connexin genes: connexin species specificity.

K. Willecke, V. Krutovskikh, H. Yamasaki, 1995, Cancer research.

Connexin32 as a tumor suppressor gene in a metastatic renal cell carcinoma cell line

H. Yamasaki, Y. Nagashima, T. Yano, 2005, Oncogene.

Connexin43 suppresses MFG-E8 while inducing contact growth inhibition of glioma cells.

C. Naus, H. Yamasaki, G. Goldberg, 2000, Cancer research.

Effects on intercellular communication in human keratinocytes and liver-derived cells of polychlorinated biphenyl congeners with differing in vivo promotion activities.

H. Yamasaki, N. Marceau, L. Bourgon, 1990, Carcinogenesis.

YAMASAKI ET AL . The first attempt to amplify the effect of exposure in utero to a carcinogen by postnatal exposure to another

H. Yamasaki, L. Tomatis, A. Loktionov, 2006 .

Alterations of (CA)n DNA repeats and tumor suppressor genes in human gastric cancer.

H. Yamasaki, Y. Omori, N. Mironov, 1994, Cancer research.

Transplacental induction of a specific mutation in fetal Ha‐ras and its critical role in post‐natal carcinogenesis

L. Tomatis, M. Hollstein, M. Hollstein, 1987, International journal of cancer.

Identification of two transcripts of AMLI/ETO‐fused gene in t(8;21) leukemic cells and expression of wild‐type ETO gene in hematopoietic cells

S. Nishikawa, N. Kamada, K. Yamaguchi, 1995, Genes, chromosomes & cancer.

A role for heterologous gap junctions between melanoma and endothelial cells in metastasis.

N. Tsubota, H. Asada, K. Yoshikawa, 2000, The Journal of clinical investigation.

DNA Repeats and Tumor Suppressor Genes in Human Gastric Cancer I

H. Yamasaki, Y. Omori, G. Potapova, 2007 .

Advances in Brief Alterations of ( C A ) n DNA Repeats and Tumor Suppressor Genes in Human Gastric Cancer

H. Yamasaki, Y. Omori, G. Potapova, 2007 .

Gap junctional intercellular communication and carcinogenesis.

H. Yamasaki, H Yamasaki, 1990, Carcinogenesis.

Reduction of malignant phenotype of HEPG2 cell is associated with the expression of connexin 26 but not connexin 32.

H. Yamasaki, T. Yano, Y. Omori, 2001, Carcinogenesis.

Connexin 43 Suppresses MFG-E 8 While Inducing Contact Growth Inhibition of Glioma Cells 1

C. Naus, H. Yamasaki, G. Goldberg, 2000 .

Nongenotoxic carcinogens: development of detection methods based on mechanisms: a European project.

S Parodi, J Ashby, J. Ashby, 1996, Mutation research.

Mutated connexin43 proteins inhibit rat glioma cell growth suppression mediated by wild‐type connexin43 in a dominant‐negative manner

H. Yamasaki, Y. Omori, 1998, International journal of cancer.

Connexin hemichannels and gap junction channels are differentially influenced by lipopolysaccharide and basic fibroblast growth factor.

C. Naus, H. Yamasaki, E. De Vuyst, 2006, Molecular biology of the cell.

Microsatellite alterations in human and rat esophageal tumors at selective loci

H. Yamasaki, J. Lozano, E. Hollams, 1995, Molecular carcinogenesis.

CC to TT mutation in the mitochondrial DNA of normal skin: relationship to ultraviolet light exposure.

H. Yamasaki, M. Ueda, M. Ichihashi, 2000, Mutation research.

UV-radiation-specific p53 mutation frequency in normal skin as a predictor of risk of basal cell carcinoma.

D. English, A. Ouhtit, H. Yamasaki, 1998, Journal of the National Cancer Institute.

Expression of AML1 and ETO Transcripts in hematopoietic cells.

N. Kamada, K. Yamaguchi, H. Yamasaki, 1995, Leukemia.

Connexin 37 mutations in rat hepatic angiosarcomas induced by vinyl chloride.

H. Yamasaki, A. Barbin, Y. Omori, 1997, Cancer research.

Role of connexin genes in growth control.

C. Naus, H. Yamasaki, 1996, Carcinogenesis.

Metsovo-tremolite asbestos fibres: in vitro effects on mutation, chromosome aberration, cell transformation and intercellular communication.

H. Yamasaki, D. Fitzgerald, H Yamasaki, 1992, Mutagenesis.

Altered gap junctional communication, intercellular signaling, and growth in cultured astrocytes deficient in connexin43

Yin Zhang, C. Naus, L. Venance, 1997, Journal of neuroscience research.

