D. Prou

Evolution and cell biology of dopamine receptors in vertebrates

J. Vincent, P. Vernier, S. Callier, 2003, Biology of the cell.

α-Synuclein Overexpression Increases Cytosolic Catecholamine Concentration

D. Sulzer, R. Edwards, E. Mosharov, 2006, The Journal of Neuroscience.

Autocrine activation of adenosine A1 receptors blocks D1A but not D1B dopamine receptor desensitization

P. Vernier, D. Prou, S. Le Crom, 2002, Journal of neurochemistry.

Oral noribogaine shows high brain uptake and anti-withdrawal effects not associated with place preference in rodents

D. Mash, D. Prou, Emeline L. Maillet, 2016, Journal of psychopharmacology.

Toxin-induced models of Parkinson’s disease

D. Prou, C. Perier, S. Przedborski, 2005, NeuroRX.

Metabolism of 3-Nitrotyrosine Induces Apoptotic Death in Dopaminergic Cells

S. Hazen, D. Prou, S. Przedborski, 2006, The Journal of Neuroscience.

The kinase domain of mitochondrial PINK1 faces the cytoplasm

E. Schon, D. Prou, C. Perier, 2008, Proceedings of the National Academy of Sciences.

Modulation of dopamine transporter function by α‐synuclein is altered by impairment of cell adhesion and by induction of oxidative stress

A. Sidhu, P. Vernier, D. Prou, 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Mutations in the lipid-binding domain of α-synuclein confer overlapping, yet distinct, functional properties in the regulation of dopamine transporter activity

A. Sidhu, H. Niznik, P. Vernier, 2003, Molecular and Cellular Neuroscience.

Neuronal NOS and cyclooxygenase-2 contribute to DNA damage in a mouse model of Parkinson disease.

M. Chesselet, M. Vila, T. Dawson, 2009, Free radical biology & medicine.

Intracellular retention of the two isoforms of the D(2) dopamine receptor promotes endoplasmic reticulum disruption.

J. Salamero, J. Vincent, P. Vernier, 2001, Journal of cell science.