S. Taylor

发表

Regulation of pyruvate dehydrogenase in isolated rat liver mitochondria. Effects of octanoate, oxidation-reduction state, and adenosine triphosphate to adenosine diphosphate ratio.

S. Taylor, C. Mukherjee, R. Jungas, 1975, The Journal of biological chemistry.

Cloning of the chicken insulin receptor substrate 1 gene.

M. Reitman, S. Taylor, M. Taouis, 1996, Gene.

Roles of 1-phosphatidylinositol 3-kinase and ras in regulating translocation of GLUT4 in transfected rat adipose cells

M. Kasuga, M. Quon, S. Taylor, 1995, Molecular and cellular biology.

Insulin-stimulated phosphorylation of recombinant pp120/HA4, an endogenous substrate of the insulin receptor tyrosine kinase.

D. Accili, Simeon I. Taylor, S. Taylor, 1995, Biochemistry.

Mutations in the insulin receptor gene.

D. Accili, M. Quon, M. de la Luz Sierra, 1992, Endocrine reviews.

Lilly Lecture: Molecular Mechanisms of Insulin Resistance: Lessons From Patients With Mutations in the Insulin-Receptor Gene

Simeon I. Taylor, S. Taylor, 1992, Diabetes.

Insulin resistance due to mutations of the insulin receptor gene: An overview

D. Accili, Simeon I. Taylor, T. Kadowaki, 1992, Journal of endocrinological investigation.

Physiological role of Akt in insulin-stimulated translocation of GLUT4 in transfected rat adipose cells.

M. Quon, S. Taylor, L. Cong, 1997, Molecular endocrinology.

Homozygosity for a new mutation (Ile119-->Met) in the insulin receptor gene in five sibs with familial insulin resistance.

D. Accili, S. Taylor, L. Al-Gazali, 1994, Journal of medical genetics.

Variant sequences of insulin receptor substrate-1 in patients with noninsulin-dependent diabetes mellitus.

D. Accili, G. Sesti, S. Taylor, 1994, The Journal of clinical endocrinology and metabolism.

Tyrosine kinase-deficient mutant human insulin receptors (Met1153-->Ile) overexpressed in transfected rat adipose cells fail to mediate translocation of epitope-tagged GLUT4.

A. Butte, M. Quon, S. Taylor, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Insulin Receptor Substrate-2 (IRS-2) Can Mediate the Action of Insulin to Stimulate Translocation of GLUT4 to the Cell Surface in Rat Adipose Cells*

M. Quon, S. Sciacchitano, S. Taylor, 1997, The Journal of Biological Chemistry.

D. Accili, T. Kadowaki, S. Taylor, 1990, The Journal of clinical endocrinology and metabolism.

Specific glycosylation site mutations of the insulin receptor alpha subunit impair intracellular transport.

Simeon I. Taylor, S. Taylor, J. Carpentier, 1993, Biochemistry.

Normal Coding Sequence of Insulin Gene in Pima Indians and Nauruans, Two Groups With Highest Prevalence of Type II Diabetes

S. Lillioja, P. Zimmet, Simeon I. Taylor, 1991, Diabetes.

Expression of variant forms of insulin receptor substrate-1 identified in patients with noninsulin-dependent diabetes mellitus.

D. Accili, G. Sesti, S. Taylor, 1997, The Journal of clinical endocrinology and metabolism.

Does Leptin Contribute to Diabetes Caused by Obesity?

M. Reitman, V. Barr, Simeon I. Taylor, 1996, Science.

Insulin stimulates both leptin secretion and production by rat white adipose tissue.

V. Barr, D. Malide, S. Taylor, 1997, Endocrinology.

Deletion of C-terminal 113 amino acids impairs processing and internalization of human insulin receptor: comparison of receptors expressed in CHO and NIH-3T3 cells.

D. Accili, S. Taylor, R. Levy-Toledano, 1993, Biochimica et biophysica acta.

Lipoatrophic-lipodystrophic syndromes: the spectrum of findings on MR imaging.

Simeon I. Taylor, S. Taylor, C. Chow, 2002, AJR. American journal of roentgenology.

The amino acid sequence of the insulin receptor is normal in an insulin-resistant Pima Indian.

S. Lillioja, T. Kadowaki, S. Taylor, 1990, The Journal of clinical endocrinology and metabolism.

Two mutant alleles of the insulin receptor gene in a patient with extreme insulin resistance.

