F. Sari

14-3-3 protein protects against cardiac endoplasmic reticulum stress (ERS) and ERS-initiated apoptosis in experimental diabetes.

Anthony J. Muslin, R. Thandavarayan, Kenichi Watanabe, 2010, Journal of pharmacological sciences.

Partial Inactivation of Cardiac 14-3-3 Protein in vivo Elicits Endoplasmic Reticulum Stress (ERS) and Activates ERS-initiated Apoptosis in ERS-induced Mice

Anthony J. Muslin, R. Thandavarayan, Kenichi Watanabe, 2006, Cellular Physiology and Biochemistry.

Modulation of AT-1R/MAPK cascade by an olmesartan treatment attenuates diabetic nephropathy in streptozotocin-induced diabetic mice

Kenji Suzuki, R. Thandavarayan, V. Sukumaran, 2012, Molecular and Cellular Endocrinology.

The Role of Carvedilol in the Treatment of Dilated and Anthracyclines-Induced Cardiomyopathy

Kenji Suzuki, R. Thandavarayan, Kenichi Watanabe, 2011, Pharmaceuticals.

Depletion of 14-3-3 Protein Exacerbates Cardiac Oxidative Stress, Inflammation and Remodeling Process via Modulation of MAPK/NF-ĸB Signaling Pathways after Streptozotocin-induced Diabetes Mellitus

Kenji Suzuki, P. Veeraveedu, S. Palaniyandi, 2011, Cellular Physiology and Biochemistry.

Carvedilol-Afforded Protection against Daunorubicin-Induced Cardiomyopathic Rats In Vivo: Effects on Cardiac Fibrosis and Hypertrophy

Kenji Suzuki, P. Veeraveedu, R. Thandavarayan, 2011, ISRN pharmacology.

Modulation of AT-1R/CHOP-JNK-Caspase12 pathway by olmesartan treatment attenuates ER stress-induced renal apoptosis in streptozotocin-induced diabetic mice.

Kenji Suzuki, S. Palaniyandi, R. Thandavarayan, 2011, European journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences.