B. Rodrigues

发表

The increase in cardiac pyruvate dehydrogenase kinase-4 after short-term dexamethasone is controlled by an Akt-p38-forkhead box other factor-1 signaling axis.

F. Wang, P. Puthanveetil, Ying Wang, 2010, Endocrinology.

AMPK Regulation of Cardiac Metabolism in Heart Disease

Min Suk Kim, B. Rodrigues, D. An, 2008 .

Free fatty acids do not release lipoprotein lipase from isolated cardiac myocytes or perfused hearts.

D. Severson, B. Rodrigues, M. Spooner, 1992, The American journal of physiology.

Cardiomyocyte VEGF Regulates Endothelial Cell GPIHBP1 to Relocate Lipoprotein Lipase to the Coronary Lumen During Diabetes Mellitus

B. Rodrigues, I. Vlodavsky, Amy P Chiu, 2016, Arteriosclerosis, thrombosis, and vascular biology.

Metabolic disturbances in diabetic cardiomyopathy

B. Rodrigues, J. McNeill, M. Cam, 1998 .

Myocardial substrate metabolism: implications for diabetic cardiomyopathy.

B. Rodrigues, J. McNeill, M. Cam, 1995, Journal of molecular and cellular cardiology.

Hypertriglyceridemia in experimental diabetes: relationship to cardiac dysfunction.

B. Rodrigues, M. Battell, J. McNeill, 1994, Canadian journal of physiology and pharmacology.

AMP-activated protein kinase confers protection against TNF-{alpha}-induced cardiac cell death.

James D. Johnson, F. Wang, P. Puthanveetil, 2009, Cardiovascular research.

Adiponectin increases LPL activity via RhoA/ROCK-mediated actin remodelling in adult rat cardiomyocytes.

F. Thong, B. Rodrigues, G. Sweeney, 2011, Endocrinology.

Cardiac lipoprotein lipase: metabolic basis for diabetic heart disease.

T. Pulinilkunnil, B. Rodrigues, 2006, Cardiovascular research.

Circulating triglyceride lipolysis facilitates lipoprotein lipase translocation from cardiomyocyte to myocardial endothelial lining.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2003, Cardiovascular research.

DIABETIC CARDIOMYOPATHY The central basis for diabetic heart failure is coronary disease associated with atherosclerosis

B. Rodrigues, D. An, Brian Rodrigues, 2006 .

Endothelial cells respond to hyperglycemia by increasing the LPL transporter GPIHBP1.

Ying Wang, B. Rodrigues, I. Vlodavsky, 2014, American journal of physiology. Endocrinology and metabolism.

Hepatic cytochrome P-450j induction in the spontaneously diabetic BB rat.

B. Rodrigues, P. Thomas, J. McNeill, 1988, Molecular pharmacology.

Loss of VEGFB and its signaling in the diabetic heart is associated with increased cell death signaling.

B. Rodrigues, I. Vlodavsky, Amy P Chiu, 2017, American journal of physiology. Heart and circulatory physiology.

AMPK control of myocardial fatty acid metabolism fluctuates with the intensity of insulin-deficient diabetes.

Sanjoy Ghosh, T. Pulinilkunnil, Min Suk Kim, 2007, Journal of molecular and cellular cardiology.

Acute dexamethasone-induced increase in cardiac lipoprotein lipase requires activation of both Akt and stress kinases.

F. Wang, P. Puthanveetil, Min Suk Kim, 2008, American journal of physiology. Endocrinology and metabolism.

Lipoprotein lipase and angiopoietin-like 4 – Cardiomyocyte secretory proteins that regulate metabolism during diabetic heart disease

P. Puthanveetil, B. Rodrigues, Andrea Wan, 2015, Critical reviews in clinical laboratory sciences.

Localization of lipoprotein lipase in the diabetic heart: regulation by acute changes in insulin.

B. Rodrigues, M. Cam, N. Sambandam, 1999, Arteriosclerosis, thrombosis, and vascular biology.

Distinct glucose lowering and beta cell protective effects of vanadium and food restriction in streptozotocin-diabetes.

