G. Cieslewicz

发表

Anti-interleukin 5 but not anti-IgE prevents airway inflammation and airway hyperresponsiveness.

T. Ishizuka, E. Gelfand, E. Hamelmann, 1999, American journal of respiratory and critical care medicine.

Local treatment with IL-12 is an effective inhibitor of airway hyperresponsiveness and lung eosinophilia after airway challenge in sensitized mice.

E. Gelfand, E. Hamelmann, G. Cieslewicz, 1998, The Journal of allergy and clinical immunology.

Temporal association between airway hyperresponsiveness and airway eosinophilia in ovalbumin-sensitized mice.

E. Gelfand, G. Cieslewicz, C. Duez, 2001, American journal of respiratory and critical care medicine.

Hyperresponsiveness Eosinophilia and Airway Prevents Antigen-Induced Airway Inhibits IL-4-and IL-13-Induced Responses A Murine IL-4 Receptor Antagonist

J. C. Pratt, E. Gelfand, G. Cieslewicz, 2001 .

A Murine IL-4 Receptor Antagonist That Inhibits IL-4- and IL-13-Induced Responses Prevents Antigen-Induced Airway Eosinophilia and Airway Hyperresponsiveness1

J. C. Pratt, E. Gelfand, G. Cieslewicz, 2001, The Journal of Immunology.

Pathologies in Mouse Models of Asthma Contribute to Allergen-Induced Airway Eosinophil Major Basic Protein-1 Does Not

James J. Lee, G. Cieslewicz, M. Borchers, 2000 .

Eosinophil Major Basic Protein-1 Does Not Contribute to Allergen-Induced Airway Pathologies in Mouse Models of Asthma1

James J. Lee, G. Cieslewicz, M. Borchers, 2000, The Journal of Immunology.

Extensive Eosinophil Degranulation and Peroxidase-Mediated Oxidation of Airway Proteins Do Not Occur in a Mouse Ovalbumin-Challenge Model of Pulmonary Inflammation1

S. Hazen, James J. Lee, G. Cieslewicz, 2001, The Journal of Immunology.

InflammationOvalbumin-Challenge Model of Pulmonary Proteins Do Not Occur in a Mouse Peroxidase-Mediated Oxidation of Airway Extensive Eosinophil Degranulation and

S. Hazen, James J. Lee, G. Cieslewicz, 2001 .

The late, but not early, asthmatic response is dependent on IL-5 and correlates with eosinophil infiltration.

G. Cieslewicz, J. Schwarze, A. Adler, 1999, The Journal of clinical investigation.

Anti-CD86 (B7.2) treatment abolishes allergic airway hyperresponsiveness in mice.

E. Hamelmann, G. Cieslewicz, A. Joetham, 1999, American journal of respiratory and critical care medicine.

Eosinophilia and Airway Hyperresponsiveness of Respiratory Syncytial Virus-Induced Lung CD 8 T Cells Are Essential in the Development

Gelfand, E. Hamelmann, G. Cieslewicz, 1999 .

Infection Following Acute Respiratory Syncytial Virus Development of Airway Hyperresponsiveness IL-5 and Eosinophils Are Essential for the and

E. Gelfand, E. Hamelmann, G. Cieslewicz, 1999 .

CD8 T cells are essential in the development of respiratory syncytial virus-induced lung eosinophilia and airway hyperresponsiveness.

E. Gelfand, E. Hamelmann, G. Cieslewicz, 1999, Journal of immunology.

The failure of STAT6-deficient mice to develop airway eosinophilia and airway hyperresponsiveness is overcome by interleukin-5.

E. Gelfand, N. Rabinovitch, G. Cieslewicz, 1999, American journal of respiratory and critical care medicine.

Early Phase Bronchoconstriction in the Mouse Requires Allergen-Specific IgG1

James J. Lee, G. Cieslewicz, M. Borchers, 2002, The Journal of Immunology.

Mouse Requires Allergen-Specific IgG Early Phase Bronchoconstriction in the

James J. Lee, G. Cieslewicz, M. Borchers, 2002 .

Eotaxin-1-Deficient Mice Develop Airway Eosinophilia and Airway Hyperresponsiveness

E. Gelfand, G. Cieslewicz, C. Duez, 2001, International Archives of Allergy and Immunology.

IL-5 and eosinophils are essential for the development of airway hyperresponsiveness following acute respiratory syncytial virus infection.

E. Gelfand, E. Hamelmann, G. Cieslewicz, 1999, Journal of immunology.

[Course of non-specific bronchial reactivity to histamine after bronchospasm induced by allergen challenge in patients with bronchial asthma].

G. Cieslewicz, I. Grzelewska-Rzymowska, J. Rozniecki, 1995, Pneumonologia i alergologia polska.

Antigen-specific immunoglobulin-A prevents increased airway responsiveness and lung eosinophilia after airway challenge in sensitized mice.

E. Gelfand, E. Hamelmann, G. Cieslewicz, 1998, American journal of respiratory and critical care medicine.

Critical roles for interleukin-4 and interleukin-5 during respiratory syncytial virus infection in the development of airway hyperresponsiveness after airway sensitization.

M. Mäkelä, E. Gelfand, G. Cieslewicz, 2000, American journal of respiratory and critical care medicine.

Transfer of the enhancing effect of respiratory syncytial virus infection on subsequent allergic airway sensitization by T lymphocytes.

M. Mäkelä, E. Gelfand, G. Cieslewicz, 1999, Journal of immunology.

Assessment of IgE allergen specificity among latex-allergic health care workers: review of IgE-binding components of various latex extracts.

A. Sheffer, G. Cieslewicz, H. Nolte, 2000, Annals of allergy, asthma & immunology : official publication of the American College of Allergy, Asthma, & Immunology.

Clinical Assessment of Latex Allergy among Hospital Health Care Providers

A. Sheffer, G. Cieslewicz, H. Nolte, 1999, International Archives of Allergy and Immunology.

From Molecular Science to the Treatment of Allergy

M. Manns, Jane Wilkinson, P. Hufnagl, 1999, International Archives of Allergy and Immunology.

Interleukin (IL)-5 but not immunoglobulin E reconstitutes airway inflammation and airway hyperresponsiveness in IL-4-deficient mice.

E. Gelfand, Q. Hamid, E. Hamelmann, 2000, American journal of respiratory cell and molecular biology.

Inhaled corticosteroid improves bronchial reactivity and decreases symptoms in patients with mitral stenosis.

M. Zembala, E. Gelfand, E. Hamelmann, 1998, Chest.