D. Huynh

S. Pulst, D. Huynh, T. Nechiporuk, 1996, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

Nuclear localization or inclusion body formation of ataxin-2 are not necessary for SCA2 pathogenesis in mouse or human

S. Pulst, D. Huynh, K. Figueroa, 2000, Nature Genetics.

Cellular distribution of torsin A and torsin B in normal human brain.

S. Pulst, D. Huynh, W. Yong, 2001, Archives of neurology.

The neurofibromatosis 2 tumor suppressor protein interacts with hepatocyte growth factor-regulated tyrosine kinase substrate.

S. Pulst, D. Gutmann, D. Huynh, 2000, Human molecular genetics.

Deranged Calcium Signaling and Neurodegeneration in Spinocerebellar Ataxia Type 2

S. Pulst, I. Bezprozvanny, D. Huynh, 2009, The Journal of Neuroscience.

Immunohistochemical Detection of Schwannomin and Neurofibromin in Vestibular Schwannomas, Ependymomas and Meningiomas

S. Pulst, D. Huynh, V. Mautner, 1997, Journal of neuropathology and experimental neurology.

Expression of ataxin‐2 in brains from normal individuals and patients with Alzheimer's disease and spinocerebellar ataxia 2

S. Pulst, M. D. Del Bigio, D. Huynh, 1999, Annals of neurology.

Dissociated Fear and Spatial Learning in Mice with Deficiency of Ataxin-2

Alcino J. Silva, S. Pulst, D. Huynh, 2009, PloS one.

Generation and characterization of Sca2 (ataxin-2) knockout mice.

Tim-Rasmus Kiehl, Alex Nechiporuk, S. Pulst, 2006, Biochemical and biophysical research communications.

Expansion of the polyQ repeat in ataxin-2 alters its Golgi localization, disrupts the Golgi complex and causes cell death.

Stefan M Pulst, Hai-Tao Yang, S. Pulst, 2003, Human molecular genetics.

Generation of SNCA Cell Models Using Zinc Finger Nuclease (ZFN) Technology for Efficient High-Throughput Drug Screening

S. Pulst, D. Huynh, D. Scoles, 2015, PloS one.

A novel protein with RNA-binding motifs interacts with ataxin-2.

S. Pulst, D. Huynh, H. Shibata, 2000, Human molecular genetics.

Ataxin-2 mediated cell death is dependent on domains downstream of the polyQ repeat

S. Pulst, D. Huynh, Hiushan Ng, 2007, Experimental Neurology.

Identification and expression of a mouse ortholog of A2BP1

S. Pulst, T. Kiehl, D. Huynh, 2001, Mammalian Genome.

The autosomal recessive juvenile Parkinson disease gene product, parkin, interacts with and ubiquitinates synaptotagmin XI.

S. Pulst, D. Huynh, D. Scoles, 2003, Human molecular genetics.

Spinocerebellar ataxia type 2: polyQ repeat variation in the CACNA1A calcium channel modifies age of onset.

Luis Velazquez, S. Pulst, D. Huynh, 2005, Brain : a journal of neurology.

Screening for mutations in synaptotagmin XI in Parkinson's disease.

S. Pulst, R. Krüger, D. Berg, 2004, Journal of neural transmission. Supplementum.

The mouse SCA2 gene: cDNA sequence, alternative splicing and protein expression.

S. Pulst, D. Huynh, T. Nechiporuk, 1998, Human molecular genetics.

Parkin is associated with actin filaments in neuronal and nonneural cells

S. Pulst, M. D. Del Bigio, D. Huynh, 2000, Annals of neurology.

The AKT modulator A-443654 reduces α-synuclein expression and normalizes ER stress and autophagy

S. Pulst, A. Zakharov, A. Jadhav, 2021, The Journal of biological chemistry.

Spinocerebellar ataxia type 2: polyQ repeat variation in the CACNA1A calcium channel modifies age of onset.

S. Pulst, D. Huynh, K. Figueroa, 2005, Brain : a journal of neurology.

KCNC3R420H , a K+ channel mutation causative in spinocerebellar ataxia 13 displays aberrant intracellular trafficking

S. Pulst, D. Rincon-Limas, D. Huynh, 2014, Neurobiology of Disease.