Metabolism of the carcinogenic hydrocarbon benzo(a)pyrene in human fibroblast and epithelial cells. II. Differences in metabolism to water‐soluble products and aryl hydrocarbon hydroxylase activity

L. Sachs, H. Yamasaki, E. Huberman, 1977, International journal of cancer.

Independent regulation of two types of aryl hydrocarbon (benzo(a)pyrene) hydroxylase in mammalian cells

L. Sachs, H. Yamasaki, E. Huberman, 1976, International journal of cancer.

Inhibition of Balb/c 3T3 cell transformation by synthetic acyclic retinoid NIK-333; possible involvement of enhanced gap junctional intercellular communication.

H. Yamasaki, F. Katoh, T. Nagata, 2007, Cancer detection and prevention.

Tumor promotion: models and assay systems.

H. Yamasaki, D. Fitzgerald, D. JamesFitzgerald, 1990, Teratogenesis, carcinogenesis, and mutagenesis.

Myelomonoblastic leukaemia cells carrying the PEBP2β/MYH11 fusion gene are CD34+, c‐KIT+ immature cells

H. Yamasaki, S. Bae, Motomi Osato, 1997, British journal of haematology.

Tumour-promoting phorbol esters rapidly inhibit bud formation in hydra

H. Yamasaki, Y. Kanno, Y. Shiba, 1987, Roux's archives of developmental biology.

Inhibition of Src activity enhances the tumor-suppressive effect of the connexin 32 gene in Caki-1 renal cancer cells.

H. Yamasaki, Y. Nagashima, T. Yano, 2006, Oncology reports.

Tumor promoters specifically and reversibly disturb development and behavior of Caenorhabditis elegans

M. Furusawa, H. Yamasaki, Y. Tabuse, 2004, Journal of Cancer Research and Clinical Oncology.

Growth inhibition by expression of connexin 26 in HeLa cells

K. Willecke, V. Krutovskikh, H. Yamasaki, 1995 .

Bystander effect from cytosine deaminase and uracil phosphoribosyl transferase genes in vitro: a partial contribution of gap junctions.

Toshiaki Tanaka, H. Yamasaki, G. Tiraby, 2009, Cancer letters.

Expression and function of connexin in normal and transformed human keratinocytes in culture.

N. Fusenig, H. Yamasaki, D. Fitzgerald, 1994, Carcinogenesis.

Inhibition of intercellular communication and enhancement of morphological transformation of Syrian hamster embryo cells by TPA. Use of TPA-sensitive and TPA-resistant cell lines.

H. Yamasaki, T. Sanner, T. Enomoto, 1985, Carcinogenesis.

Classical cadherins in urological cancers.

J. Schalken, H. Yamasaki, P. Bringuier, 2000, Morphologie : bulletin de l'Association des anatomistes.

Interaction and distinction of genotoxic and non-genotoxic events in carcinogenesis.

H. Yamasaki, H. Nakazawa, H Yamasaki, 1992, Toxicology letters.

Assignment of connexin 26 (GJB2) and 46 (GJA3) genes to human chromosome 13q11-->q12 and mouse chromosome 14D1-E1 by in situ hybridization.

D. Gros, M. Mattei, H. Yamasaki, 1996, Cytogenetics and cell genetics.

Negative growth control of renal cell carcinoma cell by connexin 32: Possible involvement of Her‐2

H. Yamasaki, Y. Nagashima, T. Yano, 2004, Molecular carcinogenesis.

Genomic instability in multistage carcinogenesis.

H. Yamasaki, N. Mironov, Nikolai Mironov, 2000, Toxicology letters.

Selective gap-junctional communication capacity of transformed and non-transformed rat liver epithelial cell lines.

H. Yamasaki, M. Mesnil, Hiroshi Yamasaki, 1988, Carcinogenesis.

Rapid inhibition of intercellular communication between BALB/c 3T3 cells by diacylglycerol, a possible endogenous functional analogue of phorbol esters.

H. Yamasaki, T. Enomoto, 1985, Cancer research.

Specific binding of phorbol esters to Friend erythroleukemia cells--general properties, down regulation and relationship to cell differentiation.

C. Drevon, N. Martel, H. Yamasaki, 1982, Carcinogenesis.

Dynamics of light-induced conformational changes of the phoborhodopsin/transducer complex formed in the n-dodecyl beta-D-maltoside micelle.

