F. Das

发表

Raptor-rictor axis in TGFβ-induced protein synthesis

D. Riley, F. Das, N. Ghosh-Choudhury, 2008 .

Mesangial Cell Hypertrophy by High Glucose Is Mediated by Downregulation of the Tumor Suppressor PTEN

F. Das, L. Mahimainathan, B. Venkatesan, 2006, Diabetes.

TGFβ intercepts nuclear glycogen synthase kinase 3β to inhibit PDGF‐induced DNA synthesis in mesangial cells

H. Abboud, F. Das, N. Ghosh-Choudhury, 2007 .

Akt kinase targets association of CBP with SMAD 3 to regulate TGFβ‐induced expression of plasminogen activator inhibitor‐1

F. Das, N. Ghosh-Choudhury, L. Mahimainathan, 2008, Journal of cellular physiology.

TGFβ enforces activation of eukaryotic elongation factor‐2 (eEF2) via inactivation of eEF2 kinase by p90 ribosomal S6 kinase (p90Rsk) to induce mesangial cell hypertrophy

F. Das, N. Ghosh-Choudhury, B. Kasinath, 2010, FEBS letters.

Bone Morphogenetic Protein-2 (BMP-2) Activates NFATc1 Transcription Factor via an Autoregulatory Loop Involving Smad/Akt/Ca2+ Signaling*

S. Ganapathy, F. Das, N. Ghosh-Choudhury, 2015, The Journal of Biological Chemistry.

PRAS40 acts as a nodal regulator of high glucose‐induced TORC1 activation in glomerular mesangial cell hypertrophy

F. Das, B. Venkatesan, B. Kasinath, 2010, Journal of cellular physiology.

TGFβ‐induced PI 3 kinase‐dependent Mnk‐1 activation is necessary for Ser‐209 phosphorylation of eIF4E and mesangial cell hypertrophy

F. Das, B. Kasinath, G. G. Choudhury, 2013, Journal of cellular physiology.

Tyrosines-740/751 of PDGFRβ contribute to the activation of Akt/Hif1α/TGFβ nexus to drive high glucose-induced glomerular mesangial cell hypertrophy.

F. Das, B. Kasinath, G. G. Choudhury, 2018, Cellular signalling.

TSC2 Deficiency Increases PTEN via HIF1α*

H. Abboud, S. Habib, F. Das, 2009, The Journal of Biological Chemistry.

Oncoprotein DJ-1 interacts with mTOR complexes to effect transcription factor Hif1α-dependent expression of collagen I (α2) during renal fibrosis

F. Das, B. Kasinath, G. G. Choudhury, 2022, The Journal of biological chemistry.

Akt2 causes TGFβ-induced deptor downregulation facilitating mTOR to drive podocyte hypertrophy and matrix protein expression

F. Das, B. Kasinath, G. G. Choudhury, 2018, PloS one.

High Glucose Forces a Positive Feedback Loop Connecting Akt Kinase and FoxO1 Transcription Factor to Activate mTORC1 Kinase for Mesangial Cell Hypertrophy and Matrix Protein Expression*

F. Das, B. Kasinath, Meenalakshmi M. Mariappan, 2014, The Journal of Biological Chemistry.

High glucose upregulation of early-onset Parkinson's disease protein DJ-1 integrates the PRAS40/TORC1 axis to mesangial cell hypertrophy.

F. Das, B. Venkatesan, B. Kasinath, 2011, Cellular signalling.

Raptor-rictor axis in TGFbeta-induced protein synthesis.

D. Riley, F. Das, L. Mahimainathan, 2008, Cellular signalling.

TGFβ-Induced Deptor Suppression Recruits mTORC1 and Not mTORC2 to Enhance Collagen I (α2) Gene Expression

H. Abboud, F. Das, B. Kasinath, 2014, PloS one.

Identify the Underlying Mechanisms Driving the Pathogenesis of Diabetic Nephropathy

F. Das, dasf, 2016 .

β2-Adrenergic Receptor Agonist Induced Hepatic Steatosis in Mice: Modeling Nonalcoholic Fatty Liver Disease in Hyperadrenergic States.

Mengwei Zang, S. Weintraub, F. Das, 2021, American journal of physiology. Endocrinology and metabolism.

Transforming Growth Factor β Integrates Smad 3 to Mechanistic Target of Rapamycin Complexes to Arrest Deptor Abundance for Glomerular Mesangial Cell Hypertrophy*

H. Abboud, F. Das, B. Kasinath, 2013, The Journal of Biological Chemistry.

TGFβ acts through PDGFRβ to activate mTORC1 via the Akt/PRAS40 axis and causes glomerular mesangial cell hypertrophy and matrix protein expression

F. Das, B. Kasinath, G. Ghosh Choudhury, 2020, The Journal of Biological Chemistry.

MicroRNA-214 Reduces Insulin-like Growth Factor-1 (IGF-1) Receptor Expression and Downstream mTORC1 Signaling in Renal Carcinoma Cells*

F. Das, B. Kasinath, G. G. Choudhury, 2016, The Journal of Biological Chemistry.

microRNA-21-induced dissociation of PDCD4 from rictor contributes to Akt-IKKβ-mTORC1 axis to regulate renal cancer cell invasion.

