C. Sunyach

发表

Loss of function of DJ-1 triggered by Parkinson's disease-associated mutation is due to proteolytic resistance to caspase-6

P. Heutink, J. Pouysségur, G. Pagès, 2010, Cell Death and Differentiation.

The α-Secretase-derived N-terminal Product of Cellular Prion, N1, Displays Neuroprotective Function in Vitro and in Vivo*

F. Checler, C. Sunyach, C. Druon, 2009, The Journal of Biological Chemistry.

The C-terminal Products of Cellular Prion Protein Processing, C1 and C2, Exert Distinct Influence on p53-dependent Staurosporine-induced Caspase-3 Activation*

F. Checler, C. Sunyach, C. A. da Costa, 2007, Journal of Biological Chemistry.

Combined pharmacological, mutational and cell biology approaches indicate that p53‐dependent caspase 3 activation triggered by cellular prion is dependent on its endocytosis

F. Checler, C. Sunyach, 2005, Journal of neurochemistry.

Primary Cultured Neurons Devoid of Cellular Prion Display Lower Responsiveness to Staurosporine through the Control of p53 at Both Transcriptional and Post-transcriptional Levels*

J. Bourdon, F. Checler, C. A. D. Alves da Costa, 2004, Journal of Biological Chemistry.

A Rare Motor Neuron Deleterious Missense Mutation in the DPYSL3 (CRMP4) Gene is Associated with ALS

B. Pettmann, P. Andersen, V. Meininger, 2013, Human mutation.

The mechanism of internalization of glycosylphosphatidylinositol‐anchored prion protein

C. Sunyach, J. Grassi, M. Mccaffrey, 2003, The EMBO journal.

Calreticulin levels determine onset of early muscle denervation by fast motoneurons of ALS model mice

B. Pettmann, T. Marissal, C. Sunyach, 2015, Neurobiology of Disease.

Reduced Calreticulin Levels Link Endoplasmic Reticulum Stress and Fas-Triggered Cell Death in Motoneurons Vulnerable to ALS

B. Pettmann, C. Henderson, C. Sunyach, 2012, The Journal of Neuroscience.

Presenilin-dependent transcriptional control of the Abeta-degrading enzyme neprilysin by intracellular domains of betaAPP and APLP.

F. Checler, C. A. D. Alves da Costa, C. Sunyach, 2005, Neuron.

The gamma/epsilon-secretase-derived APP intracellular domain fragments regulate p53.

P. S. St George-Hyslop, F. Checler, C. Sunyach, 2007, Current Alzheimer research.

APPε, the ε‐secretase‐derived N‐terminal product of the β‐amyloid precursor protein, behaves as a type I protein and undergoes α‐, β‐, and γ‐secretase cleavages

F. Checler, C. Sunyach, S. Lefranc-Jullien, 2006, Journal of neurochemistry.

Presenilin-Dependent (cid:1) -Secretase-Mediated Control of p53-Associated Cell Death in Alzheimer’s Disease

P. George-Hyslop, F. Checler, C. Sunyach, 2006 .

Enhanced neuronal Met signalling levels in ALS mice delay disease onset

B. Pettmann, C. Sunyach, C. Raoul, 2011, Cell Death and Disease.

The Disintegrin ADAM9 Indirectly Contributes to the Physiological Processing of Cellular Prion by Modulating ADAM10 Activity*

F. Checler, C. Sunyach, M. Cissé, 2005, Journal of Biological Chemistry.

(cid:1) -Secretase-derived Fragment of Cellular Prion, N1, Protects against Monomeric and Oligomeric Amyloid (cid:2) (A (cid:2) )-associated Cell Death

F. Checler, C. Sunyach, M. Guillot-Sestier, 2020 .

Environmental Health in Perinatal and Early Childhood: Awareness, Representation, Knowledge and Practice of Southern France Perinatal Health Professionals

C. Sunyach, S. Tardieu, J. Perrin, 2018, International journal of environmental research and public health.

Subfertile patients underestimate their risk factors of reprotoxic exposure

C. Sunyach, C. Metzler-Guillemain, J. Perrin, 2022, Basic and Clinical Andrology.

Protective and therapeutic effect of DNA-based immunization against hepadnavirus large envelope protein.

C. Sunyach, C. Trépo, L. Cova, 1999, Gastroenterology.

Phosphorylation of DHBV pre-S: identification of the major site of phosphorylation and effects of mutations on the virus life cycle.