H. Yamasaki, N. Kamo, Y. Toyoshima, 2007, Biochemistry.

Frequency of HPRT gene mutations induced by N-methyl-N'-nitro-N-nitrosoguanidine corresponds to replication error phenotypes of cell lines.

W. Zhu, H. Yamasaki, N. Mironov, 1998, Mutation research.

Gap junctional intercellular communication of primary and asbestos-associated malignant human mesothelial cells.

K. Pelin, H. Yamasaki, D. Fitzgerald, 1993, Carcinogenesis.

Action of phorbol esters in cell culture: mimicry of transformation, altered differentiation, and effects on cell membranes.

P. Fisher, I. Weinstein, H. Yamasaki, 1979, Journal of supramolecular structure.

Lack of the expression of EBNA-2 and LMP-1 in T-cell neoplasms possessing Epstein-Barr virus.

M. Matsuoka, H. Yamasaki, Motomi Osato, 1995, Blood.

Cell-to-cell communication in cultured rat thyroid monolayer cells is inhibited dose-dependently by methimazole.

H. Yamasaki, T. Hanafusa, S. Tarui, 1992, Research communications in chemical pathology and pharmacology.

Transforming growth factor beta as a potent promoter in two-stage BALB/c 3T3 cell transformation.

W. Birchmeier, H. Yamasaki, F. Katoh, 1988, Cancer research.

Detection of non-genotoxic carcinogens in the BALB/c 3T3 cell transformation/mutation assay system.

H. Yamasaki, C. Piccoli, D. Fitzgerald, 1989, Mutagenesis.

Differential effect of subcellular localization of communication impairing gap junction protein connexin43 on tumor cell growth in vivo

V. Krutovskikh, H. Yamasaki, C. Piccoli, 2000, Oncogene.

Use of primary keratinocyte cultures from plucked human hairs for analysis of gap junctional intercellular communication.

H. Yamasaki, S. Swierenga, C. Piccoli, 1991, Toxicology in vitro : an international journal published in association with BIBRA.

Gap junction intercellular communication propagates cell death in cancerous cells

V. Krutovskikh, H. Yamasaki, C. Piccoli, 2002, Oncogene.

Ex-vivo dye transfer assay as an approach to study gap junctional intercellular communication disorders in hepatocarcinogenesis

V. Krutovskikh, H. Yamasaki, 1995 .

Gap junctional intercellular communication and cell proliferation during rat liver carcinogenesis.

V. Krutovskikh, H. Yamasaki, A. Columbano, 1993, Environmental health perspectives.

Altered expression of connexins in urethane-induced mouse lung adenomas.

H. Yamasaki, H. Fukumasu, M. Mesnil, 2006, Life sciences.

Tumor-suppressive effect of connexin 32 in renal cell carcinoma from maintenance hemodialysis patients.

H. Yamasaki, T. Yano, F. Ito, 2003, Kidney international.

Mutagenicity, sister chromatid exchange inducibility andin vitro cell transforming ability of particulates from Athens air

H. Yamasaki, I. Arzimanoglou, C. Piccoli, 1987, Cell Biology and Toxicology.

Dose and FrequencyEffect in Mouse Skin TumorPromotion

H. Becher, H. Yamasaki, J. Wahrendorf, 1989 .

Connexin Species Specificity Negative Growth Control of HeLa Cells by Connexin Genes : Updated

K. Willecke, V. Krutovskikh, H. Yamasaki, 2006 .

Perinatal and multigenerational effect of carcinogens: possible contribution to determination of cancer susceptibility.

H. Yamasaki, L. Tomatis, A. Loktionov, 1992, Environmental health perspectives.

Enhancement of transformation and continuous inhibition of intercellular communication by 1-oleoyl-2-acetyl glycerol in BALB/c 3T3 cells.

U. Frixen, H. Yamasaki, 1987, Carcinogenesis.

Role of intercellular communication in BALB/c 3T3 cell transformation.

H. Yamasaki, T. Enomoto, 1985, Carcinogenesis; a comprehensive survey.

Effects on intercellular communication in human keratinocytes and liver-derived cells of polychlorinated biphenyl congeners with differing in vivo promotion activities.

H. Yamasaki, N. Marceau, L. Bourgon, 1990, Carcinogenesis.

TEL/AML1 fusion gene resulting from a cryptic t(12;21) is uncommon in adult patients with B-cell lineage ALL and CML lymphoblastic transformation.

H. Yamasaki, Motomi Osato, K. Takatsuki, 1997, International journal of hematology.

Connexin gene mutations in human genetic diseases.

V. Krutovskikh, H. Yamasaki, H. Yamasaki, 2000, Mutation research.