J. Pippin

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CDK inhibitor p21 is prosurvival in adriamycin-induced podocyte injury, in vitro and in vivo.

S. Shankland, C. B. Marshall, R. Krofft, 2010, American journal of physiology. Renal physiology.

CDK inhibitor p 21 is prosurvival in adriamycin-induced podocyte injury , in vitro and in vivo

S. Shankland, C. B. Marshall, R. Krofft, 2010 .

Puromycin aminonucleoside induces oxidant-dependent DNA damage in podocytes in vitro and in vivo.

S. Shankland, C. B. Marshall, R. Krofft, 2006, Kidney international.

Direct in vivo inhibition of the nuclear cell cycle cascade in experimental mesangial proliferative glomerulonephritis with Roscovitine, a novel cyclin-dependent kinase antagonist.

L. Meijer, S. Shankland, J. Pippin, 1997, The Journal of clinical investigation.

The mitochondrial-targeted peptide, SS-31, improves glomerular architecture in mice of advanced age.

P. Rabinovitch, C. Alpers, Y. Chiao, 2017, Kidney international.

Complement membrane attack complex (C5b-9) mediates interstitial disease in experimental nephrotic syndrome.

M. Nangaku, W. Couser, J. Pippin, 1999, Journal of the American Society of Nephrology : JASN.

Feature-rich covalent stains for super-resolution and cleared tissue fluorescence microscopy

Aaron R. Halpern, Min Yen Lee, Jonathan T. C. Liu, 2020, Science Advances.

C5b-9 does not mediate chronic tubulointerstitial disease in the absence of proteinuria.

W. Couser, G. Rangan, J. Pippin, 2005, Kidney international.

Nanoparticles exhibit greater accumulation in kidney glomeruli during experimental glomerular kidney disease

S. Pun, S. Shankland, J. Pippin, 2020, Physiological reports.

The cyclin-dependent kinase inhibitor p21 is required for TGF-β1-induced podocyte apoptosis

S. Shankland, J. Pippin, Y. Terada, 2005 .

Mechanical stretch induces podocyte hypertrophy in vitro.

W. Couser, S. Shankland, J. Pippin, 2005, Kidney international.

Cells of renin lineage express hypoxia inducible factor 2α following experimental ureteral obstruction

J. Duffield, S. Shankland, J. Pippin, 2016, BMC Nephrology.

Differential Expression of Parietal Epithelial Cell and Podocyte Extracellular Matrix Proteins in Focal Segmental Glomerulosclerosis and Diabetic Nephropathy.

C. Alpers, S. Shankland, J. Pippin, 2019, American journal of physiology. Renal physiology.

Reducing mTOR augments parietal epithelial cell density in a model of acute podocyte depletion and in aged kidneys.

B. McNicholas, S. Shankland, J. Pippin, 2016, American journal of physiology. Renal physiology.

Reversibility of structural and functional damage in a model of advanced diabetic nephropathy.

C. Alpers, S. Shankland, J. Pippin, 2013, Journal of the American Society of Nephrology : JASN.

Podocyte repopulation by renal progenitor cells following glucocorticoids treatment in experimental FSGS.

S. Shankland, R. Krofft, J. Pippin, 2013, American journal of physiology. Renal physiology.

The emergence of the glomerular parietal epithelial cell

B. Smeets, S. Shankland, J. Pippin, 2014, Nature Reviews Nephrology.

Cells of renin lineage take on a podocyte phenotype in aging nephropathy.

C. Alpers, S. Glenn, J. Duffield, 2014, American journal of physiology. Renal physiology.

Progenitor cells and podocyte regeneration.

J. Duffield, S. Shankland, J. Pippin, 2014, Seminars in nephrology.

Mechanical stress reduces podocyte proliferation in vitro.

W. Couser, S. Shankland, J. Pippin, 2002, Kidney international.

Podocytes that detach in experimental membranous nephropathy are viable.

