I. Grgic

I. Grgic, C. Busch, R. Köhler, 2007, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

Mitogenic modulation of Ca2+‐activated K+ channels in proliferating A7r5 vascular smooth muscle cells

I. Grgic, R. Köhler, J. Hoyer, 2006, British journal of pharmacology.

Blockade of T-lymphocyte KCa3.1 and Kv1.3 channels as novel immunosuppression strategy to prevent kidney allograft rejection.

I. Grgic, H. Wulff, R. Köhler, 2009, Transplantation proceedings.

Disruption of the Gardos channel (KCa3.1) in mice causes subtle erythrocyte macrocytosis and progressive splenomegaly

I. Grgic, B. P. Kaistha, C. Busch, 2009, Pflügers Archiv - European Journal of Physiology.

Endothelial Ca2+‐activated K+ channels in normal and impaired EDHF–dilator responses – relevance to cardiovascular pathologies and drug discovery

I. Grgic, B. P. Kaistha, R. Köhler, 2009, British journal of pharmacology.

Evidence for a Functional Role of Endothelial Transient Receptor Potential V4 in Shear Stress–Induced Vasodilatation

I. Grgic, C. Busch, R. Köhler, 2006, Arteriosclerosis, thrombosis, and vascular biology.

Disease modeling in genetic kidney diseases: mice

I. Grgic, S. Weber, M. Kömhoff, 2017, Cell and Tissue Research.

Development of a new macrophage-specific TRAP mouse (MacTRAP) and definition of the renal macrophage translational signature

I. Grgic, H. Gröne, J. Hoyer, 2020, Scientific Reports.

Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

Vanesa Bijol, Joseph V. Bonventre, J. Bonventre, 2012, Kidney international.

Discovery of new glomerular disease-relevant genes by translational profiling of podocytes in vivo

G. Genovese, I. Grgic, B. Humphreys, 2014, Kidney international.

Imaging of podocyte foot processes by fluorescence microscopy.

J. Bonventre, I. Grgic, C. Brooks, 2012, Journal of the American Society of Nephrology : JASN.

Disease modeling in genetic kidney diseases: mice

I. Grgic, S. Weber, M. Kömhoff, 2017, Cell and Tissue Research.

Erratum: Cell-specific translational profiling in acute kidney injury (Journal of Clinical Investigation (2014) 124:3 (1242-1254) DOI:10.1172/ JCI72126)

A. McMahon, Winston A Hide, S. Sui, 2014 .

Genetic Deficit of SK3 and IK1 Channels Disrupts the Endothelium-Derived Hyperpolarizing Factor Vasodilator Pathway and Causes Hypertension

J. Adelman, I. Grgic, B. P. Kaistha, 2009, Circulation.

Impaired Endothelium-Derived Hyperpolarizing Factor–Mediated Dilations and Increased Blood Pressure in Mice Deficient of the Intermediate-Conductance Ca2+-Activated K+ Channel

V. Gross, I. Grgic, R. Köhler, 2006, Circulation research.

I. Grgic, C. Busch, J. Hoyer, 2008, Cardiovascular research.

THEMED SECTION: ENDOTHELIUM IN PHARMACOLOGY REVIEW Endothelial Ca2+-activated K+ channels in normal and impaired EDHF–dilator responses – relevance to cardiovascular pathologies and drug discovery

I. Grgic, B. P. Kaistha, R. Köhler, 2009 .

Chronic epithelial kidney injury molecule-1 expression causes murine kidney fibrosis.

A. McMahon, V. Kuchroo, S. Xiao, 2013, The Journal of clinical investigation.