H. Díez

发表

Specific Roles of Akt iso Forms in Apoptosis and Axon Growth Regulation in Neurons

J. Garrido, H. Díez, F. Wandosell, 2012, PloS one.

Assessment of Autophagy in Neurons and Brain Tissue

H. Díez, F. Wandosell, Irene Benito-Cuesta, 2017, Cells.

DDC (AADC) expression is not regulated by NFAT5 (TonEBP) in dopaminergic neural cell lines.

N. Fernàndez-Castillo, B. Cormand, H. Díez, 2020, Gene.

Severe infantile parkinsonism because of a de novo mutation on DLP1 mitochondrial‐peroxisomal protein

J. Armstrong, A. Ormazabal, R. Artuch, 2017, Movement disorders : official journal of the Movement Disorder Society.

Oestradiol signalling through the Akt-mTORC1-S6K1.

J. Garrido, H. Díez, F. Wandosell, 2013, Biochimica et biophysica acta.

Protein Nanomechanics — as Studied by AFM Single-Molecule Force Spectroscopy

A. Oberhauser, M. Carrión-Vázquez, H. Díez, 2006 .

Class I PI3-kinase or Akt inhibition do not impair axonal polarization, but slow down axonal elongation.

J. Garrido, I. Torres-Aleman, S. Fernández, 2016, Biochimica et biophysica acta.

Study of a fetal brain affected by a severe form of tyrosine hydroxylase deficiency, a rare cause of early parkinsonism

A. Ormazabal, R. Artuch, M. Pineda, 2016, Metabolic brain disease.

Discovery of compounds that protect tyrosine hydroxylase activity through different mechanisms.

N. Fernàndez-Castillo, Aurora Martínez, H. Díez, 2015, Biochimica et biophysica acta.

Study of a fetal brain affected by a severe form of tyrosine hydroxylase deficiency, a rare cause of early parkinsonism

A. Ormazabal, R. Artuch, M. Pineda, 2015, Metabolic Brain Disease.

Discovery of compounds that protect tyrosine hydroxylase activity through different mechanisms.

N. Fernàndez-Castillo, Aurora Martínez, H. Díez, 2015, Biochimica et biophysica acta.