T. Meyer

发表

Enzymes and signal pathways in the pathogenesis of alcoholic cardiomyopathy

E. Leibing, T. Meyer, Prof. Dr. mult. T. Meyer, 2016, Herz.

Nucleocytoplasmic shuttling by nucleoporins Nup153 and Nup214 and CRM1-dependent nuclear export control the subcellular distribution of latent Stat1

Andreas Marg, U. Vinkemeier, T. Meyer, 2004, The Journal of cell biology.

Pattern-Recognition Receptors Sensing Viral Infection in Myocarditis and Inflammatory Heart Disease

T. Meyer, V. Ruppert, 2011 .

Constitutive and IFN‐γ‐induced nuclear import of STAT1 proceed through independent pathways

I. Lödige, U. Vinkemeier, T. Meyer, 2002 .

Nuclear localization and phosphorylation modulate pathological effects of alpha‐synuclein

S. Kügler, J. Attems, Cemil Kerimoglu, 2018, Human molecular genetics.

An inhibitory effect on the nuclear accumulation of phospho-STAT1 by its unphosphorylated form

O. Wirths, T. Meyer, P. Menon, 2022, Cell Communication and Signaling.

Novel Insights into the Pathophysiology of Chagas' Cardiomyopathy

T. Meyer, P. Stahl, 2017 .

Novel mutations in the sarcomeric protein myopalladin in patients with dilated cardiomyopathy

T. Meyer, S. Sperling, V. Ruppert, 2012, European Journal of Human Genetics.

Polymorphisms in genes encoding nonsarcomeric proteins and their role in the pathogenesis of dilated cardiomyopathy

T. Meyer, V. Ruppert, S. Pankuweit, 2012, Herz.

Evidence for CTLA4 as a susceptibility gene for dilated cardiomyopathy

T. Meyer, V. Ruppert, B. Maisch, 2010, European Journal of Human Genetics.

Identification of a missense mutation in the melusin-encoding ITGB1BP2 gene in a patient with dilated cardiomyopathy.

T. Meyer, V. Ruppert, B. Maisch, 2013, Gene.

Genotype‐specific effects on left ventricular function in parvovirus B19‐positive patients with dilated cardiomyopathy

T. Meyer, V. Ruppert, B. Maisch, 2011, Journal of medical virology.

Isolated Cardiac Sarcoidosis Associated with the Expression of a Splice Variant Coding for a Truncated BTNL2 Protein

T. Meyer, V. Ruppert, B. Maisch, 2007, Cardiology.

Trypomastigotes and amastigotes of Trypanosoma cruzi induce apoptosis and STAT3 activation in cardiomyocytes in vitro

R. Schwarz, T. Meyer, R. Gazzinelli, 2013, Apoptosis.

Detection of a large duplication mutation in the myosin-binding protein C3 gene in a case of hypertrophic cardiomyopathy.

T. Meyer, V. Ruppert, B. Maisch, 2013, Gene.

The HLA class II allele DQB1 0309 is associated with dilated cardiomyopathy.

T. Meyer, V. Ruppert, H. Müller, 2013, Gene.

Evaluation of the putative lymphoma-associated point mutation D427H in the STAT3 transcription factor

O. Wirths, T. Meyer, P. Menon, 2022, BMC molecular and cell biology.

Activated STAT1 and STAT5 transcription factors in extramedullary hematopoietic tissue in a polycythemia vera patient carrying the JAK2 V617F mutation

T. Meyer, C. Görg, V. Ruppert, 2010, International journal of hematology.

Common variable immunodeficiency, impaired neurological development and reduced numbers of T regulatory cells in a 10-year-old boy with a STAT1 gain-of-function mutation.

S. Fuchs, T. Meyer, R. Kobbe, 2016, Gene.

Do the two transcription factors form a positive feedback amplifier circuit in their common fight against pathogens?

T. Meyer, J. Staab, R. Nast, 2018, Immunology and cell biology.

Trypanosoma cruzi parasites fight for control of the JAK-STAT pathway by disarming their host.

