B. Maiti

发表

E2f1, E2f2, and E2f3 Control E2F Target Expression and Cellular Proliferation via a p53-Dependent Negative Feedback Loop

Daniel A. Orringer, Baidehi Maiti, Nidhi Sharma, 2007, Molecular and Cellular Biology.

Inactivation of E2F3 results in centrosome amplification.

R. Altura, K. Fukasawa, H. Saavedra, 2003, Cancer cell.

3-07-24 Study on cerebral venous thrombosis with special reference to efficacy of heparin

B. Maiti, I. Chakrabarti, 1997, Journal of the Neurological Sciences.

The association of metabolic syndrome with triple-negative breast cancer

B. Maiti, T. Spiro, H. Daw, 2010, Breast Cancer Research and Treatment.

Hepatic iron overload, a possible consequence of treatment with imatinib mesylate: a case report

B. Maiti, H. Daw, S. Setrakian, 2009, Cases journal.

E2f1, E2f2, and E2f3 Control E2F Target Expression and Cellular Proliferation via a p53-Dependent Negative Feedback Loop

D. Orringer, H. Saavedra, C. Timmers, 2012, Molecular and Cellular Biology.

Identification and Characterization of E2F7, a Novel Mammalian E2F Family Member Capable of Blocking Cellular Proliferation*

A. de Bruin, C. Timmers, B. Maiti, 2003, Journal of Biological Chemistry.

essential for cellular proliferation

J. Nevins, M. Greenberg, S. Orkin, 2001 .

Cloning and Characterization of Mouse E2F8, a Novel Mammalian E2F Family Member Capable of Blocking Cellular Proliferation*

A. de Bruin, C. Timmers, R. Opavsky, 2005, Journal of Biological Chemistry.

The E2F1–3 transcription factors are essential for cellular proliferation

J. Nevins, M. Greenberg, S. Orkin, 2001, Nature.

E2F-8: an E2F family member with a similar organization of DNA-binding domains to E2F-7

B. Maiti, G. Leone, N. L. Thangue, 2005, Oncogene.