D. Burgess

发表

Long QT Syndrome Type 2: Emerging Strategies for Correcting Class 2 KCNH2 (hERG) Mutations and Identifying New Patients

C. January, C. Elayi, C. Anderson, 2020, Biomolecules.

Coupling of sympathetic nerve traffic and BP at very low frequencies is mediated by large-amplitude events.

D. Randall, D. Burgess, R. Speakman, 2003, American journal of physiology. Regulatory, integrative and comparative physiology.

First-order differential-delay equation for the baroreflex predicts the 0.4-Hz blood pressure rhythm in rats.

D. Randall, D. Burgess, David R. Brown, 1997, American journal of physiology. Regulatory, integrative and comparative physiology.

Steady-state coupling of plasma membrane calcium entry to extrusion revealed by novel L-type calcium channel block.

D. Burgess, E. Schroder, D. Andres, 2008, Cell calcium.

Title: Blood Pressure Power within Frequency Range around 0.4 Hz in Rat Conforms to Self-Similar Scaling following Spinal Cord Transection Authors:

D. Randall, D. Burgess, A. Patwardhan, 2004 .

Heart rate-arterial blood pressure relationship in conscious rat before vs. after spinal cord transection.

Robert F. Taylor, D. Randall, D. Burgess, 2002, American journal of physiology. Regulatory, integrative and comparative physiology.

A KCNQ1 mutation contributes to the concealed type 1 long QT phenotype by limiting the Kv7.1 channel conformational changes associated with protein kinase A phosphorylation.

Daniel C Bartos, D. Tester, M. Ackerman, 2014, Heart rhythm.

Current Opinion in Pharmacology: Circadian Clocks Regulate Cardiac Arrhythmia Susceptibility, Repolarization, and Ion Channels

B. Delisle, D. Burgess, E. Schroder, 2021 .

Functional Invalidation of Putative Sudden Infant Death Syndrome–Associated Variants in the KCNH2-Encoded Kv11.1 Channel

Dustin N. Hartzel, C. January, M. Murray, 2018, Circulation. Arrhythmia and electrophysiology.

The cardiomyocyte molecular clock regulates the circadian expression of Kcnh2 and contributes to ventricular repolarization.

Daniel C Bartos, K. Esser, C. Elayi, 2015, Heart rhythm.

Mechanistic Basis for Type 2 Long QT Syndrome Caused by KCNH2 Mutations that Disrupt Conserved Arginine Residues in the Voltage Sensor

Daniel C Bartos, Ashley M. Smith, C. Elayi, 2013, The Journal of Membrane Biology.

Mutation-Specific Differences in Kv7.1 (KCNQ1) and Kv11.1 (KCNH2) Channel Dysfunction and Long QT Syndrome Phenotypes

Daniel C Bartos, C. January, C. Anderson, 2022, International journal of molecular sciences.

Cardiomyocyte Deletion of Bmal1 Exacerbates QT- and RR-Interval Prolongation in Scn5a+/ΔKPQ Mice

K. Esser, C. Elayi, B. Delisle, 2021, Frontiers in Physiology.

Light phase-restricted feeding slows basal heart rate to exaggerate the type-3 long QT syndrome phenotype in mice.

A. Moss, K. Esser, C. Elayi, 2014, American journal of physiology. Heart and circulatory physiology.

The Sympathetic Nervous System ’ s Role in Regulating Blood Pressure Variability Understanding How SNA Controls Vascular Tone Is an Important Step in Determining the Pathogenesis of Hypertension

M. Navakatikyan, J. Ringwood, D. Burgess, 2001 .

Computer simulation of healthy subjects and heart failure patients Baroreflex sensitivity, blood pressure buffering, and resonance: what are the links?

J. Saul, M. Schalij, D. Randall, 2007 .

First-order differential-delay equation for the baroreflex predicts the 0.4-Hz blood pressure rhythm in rats.

D. Randall, D. Burgess, J. C. Hundley, 1997, The American journal of physiology.

Circadian clocks regulate cardiac arrhythmia susceptibility, repolarization, and ion channels.