B. Rodrigues, J. McNeill, M. Cam, 1999, European journal of endocrinology.

Myocardial Insulin Signaling and Autophagy

P. Trivedi, J. Bartlett, T. Pulinilkunnil, 2018 .

Insulin and the Heart

S. Verma, B. Rodrigues, J. McNeill, 1999 .

Dual effects of hyperglycemia on endothelial cells and cardiomyocytes to enhance coronary LPL activity.

B. Rodrigues, I. Vlodavsky, Amy P Chiu, 2018, American journal of physiology. Heart and circulatory physiology.

Single-dose dexamethasone induces whole-body insulin resistance and alters both cardiac fatty acid and carbohydrate metabolism.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2004, Diabetes.

Brief episode of STZ-induced hyperglycemia produces cardiac abnormalities in rats fed a diet rich in n-6 PUFA.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2004, American journal of physiology. Heart and circulatory physiology.

Physiological and Pathological Consequences of Streptozotocin Diabetes on the Heart

B. Rodrigues, J. McNeill, 2018 .

Interventions in Experimentally Induced Diabetic Cardiomyopathy

B. Rodrigues, J. McNeill, M. Cam, 1996 .

Therapeutic Interventions in the Diabetic Heart

B. Rodrigues, J. McNeill, 1996 .

Cardiac glycogen accumulation following dexamethasone is regulated by AMPK.

P. Puthanveetil, B. Rodrigues, 2008 .

Cardiac glycogen accumulation after dexamethasone is regulated by AMPK.

T. Pulinilkunnil, P. Puthanveetil, Min Suk Kim, 2008, American journal of physiology. Heart and circulatory physiology.

Loss of function of transcription factor EB remodels lipid metabolism and cell death pathways in the cardiomyocyte.

Petra C. Kienesberger, A. Ballabio, S. Flibotte, 2020, Biochimica et biophysica acta. Molecular basis of disease.

The role of lipid abnormalities in the development of diabetic cardiomyopathy

B. Rodrigues, 1989 .

Diabetes Mellitus Severity and a Switch From Using Lipoprotein Lipase to Adipose‐Derived Fatty Acid Results in a Cardiac Metabolic Signature That Embraces Cell Death

S. Flibotte, Sanjoy Ghosh, B. Rodrigues, 2019, Journal of the American Heart Association.

Role of dietary fatty acids and acute hyperglycemia in modulating cardiac cell death.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2004, Nutrition.

Streptozotocin- and alloxan-induced diabetes modifies total plasma and lipoprotein lipid concentration and composition without altering cholesteryl ester transfer activity.

B. Rodrigues, K. Wasan, W. Wong, 1998, Pharmacology & toxicology.

Intrinsic and extrinsic regulation of cardiac lipoprotein lipase following diabetes.

Ying Wang, B. Rodrigues, 2015, Biochimica et biophysica acta.

Cardiomyocyte-endothelial cell control of lipoprotein lipase.

B. Rodrigues, Amy P Chiu, Andrea Wan, 2016, Biochimica et biophysica acta.

Cardiomyocyte apoptosis induced by short-term diabetes requires mitochondrial GSH depletion.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2005, American journal of physiology. Heart and circulatory physiology.

Diabetes triggers a PARP1 mediated death pathway in the heart through participation of FoxO1.

F. Wang, P. Puthanveetil, Ying Wang, 2012, Journal of molecular and cellular cardiology.

Lipoprotein Lipase and Its Delivery of Fatty Acids to the Heart

B. Rodrigues, Rui Shang, 2021, Biomolecules.

Cleavage of Protein Kinase D After Acute Hypoinsulinemia Prevents Excessive Lipoprotein Lipase–Mediated Cardiac Triglyceride Accumulation

F. Wang, P. Puthanveetil, Min Suk Kim, 2009, Diabetes.

Acute Diabetes Moderates Trafficking of Cardiac Lipoprotein Lipase Through p38 Mitogen-Activated Protein Kinase–Dependent Actin Cytoskeleton Organization

P. Puthanveetil, Min Suk Kim, A. Abrahani, 2008, Diabetes.

Acute diabetes moderates trafficking of cardiac lipoprotein lipase through p38 MAPK dependent actin cytoskeleton organization

P. Puthanveetil, Min Suk Kim, A. Abrahani, 2007 .

The metabolic "switch" AMPK regulates cardiac heparin-releasable lipoprotein lipase.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2005, American journal of physiology. Endocrinology and metabolism.