S. Shankland, R. Krofft, J. Pippin, 2003, Kidney international.

Viable Podocytes Detach in Experimental Diabetic Nephropathy: Potential Mechanism Underlying Glomerulosclerosis

S. Shankland, R. Krofft, J. Pippin, 2004, Nephron Experimental Nephrology.

Erosion of the Epigenetic Landscape and Loss of Cellular Identity as a Cause of Aging in Mammals

Andreas R. Pfenning, Brendan L. O’Connell, Margarita V. Meer, 2019, bioRxiv.

Erosion of the Epigenetic Landscape and Loss of Cellular Identity as a Cause of Aging in Mammals

Andreas R. Pfenning, Brendan L. O’Connell, Margarita V. Meer, 2019, bioRxiv.

Global transcriptomic changes occur in aged mouse podocytes.

S. Shankland, J. Pippin, S. Akilesh, 2020, Kidney international.

Glomerular parietal epithelial cells contribute to adult podocyte regeneration in experimental focal segmental glomerulosclerosis

S. Shankland, J. Pippin, Diana G. Eng, 2015, Kidney international.

Renin-Angiotensin-Aldosterone System Inhibition Increases Podocyte Derivation from Cells of Renin Lineage.

S. Shankland, J. Kutz, J. Pippin, 2016, Journal of the American Society of Nephrology : JASN.

Modulation of apoptosis by the cyclin-dependent kinase inhibitor p27(Kip1).

S. Shankland, J. Pippin, J. Roberts, 1999, The Journal of clinical investigation.

Tubulointerstitial disease in glomerulonephritis. Potential role of osteopontin (uropontin).

C. Alpers, S. Schwartz, J. Pippin, 1994, The American journal of pathology.

Role of intrinsic renal cells versus infiltrating cells in glomerular crescent formation.

W. Couser, S. Shankland, J. Pippin, 1998, Kidney international.

Volumetric, Nanoscale Optical Imaging of Mouse and Human Kidney via Expansion Microscopy

Aaron R. Halpern, C. Alpers, J. Vaughan, 2018, Scientific Reports.

Limitation of podocyte proliferation improves renal function in experimental crescentic glomerulonephritis.

S. Shankland, R. Krofft, J. Pippin, 2005, Kidney international.

The Cyclin-Dependent Kinase Inhibitor p21 Limits Murine Mesangial Proliferative Glomerulonephritis

C. Alpers, S. Shankland, J. Pippin, 2005, Nephron Experimental Nephrology.

DNA damage is a novel response to sublytic complement C5b-9-induced injury in podocytes.

W. Couser, S. Shankland, J. Pippin, 2003, The Journal of clinical investigation.

Complement (C5b-9) induces DNA synthesis in rat mesangial cells in vitro.

W. Couser, S. Shankland, J. Pippin, 2001, Kidney international.

Cyclin kinase inhibitors are increased during experimental membranous nephropathy: potential role in limiting glomerular epithelial cell proliferation in vivo.

S. E. Thomas, M. Nangaku, W. Couser, 1997, Kidney international.

The enigmatic parietal epithelial cell is finally getting noticed: a review.

S. Shankland, R. Krofft, J. Pippin, 2009, Kidney international.

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

C. Alpers, A. Attie, Mark P. Keller, 2010, Journal of the American Society of Nephrology : JASN.

De novo expression of podocyte proteins in parietal epithelial cells in experimental aging nephropathy.

S. Shankland, R. Krofft, J. Pippin, 2012, American journal of physiology. Renal physiology.

ACE-inhibition increases podocyte number in experimental glomerular disease independent of proliferation

S. Shankland, R. Krofft, J. Pippin, 2014, Journal of the renin-angiotensin-aldosterone system : JRAAS.

C6 mediates chronic progression of tubulointerstitial damage in rats with remnant kidneys.

M. Nangaku, W. Couser, J. Pippin, 2002, Journal of the American Society of Nephrology : JASN.

Interstitial pericytes decrease in aged mouse kidneys

P. Rabinovitch, J. Duffield, S. Shankland, 2015, Aging.

Mitotic cell cycle proteins increase in podocytes despite lack of proliferation.