H. Abboud, F. Das, B. Kasinath, 2014, Experimental cell research.

NFκB-mediated cyclin D1 expression by microRNA-21 influences renal cancer cell proliferation.

H. Abboud, F. Das, B. Kasinath, 2013, Cellular signalling.

MicroRNA-21 Orchestrates High Glucose-induced Signals to TOR Complex 1, Resulting in Renal Cell Pathology in Diabetes*

Falguni Das, Goutam Ghosh Choudhury, F. Das, 2011, The Journal of Biological Chemistry.

microRNA-21 Governs TORC1 Activation in Renal Cancer Cell Proliferation and Invasion

Falguni Das, Goutam Ghosh Choudhury, Hanna E. Abboud, 2012, PloS one.

Unrestrained Mammalian Target of Rapamycin Complexes 1 and 2 Increase Expression of Phosphatase and Tensin Homolog Deleted on Chromosome 10 to Regulate Phosphorylation of Akt Kinase*

H. Abboud, F. Das, L. Mahimainathan, 2011, The Journal of Biological Chemistry.

c-Abl-dependent Molecular Circuitry Involving Smad5 and Phosphatidylinositol 3-Kinase Regulates Bone Morphogenetic Protein-2-induced Osteogenesis*

S. Ganapathy, F. Das, C. Mandal, 2013, The Journal of Biological Chemistry.

Reciprocal regulation of miR-214 and PTEN by high glucose regulates renal glomerular mesangial and proximal tubular epithelial cell hypertrophy and matrix expansion.

F. Das, B. Kasinath, Meenalakshmi M. Mariappan, 2017, American journal of physiology. Cell physiology.

PDGF receptor-β uses Akt/mTORC1 signaling node to promote high glucose-induced renal proximal tubular cell collagen I (α2) expression.

F. Das, B. Venkatesan, B. Kasinath, 2017, American journal of physiology. Renal physiology.

c-Src couples PI 3 kinase/Akt and MAPK signaling to PDGF-induced DNA synthesis in mesangial cells.

F. Das, L. Mahimainathan, B. Venkatesan, 2006, Cellular signalling.

microRNA‐181a downregulates deptor for TGF&bgr;‐induced glomerular mesangial cell hypertrophy and matrix protein expression

F. Das, B. Kasinath, G. G. Choudhury, 2018, Experimental cell research.

Resveratrol inhibits PDGF receptor mitogenic signaling in mesangial cells: role of PTP1B

H. Abboud, F. Das, L. Mahimainathan, 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Deacetylation of S6 kinase promotes high glucose–induced glomerular mesangial cell hypertrophy and matrix protein accumulation

F. Das, B. Kasinath, G. Ghosh Choudhury, 2019, The Journal of Biological Chemistry.

Downregulation of catalase by reactive oxygen species via PI 3 kinase/Akt signaling in mesangial cells

F. Das, L. Mahimainathan, B. Venkatesan, 2007, Journal of cellular physiology.

High glucose enhances microRNA-26a to activate mTORC1 for mesangial cell hypertrophy and matrix protein expression.

F. Das, B. Kasinath, G. G. Choudhury, 2015, Cellular signalling.

A positive feedback loop involving Erk5 and Akt turns on mesangial cell proliferation in response to PDGF.

H. Abboud, F. Das, B. Kasinath, 2014, American journal of physiology. Cell physiology.

High glucose-stimulated enhancer of zeste homolog-2 (EZH2) forces suppression of deptor to cause glomerular mesangial cell pathology.

F. Das, B. Kasinath, G. G. Choudhury, 2021, Cellular Signalling.

TGFβ instructs mTORC2 to activate PKCβII for increased TWIST1 expression in proximal tubular epithelial cell injury

F. Das, B. Kasinath, G. Ghosh Choudhury, 2023, FEBS letters.

Hydrophobic motif site-phosphorylated protein kinase CβII between mTORC2 and Akt regulates high glucose-induced mesangial cell hypertrophy.

F. Das, B. Kasinath, G. G. Choudhury, 2016, American journal of physiology. Cell physiology.

microRNA-21 ORCHESTRATES HIGH GLUCOSE-INDUCED SIGNALS TO TORC 1 FOR RENAL CELL PATHOLOGY IN DIABETES

F. Das, B. Kasinath, G. G. Choudhury, 2011 .

Transforming Growth Factor β Integrates Smad 3 to Mechanistic Target of Rapamycin Complexes to Arrest Deptor Abundance for Glomerular Mesangial Cell Hypertrophy*

H. Abboud, F. Das, B. Kasinath, 2013, The Journal of Biological Chemistry.

High glucose-induced downregulation of PTEN-Long is sufficient for proximal tubular cell injury in diabetic kidney disease.

F. Das, B. Kasinath, G. G. Choudhury, 2024, Experimental cell research.