Simeon I. Taylor, B. Marcus-Samuels, T. Kadowaki, 1988, Science.

Genetic basis of endocrine disease. 1. Molecular genetics of insulin resistant diabetes mellitus.

D. Accili, Simeon I. Taylor, T. Kadowaki, 1991, The Journal of clinical endocrinology and metabolism.

Insulin receptor substrate 1 mediates the stimulatory effect of insulin on GLUT4 translocation in transfected rat adipose cells.

A. Butte, M. Quon, G. Sesti, 1994, The Journal of biological chemistry.

Assay of antibodies directed against cell surface receptors.

B. Marcus-Samuels, S. Taylor, L. Underhill, 1985, Methods in enzymology.

Four mutant alleles of the insulin receptor gene associated with genetic syndromes of extreme insulin resistance.

Y. Yazaki, T. Kadowaki, S. Taylor, 1997, Biochemical and biophysical research communications.

Absence of insulin receptor gene mutations in three insulin-resistant women with the polycystic ovary syndrome.

S. Taylor, E. Koller, R. Kohanski, 1994, Metabolism: clinical and experimental.

Postbinding characterization of five naturally occurring mutations in the human insulin receptor gene: impaired insulin-stimulated c-jun expression and thymidine incorporation despite normal receptor autophosphorylation.

M. Quon, S. Taylor, A. Cama, 1992, Biochemistry.

A mutation in the insulin receptor gene that impairs transport of the receptor to the plasma membrane and causes insulin‐resistant diabetes.

E. Lander, M. Permutt, A. Ullrich, 1989, The EMBO journal.

A nonsense mutation causing decreased levels of insulin receptor mRNA: detection by a simplified technique for direct sequencing of genomic DNA amplified by the polymerase chain reaction.

Simeon I. Taylor, T. Kadowaki, S. Taylor, 1990, Proceedings of the National Academy of Sciences of the United States of America.

Investigation of the mechanism of the dominant negative effect of mutations in the tyrosine kinase domain of the insulin receptor.

D. Accili, S. Taylor, L. Caro, 1994, The EMBO journal.

Insulin receptors in normal and disease states.

G. Grunberger, S. Taylor, P. Gorden, 1983, Clinics in endocrinology and metabolism.

Lipoatrophy syndromes: when ‘too little fat’ is a clinical problem

M. Reitman, Simeon I. Taylor, K. Rother, 2000, Pediatric diabetes.

Insulin receptor regulation of cell surface integrins: a possible mechanism contributing to the development of diabetic complications.

S. Taylor, T. Tennenbaum, E. Wertheimer, 1998, Proceedings of the Association of American Physicians.

Insulin-induced activation of a phosphatidyl inositol 3-kinase. Demonstration that the P85 subunit binds directly to the COOH terminus of the insulin receptor in intact cells

Simeon I. Taylor, S. Taylor, M. Taouis, 2022 .

Hypoglycemia associated with antibodies to the insulin receptor.

G. Grunberger, B. Marcus-Samuels, S. Taylor, 1982, The New England journal of medicine.

Deletion of 343 amino acids from the carboxyl terminus of the beta-subunit of the insulin receptor inhibits insulin signaling.

C. Haft, S. Taylor, 1994, Biochemistry.

Insulin receptor degradation is accelerated in cultured lymphocytes from patients with genetic syndromes of extreme insulin resistance.

S. Taylor, J. Roth, P. Gorden, 1984, The Journal of clinical investigation.

pp120/ecto-ATPase, an endogenous substrate of the insulin receptor tyrosine kinase, is expressed as two variably spliced isoforms.

D. Accili, R. Margolis, Simeon I. Taylor, 1993, The Journal of biological chemistry.

Hepatocyte plasma membrane ECTO-ATPase (pp120/HA4) is a substrate for tyrosine kinase activity of the insulin receptor.

R. Margolis, S. Taylor, A. Hubbard, 1990, Biochemical and biophysical research communications.

Insulin stimulates phosphorylation of a 120-kDa glycoprotein substrate (pp120) for the receptor-associated protein kinase in intact H-35 hepatoma cells.

D. Accili, B. Marcus-Samuels, S. Taylor, 1987, Proceedings of the National Academy of Sciences of the United States of America.