W. Couser, S. Shankland, J. Pippin, 2003, Kidney international.

Cyclin I Protects Podocytes from Apoptosis*

James M. Roberts, S. Shankland, J. Pippin, 2006, Journal of Biological Chemistry.

Cyclin I and p35 determine the subcellular distribution of Cdk5.

T. Benzing, S. Shankland, J. Pippin, 2015, American journal of physiology. Cell physiology.

Cyclin I and p 35 determine the subcellular distribution of Cdk 5

T. Benzing, S. Shankland, J. Pippin, 2015 .

Inducible rodent models of acquired podocyte diseases.

S. Shankland, C. B. Marshall, R. Krofft, 2009, American journal of physiology. Renal physiology.

De novo expression of podocyte proteins in parietal epithelial cells during experimental glomerular disease.

C. Alpers, S. Shankland, C. B. Marshall, 2010, American journal of physiology. Renal physiology.

CD59 protects glomerular endothelial cells from immune-mediated thrombotic microangiopathy in rats.

C. Alpers, M. Nangaku, W. Couser, 1998, Journal of the American Society of Nephrology : JASN.

Dexamethasone’s Prosurvival Benefits in Podocytes Require Extracellular Signal-Regulated Kinase Phosphorylation

M. Nangaku, S. Shankland, J. Pippin, 2008, Nephron Experimental Nephrology.

TLR4 links podocytes with the innate immune system to mediate glomerular injury.

E. Bock, C. Alpers, B. Banas, 2008, Journal of the American Society of Nephrology : JASN.

Renal microvascular injury induced by antibody to glomerular endothelial cells is mediated by C5b-9.

C. Alpers, M. Nangaku, W. Couser, 1997, Kidney international.

Amelioration of diabetic nephropathy in SPARC-null mice.

C. Alpers, W. Couser, J. Pippin, 2003, Journal of the American Society of Nephrology : JASN.

SPARC is expressed by mesangial cells in experimental mesangial proliferative nephritis and inhibits platelet-derived-growth-factor-medicated mesangial cell proliferation in vitro.

M. Reed, C. Alpers, W. Couser, 1996, The American journal of pathology.

p35, the non-cyclin activator of Cdk5, protects podocytes against apoptosis in vitro and in vivo.

B. Schermer, T. Benzing, S. Shankland, 2010, Kidney international.

Cyclin I activates Cdk5 and regulates expression of Bcl-2 and Bcl-XL in postmitotic mouse cells.

James M. Roberts, S. Shankland, R. Krofft, 2009, The Journal of clinical investigation.

Rosuvastatin protects against podocyte apoptosis in vitro.

S. Shankland, J. Pippin, R. Durvasula, 2008, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

Induction of TRPC6 channel in acquired forms of proteinuric kidney disease.

M. Rastaldi, M. Kretzler, S. Shankland, 2007, Journal of the American Society of Nephrology : JASN.

Can podocytes be regenerated in adults?

Benjamin S. Freedman, S. Shankland, J. Pippin, 2017, Current opinion in nephrology and hypertension.

Partial podocyte replenishment in experimental FSGS derives from nonpodocyte sources.

S. Shankland, J. Pippin, Diana G. Eng, 2016, American journal of physiology. Renal physiology.

The glomerular parietal epithelial cell’s responses are influenced by SM22 alpha levels

S. Shankland, J. Pippin, Shokichi Naito, 2014, BMC Nephrology.

Aberrant Notch1-dependent effects on glomerular parietal epithelial cells promotes collapsing focal segmental glomerulosclerosis with progressive podocyte loss.

I. Pastan, S. Shankland, J. Pippin, 2013, Kidney international.

Metalloprotease Meprinβ in Rat Kidney: Glomerular Localization and Differential Expression in Glomerulonephritis

J. Pippin, H. Marti, B. Oneda, 2008, PloS one.

CD44 impacts glomerular parietal epithelial cell changes in the aged mouse kidney

S. Shankland, J. Pippin, K. Hiromura, 2020, Physiological reports.

Transfected CD59 protects mesangial cells from injury induced by antibody and complement.

