J. Thiery

发表

Granzyme B–Activated p53 Interacts with Bcl-2 To Promote Cytotoxic Lymphocyte–Mediated Apoptosis

Thouraya Ben Safta, Linda Ziani, Loëtitia Favre, 2015, The Journal of Immunology.

A three-dimensional tumor cell defect in activating autologous CTLs is associated with inefficient antigen presentation correlated with heat shock protein-70 down-regulation.

S. Richon, V. Dangles-Marie, P. Validire, 2003, Cancer research.

Perforin pores in the endosomal membrane trigger release of endocytosed granzyme B to the cytosol of target cells

J. Lieberman, T. Kirchhausen, D. Martinvalet, 2011, Nature Immunology.

Isolation of Cytotoxic T Cell and NK Granules and Purification of Their Effector Proteins

J. Lieberman, D. Martinvalet, J. Thiery, 2010, Current protocols in cell biology.

Perforin: a key pore-forming protein for immune control of viruses and cancer.

J. Lieberman, J. Thiery, 2014, Sub-cellular biochemistry.

Arf-like GTPase Arl8b regulates lytic granule polarization and natural killer cell–mediated cytotoxicity

J. Lieberman, J. Orange, J. Thiery, 2013, Molecular biology of the cell.

The pharmalogical reactivation of p53 function improves breast tumor cell lysis by granzyme B and NK cells through induction of autophagy

S. Chouaib, J. Thiery, G. Meurice, 2019, Cell Death & Disease.

Opposite effects of estrogen receptors alpha and beta on MCF-7 sensitivity to the cytotoxic action of TNF and p53 activity

G. Leclercq, S. Chouaib, J. Thiery, 2005, Oncogene.

Granzyme B-induced mitochondrial ROS are required for apoptosis

J. Lieberman, D. Martinvalet, J. Thiery, 2014, Cell Death and Differentiation.

Editor's Note: ITPR1 Protects Renal Cancer Cells against Natural Killer Cells by Inducing Autophagy.

D. Olive, C. Richon, B. Janji, 2022, Cancer research.

Dual effect of autophagy in the regulation of cell-mediated cytotoxicity

S. Chouaib, J. Thiery, 2020 .

inducing autophagy ITPR 1 protects renal cancer cells against natural killer cells

Katy, Catherine, Betty, 2014 .

Perforin activates clathrin- and dynamin-dependent endocytosis, which is required for plasma membrane repair and delivery of granzyme B for granzyme-mediated apoptosis.

J. Lieberman, T. Kirchhausen, D. Martinvalet, 2010, Blood.

Cytotoxic Cells Kill Intracellular Bacteria through Granulysin-Mediated Delivery of Granzymes

J. Lieberman, T. Kirchhausen, A. Krensky, 2015, Cell.

Release of interleukin-6 (IL-6) from human vascular smooth muscle cells

J. Thiery, W. Kübler, E. Hodenberg, 1993 .

Tumor escape from killing: role of killer inhibitory receptors and acquisition of tumor resistance to cell death.

F. Mami-Chouaib, S. Chouaib, J. Thiery, 2002, Tissue antigens.

Wild-Type p53 Function Autologous CTL Killing by Restoration of Potentiation of a Tumor Cell Susceptibility to

Isabelle, C. Richon, H. Haddada, 2003 .

The sensitivity of renal cell carcinoma cells to interferon alpha correlates with p53-induction and involves Bax.

S. Chouaib, J. Thiery, B. Escudier, 2005, European cytokine network.

Modulation of the antitumor immune response by cancer-associated fibroblasts: mechanisms and targeting strategies to hamper their immunosuppressive functions

J. Thiery, 2022, Exploration of targeted anti-tumor therapy.

Down-Regulation Presentation Correlated with Heat Shock Protein-70 Autologous CTLs Is Associated with Inefficient Antigen A Three-Dimensional Tumor Cell Defect in Activating

S. Richon, V. Dangles-Marie, P. Validire, 2003 .

p53 Potentiation of Tumor Cell Susceptibility to CTL Involves Fas and Mitochondrial Pathways1

C. Richon, F. Mami-Chouaib, S. Chouaib, 2005, The Journal of Immunology.

Analysis of the mechanisms of human cytotoxic T lymphocyte response inhibition by NO.

F. Mami-Chouaib, S. Chouaib, J. Thiery, 2002, International immunology.

Tumor escape from killing: Role of killer

F. Mami-Chouaib, S. Chouaib, J. Thiery, 2002 .

The Effect of Hypoxia and Hypoxia-Associated Pathways in the Regulation of Antitumor Response: Friends or Foes?

S. Terry, S. Chouaib, J. Thiery, 2022, Frontiers in Immunology.

Hypoxia increases melanoma-associated fibroblasts immunosuppressive potential and inhibitory effect on T cell-mediated cytotoxicity

Linda Ziani, S. Chouaib, J. Thiery, 2021, Oncoimmunology.