Evidence for rapid "metabolic switching" through lipoprotein lipase occupation of endothelial-binding sites.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2003, Journal of molecular and cellular cardiology.

Aggravated restenosis and atherogenesis in ApoCIII transgenic mice but lack of protection in ApoCIII knockouts: the effect of authentic triglyceride-rich lipoproteins with and without ApoCIII.

Mei Han, B. Rodrigues, Yingchun Han, 2015, Cardiovascular research.

Proatherogenic and antiatherogenic effects of probucol and phytosterols in apolipoprotein E-deficient mice: possible mechanisms of action.

B. McManus, B. Rodrigues, J. Frohlich, 1999, Circulation.

Cardiac heparin-releasable lipoprotein lipase activity in fructose-hypertensive rats: effect of coronary vasodilation.

B. Rodrigues, M. Cam, N. Sambandam, 1997, Journal of cardiovascular pharmacology.

Altered cardiac fatty acid composition and utilization following dexamethasone-induced insulin resistance.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2006, American journal of physiology. Endocrinology and metabolism.

Altered Substrate Utilization in the Diabetic Heart: Role of Lipoprotein Lipase

T. Pulinilkunnil, B. Rodrigues, N. Sambandam, 2003 .

Metabolism of VLDL is increased in streptozotocin-induced diabetic rat hearts.

B. Rodrigues, N. Sambandam, M. A. Abrahani, 2000, American journal of physiology. Heart and circulatory physiology.

Regulation of lipoprotein lipase activity in cardiac myocytes from control and diabetic rat hearts by plasma lipids.

D. Severson, B. Rodrigues, M. Spooner, 1992, Canadian journal of physiology and pharmacology.

Role of Lipoprotein Lipase in Fatty Acid Delivery to the Heart

B. Rodrigues, Andrea Wan, 2014 .

Hyperglycemia-Induced Protein Kinase C β2 Activation Induces Diastolic Cardiac Dysfunction in Diabetic Rats by Impairing Caveolin-3 Expression and Akt/eNOS Signaling

Haobo Li, M. Irwin, Z. Xia, 2013, Diabetes.

Metformin influences cardiomyocyte cell death by pathways that are dependent and independent of caspase-3

T. Pulinilkunnil, A. Abrahani, B. Rodrigues, 2006, Diabetologia.

Cardiac dysfunction in isolated perfused hearts from spontaneously diabetic BB rats.

B. Rodrigues, J. McNeill, 1990, Canadian journal of physiology and pharmacology.

Lipoprotein lipase mediated fatty acid delivery and its impact in diabetic cardiomyopathy.

Ying Wang, Min Suk Kim, B. Rodrigues, 2012, Biochimica et biophysica acta.

beta-Agonist stimulation produces changes in cardiac AMPK and coronary lumen LPL only during increased workload.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2005, American journal of physiology. Endocrinology and metabolism.

Protein Kinase D Is a Key Regulator of Cardiomyocyte Lipoprotein Lipase Secretion After Diabetes

C. Proud, F. Wang, P. Puthanveetil, 2008, Circulation research.

Cardiac lipoprotein lipase in the spontaneously hypertensive rat.

B. Rodrigues, X. Chen, M. Cam, 1997, Cardiovascular research.

Cardiac function in spontaneously hypertensive diabetic rats.

B. Rodrigues, J. McNeill, 1986, The American journal of physiology.

Effect of tri-iodothyronine on cardiac responses to adrenergic-agonists in STZ-induced diabetic rats.

B. Rodrigues, J. McNeill, R. Goyal, 1987, General pharmacology.

Endothelial Heparanase Regulates Heart Metabolism by Stimulating Lipoprotein Lipase Secretion From Cardiomyocytes

Ying Wang, B. Rodrigues, I. Vlodavsky, 2013, Arteriosclerosis, thrombosis, and vascular biology.