M. Nangaku, W. Couser, J. Pippin, 1996, Kidney international.

Albumin-induced apoptosis of glomerular parietal epithelial cells is modulated by extracellular signal-regulated kinase 1/2.

S. Shankland, R. Krofft, J. Pippin, 2012, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

Modulation of experimental mesangial proliferative nephritis by interferon-gamma.

C. Alpers, W. Couser, J. Floege, 1995, Kidney international.

C5b-9 regulates peritubular myofibroblast accumulation in experimental focal segmental glomerulosclerosis.

W. Couser, G. Rangan, J. Pippin, 2004, Kidney international.

Role of smooth muscle protein SM22α in glomerular epithelial cell injury.

C. Alpers, R. Feil, S. Shankland, 2011, American journal of physiology. Renal physiology.

Establishment of conditionally immortalized mouse glomerular parietal epithelial cells in culture.

S. Shankland, R. Krofft, J. Pippin, 2008, Journal of the American Society of Nephrology : JASN.

Faulty Podocyte Hypoxia Sensing—A Novel Pathway for Rapidly Progressive Glomerulonephritis Loss of the Tumor Suppressor Vhlh Leads to Upregulation of Cxcr4 and Rapidly Progressive Glomerulonephritis. Nat Med 12: 1081–1087, 2006

M. Rastaldi, C. Cohen, M. Kretzler, 2006 .

TRPC6 protects podocytes from complement-mediated injury

Dinda, S. Dryer, A. Kistler, 2013 .

Lineage tracing aged mouse kidneys shows lower number of cells of renin lineage and reduced responsiveness to RAAS inhibition.

Benjamin S. Freedman, S. Shankland, J. Pippin, 2018, American journal of physiology. Renal physiology.

Upregulated PD-1 signaling antagonizes glomerular health in aged kidneys and disease

M. Bitzer, J. Vaughan, S. Shankland, 2022, The Journal of clinical investigation.

Characterization of Glomerular Sox9+ Cells in Anti-Glomerular Basement Membrane Nephritis in the Rat.

M. Wegner, S. Shankland, J. Pippin, 2018, The American journal of pathology.

The Contribution of Podocytes to Chronic Allograft Nephropathy

P. Jablonski, S. Shankland, J. Pippin, 2008, Nephron Experimental Nephrology.

Urinary Podocyte Loss Is Increased in Patients with Fabry Disease and Correlates with Clinical Severity of Fabry Nephropathy

D. Warnock, S. Shankland, J. Pippin, 2016, PloS one.

Experimental Models of Membranous Nephropathy.

S. Shankland, J. Pippin, J. A. Jefferson, 2010, Drug discovery today. Disease models.

Beneficial effects of systemic immunoglobulin in experimental membranous nephropathy.

C. Alpers, M. Nangaku, W. Couser, 1996, Kidney international.

Differential expression of d-type cyclins in podocytes in vitro and in vivo.

W. Couser, S. Shankland, J. Pippin, 2004, The American journal of pathology.

Intravenous immunoglobulin protects against experimental thrombotic microangiopathy.

Y. G. Kim, W. Couser, J. Pippin, 2001, Kidney international.

A new function for parietal epithelial cells: a second glomerular barrier.

C. Alpers, S. Shankland, J. Pippin, 2009, American journal of physiology. Renal physiology.

Boronic acid copolymers for direct loading and acid-triggered release of Bis-T-23 in cultured podocytes.

S. Pun, S. Shankland, J. Pippin, 2018, ACS biomaterials science & engineering.

Podocyte Aging: Why and How Getting Old Matters.