Two Patients With Insulin Resistance Due to Decreased Levels of Insulin-Receptor mRNA

Simeon I. Taylor, T. Kadowaki, R. Kawamori, 1991, Diabetes.

Genetics of the insulin receptor defect in a patient with extreme insulin resistance.

B. Marcus-Samuels, S. Taylor, M. Elders, 1986, The Journal of clinical endocrinology and metabolism.

Glucose metabolism in perfused skeletal muscle. Pyruvate dehydrogenase activity in starvation, diabetes and exercise.

S. Taylor, S. Hagg, N. B. Ruberman, 1976, The Biochemical journal.

Decreased insulin binding in cultured lymphocytes from two patients with extreme insulin resistance.

M. Kasuga, S. Taylor, J. Roth, 1982, The Journal of clinical endocrinology and metabolism.

Insulin resistance associated with androgen excess in women with autoantibodies to the insulin receptor.

S. Taylor, J. Roth, P. Gorden, 1982, Annals of internal medicine.

Characterization of receptors for insulin and insulin-like growth factor-1 on FRTL-5 thyroid cells.

S. Taylor, C. Rotella, N. Perrotti, 1989, Advances in experimental medicine and biology.

Mutations in the insulin receptor gene in patients with genetic syndromes of insulin resistance.

D. Accili, Simeon I. Taylor, T. Kadowaki, 1991, Advances in experimental medicine and biology.

Unusual forms of insulin resistance.

D. Accili, T. Kadowaki, S. Taylor, 1991, Annual review of medicine.

Insulin stimulates phosphorylation of serine residues in soluble insulin receptors.

G. Grunberger, S. Taylor, Y. Zick, 1983, Biochemical and biophysical research communications.

The role of antireceptor antibodies in stimulating phosphorylation of the insulin receptor.

S. Taylor, Y. Zick, J. Roth, 1984, The Journal of biological chemistry.

Tyrosine kinase activity of the insulin receptor of patients with type A extreme insulin resistance: studies with circulating mononuclear cells and cultured lymphocytes.

G. Grunberger, S. Taylor, P. Gorden, 1984, The Journal of clinical endocrinology and metabolism.

Anti-receptor antibodies mimic the effect of insulin to down-regulate insulin receptors in cultured human lymphoblastoid (IM-9) cells.

B. Marcus-Samuels, S. Taylor, B. Marcus‐Samuels, 1984, The Journal of clinical endocrinology and metabolism.

Mechanisms of hormone resistance: lessons from insulin‐resistant patients

D. Accili, M. Quon, C. Haft, 1994, Acta paediatrica (Oslo, Norway : 1992). Supplement.

Homozygosity for a null allele of the insulin receptor gene in a patient with leprechaunism

D. Accili, Simeon I. Taylor, S. Taylor, 1995, Human mutation.

Paradoxical biological effects of overexpressed insulin‐like growth factor‐1 receptors in chinese hamster ovary cells

Simeon I. Taylor, C. Roberts, D. Leroith, 1993, Journal of cellular physiology.

Defects in human insulin receptor gene expression.

A. Ullrich, C. Roberts, S. Taylor, 1988, Molecular endocrinology.

Deconstructing Type 2 Diabetes

Simeon I. Taylor, S. Taylor, S I Taylor, 1999, Cell.

Downregulation Occurs Normally in Cultured Epstein-Barr Virus-transformed Lymphocytes from Patients with Extreme Insulin Resistance: Discrepancy Between Downregulation In Vivo and In Vitro

S. Taylor, J. Roth, P. Gorden, 1984, Diabetes.

Genetically defined forms of diabetes in children.

Simeon I. Taylor, S. Taylor, E. Arioglu, 1999, The Journal of clinical endocrinology and metabolism.

Substitution of isoleucine for methionine at position 1153 in the beta-subunit of the human insulin receptor. A mutation that impairs receptor tyrosine kinase activity, receptor endocytosis, and insulin action.

M. Quon, M. de la Luz Sierra, S. Taylor, 1992, The Journal of biological chemistry.

Mutations in the insulin receptor gene in patients with genetic syndromes of insulin resistance and acanthosis nigricans.

D. Accili, Simeon I. Taylor, T. Kadowaki, 1992, The Journal of investigative dermatology.

Mutagenesis of lysine 460 in the human insulin receptor. Effects upon receptor recycling and cooperative interactions among binding sites.

