D. Kramsch

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Cardiovascular Effects of Exercise in Primate Atherosclerosis

D. Kramsch, A. Aspen, B. Abramowitz, 1983 .

Atherosclerotic Disease in Non-Human Primates: Its Prevention and Regression by Moderate Conditioning Exercise

D. Kramsch, A. Aspen, R. Spicer, 1983 .

Atherosclerosis: Prevention by agents not affecting abnormal levels of blood lipids.

D. Kramsch, A. Aspen, L. Rozler, 1981, Science.

Immunolocalization of native antioxidant scavenger enzymes in early hypertensive and atherosclerotic arteries. Role of oxygen free radicals.

R. Sharma, D. Kramsch, A. Sevanian, 1992, Arteriosclerosis and thrombosis : a journal of vascular biology.

Occlusive atheroslcerotic disease of the coronary arteries in monkey (Macaca irus) induced by diet.

D. Kramsch, W. Hollander, 1968, Experimental and molecular pathology.

Effects of lovastatin on ApoA- and ApoB-containing lipoproteins. Families in a subpopulation of patients participating in the Monitored Atherosclerosis Regression Study (MARS).

L. Labree, W. Mack, D. Kramsch, 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.

Body fat distribution affects responses to high fat cholesterol containing diet in male cynomolgus monkeys.

D. Kramsch, R. Sharma, H. Chin, 1991, Atherosclerosis.

Biochemistry of collagen and elastic fibers in atherosclerotic lesions.

D. Kramsch, C. Franzblau, 1977, Advances in experimental medicine and biology.

Limits of lipid-lowering therapy: the potential benefits of amlodipine as an antiatherosclerotic agent.

D. Kramsch, 1997, International journal of cardiology.

Lessons from the Monitored Atherosclerosis Regression Study (MARS) and beyond: atherosclerosis progression/regression responses to therapy depend on lesion size and composition

D. Kramsch, 1996 .

Triglyceride‐ and Cholesterol‐Rich Lipoproteins Have a Differential Effect on Mild/Moderate and Severe Lesion Progression as Assessed by Quantitative Coronary Angiography in a Controlled Trial of Lovastatin

S. Azen, L. Labree, W. Mack, 1994, Circulation.

Quantitation and localization of regional body fat distribution--a comparison between magnetic resonance imaging and somatometry.

P. Colletti, P. Lee, D. Kramsch, 1996, Obesity research.

The Effect of Agents Interfering with Soft Tissue Calcification and Cell Proliferation on Calcific Fibrous‐Fatty Plaques in Rabbits

D. Kramsch, C. Chan, 1978, Circulation research.

The Monitored Atherosclerosis Regression Study (MARS). Design, methods and baseline results.

D. DeMets, S. Azen, W. Mack, 1992, The Online journal of current clinical trials.

Calcium antagonists and atherosclerosis.

D. Kramsch, 1985, Advances in experimental medicine and biology.

Probucol suppresses oxidant stress in hypertensive arteries. Immunohistochemical evidence.

W. Mack, D. Kramsch, A. Sevanian, 1996, American journal of hypertension.

Oxidative Damage to Circulating LDL

D. Kramsch, A. Sevanian, H. Hodis, 1993 .

Biochemical and cytotoxic in vivo circulating oxidized (LDL-) characteristics of an low density lipoprotein

D. Kramsch, A. Sevanian, H. Hodis, 1994 .

Biochemical and cytotoxic characteristics of an in vivo circulating oxidized low density lipoprotein (LDL-).

D. Kramsch, A. Sevanian, H. Hodis, 1994, Journal of lipid research.

Prevention and Therapy of Induced Atherosclerosis in Rabbits and Monkeys Regardless of Serum Cholesterol Levels

D. Kramsch, A. Aspen, C. Chan, 1978 .

The distribution of intravenously administered [3H]Chole-sterol in the arteries and other tissues

D. Kramsch, I. Gore, W. Hollander, 1967 .

The distribution of intravenously administered [3H] cholesterol in the arteries and other tissues. 2. Radioautographic findings.

D. Kramsch, I. Gore, W. Hollander, 1967, Journal of atherosclerosis research.

Metabolism and Distribution of Intravenously Administered d‐Aldosterone‐l,2‐H3 in the Arteries, Kidneys, and Heart of Dog

A. Chobanian, D. Kramsch, J. Melby, 1966 .

The role of connective tissue in atherosclerosis.

