V. Dolcini

发表

p53-dependent Aph-1 and Pen-2 Anti-apoptotic Phenotype Requires the Integrity of the γ-Secretase Complex but Is Independent of Its Activity*

P. George-Hyslop, J. Dunys, F. Checler, 2007, Journal of Biological Chemistry.

Apoptotic cell death influences the signaling activity of the amyloid precursor protein through ShcA and Grb2 adaptor proteins in neuroblastoma SH‐SY5Y cells

V. Dolcini, G. Schettini, M. Nizzari, 2004, Journal of neurochemistry.

Biological targets of neurotoxic pesticides analysed by alteration of developmental events in the Mediterranean sea urchin, Paracentrotus lividus.

V. Dolcini, P. Guidetti, C. Falugi, 2003, Marine environmental research.

Identification of Amino-Terminally and Phosphotyrosine-Modified Carboxy-Terminal Fragments of the Amyloid Precursor Protein in Alzheimer's Disease and Down's Syndrome Brain

V. Dolcini, G. Schettini, C. Russo, 2001, Neurobiology of Disease.

TMP21 regulates Abeta production but does not affect caspase-3, p53, and neprilysin.

P. S. St George-Hyslop, P. Fraser, Fusheng Chen, 2008, Biochemical and biophysical research communications.

Caulerpenyne interferes with microtubuledependent events during the first mitotic cycle of sea urchin eggs

V. Dolcini, J. Girard, P. Huitorel, 1998 .

Caulerpenyne interferes with microtubule-dependent events during the first mitotic cycle of sea urchin eggs.

V. Dolcini, J. Girard, P. Huitorel, 1998, European journal of cell biology.

Signal Transduction through Tyrosine‐Phosphorylated Carboxy‐Terminal Fragments of APP via an Enhanced Interaction with Shc/Grb2 Adaptor Proteins in Reactive Astrocytes of Alzheimer's Disease Brain

T. Russo, N. Zambrano, V. Dolcini, 2002, Annals of the New York Academy of Sciences.

Pyroglutamate‐modified amyloid β‐peptides – AβN3(pE) – strongly affect cultured neuron and astrocyte survival

G. Damonte, V. Dolcini, G. Schettini, 2002 .

Molecular aspects of neurodegeneration in Alzheimer's disease.

V. Dolcini, G. Schettini, C. Russo, 2002, Functional neurology.

Pyroglutamate-modified amyloid beta-peptides--AbetaN3(pE)--strongly affect cultured neuron and astrocyte survival.

G. Damonte, V. Dolcini, G. Schettini, 2002, Journal of neurochemistry.

Amino-terminal modification and tyrosine phosphorylation of [corrected] carboxy-terminal fragments of the amyloid precursor protein in Alzheimer's disease and Down's syndrome brain.

V. Dolcini, G. Schettini, C. Russo, 2001, Neurobiology of disease.

Signal Transduction through Tyrosine-phosphorylated C-terminal Fragments of Amyloid Precursor Protein via an Enhanced Interaction with Shc/Grb2 Adaptor Proteins in Reactive Astrocytes of Alzheimer's Disease Brain*

T. Russo, N. Zambrano, V. Dolcini, 2002, The Journal of Biological Chemistry.