C. Busch

发表

Renal fibrosis is attenuated by targeted disruption of KCa3.1 potassium channels

I. Grgic, É. Kiss, B. P. Kaistha, 2009, Proceedings of the National Academy of Sciences.

I. Grgic, C. Busch, R. Köhler, 2008, Cardiovascular research.

Genetic Deficit of SK3 and IK1 Channels Disrupts the Endothelium-Derived Hyperpolarizing Factor Vasodilator Pathway and Causes Hypertension

J. Adelman, I. Grgic, B. P. Kaistha, 2009, Circulation.

Evidence for functional and dynamic microcompartmentation of Cav-1/TRPV4/K(Ca) in caveolae of endothelial cells.

I. Grgic, C. Busch, Anuradha Kaistha, 2015, European journal of cell biology.

Hyperkalaemia in a tetraplegic adolescent due to de novo sodium channel mutation.

I. Grgic, C. Busch, R. Köhler, 2007, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

Disruption of the Gardos channel (KCa3.1) in mice causes subtle erythrocyte macrocytosis and progressive splenomegaly

I. Grgic, B. P. Kaistha, C. Busch, 2009, Pflügers Archiv - European Journal of Physiology.

Naphtho[1,2-d]thiazol-2-ylamine (SKA-31), a New Activator of KCa2 and KCa3.1 Potassium Channels, Potentiates the Endothelium-Derived Hyperpolarizing Factor Response and Lowers Blood Pressure

C. Busch, G. Raman, H. Wulff, 2009, Molecular Pharmacology.

Rings of anionic amino acids as structural determinants of ion selectivity in the acetylcholine receptor channel

B. Sakmann, J. Nakai, M. Mishina, 1991, Proceedings of the Royal Society of London. Series B: Biological Sciences.

Rings of negatively charged amino acids determine the acetylcholine receptor channel conductance

B. Sakmann, J. Nakai, Y. Mori, 1988, Nature.

Evidence for a Functional Role of Endothelial Transient Receptor Potential V4 in Shear Stress–Induced Vasodilatation

I. Grgic, C. Busch, R. Köhler, 2006, Arteriosclerosis, thrombosis, and vascular biology.

SKA-31, A New Activator of KCa2 And KCa3.1 Potassium Channels, Potentiates the EDHF Response and Lowers Blood Pressure

C. Busch, G. Raman, H. Wulff, 2009 .

Genetic Deficit of SK3 and IK1 Channels Disrupts the Endothelium-Derived Hyperpolarizing Factor Vasodilator Pathway and Causes Hypertension

J. Adelman, I. Grgic, B. P. Kaistha, 2009, Circulation.

Naphtho[1,2-d]thiazol-2-ylamine (SKA-31), a New Activator of KCa2 and KCa3.1 Potassium Channels, Potentiates the Endothelium-Derived Hyperpolarizing Factor Response and Lowers Blood Pressure

C. Busch, G. Raman, H. Wulff, 2009, Molecular Pharmacology.

I. Grgic, C. Busch, J. Hoyer, 2008, Cardiovascular research.

Abstract of the 68th Meeting (Spring Meeting) 6–9 March 1990, Heidelberg

A. Konnerth, B. Sakmann, P. Jonas, 1990, Pflügers Archiv.