M. Huang

发表

NLRP 3 Plays a Critical Role in the Development of Experimental Autoimmune Encephalomyelitis by Mediating Th 1 and Th 17 Responses

Denis Gris, Z. Ye, H. Iocca, 2010 .

NLRP3 Plays a Critical Role in the Development of Experimental Autoimmune Encephalomyelitis by Mediating Th1 and Th17 Responses

Monika Schneider, Denis Gris, Z. Ye, 2010, The Journal of Immunology.

Fatty acid–induced NLRP3-ASC inflammasome activation interferes with insulin signaling

Denis Gris, Haitao Wen, M. Huang, 2011, Nature Immunology.

NLRP3 (NALP3, Cryopyrin) Facilitates In Vivo Caspase-1 Activation, Necrosis, and HMGB1 Release via Inflammasome-Dependent and -Independent Pathways1

M. Huang, J. Ting, I. C. Allen, 2009, The Journal of Immunology.

NLRP3 (NALP3, cryopyrin) facilitates in vivo caspase-1, necrosis, & HMGB1 release via inflammasome-dependent and – independent pathways 1

M. Huang, I. C. Allen, D. Bergstralh, 2013 .

Lung Injury and Morbidity in Mice NLRP1-Dependent Pyroptosis Leads to Acute

M. Huang, J. Snouwaert, J. Ting, 2012 .

NLRC5-dependent activation of the inflammasome

Brianne R. Barker, Conti, M. Huang, 2013 .

Cutting Edge: NLRC5-Dependent Activation of the Inflammasome

Brianne R. Barker, M. Accavitti-Loper, M. Huang, 2011, The Journal of Immunology.

of the Inflammasome Cutting Edge: NLRC5-Dependent Activation

Stephen, M. Huang, Mildred Kwan, 2022 .

NLRP3 Plays a Critical Role in the Development of Experimental Autoimmune Encephalomyelitis by Mediating Th1 and Th17 Responses

Denis Gris, Z. Ye, H. Iocca, 2010, The Journal of Immunology.

in Human Cells Porphyromonas gingivalis ASC Speck Formation Induced by (ASC) and NLRP3 in Causing Necrosis and Containing a Caspase Recruitment Domain Critical Role of Apoptotic Speck Protein

M. Huang, J. Ting, R. Arnold, 2009 .

Porphyromonas gingivalis Mediates Inflammasome Repression in Polymicrobial Cultures through a Novel Mechanism Involving Reduced Endocytosis*

Haitao Wen, M. Huang, J. Ting, 2012, The Journal of Biological Chemistry.

NLRP3 (NALP3, Cryopyrin) Facilitates In Vivo Caspase-1 Activation, Necrosis, and HMGB1 Release via Inflammasome-Dependent and -Independent Pathways1

M. Huang, J. Ting, I. C. Allen, 2009, The Journal of Immunology.

Short hairpin RNA (shRNA): design, delivery, and assessment of gene knockdown.

M. Huang, C. Moore, E. Guthrie, 2010, Methods in molecular biology.

NLRP1-Dependent Pyroptosis Leads to Acute Lung Injury and Morbidity in Mice

B. Koller, M. Huang, J. Ting, 2012, The Journal of Immunology.

Inflammasome inhibition as a pathogenic stealth mechanism.

M. Huang, J. Ting, D. Taxman, 2010, Cell host & microbe.

The NLR Adaptor ASC/PYCARD Regulates DUSP10, Mitogen-activated Protein Kinase (MAPK), and Chemokine Induction Independent of the Inflammasome*

Denis Gris, M. Huang, J. Ting, 2011, The Journal of Biological Chemistry.

Critical Roles of Inflammasome Components NLRs, PYCARD, and Caspase-1 in Host - Pathogen Interactions

M. Huang, 2010 .

Neisseria gonorrhoeae Activates the Proteinase Cathepsin B to Mediate the Signaling Activities of the NLRP3 and ASC-Containing Inflammasome1

M. Huang, J. Ting, B. O’Connor, 2009, The Journal of Immunology.

Critical Role of Apoptotic Speck Protein Containing a Caspase Recruitment Domain (ASC) and NLRP3 in Causing Necrosis and ASC Speck Formation Induced by Porphyromonas gingivalis in Human Cells1

M. Huang, J. Ting, R. Arnold, 2009, The Journal of Immunology.

Deletion of ripA Alleviates Suppression of the Inflammasome and MAPK by Francisella tularensis

Denis Gris, Haitao Wen, Robin R. Craven, 2010, The Journal of Immunology.

Effects of the Putative Transcriptional Regulator IclR on Francisella tularensis Pathogenesis

M. Huang, Cheryl N. Miller, T. Kawula, 2010, Infection and Immunity.

NLRP 3 Plays a Critical Role in the Development of Experimental Autoimmune Encephalomyelitis by Mediating Th 1 and Th 17 Responses

Denis Gris, Z. Ye, H. Iocca, 2010 .