J. Ham

发表

Trim17, a novel E3 ubiquitin-ligase, initiates neuronal apoptosis

B. Pettmann, I. Lassot, I. Robbins, 2010, Cell Death and Differentiation.

Genome-wide analysis of alternative splicing in medulloblastoma identifies splicing patterns characteristic of normal cerebellar development.

M. Barenco, F. Menghi, T. Jacques, 2011, Cancer research.

The bovine papillomavirus 1 E2 protein contains two activation domains: one that interacts with TBP and another that functions after TBP binding.

M. Yaniv, J. Ham, G. Steger, 1995, The EMBO journal.

Stress‐activated protein kinases are negatively regulated by cell density

M. Yaniv, S. Garbay, J. Ham, 1998, The EMBO journal.

Comprehensive molecular characterisation of epilepsy-associated glioneuronal tumours

M. Thom, W. Harkness, J. Cross, 2017, Acta Neuropathologica.

Role of the Jun Kinase Pathway in the Regulation of c-Jun Expression and Apoptosis in Sympathetic Neurons

L. Rubin, J. Ham, J. Whitfield, 1998, The Journal of Neuroscience.

Characterization of response elements for androgens, glucocorticoids and progestins in mouse mammary tumour virus.

P. Webb, J. Ham, M. Parker, 1988, Nucleic acids research.

Cloning of the CDC7 gene of Saccharomyces cerevisiae in association with centromeric DNA.

J. Ham, I. Johnston, D. A. Hughes, 1985, Gene.

The MEK-ERK pathway negatively regulates bim expression through the 3' UTR in sympathetic neurons

M. Kristiansen, J. Ham, J. Gilley, 2011, BMC Neuroscience.

Transcriptional analysis of the CDC7 protein kinase gene of Saccharomyces cerevisiae.

J. Ham, I. Johnston, J. Rosamond, 1989, Nucleic acids research.

Identification of androgen response elements in mouse mammary tumour virus and the rat prostate C3 gene

P. Webb, J. Ham, M. Parker, 1987, Journal of cellular biochemistry.

Evidence for positive and negative regulatory elements in the 5'-flanking sequence of the mouse sparc (osteonectin) gene.

B. Hogan, S. Nomura, J. Ham, 1989, The Journal of biological chemistry.

Evidence for increased complexity in the regulation of Bim expression in sympathetic neurons: involvement of novel transcriptional and translational mechanisms.

J. Ham, J. Gilley, 2005, DNA and cell biology.

E2 proteins: modulators of papillomavirus transcription and replication.

M. Yaniv, J. Ham, G. Steger, 1996, Methods in Enzymology.

NF-Y is essential for expression of the proapoptotic bim gene in sympathetic neurons

R. Mantovani, I. Lassot, M. Kristiansen, 2011, Cell Death and Differentiation.

Methods for culturing primary sympathetic neurons and for determining neuronal viability.

J. Ham, J. Whitfield, S. Neame, 2004, Methods in molecular biology.

c-Jun and the transcriptional control of neuronal apoptosis.

J. Ham, J. Whitfield, A. Eilers, 2000, Biochemical pharmacology.

Immunocytochemical techniques for studying apoptosis in primary sympathetic neurons.

J. Ham, J. Whitfield, S. Neame, 2004, Methods in molecular biology.

The functional BPV-1 E2 trans-activating protein can act as a repressor by preventing formation of the initiation complex.

P. Howley, M. Yaniv, N. Dostatni, 1991, Genes & development.

The progesterone receptor can regulate transcription in the absence of a functional TATA box element.

J. Ham, M. Parker, O. Bakker, 1990, The Journal of biological chemistry.

The papillomavirus E2 protein: a factor with many talents.

J. Gauthier, M. Yaniv, N. Dostatni, 1991, Trends in biochemical sciences.

Several different upstream promoter elements can potentiate transactivation by the BPV‐1 E2 protein.

M. Yaniv, N. Dostatni, J. Ham, 1991, The EMBO journal.

Dominant-Negative c-Jun Promotes Neuronal Survival by Reducing BIM Expression and Inhibiting Mitochondrial Cytochrome c Release

O. Bernard, J. Ham, J. Whitfield, 2001, Neuron.

Structural organization and expression of the mouse estrogen receptor.

J. Ham, J. Lees, M. Parker, 1987, Molecular endocrinology.

Phosphorylation of c-Jun Is Necessary for Apoptosis Induced by Survival Signal Withdrawal in Cerebellar Granule Neurons

L. Rubin, J. Kyriakis, J. Ham, 1998, The Journal of Neuroscience.

Direct inhibition of c‐Jun N‐terminal kinase in sympathetic neurones prevents c‐jun promoter activation and NGF withdrawal‐induced death

J. Ham, J. Whitfield, H. Desmond, 2001, Journal of neurochemistry.

FOXO transcription factors directly activate bim gene expression and promote apoptosis in sympathetic neurons

P. Coffer, J. Ham, J. Gilley, 2003, The Journal of cell biology.

A c-jun dominant negative mutant protects sympathetic neurons against programmed cell death

L. Rubin, M. Yaniv, J. Ham, 1995, Neuron.

Mkp1 Is a c-Jun Target Gene That Antagonizes JNK-Dependent Apoptosis in Sympathetic Neurons

T. Jacques, M. Kristiansen, J. Ham, 2010, The Journal of Neuroscience.

Bax promotes neuronal cell death and is downregulated during the development of the nervous system.

L. Rubin, J. Ham, J. Whitfield, 1997, Development.

Hrk/DP5 contributes to the apoptosis of select neuronal populations but is dispensable for haematopoietic cell apoptosis

E. Stanley, A. Strasser, C. Scott, 2007, Journal of Cell Science.

How do eukaryotic activator proteins stimulate the rate of transcription by RNA polymerase II?

M. Yaniv, J. Ham, G. Steger, 1992, FEBS letters.

The proapoptotic dp5 gene is a direct target of the MLK-JNK-c-Jun pathway in sympathetic neurons

M. Kristiansen, J. Ham, E. Towers, 2009, Nucleic acids research.

Programmed cell death during neuronal development: the sympathetic neuron model

J Ham, M. Kristiansen, J. Ham, 2014, Cell Death and Differentiation.

Regulation of gene expression by nuclear hormone receptors.

J. Ham, M. Parker, 1989, Current opinion in cell biology.

Global analysis of gene expression in NGF-deprived sympathetic neurons identifies molecular pathways associated with cell death

F. Menghi, M. Hubank, M. Kristiansen, 2011, BMC Genomics.

c-Jun and Bax: regulators of programmed cell death in developing neurons.

J. Ham, J. Whitfield, A. Eilers, 1999, Biochemical Society transactions.

Cooperativity in vivo between the E2 transactivator and the TATA box binding protein depends on core promoter structure.

M. Yaniv, J. Ham, G. Steger, 1994, The EMBO journal.

HRK (harakiri, BCL2 interacting protein (contains only BH3 domain))

J. Ham, 2012 .

Genome-wide analysis of alternative splicing in medulloblastoma identifies splicing patterns characteristic of normal cerebellar development.

M. Barenco, F. Menghi, T. Jacques, 2011, Cancer Research.