Changes in lipoprotein lipase in the heart following diabetes onset

B. Rodrigues, Yajie Zhai, Chae Syng Lee, 2022, Engineering.

Acute diabetes does not reduce heparin-releasable lipoprotein lipase activity in perfused hearts from Wistar-Kyoto rats.

D. Severson, B. Rodrigues, 1993, Canadian journal of physiology and pharmacology.

Endothelial heparanase secretion after acute hypoinsulinemia is regulated by glucose and fatty acid.

Sanjoy Ghosh, F. Wang, P. Puthanveetil, 2009, American journal of physiology. Heart and circulatory physiology.

Pyruvate dehydrogenase and the regulation of glucose oxidation in hypertrophied rat hearts.

K. M. Popov, Robert A. Harris, B. Rodrigues, 2002, Cardiovascular research.

Palmitoyl lysophosphatidylcholine mediated mobilization of LPL to the coronary luminal surface requires PKC activation.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2004, Journal of molecular and cellular cardiology.

The ischemic metabolite lysophosphatidylcholine increases rat coronary arterial tone by endothelium-dependent mechanisms.

I. Laher, B. Rodrigues, K. Macleod, 2009, Journal of molecular and cellular cardiology.

Cardiac-specific VEGFB overexpression reduces lipoprotein lipase activity and improves insulin action in rat heart.

K. Alitalo, R. Boushel, B. Rodrigues, 2021, American journal of physiology. Endocrinology and metabolism.

Severity of Diabetes Governs Vascular Lipoprotein Lipase by Affecting Enzyme Dimerization and Disassembly

F. Wang, P. Puthanveetil, Ying Wang, 2011, Diabetes.

Susceptibility of spontaneously hypertensive rats to the diabetogenic effects of streptozotocin.

B. Rodrigues, M. Cam, E. Fok, 1996, Canadian journal of physiology and pharmacology.

Endothelial Cell Heparanase Taken Up by Cardiomyocytes Regulates Lipoprotein Lipase Transfer to the Coronary Lumen After Diabetes

Ying Wang, B. Rodrigues, George Liu, 2014, Diabetes.

Endothelial Cell Heparanase Taken Up by Cardiomyocytes Regulates Lipoprotein Lipase Transfer to the Coronary Lumen following Diabetes

Ying Wang, B. Rodrigues, George Liu, 2014 .

Streptozotocin-induced diabetes enhances cardiac heparin-releasable lipoprotein lipase activity in spontaneously hypertensive rats.

B. Rodrigues, M. Cam, N. Sambandam, 1998, Hypertension.

Differential Effects of Streptozotocin-Induced Diabetes on Cardiac Lipoprotein Lipase Activity

B. Rodrigues, M. Cam, N. Sambandam, 1997, Diabetes.

Long term incubation of cardiac myocytes with oleic acid and very-low density lipoprotein reduces heparin-releasable lipoprotein lipase activity.

D. Severson, B. Rodrigues, M. Spooner, 1992 .

Ischemia-reperfusion alters cardiac lipoprotein lipase.

T. Pulinilkunnil, F. Wang, P. Puthanveetil, 2010, Biochimica et biophysica acta.

High glucose facilitated endothelial heparanase transfer to the cardiomyocyte modifies its cell death signature.

B. Rodrigues, I. Vlodavsky, Amy P Chiu, 2016, Cardiovascular research.

A role for hepatic leptin signaling in lipid metabolism via altered very low density lipoprotein composition and liver lipase activity in mice

Ying Wang, B. Rodrigues, T. Kieffer, 2013, Hepatology.

Effect of Choline and Methionine Treatment on Cardiac Dysfunction of Diabetic Rats

B. Rodrigues, J. McNeill, C. Heyliger, 1986, Diabetes.

Effects of in vivo and in vitro treatment with L-carnitine on isolated hearts from chronically diabetic rats.

B. Rodrigues, J. McNeill, J. Ross, 1990, Canadian journal of physiology and pharmacology.

Fatty Acid-Induced Nuclear Translocation of Heparanase Uncouples Glucose Metabolism in Endothelial Cells

James D. Johnson, F. Wang, P. Puthanveetil, 2012, Arteriosclerosis, thrombosis, and vascular biology.