Simeon I. Taylor, B. Marcus-Samuels, A. Rovira, 1990, The Journal of biological chemistry.

Mutations in the insulin receptor gene in genetic forms of insulin resistance.

D. Accili, B. Marcus-Samuels, T. Kadowaki, 1990, Recent progress in hormone research.

Studies on the mechanism of activation of adipose tissue pyruvate dehydrogenase by insulin.

S. Taylor, C. Mukherjee, R. Jungas, 1973, The Journal of biological chemistry.

Extreme insulin resistance in association with abnormally high binding affinity of insulin receptors from a patient with leprechaunism: evidence for a defect intrinsic to the receptor.

M. Kasuga, S. Taylor, J. Roth, 1982, The Journal of clinical endocrinology and metabolism.

Receptors for peptide hormones: alterations in diseases of humans.

S. Taylor, J. Roth, 1982, Annual review of physiology.

Action of insulin receptor substrate-3 (IRS-3) and IRS-4 to stimulate translocation of GLUT4 in rat adipose cells.

M. Quon, S. Sciacchitano, S. Taylor, 1999, Molecular endocrinology.

Insulin receptor biosynthesis in cultured lymphocytes from insulin-resistant patients.

S. Taylor, J. A. Hedo, V. Moncada, 1985, The Journal of clinical investigation.

Cloning, tissue expression, and chromosomal localization of the mouse IRS-3 gene.

S. Sciacchitano, S. Taylor, 1997, Endocrinology.

Regulation of lipogenesis in adipose tissue: the significance of the activation of pyruvate dehydrogenase by insulin.

S. Taylor, R. Jungas, 1974, Archives of biochemistry and biophysics.

Insulin Receptor Substrate-1 Pleckstrin Homology and Phosphotyrosine-binding Domains Are Both Involved in Plasma Membrane Targeting*

D. Leroith, S. Taylor, A. R. Jacobs, 2001, The Journal of Biological Chemistry.

An endogenous substrate for the insulin receptor-associated tyrosine kinase.

S. Taylor, R. Rees-Jones, 1985, The Journal of biological chemistry.

Decreased insulin binding to cultured cells from a patient with the Rabson-Mendenhall syndrome: dichotomy between studies with cultured lymphocytes and cultured fibroblasts.

S. Taylor, J. Roth, M. S. Ríos, 1983, The Journal of clinical endocrinology and metabolism.

Biosynthesis of ergosta-4,6,8(14),22-tetraen-3-one. In vivo incorporation of a 1,4-dioxide.

S. Taylor, J. D. White, 1970, Journal of the American Chemical Society.

Tissue distribution and subcellular localization of an endogenous substrate (pp 120) for the insulin receptor-associated tyrosine kinase.

D. Accili, Simeon I. Taylor, S. Taylor, 1986, Endocrinology.

Qualitative abnormalities in insulin binding in a patient with extreme insulin resistance: decreased sensitivity to alterations in temperature and pH.

S. Taylor, J. Roth, R. Blizzard, 1981, Proceedings of the National Academy of Sciences of the United States of America.

A mutation in the extracellular domain of the insulin receptor impairs the ability of insulin to stimulate receptor autophosphorylation.

A. Ullrich, D. Accili, S. Taylor, 1991, The Journal of biological chemistry.

Tyrosine kinase activity of insulin receptors from an insulin-resistant patient with leprechaunism

S. Taylor, A. Cama, 1987, Diabetologia.

Defect in cooperativity in insulin receptors from a patient with a congenital form of extreme insulin resistance.

S. Taylor, S. Leventhal, 1983, The Journal of clinical investigation.

Mutagenesis of Phe381 and Phe382 in the extracellular domain of the insulin receptor: effects on receptor biosynthesis, processing, and ligand‐dependent internalization

A. Ullrich, D. Accili, Simeon I. Taylor, 1994, FEBS letters.

Unique Features of the Insulin Receptor in Rat Brain

C. Agardh, Simeon I. Taylor, S. Taylor, 1984, Journal of neurochemistry.

Analysis of insulin receptors on human lymphoblastoid cell lines by flow cytometry

C. Kahn, S. Taylor, R. Jackson, 1984, Diabetologia.

Binding of hormones to receptors. An alternative explanation of nonlinear Scatchard plots.