R. Schwarz, T. Meyer, P. Stahl, 2014, JAK-STAT.

Cell-Type-Specific Expression of STAT Transcription Factors in Tissue Samples from Patients with Lymphocytic Thyroiditis

T. Meyer, P. Barth, J. Staab, 2012, Endocrine Pathology.

STAT1-cooperative DNA binding distinguishes type 1 from type 2 interferon signaling

Ronald Naumann, Filipa Antunes, Markus R Owen, 2014, Nature Immunology.

Posttranscriptional Gene Silencing

T. Meyer, V. Ruppert, B. Maisch, 2009 .

Nucleocytoplasmic translocation of Stat1 is regulated by a leucine-rich export signal in the coiled-coil domain.

U. Vinkemeier, T. Meyer, A. Begitt, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Hereditary long QT syndrome due to autoimmune hypoparathyroidism in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy syndrome.

T. Meyer, V. Ruppert, B. Maisch, 2007, Journal of electrocardiology.

Microinjected antibodies interfere with protein nucleocytoplasmic shuttling by distinct molecular mechanisms

M. Vigneron, Andreas Marg, U. Vinkemeier, 2008, Cytometry. Part A : the journal of the International Society for Analytical Cytology.

STAT nuclear translocation: potential for pharmacological intervention

U. Vinkemeier, T. Meyer, 2007, Expert opinion on therapeutic targets.

Cytokine Signaling in Tissue Engineering

T. Meyer, V. Ruppert, B. Maisch, 2009 .

JAK-STAT Signaling Circuits in Myocarditis and Dilated Cardiomyopathy*

T. Meyer, V. Ruppert, 2007, Herz Kardiovaskuläre Erkrankungen.

Signal Pathways from the Plasma Membrane to the Nucleus Regulating Craniofacial Pattern Formation

T. Meyer, 2021, Fundamentals of Craniofacial Malformations.

Toll-Like Receptors: Potential Targets for Therapeutic Interventions

T. Meyer, V. Ruppert, B. Maisch, 2009 .

DNA binding controls inactivation and nuclear accumulation of the transcription factor Stat1.

Andreas Marg, B. Wiesner, U. Vinkemeier, 2003, Genes & development.

A Weak TATA Box Is a Prerequisite for Glucocorticoid-dependent Repression of the Osteocalcin Gene*

T. Meyer, J. Carlstedt-Duke, T. Meyer, 1997, The Journal of Biological Chemistry.

Length Polymorphisms in the Angiotensin I-Converting Enzyme Gene and the Serotonin-Transporter-Linked Polymorphic Region Constitute a Risk Haplotype for Depression in Patients with Coronary Artery Disease

M. Hellmich, M. Michal, W. Söllner, 2020, Biochemical Genetics.

CDK8 as the STAT1 serine 727 kinase?

C. Herrmann-Lingen, T. Meyer, J. Staab, 2013, JAK-STAT.

Gene Activation by the Cytokine-Driven Transcription Factor STAT1

T. Meyer, J. Staab, R. Nast, 2019, Gene Regulation.

Toxoplasma gondii stabilises tetrameric complexes of tyrosine‐phosphorylated signal transducer and activator of transcription‐1 and leads to its sustained and promiscuous DNA binding

T. Meyer, C. Lüder, J. Staab, 2018, Cellular microbiology.

Mechanisms of Mitochondrial Redox Signaling in Psychosocial Stress-Responsive Systems: New Insights into an Old Story.

T. Meyer, P. Wirtz, 2017, Antioxidants & redox signaling.

The two interfaces of the STAT1 N-terminus exhibit opposite functions in IFNγ-regulated gene expression.

C. Herrmann-Lingen, T. Meyer, Theresa Riebeling, 2015, Molecular immunology.

Trypanosoma cruzi Evades the Protective Role of Interferon-Gamma-Signaling in Parasite-Infected Cells

R. Schwarz, T. Meyer, P. Stahl, 2014, PloS one.

A Conserved Motif in the Linker Domain of STAT1 Transcription Factor Is Required for Both Recognition and Release from High-Affinity DNA-Binding Sites

C. Herrmann-Lingen, T. Meyer, J. Staab, 2014, PloS one.

Clinically Relevant Dimer Interface Mutants of STAT1 Transcription Factor Exhibit Differential Gene Expression

C. Herrmann-Lingen, T. Meyer, J. Staab, 2013, PloS one.

A rapid conformational rearrangement of STAT1 dimers is required for termination rather than for amplification of interferon-γ signaling

C. Herrmann-Lingen, T. Meyer, J. Staab, 2013, JAK-STAT.

Two glutamic acid residues in the DNA-binding domain are engaged in the release of STAT1 dimers from DNA

T. Meyer, V. Ruppert, V. Koch, 2012, BMC Cell Biology.

Assessing sequence-specific DNA binding and transcriptional activity of STAT1 transcription factor.

