Daniela Ponce-Balbuena

发表

TGF-β1, Released by Myofibroblasts, Differentially Regulates Transcription and Function of Sodium and Potassium Channels in Adult Rat Ventricular Myocytes

J. Jalife, K. Kaur, M. Zarzoso, 2013, PloS one.

Extracellular Matrix–Mediated Maturation of Human Pluripotent Stem Cell–Derived Cardiac Monolayer Structure and Electrophysiological Function

José Jalife, Alexandra Bizy, Todd J Herron, 2016, Circulation. Arrhythmia and electrophysiology.

Kir2.1 Interactome Mapping Uncovers PKP4 as a Modulator of the Kir2.1-Regulated Inward Rectifier Potassium Currents

R. Kuick, A. Nesvizhskii, J. Rual, 2020, Molecular & Cellular Proteomics.

In vitro model of ischemic heart failure using human induced pluripotent stem cell–derived cardiomyocytes

Daniela Ponce-Balbuena, N. Madamanchi, T. Herron, 2021, JCI insight.

Human influenza A virus causes myocardial and cardiac-specific conduction system infections associated with early inflammation and premature death

J. Jalife, M. Desco, Daniela Ponce-Balbuena, 2020, Cardiovascular research.

SNTA1 gene rescues ion channel function and is antiarrhythmic in cardiomyocytes derived from induced pluripotent stem cells from muscular dystrophy patients

J. Jalife, Daniela Ponce-Balbuena, G. Guerrero-Serna, 2022, eLife.

SNTA1 Gene Rescues Ion Channel Function in Cardiomyocytes Derived from Induced Pluripotent Stem Cells Reprogrammed from Muscular Dystrophy Patients with Arrhythmias

J. Jalife, Daniela Ponce-Balbuena, G. Guerrero-Serna, 2022, bioRxiv.

Paclitaxel mitigates structural alterations and cardiac conduction system defects in a mouse model of Hutchinson–Gilford progeria syndrome

J. Jalife, Daniela Ponce-Balbuena, V. Andrés, 2021, Cardiovascular research.

Human perinatal stem cell derived extracellular matrix enables rapid maturation of hiPSC-CM structural and functional phenotypes

S. Griffey, Daniela Ponce-Balbuena, T. Herron, 2020, Scientific Reports.

Mechanisms for Kir channel inhibition by quinacrine: acute pore block of Kir2.x channels and interference in PIP2 interaction with Kir2.x and Kir6.2 channels

Daniela Ponce-Balbuena, J. Sánchez-Chapula, A. Lopez-Izquierdo, 2011, Pflügers Archiv - European Journal of Physiology.

Tbx20 controls the expression of the KCNH2 gene and of hERG channels

J. Tamargo, J. López-Sendón, J. Jalife, 2017, Proceedings of the National Academy of Sciences.

Cardiac Kir2.1 and NaV1.5 Channels Traffic Together to the Sarcolemma to Control Excitability

Francisco J. Alvarado, R. Ramirez, J. Jalife, 2018, Circulation research.

The Antimalarial Drug Mefloquine Inhibits Cardiac Inward Rectifier K+ Channels: Evidence for Interference in PIP2-Channel Interaction

Daniela Ponce-Balbuena, J. Sánchez-Chapula, A. Lopez-Izquierdo, 2011, Journal of cardiovascular pharmacology.

Brugada syndrome trafficking-defective Nav1.5 channels can trap cardiac Kir2.1/2.2 channels.

J. Tamargo, J. Jalife, Daniela Ponce-Balbuena, 2018, JCI insight.

Myosin light chain 2-based selection of human iPSC-derived early ventricular cardiac myocytes.

R. Ramirez, J. Jalife, M. Zarzoso, 2013, Stem cell research.

Mutated KCNJ5 activates the acute and chronic regulatory steps in aldosterone production.

J. Jalife, Daniela Ponce-Balbuena, C. Gomez-Sanchez, 2016, Journal of molecular endocrinology.

Thiopental inhibits function of different inward rectifying potassium channel isoforms by a similar mechanism.

Daniela Ponce-Balbuena, J. Sánchez-Chapula, A. Lopez-Izquierdo, 2010, European journal of pharmacology.

Molecular and Electrophysiological Analyses of ATP2B4 Gene Variants in Bilateral Adrenal Hyperaldosteronism

Daniela Ponce-Balbuena, P. Mulatero, S. Monticone, 2020, Hormones and Cancer.