B. Delisle, M. Ono, D. Burgess, 2020, Current opinion in pharmacology.

Extended longitudinal analysis of arterial pressure and heart rate control in unanesthetized rats with type 1 diabetes

D. Randall, D. Burgess, A. Patwardhan, 2012, Autonomic Neuroscience.

Longitudinal Analysis of Arterial Blood Pressure and Heart Rate Response to Acute Behavioral Stress in Rats with Type 1 Diabetes Mellitus and in Age-Matched Controls

D. Randall, D. Burgess, A. Patwardhan, 2011, Front. Physio..

Blood pressure power within frequency range ∼0.4 Hz in rat conforms to self-similar scaling following spinal cord transection

Robert F. Taylor, D. Randall, D. Burgess, 2005 .

The sympathetic nervous system's role in regulating blood pressure variability

M. Navakatikyan, S. Malpas, J. Ringwood, 2001, IEEE Engineering in Medicine and Biology Magazine.

Renal SNA as the primary mediator of slow oscillations in blood pressure during hemorrhage.

S. Malpas, D. Burgess, S C Malpas, 2000, American journal of physiology. Heart and circulatory physiology.

The role of the cardiomyocyte circadian clocks in ion channel regulation and cardiac electrophysiology

K. Esser, B. Delisle, M. Ono, 2022, The Journal of physiology.

Timing of food intake in mice unmasks a role for the cardiomyocyte circadian clock mechanism in limiting QT-interval prolongation

K. Esser, C. Elayi, B. Delisle, 2021, Chronobiology international.

Molecular pathogenesis of long QT syndrome type 2

C. January, C. Elayi, C. Anderson, 2016, Journal of arrhythmia.

Multifiber renal SNA recordings predict mean arterial blood pressure in unanesthetized rat.

D. Randall, D. Burgess, J. C. Hundley, 1997, The American journal of physiology.

Low-frequency renal sympathetic nerve activity, arterial BP, stationary "1/f noise," and the baroreflex.

D. Randall, D. Burgess, D. Brown, 1999, The American journal of physiology.

Low-frequency renal sympathetic nerve activity, arterial BP, stationary "1/ f noise," and the baroreflex.

D. Randall, D. Burgess, David R. Brown, 1999, American journal of physiology. Regulatory, integrative and comparative physiology.

Title : Coupling of Sympathetic Nerve Traffic and BP at Very Low Frequencies is Mediated by Large Amplitude Events Authors :

D. Randall, D. Burgess, R. Speakman, 2002 .

Mechanism of inactivation gating of human T-type (low-voltage activated) calcium channels.

B. Delisle, D. Burgess, J. Satin, 2002, Biophysical journal.

Mechanistic Basis for Type 2 Long QT Syndrome Caused by KCNH2 Mutations that Disrupt Conserved Arginine Residues in the Voltage Sensor

Daniel C Bartos, Ashley M. Smith, C. Elayi, 2013, The Journal of Membrane Biology.

Chronic verapamil treatment remodels ICa,L in mouse ventricle.

D. Burgess, E. Schroder, D. Andres, 2007, American journal of physiology. Heart and circulatory physiology.

A KCNQ1 Mutation Causes a High Penetrance for Familial Atrial Fibrillation

Daniel C Bartos, E. Behr, D. Tester, 2013, Journal of cardiovascular electrophysiology.

High-risk long QT syndrome mutations in the Kv7.1 (KCNQ1) pore disrupt the molecular basis for rapid K(+) permeation.

Daniel C Bartos, A. Moss, K. Campbell, 2012, Biochemistry.

R231C mutation in KCNQ1 causes long QT syndrome type 1 and familial atrial fibrillation.

Michael John Ackerman, Craig T. January, Brian P. Delisle, 2011, Heart rhythm.

A KCNQ1 Mutation Causes a High Penetrance for Familial Atrial Fibrillation

Daniel C Bartos, E. Behr, D. Tester, 2013, Journal of cardiovascular electrophysiology.

Circadian Regulation of Cardiac Arrhythmias and Electrophysiology

B. Delisle, D. Burgess, Abhilash Prabhat, 2024, Circulation research.