S. Shankland, O. Wessely, J. Pippin, 2021, Journal of the American Society of Nephrology : JASN.

SSeCKS sequesters cyclin D1 in glomerular parietal epithelial cells and influences proliferative injury in the glomerulus

C. Alpers, Kelly D. Smith, P. Nelson, 2012, Laboratory Investigation.

WT1 Is Necessary for the Proliferation and Migration of Cells of Renin Lineage Following Kidney Podocyte Depletion

S. Shankland, J. Pippin, Diana G. Eng, 2017, Stem cell reports.

Tracking the stochastic fate of cells of the renin lineage after podocyte depletion using multicolor reporters and intravital imaging

S. Shankland, J. Pippin, Diana G. Eng, 2017, PloS one.

ATRA induces podocyte differentiation and alters nephrin and podocin expression in vitro and in vivo.

S. Shankland, R. Krofft, J. Pippin, 2005, Kidney international.

Retinoids Augment the Expression of Podocyte Proteins by Glomerular Parietal Epithelial Cells in Experimental Glomerular Disease

P. Nelson, S. Shankland, R. Krofft, 2012, Nephron Experimental Nephrology.

The Rapamycin derivative RAD inhibits mesangial cell migration through the CDK-inhibitor p27KIP1

S. Shankland, J. Pippin, C. Daniel, 2004, Laboratory Investigation.

Edinburgh Research Explorer Wt1 is necessary for the proliferation and migration of cells of renin lineage following podocyte depletion

S. Shankland, J. Pippin, Diana G. Eng, 2018 .

WT1 Is Necessary for the Proliferation and Migration of Cells of Renin Lineage Following Kidney Podocyte Depletion

S. Shankland, J. Pippin, Diana G. Eng, 2017, Stem cell reports.

Microarray and Bioinformatics Analysis of Gene Expression in Experimental Membranous Nephropathy

B. Mayer, C. Alpers, P. Perco, 2009, Nephron Experimental Nephrology.

High-Throughput Screening Enhances Kidney Organoid Differentiation from Human Pluripotent Stem Cells and Enables Automated Multidimensional Phenotyping.

Matthias Kretzler, Rajasree Menon, Benjamin S. Freedman, 2018, Cell stem cell.

Gene‐Edited Human Kidney Organoids Reveal Mechanisms of Disease in Podocyte Development

Benjamin S. Freedman, Lih-Yuan Lin, A. Churchill, 2017, Stem cells.

Resource High-Throughput Screening Enhances KidneyOrganoid Differentiation fromHuman Pluripotent StemCells and Enables AutomatedMultidimensional Phenotyping Graphical

Benjamin S. Freedman, L. Tran, R. Moon, 2018 .

Loss of epigenetic information as a cause of mammalian aging

Andreas R. Pfenning, Brendan L. O’Connell, Margarita V. Meer, 2023, Cell.

Intravital imaging of podocyte calcium in glomerular injury and disease.

S. Shankland, J. Pippin, J. Peti-Peterdi, 2014, The Journal of clinical investigation.

Changes in cell-cycle protein expression during experimental mesangial proliferative glomerulonephritis.

M. Nangaku, W. Couser, S. Shankland, 1996, Kidney international.

Loss of the tumor suppressor Vhlh leads to upregulation of Cxcr4 and rapidly progressive glomerulonephritis in mice

Matthias Kretzler, Shiying Cui, M. Rastaldi, 2006, Nature Medicine.

Novel siRNA Delivery System to Target Podocytes In Vivo

C. Alpers, S. Shankland, R. Krofft, 2010, PloS one.

Podocytes in culture: past, present, and future.

S. Shankland, J. Pippin, S J Shankland, 2007, Kidney international.

Dual lineage tracing shows that glomerular parietal epithelial cells can transdifferentiate toward the adult podocyte fate.

Benjamin S. Freedman, J. Vaughan, S. Shankland, 2019, Kidney international.

Podocyte Aging: Why and How Getting Old Matters.

J. Vaughan, S. Shankland, O. Wessely, 2021, Journal of the American Society of Nephrology : JASN.

Cyclin I-Cdk5 governs survival in post-mitotic cells.

S. Shankland, J. Pippin, P. Brinkkoetter, 2010, Cell cycle.

A new model of renal microvascular endothelial injury.