C. Borel, C. Sunyach, C. Trépo, 1998, Virology.

Residues Critical for Duck Hepatitis B Virus Neutralization Are Involved in Host Cell Interaction

C. Borel, H. Will, C. Sunyach, 1999, Journal of Virology.

In vivo selection of duck hepatitis B virus pre-S variants which escape from neutralization.

C. Sunyach, C. Trépo, L. Cova, 1997, Virology.

Transcriptional repression of p53 by parkin and impairment by mutations associated with autosomal recessive juvenile Parkinson's disease

Patrick M. Abou-Sleiman, Nicholas W. Wood, Andrew West, 2009, Nature Cell Biology.

Presenilin-Dependent Transcriptional Control of the Aβ-Degrading Enzyme Neprilysin by Intracellular Domains of βAPP and APLP

F. Checler, C. Sunyach, C. A. D. Costa, 2005, Neuron.

The γ /η-Secretase-Derived APP Intracellular Domain Fragments Regulate p53

P. George-Hyslop, F. Checler, C. Sunyach, 2007 .

LRP1 controls biosynthetic and endocytic trafficking of neuronal prion protein

S. Moestrup, C. Oxvig, G. Bu, 2008, Journal of Cell Science.

Presenilin-Dependent γ-Secretase-Mediated Control of p53-Associated Cell Death in Alzheimer's Disease

P. S. St George-Hyslop, F. Checler, C. Sunyach, 2006, The Journal of Neuroscience.

Olesoxime delays muscle denervation, astrogliosis, microglial activation and motoneuron death in an ALS mouse model

B. Pettmann, C. Sunyach, C. Raoul, 2012, Neuropharmacology.

Optimised and Rapid Pre-clinical Screening in the SOD1G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis (ALS)

Aneurin J. Kennerley, Jason Berwick, Mimoun Azzouz, 2011, PloS one.

Caspase-3-derived C-terminal Product of Synphilin-1 Displays Antiapoptotic Function via Modulation of the p53-dependent Cell Death Pathway*

P. McLean, F. Checler, E. Giaime, 2006, Journal of Biological Chemistry.

α-Secretase-derived Fragment of Cellular Prion, N1, Protects against Monomeric and Oligomeric Amyloid β (Aβ)-associated Cell Death*

F. Checler, C. Sunyach, M. Guillot-Sestier, 2011, The Journal of Biological Chemistry.

M1 and M3 Muscarinic Receptors Control Physiological Processing of Cellular Prion by Modulating ADAM17 Phosphorylation and Activity

F. Checler, C. Sunyach, B. Vincent, 2007, The Journal of Neuroscience.

Response to Correspondence: Pardossi-Piquard et al., “Presenilin-Dependent Transcriptional Control of the Aβ-Degrading Enzyme Neprilysin by Intracellular Domains of βAPP and APLP.” Neuron 46, 541–554

P. George-Hyslop, J. Dunys, F. Checler, 2007, Neuron.

«Spiromed»: a rapid and inexpensive Spiroplasma isolation technique

C. Sunyach, I. Humphery-Smith, C. Chastel, 1993 .

p53-Dependent Transcriptional Control of Cellular Prion by Presenilins

P. S. St George-Hyslop, H. Orzechowski, F. Checler, 2009, The Journal of Neuroscience.

Presenilin-dependent gamma-secretase-mediated control of p53-associated cell death in Alzheimer's disease.

P. S. St George-Hyslop, F. Checler, C. Sunyach, 2006, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Regulation of βAPP and PrPc Cleavage by α-Secretase: Mechanistic and Therapeutic Perspectives

F. Checler, C. Sunyach, M. Guillot-Sestier, 2008 .

Regulation of betaAPP and PrPc cleavage by alpha-secretase: mechanistic and therapeutic perspectives.

F. Checler, C. Sunyach, M. Guillot-Sestier, 2008, Current Alzheimer research.

Somatic and axonal LIGHT signaling elicit degenerative and regenerative responses in motoneurons, respectively

M. Bowerman, C. Sunyach, C. Raoul, 2014, EMBO reports.

α-Secretase-derived fragment of cellular prion, N1, protects against monomeric and oligomeric amyloid β (Aβ)-associated cell death.

F. Checler, C. Sunyach, M. Guillot-Sestier, 2013, The Journal of Biological Chemistry.

Somatic and axonal LIGHT signaling elicit degenerative and regenerative responses in motoneurons, respectively

M. Bowerman, C. Sunyach, C. Raoul, 2014, EMBO reports.