S. Taylor, 1975, Biochemistry.

Autoantibodies to the insulin receptor as a cause of autoimmune hypoglycemia in systemic lupus erythematosus.

D. Moller, S. Taylor, R. Ratner, 1988, American Journal of Medicine.

Clinical disorders associated with autoantibodies to the insulin receptor. Simulation by passive transfer of immunoglobulins to rats.

S. Taylor, P. Gorden, S. Chernick, 1983, The Journal of clinical investigation.

Identification of pp120, an endogenous substrate for the hepatocyte insulin receptor tyrosine kinase, as an integral membrane glycoprotein of the bile canalicular domain.

D. Seminara, R. Margolis, S. Taylor, 1988, Proceedings of the National Academy of Sciences of the United States of America.

Two mutations in a conserved structural motif in the insulin receptor inhibit normal folding and intracellular transport of the receptor.

Simeon I. Taylor, S. Taylor, E. Wertheimer, 1994, The Journal of biological chemistry.

Information transfer, cell regulation, and disease mechanisms: insights from studies of cell surface receptors.

S. Taylor, J. Roth, 1981, Harvey lectures.

Insulin stimulated protein phosphorylation in human plasma liver membranes: detection of endogenous or plasma membrane associated substrates for insulin receptor kinase.

J. Shafer, S. Taylor, M. Sinha, 1987, Biochemical and biophysical research communications.

Immunological similarity between the insulin receptor and the protein encoded by the src oncogene.

G. Grunberger, N. Richert, S. Taylor, 1986, Endocrinology.

Immunoprecipitation of insulin receptors from cultured human lymphocytes (IM-9 cells) by antibodies to pp60src.

I. Pastan, N. Richert, S. Taylor, 1985, Science.

Reevaluation of the evidence that an antibody to the insulin receptor is insulinmimetic without activating the protein tyrosine kinase activity of the receptor.

O. Rosen, S. Taylor, R. Gherzi, 1987, The Journal of biological chemistry.

Rat liver membranes contain a 120 kDa glycoprotein which serves as a substrate for the tyrosine kinases of the receptors for insulin and epidermal growth factor

Simeon I. Taylor, S. Taylor, S. Phillips, 1987, FEBS letters.

Insulin-stimulated receptor phosphorylation appears normal in cultured Epstein-Barr virus-transformed lymphocyte cell lines derived from patients with extreme insulin resistance.

S. Taylor, Y. Zick, J. Roth, 1985, The Journal of clinical endocrinology and metabolism.

Proximal Enhancer of the Human Insulin Receptor Gene Binds the Transcription Factor Sp1

Hui Chen, Simeon I. Taylor, S. Taylor, 1994, Diabetes.

Dual Role of a Dileucine Motif in Insulin Receptor Endocytosis*

C. Haft, S. Taylor, J. Carpentier, 1997, The Journal of Biological Chemistry.

Molecular cloning of pp120/ECTO-ATPase, an endogenous substrate of the insulin receptor kinase.

D. Accili, S. Taylor, S. Najjar, 1993, Advances in experimental medicine and biology.

Expression of variant forms of insulin receptor substrate-1 identified in patients with noninsulin-dependent diabetes mellitus.

D. Accili, G. Sesti, S. Taylor, 1997, The Journal of clinical endocrinology and metabolism.

Insulin bound to the insulin receptor of IM-9 lymphocytes is more accessible to antiinsulin antibodies after treatment with dithiothreitol: bound insulin is deeply buried in the receptor-binding site.

S. Taylor, A. McElduff, J. Schroer, 1984, Endocrinology.

Atypical antiinsulin receptor antibodies in a patient with type B insulin resistance and scleroderma.

B. Marcus-Samuels, S. Taylor, W. Bloise, 1989, The Journal of clinical endocrinology and metabolism.

Effects of thyroid hormone on the insulin receptor in rat liver membranes.

S. Taylor, K. Burman, L. Wartofsky, 1982, Endocrinology.

Hormonal regulation of insulin receptor gene expression. Hydrocortisone and insulin act by different mechanisms.

S. Taylor, P. Gorden, C. McKeon, 1988, The Journal of biological chemistry.

Detection of mutations in the insulin receptor gene in patients with insulin resistance by analysis of single-stranded conformational polymorphisms

Y. Yazaki, M. Kasuga, T. Kadowaki, 1992, Diabetologia.

Use of polymerase chain reaction catalyzed by Taq DNA polymerase for site-specific mutagenesis.