C. Alpers, M. Nangaku, W. Couser, 1997, Kidney international.

Activated ERK1/2 increases CD44 in glomerular parietal epithelial cells leading to matrix expansion.

S. Shankland, J. Pippin, C. Daniel, 2017, Kidney international.

Sex differences in transcriptomic profiles in aged kidney cells of renin lineage

S. Gharib, S. Shankland, J. Pippin, 2018, Aging.

Activation of a local tissue angiotensin system in podocytes by mechanical strain.

W. Couser, S. Shankland, J. Pippin, 2004, Kidney international.

Dexamethasone prevents podocyte apoptosis induced by puromycin aminonucleoside: role of p53 and Bcl-2-related family proteins.

S. Shankland, C. B. Marshall, J. Pippin, 2005, Journal of the American Society of Nephrology : JASN.

The Atypical Cyclin-Dependent Kinase 5 (Cdk5) Guards Podocytes from Apoptosis in Glomerular Disease While Being Dispensable for Podocyte Development

B. Schermer, T. Benzing, S. Shankland, 2021, Cells.

Cells of renin lineage are progenitors of podocytes and parietal epithelial cells in experimental glomerular disease.

S. Glenn, J. Duffield, S. Shankland, 2013, The American journal of pathology.

Cells of renin lineage are adult pluripotent progenitors in experimental glomerular disease.

S. Glenn, J. Duffield, S. Shankland, 2015, American journal of physiology. Renal physiology.

Dystroglycan does not contribute significantly to kidney development or function, in health or after injury.

J. Kreidberg, S. Shankland, J. Pippin, 2011, American journal of physiology. Renal physiology.

Compound effects of aging and experimental FSGS on glomerular epithelial cells

S. Shankland, J. Kutz, J. Pippin, 2017, Aging.

Focal and segmental glomerulosclerosis induced in mice lacking decay-accelerating factor in T cells.

C. Alpers, R. Quigg, M. Haas, 2009, The Journal of clinical investigation.

Cell cycle re-entry sensitizes podocytes to injury induced death

S. Shankland, J. Pippin, C. Daniel, 2016, Cell cycle.

Role of the complement membrane attack complex (C5b-9) in mediating experimental mesangioproliferative glomerulonephritis.

C. Alpers, W. Couser, J. Pippin, 1996, Kidney international.

Darbepoetin alfa protects podocytes from apoptosis in vitro and in vivo.

S. Shankland, R. Krofft, J. Pippin, 2007, Kidney international.

The subcellular localization of cyclin dependent kinase 2 determines the fate of mesangial cells: role in apoptosis and proliferation

S. Shankland, J. Pippin, J. Roberts, 2002, Oncogene.

Mesangial cell proliferation mediated by PDGF and bFGF is determined by levels of the cyclin kinase inhibitor p27Kip1.

M. Nangaku, W. Couser, S. Shankland, 1997, Kidney international.

Charting the transcriptional landscape of cells of renin lineage following podocyte depletion

S. Gharib, S. Shankland, J. Pippin, 2017, PloS one.

Cyclin-dependent kinase 5 is a regulator of podocyte differentiation, proliferation, and morphology.

A. Kulkarni, S. Shankland, J. Pippin, 2004, The American journal of pathology.

Transcriptional Landscape of Glomerular Parietal Epithelial Cells

S. Gharib, S. Shankland, R. Krofft, 2014, PloS one.

Changes in glomerular parietal epithelial cells in mouse kidneys with advanced age.

P. Rabinovitch, B. McNicholas, S. Shankland, 2015, American journal of physiology. Renal physiology.

Cells of NG2 lineage increase in glomeruli of mice following podocyte depletion.

S. Shankland, J. Pippin, Diana G. Eng, 2018, American journal of physiology. Renal physiology.

Parietal epithelial cell differentiation to a podocyte fate in the aged mouse kidney

S. Shankland, J. Pippin, Diana G. Eng, 2020, Aging.

Both cyclin I and p35 are required for maximal survival benefit of cyclin-dependent kinase 5 in kidney podocytes.