Hypoxia increases mutational load of breast cancer cells through frameshift mutations

P. Dessen, F. Rosselli, S. Chouaib, 2020, Oncoimmunology.

Melanoma-associated fibroblasts decrease tumor cell susceptibility to NK cell-mediated killing through matrix-metalloproteinases secretion

C. Robert, G. Favre, Linda Ziani, 2017, Oncotarget.

Role of Hypoxia-Mediated Autophagy in Tumor Cell Death and Survival

S. Chouaib, J. Thiery, C. Jamora, 2021, Cancers.

The Most Common VHL Point Mutation R167Q in Hereditary VHL Disease Interferes with Cell Plasticity Regulation

S. Terry, J. Adam, P. Dessen, 2021, Cancers.

Alteration of the Antitumor Immune Response by Cancer-Associated Fibroblasts

Linda Ziani, S. Chouaib, J. Thiery, 2018, Front. Immunol..

ITPR1 protects renal cancer cells against natural killer cells by inducing autophagy.

D. Olive, C. Richon, B. Janji, 2014, Cancer research.

Capture of MicroRNA–Bound mRNAs Identifies the Tumor Suppressor miR-34a as a Regulator of Growth Factor Signaling

Oliver Hofmann, Winston Hide, Judy Lieberman, 2011, PLoS genetics.

Attenuation of Soft-Tissue Sarcomas Resistance to the Cytotoxic Action of TNF-α by Restoring p53 Function

L. Vassilev, P. Terrier, S. Bonvalot, 2012, PloS one.

Role of Hypoxic Stress in Regulating Tumor Immunogenicity, Resistance and Plasticity

S. Terry, S. Chouaib, J. Thiery, 2018, International journal of molecular sciences.

Granzyme B-induced Cell Death Involves Induction of p53 Tumor Suppressor Gene and Its Activation in Tumor Target Cells*

C. Richon, S. Chouaib, J. Thiery, 2007, Journal of Biological Chemistry.

Potentiation of a Tumor Cell Susceptibility to Autologous CTL Killing by Restoration of Wild-Type p53 Function1

C. Richon, H. Haddada, F. Mami-Chouaib, 2003, The Journal of Immunology.

Critical Role of Tumor Microenvironment in Shaping NK Cell Functions: Implication of Hypoxic Stress

Linda Ziani, S. Chouaib, J. Thiery, 2015, Front. Immunol..

Cancer-associated Fibroblasts and Modulation of the Antitumor Immune Response

Linda Ziani, S. Chouaib, J. Thiery, 2015 .

Cancer-associated Fibroblasts and Modulation of the Antitumor Immune

S. Chouaib, J. Thiery, 2015 .

hSMG-1 is a granzyme B-associated stress-responsive protein kinase

J. Galon, C. Richon, F. Rosselli, 2011, Journal of Molecular Medicine.

Granzymes and cell death.

D. Martinvalet, J. Thiery, Dipanjan Chowdhury, 2008, Methods in enzymology.

Selection of tumor-resistant variants following sustained natural killer cell-mediated immune stress

S. Terry, D. Olive, B. Janji, 2020, Oncology reports.

Tumor resistance to specific lysis: a major hurdle for successful immunotherapy of cancer.

F. Mami-Chouaib, S. Chouaib, J. Thiery, 2009, Clinical immunology.

Erratum: Cytotoxic cells kill intracellular bacteria through granulysin-mediated delivery of granzymes (Cell (2014) 153 (1309-1323) DOI: 10.1016/j.cell.2015.05.021)

J. Lieberman, T. Kirchhausen, A. Krensky, 2015 .

Cytotoxic Cells Kill Intracellular Bacteria through Granulysin-Mediated Delivery of Granzymes

J. Lieberman, T. Kirchhausen, A. Krensky, 2014, Cell.

p53 reactivating small molecule PRIMA‑1MET/APR‑246 regulates genomic instability in MDA‑MB‑231 cells.

S. Chouaib, J. Thiery, B. Wojtaś, 2022, Oncology reports.

Role of p53 in the sensitization of tumor cells to apoptotic cell death.

F. Mami-Chouaib, S. Chouaib, J. Thiery, 2002, Molecular immunology.

[A novel caspase-independent apoptotic pathway triggered by Granzyme A].

D. Martinvalet, J. Thiery, 2008, Medecine sciences : M/S.

Mechanisms of Cytotoxic Lymphocyte-Mediated Apoptosis and Relationship with the Tumor Suppressor p53.

Linda Ziani, S. Chouaib, J. Thiery, 2015, Critical Reviews in Immunology.

Hypoxia as a potential inducer of immune tolerance, tumor plasticity and a driver of tumor mutational burden: Impact on cancer immunotherapy.

B. Janji, J. Thiery, P. Limani, 2023, Seminars in cancer biology.