Acute intralipid infusion reduces cardiac luminal lipoprotein lipase but recruits additional enzyme from cardiomyocytes.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2006, Cardiovascular research.

Long term incubation of cardiac myocytes with oleic acid and very-low density lipoprotein reduces heparin-releasable lipoprotein lipase activity

D. Severson, B. Rodrigues, M. Spooner, 2005, Molecular and Cellular Biochemistry.

Lysophosphatidylcholine Potentiates Phenylephrine Responses in Rat Mesenteric Arterial Bed through Modulation of Thromboxane A2

B. Rodrigues, K. Macleod, Rui Zhang, 2006, Journal of Pharmacology and Experimental Therapeutics.

Flow‐Induced Secretion of Endothelial Heparanase Regulates Cardiac Lipoprotein Lipase and Changes Following Diabetes

James D. Johnson, B. Rodrigues, H. Cen, 2022, Journal of the American Heart Association.

Glucose-induced endothelial heparanase secretion requires cortical and stress actin reorganization.

James D. Johnson, Sanjoy Ghosh, F. Wang, 2010, Cardiovascular research.

sarcoplasmic reticulum: effects of fatiguing activity Glycogen and glycogen phosphorylase associated with

I. Smith, T. Pulinilkunnil, K. Foley, 2015 .

Effects of hydralazine on streptozotocin-induced diabetic rats: prevention of hyperlipidemia and improvement in cardiac function.

B. Rodrigues, J. McNeill, R. Goyal, 1986, The Journal of pharmacology and experimental therapeutics.

Induction of mitochondrial nitrative damage and cardiac dysfunction by chronic provision of dietary omega-6 polyunsaturated fatty acids.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2006, Free radical biology & medicine.

Cardiac cell death in early diabetes and its modulation by dietary fatty acids.

Sanjoy Ghosh, B. Rodrigues, 2006, Biochimica et biophysica acta.

Role of changes in cardiac metabolism in development of diabetic cardiomyopathy.

Brian Rodrigues, Ding An, B. Rodrigues, 2006, American journal of physiology. Heart and circulatory physiology.

Cardiomyocyte ATP Production, Metabolic Flexibility, and Survival Require Calcium Flux through Cardiac Ryanodine Receptors in Vivo*

James D. Johnson, B. Rodrigues, D. Luciani, 2013, The Journal of Biological Chemistry.

AMP-activated protein kinase in the heart: role in cardiac glucose and fatty acid metabolism

B. Rodrigues, Girish Kewalramani, 2009 .

Autophagy, Metabolic Disease, and Pathogenesis of Heart Dysfunction.

B. Rodrigues, Fulong Wang, Jocelyn Jia, 2017, The Canadian journal of cardiology.

Involvement of Heparanase in Endothelial Cell-Cardiomyocyte Crosstalk.

B. Rodrigues, Nathaniel Lal, Bahira Hussein, 2020, Advances in experimental medicine and biology.

FoxO1 is crucial for sustaining cardiomyocyte metabolism and cell survival.

P. Puthanveetil, B. Rodrigues, Andrea Wan, 2013, Cardiovascular research.

Effect of hypertension on the development of diabetic cardiomyopathy.

B. Rodrigues, J. McNeill, D. Mathis, 2000, Canadian journal of physiology and pharmacology.

Endothelial cell-cardiomyocyte crosstalk in diabetic cardiomyopathy.

B. Rodrigues, Andrea Wan, 2016, Cardiovascular research.

Lack of effect of oral L-carnitine treatment on lipid metabolism and cardiac function in chronically diabetic rats.

B. Rodrigues, J. McNeill, D. Seccombe, 1990, Canadian journal of physiology and pharmacology.

Effect of L-Carnitine Treatment on Lipid Metabolism and Cardiac Performance in Chronically Diabetic Rats

B. Rodrigues, J. McNeill, H. Xiang, 1988, Diabetes.

Metabolic disturbances in diabetic cardiomyopathy

B. Rodrigues, J. McNeill, M. Cam, 1998, Molecular and Cellular Biochemistry.

Strain differences in susceptibility to streptozotocin-induced diabetes: effects on hypertriglyceridemia and cardiomyopathy.