U. Vinkemeier, T. Meyer, 2010, Methods in molecular biology.

Cell type-specific and tyrosine phosphorylation-independent nuclear presence of STAT1 and STAT3.

U. Vinkemeier, T. Meyer, K. Gavénis, 2002, Experimental cell research.

Constitutive and IFN-gamma-induced nuclear import of STAT1 proceed through independent pathways.

U. Vinkemeier, T. Meyer, A. Begitt, 2002, The EMBO journal.

Identification of a distinct subset of disease-associated gain-of-function missense mutations in the STAT1 coiled-coil domain as system mutants.

T. Buhl, T. Meyer, A. Ivetic, 2019, Molecular immunology.

DNA binding reduces the dissociation rate of STAT1 dimers and impairs the interdimeric exchange of protomers

C. Herrmann-Lingen, T. Meyer, Theresa Riebeling, 2014, BMC Biochemistry.

A Single Residue Modulates Tyrosine Dephosphorylation, Oligomerization, and Nuclear Accumulation of Stat Transcription Factors*

U. Vinkemeier, T. Meyer, S. John, 2004, Journal of Biological Chemistry.

Lack of STAT1 co-operative DNA binding protects against adverse cardiac remodelling in acute myocardial infarction

O. Wirths, U. Vinkemeier, T. Meyer, 2023, Frontiers in Cardiovascular Medicine.

Nucleocytoplasmic shuttling of STAT transcription factors.

U. Vinkemeier, T. Meyer, 2004, European journal of biochemistry.

Trypomastigotes and amastigotes of Trypanosoma cruzi induce apoptosis and STAT3 activation in cardiomyocytes in vitro

R. Schwarz, T. Meyer, R. Gazzinelli, 2013, Apoptosis.

STAM and Hrs interact sequentially with IFN-α Receptor to control spatiotemporal JAK–STAT endosomal activation

T. Meyer, J. Piehler, Daniela Chmiest, 2023, Nature Cell Biology.

The anti-parallel dimer binding interface in STAT3 transcription factor is required for the inactivation of cytokine-mediated signal transduction.

O. Wirths, T. Meyer, P. Menon, 2021, Biochimica et biophysica acta. Molecular cell research.

The pathogenic T387A missense mutation in the gene encoding signal transducer and activator of transcription 1 exhibits a differential gene expression profile.

T. Meyer, J. Staab, Till Schwämmle, 2020, Molecular immunology.

Green fluorescent protein‐tagging reduces the nucleocytoplasmic shuttling specifically of unphosphorylated STAT1

U. Vinkemeier, T. Meyer, A. Begitt, 2007, The FEBS journal.

STAT1-cooperative DNA binding distinguishes type 1 from type 2 interferon signaling

M. Owen, K. Knobeloch, U. Vinkemeier, 2014, Nature Immunology.

Gene expression profiling from endomyocardial biopsy tissue allows distinction between subentities of dilated cardiomyopathy.

T. Meyer, W. Grimm, V. Ruppert, 2008, The Journal of thoracic and cardiovascular surgery.

A novel interface between the N-terminal and coiled-coil domain of STAT1 functions in an auto-inhibitory manner

O. Wirths, T. Meyer, J. Staab, 2023, Cell Communication and Signaling.

Activation of STAT1 transcription factor precedes up-regulation of coxsackievirus-adenovirus receptor during viral myocarditis.

T. Meyer, V. Ruppert, B. Maisch, 2008, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.

Common variable immunodeficiency, impaired neurological development and reduced numbers of T regulatory cells in a 10-year-old boy with a STAT1 gain-of-function mutation.

S. Fuchs, T. Meyer, R. Kobbe, 2016, Gene.