Tamoxifen inhibits cardiac ATP-sensitive and acetylcholine-activated K+ currents in part by interfering with phosphatidylinositol 4,5-bisphosphate-channel interaction.

Daniela Ponce-Balbuena, J. Sánchez-Chapula, A. Lopez-Izquierdo, 2010, Journal of pharmacological sciences.

Long QT syndrome – Bench to bedside

Daniela Ponce-Balbuena, I. Deschênes, 2021, Heart rhythm O2.

Nav1.5 N-terminal domain binding to α1-syntrophin increases membrane density of human Kir2.1, Kir2.2 and Nav1.5 channels.

J. Tamargo, J. Jalife, Daniela Ponce-Balbuena, 2016, Cardiovascular research.

Protein assemblies of sodium and inward rectifier potassium channels control cardiac excitability and arrhythmogenesis.

José Jalife, J. Jalife, Daniela Ponce-Balbuena, 2015, American journal of physiology. Heart and circulatory physiology.

Dominant Frequency Increase Rate Predicts Transition from Paroxysmal to Long-Term Persistent Atrial Fibrillation

Michael F Swartz, M. Deo, R. Ramirez, 2014, Circulation.

Specific residues of the cytoplasmic domains of cardiac inward rectifier potassium channels are effective antifibrillatory targets

O. Berenfeld, J. Jalife, J. Stuckey, 2010, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Eplerenone Reduces Atrial Fibrillation Burden Without Preventing Atrial Electrical Remodeling.

R. Ramirez, O. Berenfeld, J. Jalife, 2017, Journal of the American College of Cardiology.

Galectin-3 Regulates Atrial Fibrillation Remodeling and Predicts Catheter Ablation Outcomes

F. Morady, R. Ramirez, O. Berenfeld, 2016, JACC. Basic to translational science.

Constitutive Intracellular Na+ Excess in Purkinje Cells Promotes Arrhythmogenesis at Lower Levels of Stress Than Ventricular Myocytes From Mice With Catecholaminergic Polymorphic Ventricular Tachycardia

M. Deo, R. Ramirez, J. Jalife, 2016, Circulation.

Chloroquine Blocks a Mutant Kir2.1 Channel Responsible for Short QT Syndrome and Normalizes Repolarization Properties in silico

Frank B. Sachse, Martin Tristani-Firouzi, F. Sachse, 2009, Cellular Physiology and Biochemistry.

ABCC9 is a novel Brugada and early repolarization syndrome susceptibility gene.

C. Antzelevitch, M. Borggrefe, A. Terzic, 2014, International journal of cardiology.

Structural bases for the different anti-fibrillatory effects of chloroquine and quinidine.

José Jalife, Sandeep V Pandit, J. Jalife, 2011, Cardiovascular research.

Carvedilol inhibits Kir2.3 channels by interference with PIP₂-channel interaction.

Daniela Ponce-Balbuena, T. D. de Boer, J. Sánchez-Chapula, 2011, European journal of pharmacology.

Decreased FAM13B Expression Increases Atrial Fibrillation Susceptibility by Regulating Sodium Current and Calcium Handling

D. V. Van Wagoner, Daniela Ponce-Balbuena, C. Moravec, 2023, JACC: Basic to Translational Science.

Molecular Mechanisms of Chloroquine Inhibition of Heterologously Expressed Kir6.2/SUR2A Channels

C. Nichols, Daniela Ponce-Balbuena, J. Sánchez-Chapula, 2012, Molecular Pharmacology.

Dominant Frequency Increase Rate Predicts Transition from Paroxysmal to Long-Term Persistent Atrial Fibrillation

Michael F Swartz, M. Deo, R. Ramirez, 2014, Circulation.

Dominant Frequency Increase Rate Predicts Transition from Paroxysmal to Long-term Persistent Atrial Fibrillation Running Title: Martins Et Al.; Transition from Paroxysmal to Persistent Af

M. Deo, O. Berenfeld, J. Jalife, 2022 .

p38γ/δ activation alters cardiac electrical activity and predisposes to ventricular arrhythmia

Daniela Ponce-Balbuena, A. Mora, D. Filgueiras-Rama, 2023, Nature Cardiovascular Research.

Molecular Mechanisms of Chloroquine Inhibition of Heterologously Expressed Kir6.2/SUR2A Channels

Daniela Ponce-Balbuena, J. Sánchez-Chapula, T. Ferrer, 2012, Molecular Pharmacology.