D. Kramsch, 1978, Advances in experimental medicine and biology.

Suppression of Calcific Fibrous‐Fatty Plaque Formation in Rabbits by Agents Not Affecting Elevated Serum Cholesterol Levels: The Effect of Thiophene Compounds

D. Kramsch, C. Chan, H. Wells, 1978, Circulation research.

The oxygen environment of the arterial media in early rabbit hypertension.

D. Kramsch, D. Crawford, 1988, Experimental and molecular pathology.

Arterial wall metabolism in experimental hypertension of coarctation of the aorta of short duration.

D. Kramsch, W. Hollander, I. Madoff, 1968, The Journal of clinical investigation.

Regression of atherosclerosis: which components regress and what influences their reversal.

D. Kramsch, D. Blankenhorn, 1992, Wiener klinische Wochenschrift.

Biochemical Changes of the Arterial Wall in Atherosclerosis with Special Reference to Connective Tissue: Promising Experimental Avenues for their Prevention

D. Kramsch, 1981 .

Components of the protein-lipid complex of arterial elastin: their role in the retention of lipid in atherosclerotic lesions.

D. Kramsch, C. Franzblau, W. Hollander, 1974, Advances in experimental medicine and biology.

A Possible Role for Elastin Ligands in the Proteolytic Degradation of Arterial Elastic Lamellae in the Rabbit

D. Kramsch, P. Milbury, H. Kagan, 1979, Circulation research.

The interaction of serum and arterial lipoproteins with elastin of the arterial intima and its role in the lipid accumulation in atherosclerotic plaques.

D. Kramsch, W. Hollander, 1973, The Journal of clinical investigation.

Plasma renin activity in hypertension associated with coarctation of the aorta.

D. Kramsch, W. Hollander, S. Yagi, 1968, The American journal of physiology.

Slome General Considerations on Vascular Reactivity

D. Kramsch, S. Rodbard, S. Hoobler, 2005 .

Suppression of experimental atherosclerosis by the Ca++-antagonist lanthanum. Possible role of calcium in atherogenesis.

D. Kramsch, A. Aspen, C. Apstein, 1980, The Journal of clinical investigation.

Coronary Angiographic Changes with Lovastatin Therapy: The Monitored Atherosclerosis Regression Study (MARS)

Stanley Azen, S. Azen, W. Mack, 1993, Annals of Internal Medicine.

Reversal of Atherosis and Sclerosis: The Two Components of Atherosclerosis

D H Blankenhorn, D. Kramsch, D. Blankenhorn, 1989, Circulation.

Reduction of coronary atherosclerosis by moderate conditioning exercise in monkeys on an atherogenic diet.

W. Hood, D. Kramsch, A. Aspen, 1981, The New England journal of medicine.

Limits of lipid-lowering therapy: the benefits of amlodipine as an anti-atherosclerotic agent.

D. Kramsch, R. Sharma, 1995, Journal of human hypertension.

The protein and lipid composition of arterial elastin and its relationship to lipid accumulation in the atherosclerotic plaque.

D. Kramsch, C. Franzblau, W. Hollander, 1971, The Journal of clinical investigation.

Prevention of Arterial Calcium Deposition with Diphosphonates and Calcium Entry Blockers

D. Kramsch, 1985 .

Tolerance and safety of enalapril.

D. Kramsch, R. Davies, J. Rush, 1984, British journal of clinical pharmacology.

Some General Considerations on Vascular Reactivity: In Vitro Effects of Vasopressor Agents on the Metabolism of the Vascular Wall

D. Kramsch, S. Rodbard, S. Hoobler, 1964, Circulation.

A Primate Model for the Study of Coronary Atherosclerotic Heart Disease

D. Kramsch, W. Hollander, P. R. Clapp, 1973 .

THE DEGRADATION OF ERGASTOPLASMIC STRUCTURES AND ITS EFFECT ON THE DNA SYNTHESIS.

D. Kramsch, W. Oehlert, V. Beck, 1963, Experimental Cell Research.

Connective Tissues in Arterial and Pulmonary Disease

D. Kramsch, A. Chandler, L. W. Carter, 1981, Springer New York.

The efficacy and safety of enalapril in moderate to severe essential hypertension.

D. Kramsch, J. Walker, S. F. Kulaga, 1984, Journal of hypertension. Supplement : official journal of the International Society of Hypertension.