S. Shankland, R. Krofft, J. Pippin, 2012, American journal of physiology. Renal physiology.

Protease nexin-1, tPA, and PAI-1 are upregulated in cryoglobulinemic membranoproliferative glomerulonephritis.

C. Alpers, S. Shankland, J. Pippin, 2008, Journal of the American Society of Nephrology : JASN.

Complement (C5b-9) induces glomerular epithelial cell DNA synthesis but not proliferation in vitro.

W. Couser, S. Shankland, J. Pippin, 1999, Kidney international.

Glomerular disease augments kidney accumulation of synthetic anionic polymers.

S. Pun, S. Shankland, J. Pippin, 2018, Biomaterials.

Retinoic acid improves nephrotoxic serum-induced glomerulonephritis through activation of podocyte retinoic acid receptor α.

D. Salant, Xiaoqiang Ding, Kyung Lee, 2017, Kidney international.

Differential expression of transforming growth factor-β isoforms and receptors in experimental membranous nephropathy

S. Friedman, W. Couser, S. Shankland, 1996 .

Feature-rich covalent stains for super-resolution and cleared tissue fluorescence microscopy.

Aaron R. Halpern, Min Yen Lee, Jonathan T. C. Liu, 2020, Science Advances.

Inhibiting NLRP3 signaling in aging podocytes improves their life- and health-span

A. Theberge, M. Bitzer, N. Hukriede, 2023, Aging.

Upregulated PD-1 Signaling is an Important Antagonist to Glomerular Health in Aged Kidneys

M. Bitzer, S. Shankland, U. Tran, 2021, bioRxiv.

Does treating with anti–PD-1 to improve glomerular health come without a cost? Reply.

S. Shankland, O. Wessely, J. Pippin, 2022, The Journal of clinical investigation.

Cyclin I activates Cdk5 and regulates expression of Bcl-2 and Bcl-XL in postmitotic mouse cells.

James M. Roberts, S. Shankland, R. Krofft, 2009, The Journal of clinical investigation.

Loss of epigenetic information as a cause of mammalian aging

Andreas R. Pfenning, Brendan L. O’Connell, Margarita V. Meer, 2023, Cell.

Focal and segmental glomerulosclerosis induced in mice lacking decay-accelerating factor in T cells.

C. Alpers, R. Quigg, M. Haas, 2009, The Journal of clinical investigation.

Partial podocyte replenishment in experimental FSGS derives from nonpodocyte sources.

S. Shankland, J. Pippin, Diana G. Eng, 2016, American journal of physiology. Renal physiology.

Reversibility of structural and functional damage in a model of advanced diabetic nephropathy.

C. Alpers, S. Shankland, J. Pippin, 2013, Journal of the American Society of Nephrology : JASN.

Changes In Glomerular Parietal Epithelial Cells In Mouse Kidneys With Advanced Age 3 4 Authors: 5

B. McNicholas, S. Shankland, J. Pippin, 2015 .

Glomerular parietal epithelial cells contribute to adult podocyte regeneration in experimental focal segmental glomerulosclerosis

S. Shankland, J. Pippin, Diana G. Eng, 2015, Kidney international.

Podocyturia correlates with proteinuria in patients with Fabry disease (FD) and is a potential biomarker of Fabry nephropathy

S. Shankland, J. Pippin, S. Uhrich, 2013 .

Podocytes from hypertensive and obese mice acquire an inflammatory, senescent, and aged phenotype

J. Pippin, Diana G. Eng, B. M. V. Pereira, 2024, American journal of physiology. Renal physiology.

Podocyte Senescence and Aging

J. Pippin, Stuart J. Shankland, J. N. Kutz, 2023, Kidney360.

Edinburgh Research Explorer Wt1 is necessary for the proliferation and migration of cells of renin lineage following podocyte depletion

S. Shankland, J. Pippin, Diana G. Eng, 2018 .