J. W. Tullai

发表

Phosphatidylinositol 3-kinase signaling in proliferating cells maintains an anti-apoptotic transcriptional program mediated by inhibition of FOXO and non-canonical activation of NFκB transcription factors

Jolyon Terragni, G. Cooper, J. Graham, 2008, BMC Cell Biology.

Identification of Transcription Factor Binding Sites Upstream of Human Genes Regulated by the Phosphatidylinositol 3-Kinase and MEK/ERK Signaling Pathways*

S. Kasif, G. Cooper, Michael Schaffer, 2004, Journal of Biological Chemistry.

Immediate-Early and Delayed Primary Response Genes Are Distinct in Function and Genomic Architecture*

S. Kasif, G. Cooper, Michael Schaffer, 2007, Journal of Biological Chemistry.

A GSK-3-mediated transcriptional network maintains repression of immediate early genes in quiescent cells

G. Cooper, J. Graham, J. W. Tullai, 2011, Cell cycle.

Glycogen Synthase Kinase-3 Represses Cyclic AMP Response Element-binding Protein (CREB)-targeted Immediate Early Genes in Quiescent Cells*

S. Kasif, G. Cooper, Michael Schaffer, 2007, Journal of Biological Chemistry.

GSK-3 Represses Growth Factor-inducible Genes by Inhibiting NF-κB in Quiescent Cells*

G. Cooper, J. Graham, J. W. Tullai, 2009, The Journal of Biological Chemistry.

The association of metalloendopeptidase EC 3.4.24.15 at the extracellular surface of the AtT-20 cell plasma membrane

J. W. Tullai, James L. Roberts, P. Crack, 1999, Brain Research.

Thiol Activation of Endopeptidase EC 3.4.24.15

J. Roberts, J. W. Tullai, M. Glucksman, 1997, The Journal of Biological Chemistry.

Secretion of metalloendopeptidase 24.15 (EC 3.4.24.15).

J. Roberts, J. W. Tullai, M. Glucksman, 1999, DNA and cell biology.

The Neuropeptide Processing Enzyme EC 3.4.24.15 Is Modulated by Protein Kinase A Phosphorylation*

J. Martignetti, J. W. Tullai, M. Glucksman, 2000, The Journal of Biological Chemistry.

AP-1 Is a Component of the Transcriptional Network Regulated by GSK-3 in Quiescent Cells

G. Cooper, J. Graham, J. W. Tullai, 2011, PloS one.

Regulated expression during development and following sciatic nerve injury of mRNAs encoding the receptor tyrosine phosphatase HPTPzeta/RPTPbeta.

J. W. Tullai, W. H. Yu, J. Li, 1998, Brain research. Molecular brain research.

Phosphatidylinositol 3-kinase signaling in proliferating cells maintains an anti-apoptotic transcriptional program mediated by inhibition of FOXO and non-canonical activation of NFkappaB transcription factors.

Jolyon Terragni, Michael Schaffer, J. W. Tullai, 2008, BMC cell biology.

Phosphatidylinositol 3-Kinase Signaling in Proliferating Cells Maintains an Anti-Apoptotic Transcriptional Program Mediated by Inhibition of Foxo and Non-Canonical Activation of NFkB Transcription Factors

Jolyon Terragni, Michael Schaffer, J. W. Tullai, 2008 .

Bmc Cell Biology Phosphatidylinositol 3-kinase Signaling in Proliferating Cells Maintains an Anti-apoptotic Transcriptional Program Mediated by Inhibition of Foxo and Non-canonical Activation of Nfb Transcription Factors

Jolyon Terragni, Michael Schaffer, J. W. Tullai, 2007 .