B. Rodrigues, J. McNeill, R. Goyal, 1997, Cardiovascular research.

Cardiac triglyceride accumulation following acute lipid excess occurs through activation of a FoxO1-iNOS-CD36 pathway.

T. Pulinilkunnil, P. Puthanveetil, Ying Wang, 2011, Free radical biology & medicine.

Rat models of cardiac insulin resistance.

Jun Ren, Sanjoy Ghosh, B. Rodrigues, 2007, Methods in molecular medicine.

β-cell ABCA1 influences insulin secretion, glucose homeostasis and response to thiazolidinedione treatment

James D. Johnson, B. Marsh, M. Hayden, 2007, Nature Medicine.

Vascular Endothelial Growth Factor B and Its Signaling

B. Rodrigues, Nathaniel Lal, Karanjit Puri, 2018, Front. Cardiovasc. Med..

Glucocorticoids produce whole body insulin resistance with changes in cardiac metabolism.

B. Rodrigues, D. Qi, 2007, American journal of physiology. Endocrinology and metabolism.

FoxO 1 is crucial for sustaining cardiomyocyte metabolism and cell survival

P. Puthanveetil, B. Rodrigues, Andrea Wan, 2013 .

Hyperglycemia-Induced Secretion of Endothelial Heparanase Stimulates a Vascular Endothelial Growth Factor Autocrine Network in Cardiomyocytes That Promotes Recruitment of Lipoprotein Lipase

James D. Johnson, Ying Wang, B. Rodrigues, 2013, Arteriosclerosis, thrombosis, and vascular biology.

Human and mouse muscle transcriptomic analyses identify insulin receptor mRNA downregulation in hyperinsulinemia‐associated insulin resistance

James D. Johnson, J. Timmons, B. Rodrigues, 2021, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

cytoskeleton reorganization cardiomyocyte lipoprotein lipase involves actin Lysophosphatidic acid-mediated augmentation of

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2016 .

Lysophosphatidic acid-mediated augmentation of cardiomyocyte lipoprotein lipase involves actin cytoskeleton reorganization.

Sanjoy Ghosh, T. Pulinilkunnil, A. Abrahani, 2005, American journal of physiology. Heart and circulatory physiology.

The diabetic heart: metabolic causes for the development of a cardiomyopathy.

B. Rodrigues, J. McNeill, 1992, Cardiovascular research.

Hypertrophied rat hearts are less responsive to the metabolic and functional effects of insulin.

B. Rodrigues, G. Bondy, C. Lydell, 2000, American journal of physiology. Endocrinology and metabolism.

Cardiac Dysfunction in the Diabetic Heart

B. Rodrigues, J. McNeill, R. Vadlamudi, 1987 .

Reducing insulin via conditional partial gene ablation in adults reverses diet-induced weight gain

James D. Johnson, C. Nislow, S. Flibotte, 2017, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Heparanase protects the heart against chemical or ischemia/reperfusion injury.

C. Nislow, S. Flibotte, T. Pulinilkunnil, 2019, Journal of molecular and cellular cardiology.

The function of heparanase in diabetes and its complications.

F. Wang, B. Rodrigues, Andrea Wan, 2013, Canadian journal of diabetes.

Comparison of cardiac function in male and female diabetic rats.

B. Rodrigues, J. McNeill, 1987, General pharmacology.

Hypertension and diabetic cardiomyopathy

B. Rodrigues, 1985 .

Increased efflux of glutathione conjugate in acutely diabetic cardiomyocytes.

Sanjoy Ghosh, T. Pulinilkunnil, B. Rodrigues, 2004, Canadian journal of physiology and pharmacology.

Heparanase Overexpression Induces Glucagon Resistance and Protects Animals From Chemically Induced Diabetes

James D. Johnson, C. Nislow, S. Flibotte, 2016, Diabetes.

Glucocorticoid excess induces accumulation of cardiac glycogen and triglyceride: suggested role for AMPK.

P. Puthanveetil, B. Rodrigues, 2013, Current pharmaceutical design.