T. Kadowaki, S. Taylor, H. Kadowaki, 1989, Gene.

Absence of functional insulin receptor substrate-3 (IRS-3) gene in humans

G. Lienhard, P. Arner, J. Zierath, 2002, Diabetologia.

Analysis of the juxtamembrane dileucine motif in the insulin receptor.

C. Haft, M. de la Luz Sierra, S. Taylor, 1998, Endocrinology.

Substitution of arginine for histidine at position 209 in the alpha-subunit of the human insulin receptor. A mutation that impairs receptor dimerization and transport of receptors to the cell surface.

D. Accili, Y. Yazaki, T. Kadowaki, 1991, The Journal of biological chemistry.

Substitution of lysine for asparagine at position 15 in the alpha-subunit of the human insulin receptor. A mutation that impairs transport of receptors to the cell surface and decreases the affinity of insulin binding.

D. Accili, T. Kadowaki, S. Taylor, 1990, The Journal of biological chemistry.

Structural and functional analysis of the insulin receptor promoter.

D. Accili, T. Kadowaki, S. Taylor, 1990, Molecular endocrinology.

Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia.

S. Aizawa, S. Sciacchitano, Y. Terauchi, 2000, Diabetes.

Expression of a cDNA encoding the human insulin receptor-related receptor.

D. Accili, S. Taylor, Y. Suzuki, 1994, The Journal of biological chemistry.

Mutational and haplotype analyses of families with familial partial lipodystrophy (Dunnigan variety) reveal recurrent missense mutations in the globular C-terminal domain of lamin A/C.

M. Lovett, A. Bowcock, Simeon I. Taylor, 2000, American journal of human genetics.

Substitution of glutamic acid for alanine 1135 in the putative "catalytic loop" of the tyrosine kinase domain of the human insulin receptor. A mutation that impairs proteolytic processing into subunits and inhibits receptor tyrosine kinase activity.

M. Quon, M. de la Luz Sierra, L. Ottini, 1993, The Journal of biological chemistry.

A mutation in the tyrosine kinase domain of the insulin receptor associated with insulin resistance in an obese woman.

M. de la Luz Sierra, Simeon I. Taylor, L. Ottini, 1991, The Journal of clinical endocrinology and metabolism.

Point mutation causing a single amino acid substitution in the hormone binding domain of the glucocorticoid receptor in familial glucocorticoid resistance.

K. Constantine, D. Accili, G. Chrousos, 1991, The Journal of clinical investigation.

Syndromes of autoimmunity and hypoglycemia. Autoantibodies directed against insulin and its receptor.

D. Accili, S. Taylor, P. Gorden, 1989, Endocrinology and metabolism clinics of North America.

Effects of overexpressing wild-type and mutant PDGF receptors on translocation of GLUT4 in transfected rat adipose cells.

M. Quon, S. Taylor, A. Kazlauskas, 1996, Biochemical and biophysical research communications.

A gene for congenital generalized lipodystrophy maps to human chromosome 9q34.

A. Bowcock, S. O’Rahilly, Simeon I. Taylor, 1999, The Journal of clinical endocrinology and metabolism.

Rescue and activation of a binding-deficient insulin receptor. Evidence for intermolecular transphosphorylation.

P. Roach, S. Taylor, M. Taouis, 1994, The Journal of biological chemistry.

Human orthologs of yeast vacuolar protein sorting proteins Vps26, 29, and 35: assembly into multimeric complexes.

C. Haft, M. de la Luz Sierra, V. Barr, 2000, Molecular biology of the cell.

Rational design of peptide agonists of cell-surface receptors.

S. Taylor, 2000, TIPS - Trends in Pharmacological Sciences.

Antibodies to the extracellular receptor domain restore the hormone-insensitive kinase and conformation of the mutant insulin receptor valine 382.

E. Van Obberghen, D. Accili, S. Taylor, 1993, The Journal of biological chemistry.

Deletion of exon 3 of the insulin receptor gene in a kindred with a familial form of insulin resistance.

R. Ebstein, D. Accili, S. Taylor, 1994, The Journal of clinical endocrinology and metabolism.

Five mutant alleles of the insulin receptor gene in patients with genetic forms of insulin resistance.

