B. Siroky

发表

Clinical and Molecular Insights into Tuberous Sclerosis Complex Renal Disease

B. Siroky, Hong Yin, J. Bissler, 2011, Pediatric Nephrology.

Human TSC-associated renal angiomyolipoma cells are hypersensitive to ER stress.

B. Siroky, Hong Yin, J. Bissler, 2012, American journal of physiology. Renal physiology.

Impaired ability of the Na+/Ca2+ exchanger from the Dahl/Rapp salt-sensitive rat to regulate cytosolic calcium.

P. D. Bell, E. Schwiebert, B. Siroky, 2003, American journal of physiology. Renal physiology.

Cystin localizes to primary cilia via membrane microdomains and a targeting motif.

J. Kappes, A. Kispert, L. Guay-Woodford, 2009, Journal of the American Society of Nephrology : JASN.

Hyperglycemia in the absence of cilia accelerates cystogenesis and induces renal damage.

P. D. Bell, M. Zile, C. Baicu, 2015, American journal of physiology. Renal physiology.

The TRPP2-dependent channel of renal primary cilia also requires TRPM3

B. Siroky, Lu Lu, B. Dixon, 2019, PloS one.

Improvement in Renal Cystic Disease of Tuberous Sclerosis Complex After Treatment with Mammalian Target of Rapamycin Inhibitor.

B. Siroky, B. Dixon, A. Towbin, 2017, The Journal of pediatrics.

Tuberous sclerosis complex, mTOR, and the kidney: report of an NIDDK-sponsored workshop.

R. Rasooly, E. Henske, B. Siroky, 2014, American journal of physiology. Renal physiology.

Glomerulocystic kidney disease

B. Siroky, Hong Yin, J. Bissler, 2010, Pediatric Nephrology.

Tuberin Regulates Prostaglandin Receptor–Mediated Viability, via Rheb, in mTORC1-Hyperactive Cells

D. Kwiatkowski, D. Plas, B. Siroky, 2017, Molecular Cancer Research.

Collecting duct cells that lack normal cilia have mislocalized vasopressin-2 receptors.

B. Siroky, R. Fenton, Takamitsu Saigusa, 2012, American journal of physiology. Renal physiology.

Tsc2 mutation induces renal tubular cell nonautonomous disease

B. Siroky, J. Bissler, P. Vogel, 2021, Genes & diseases.

Vps34-mediated macropinocytosis in Tuberous Sclerosis Complex 2-deficient cells supports tumorigenesis

E. Henske, B. Siroky, Amine Belaid, 2018, Scientific Reports.

Increased susceptibility to structural acute kidney injury in a mouse model of presymptomatic cardiomyopathy.

B. Siroky, Mahima Devarajan, J. Robbins, 2017, American journal of physiology. Renal physiology.

Regulation of mesangial cell Na+/Ca2+ exchanger isoforms

B. Siroky, I. Williams, G. Kovacs, 2004, Journal of cellular physiology.

The growing importance of mTORC1-S6K1 signaling in kidney.

M. Bitzer, B. Siroky, 2009, American journal of physiology. Renal physiology.

Apical trypsin increases ion transport and resistance by a phospholipase C-dependent rise of Ca2+.

P. Factor, S. Matalon, B. Siroky, 2005, American journal of physiology. Lung cellular and molecular physiology.

Loss of primary cilia upregulates renal hypertrophic signaling and promotes cystogenesis.

M. Bilgen, L. Guay-Woodford, B. Siroky, 2011, Journal of the American Society of Nephrology : JASN.

Amyloid beta peptide 1-40 stimulates the Na+/Ca2+ exchange activity of SNCX.

P. Komlósi, B. Siroky, G. Kovacs, 2005, Current neurovascular research.

Loss of matrix metalloproteinase-8 is associated with worsened recovery after ischemic kidney injury

H. Wong, B. Siroky, R. Basu, 2015, Renal failure.

Renal involvement in tuberous sclerosis complex and von Hippel–Lindau disease: shared disease mechanisms?

B. Siroky, J. Bissler, M. Czyzyk-Krzeska, 2009, Nature Clinical Practice Nephrology.

Na+/Ca2+ Exchanger: Target for Oxidative Stress in Salt-Sensitive Hypertension

P. Komlósi, B. Siroky, I. Williams, 2003, Hypertension.

Loss of apical monocilia on collecting duct principal cells impairs ATP secretion across the apical cell surface and ATP-dependent and flow-induced calcium signals

B. Yoder, P. Komlósi, E. Schwiebert, 2007, Purinergic Signalling.

Renal disease in tuberous sclerosis complex: pathogenesis and therapy

E. Henske, B. Siroky, H. Lam, 2018, Nature Reviews Nephrology.

Evidence for pericyte origin of TSC-associated renal angiomyolipomas and implications for angiotensin receptor inhibition therapy.

Z. Tsuchihashi, B. Siroky, Hong Yin, 2014, American journal of physiology. Renal physiology.

Tsc Gene Locus Disruption and Differences in Renal Epithelial Extracellular Vesicles

B. Siroky, J. Bissler, F. Zadjali, 2021, Frontiers in Physiology.

Tuberous sclerosis complex exhibits a new renal cystogenic mechanism

B. Siroky, J. Bissler, D. Bridges, 2019, Physiological reports.

Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells.

L. Guay-Woodford, B. Yoder, P. Komlósi, 2006, American journal of physiology. Renal physiology.

Characterization of renal toxicity in mice administered the marine biotoxin domoic Acid.

M. Janech, B. Siroky, J. Bissler, 2014, Journal of the American Society of Nephrology : JASN.

Primary cilia regulate the osmotic stress response of renal epithelial cells through TRPM3.

B. Siroky, J. Bissler, Lu Lu, 2017, American journal of physiology. Renal physiology.

Renal cystic disease: the role of the primary cilium/centrosome complex in pathogenesis.

L. Guay-Woodford, B. Siroky, 2006, Advances in chronic kidney disease.

Stressed podocytes fail to fold: a potential new role of ER in FSGS.

B. Siroky, J. Bissler, 2011, American journal of physiology. Renal physiology.

Vps34-mediated macropinocytosis in Tuberous Sclerosis Complex 2-deficient cells supports tumorigenesis

E. Henske, B. Siroky, Amine Belaid, 2018, Scientific Reports.

Cell Cycle Control and DNA Damage Response of Conditionally Immortalized Urothelial Cells

B. Siroky, J. Bissler, B. Dixon, 2011, PloS one.

Hyperglycemia in the absence of cilia accelerates cystogenesis and induces renal damage.

C. Baicu, B. Siroky, J. Bissler, 2015, American journal of physiology. Renal physiology.