Simeon I. Taylor, T. Kadowaki, S. Taylor, 1990, The Journal of clinical investigation.

The insulin-stimulated receptor kinase is a tyrosine-specific casein kinase.

G. Grunberger, S. Taylor, Y. Zick, 1983, European journal of biochemistry.

Author Index to Volume 341

S. Taylor, L. Mosthaf, R. Levy-Toledano, 2003, Neuroscience Letters.

Involvement of dileucine motifs in the internalization and degradation of the insulin receptor.

R. Klausner, C. Haft, Simeon I. Taylor, 1994, The Journal of biological chemistry.

Receptor defects in patients with extreme insulin resistance.

S. Taylor, 1985, Diabetes/metabolism reviews.

Streptavidin blotting: a sensitive technique to study cell surface proteins; application to investigate autophosphorylation and endocytosis of biotin-labeled insulin receptors.

S. Taylor, L. Caro, R. Levy-Toledano, 1993, Endocrinology.

Targeted inactivation of the insulin receptor gene in mouse 3T3-L1 fibroblasts via homologous recombination.

D. Accili, S. Taylor, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Tumor-promoting phorbol ester stimulates tyrosine phosphorylation in U-937 monocytes.

G. Grunberger, S. Taylor, Y. Zick, 1984, Proceedings of the National Academy of Sciences of the United States of America.

Case Report: Hypoglycemia Due to a Novel Activating Glucokinase Variant in an Adult – a Molecular Approach

R. MacIsaac, S. Taylor, H. Thomas, 2022, Frontiers in Endocrinology.

Non-Insulin-Mediated Glucose Disappearance in Subjects With IDDM: Discordance Between Experimental Results and Minimal Model Analysis

M. Quon, S. Taylor, R. Eastman, 1994, Diabetes.

Insulin resistance associated with decreased levels of insulin-receptor messenger ribonucleic acid: evidence of a de novo mutation in the maternal allele.

S. Taylor, H. Makino, Y. Suzuki, 1995, The Journal of clinical endocrinology and metabolism.

Cell surface alteration in Epstein-Barr virus-transformed cells from patients with extreme insulin resistance.

S. Taylor, J. Carpentier, D. Gorden, 1990, Diabetes.

Structural basis by which a recessive mutation in the alpha-subunit of the insulin receptor affects insulin binding.

P. Roach, S. Taylor, M. Taouis, 1994, The Journal of biological chemistry.

Characterization of a hybrid receptor formed by dimerization of the insulin receptor-related receptor (IRR) with the insulin receptor (IR): coexpression of cDNAs encoding human IRR and human IR in NIH-3T3 cells.

D. Accili, S. Taylor, H. Jui, 1996, Biochemistry.

Substitution of Leu for Pro-193 in the insulin receptor in a patient with a genetic form of severe insulin resistance.

R. Taramelli, M. Ferrari, S. Taylor, 1993, Human molecular genetics.

Binding of Insulin to Its Receptor Impairs Recognition by Monoclonal Anti-insulin Antibodies

S. Taylor, T. Bender, J. Schroer, 1984, Diabetes.

Protein phosphorylation and insulin action.

D. Accili, S. Taylor, S. Phillips, 1988, Advances in experimental medicine and biology.

Structure of the insulin receptor and post-receptor events in cells from a type A insulin-resistant patient with normal alpha-subunit but defective function of the beta-subunit of the insulin receptor.

G. Grunberger, S. Taylor, P. Gorden, 1985, Transactions of the Association of American Physicians.

Defects in receptor biosynthesis in patients with genetic forms of extreme insulin resistance.

S. Taylor, J. A. Hedo, A. Mcelduff, 1984, Transactions of the Association of American Physicians.

Immunoglobulin heavy chain-binding protein binds to misfolded mutant insulin receptors with mutations in the extracellular domain.

A. Ullrich, D. Accili, T. Kadowaki, 1992, The Journal of biological chemistry.

Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia.

S. Aizawa, S. Sciacchitano, Y. Terauchi, 2000, Diabetes.

Regulation of pyruvate dehydrogenase in isolated rat liver mitochondria. Effects of octanoate, oxidation-reduction state, and adenosine triphosphate to adenosine diphosphate ratio.

S. Taylor, C. Mukherjee, R. Jungas, 1975, The Journal of biological chemistry.