Y. Oka

发表

Integrin-linked kinase (ILK) regulation of the cell viability in PTEN mutant glioblastoma and in vitro inhibition by the specific COX-2 inhibitor NS-398.

J. Kuratsu, T. Asano, H. Katagiri, 2004, Cancer letters.

Hepatic Akt activation induces marked hypoglycemia, hepatomegaly, and hypertriglyceridemia with sterol regulatory element binding protein involvement.

T. Asano, H. Katagiri, Y. Oka, 2003, Diabetes.

Acyl-coenzyme A dehydrogenases are localized on GLUT4-containing vesicles via association with insulin-regulated aminopeptidase in a manner dependent on its dileucine motif.

T. Kodama, T. Asano, H. Katagiri, 2002, Molecular endocrinology.

Dexamethasone-induced insulin resistance in 3T3-L1 adipocytes is due to inhibition of glucose transport rather than insulin signal transduction.

T. Asano, M. Funaki, H. Katagiri, 2000, Diabetes.

Overexpression of Catalytic Subunit p110α of Phosphatidylinositol 3-Kinase Increases Glucose Transport Activity with Translocation of Glucose Transporters in 3T3-L1 Adipocytes*

Y. Yazaki, T. Asano, H. Katagiri, 1996, The Journal of Biological Chemistry.

Variants in KCNQ1 are associated with susceptibility to type 2 diabetes mellitus

L. Groop, Yusuke Nakamura, A. Sekine, 2008, Nature Genetics.

Impact of Plasma Oxidized Low-Density Lipoprotein Removal on Atherosclerosis

T. Sawamura, Yuko Sato, H. Katagiri, 2008, Circulation.

Chronic mild stress alters circadian expressions of molecular clock genes in the liver.

H. Katagiri, Y. Oka, Tetsuya Yamada, 2013, American journal of physiology. Endocrinology and metabolism.

Chronic mild stress alters circadian expressions

H. Katagiri, Y. Oka, Tetsuya Yamada, 2012 .

Carotid arterial elasticity is a sensitive atherosclerosis value reflecting visceral fat accumulation in obese subjects.

H. Hasegawa, H. Kanai, Y. Hinokio, 2009, Atherosclerosis.

A novel method for evaluating human carotid artery elasticity: possible detection of early stage atherosclerosis in subjects with type 2 diabetes.

Hiroshi Kanai, Hideki Katagiri, Hideyuki Hasegawa, 2008, Atherosclerosis.

metabolism in 3T3-L1 adipocytes Role of PDK1 in insulin-signaling pathway for glucose

T. Kodama, T. Asano, H. Katagiri, 2016 .

Role of PDK 1 in insulin-signaling pathway for glucose metabolism in 3 T 3L 1 adipocytes

T. Kodama, T. Asano, H. Katagiri, 2002 .

Role of PDK1 in insulin-signaling pathway for glucose metabolism in 3T3-L1 adipocytes.

T. Kodama, T. Asano, H. Katagiri, 2002, American journal of physiology. Endocrinology and metabolism.

Role of PKC isoforms in glucose transport in 3T3-L1 adipocytes: insignificance of atypical PKC.

T. Asano, H. Katagiri, Y. Oka, 2002, American journal of physiology. Endocrinology and metabolism.

The Pro12 -->Ala substitution in PPAR-gamma is associated with resistance to development of diabetes in the general population: possible involvement in impairment of insulin secretion in individuals with type 2 diabetes.

M. Kasuga, T. Sanke, K. Nanjo, 2001, Diabetes.

Identification and functional analysis of sulfonylurea receptor 1 variants in Japanese patients with NIDDM.

M. Permutt, J. Bryan, L. Aguilar-Bryan, 1998, Diabetes.

Direct stimulation of basal insulin secretion by physiological concentrations of leptin in pancreatic beta cells.

T. Asano, Y. Oka, H. Ishihara, 1997, Endocrinology.

Regulation of phosphoinositide metabolism, Akt phosphorylation, and glucose transport by PTEN (phosphatase and tensin homolog deleted on chromosome 10) in 3T3-L1 adipocytes.

T. Asano, M. Funaki, H. Katagiri, 2001, Molecular endocrinology.

Bone marrow (BM) transplantation promotes beta-cell regeneration after acute injury through BM cell mobilization.

H. Nakauchi, H. Sasano, H. Katagiri, 2007, Endocrinology.

Genetic variations at urotensin II and urotensin II receptor genes and risk of type 2 diabetes mellitus in Japanese

Y. Hinokio, Y. Oka, A. Matsutani, 2004, Peptides.

Role of urotensin II gene in genetic susceptibility to Type 2 diabetes mellitus in Japanese subjects

Y. Hinokio, J. Satoh, Y. Oka, 2003, Diabetologia.

Domains responsible for the differential targeting of glucose transporter isoforms.

Y. Yazaki, T. Asano, H. Katagiri, 1992, The Journal of biological chemistry.

Pattern recognition analysis for 1H NMR spectra of plasma from hemodialysis patients

Y. Oka, H. Sekino, Takeshi Kobayashi, 2009, Analytical and bioanalytical chemistry.

A Novel Isoform of Syntaxin-binding Protein Homologous to Yeast Sec1 Expressed Ubiquitously in Mammalian Cells (*)

J. Miyazaki, Y. Yazaki, J. Terasaki, 1995, The Journal of Biological Chemistry.

LST8 level controls basal p70 S6 kinase and Akt phosphorylations, and mTORC1 and mTORC2 negatively regulate each other by competing for association with LST8.

K. Inoki, T. Asano, H. Kurihara, 2012, Obesity research & clinical practice.

Type I Phosphatidylinositol-4-phosphate 5-Kinases

Y. Yazaki, T. Asano, Y. Oka, 1998, The Journal of Biological Chemistry.

Mitochondrial diabetes mellitus: Prevalence and clinical characterization of diabetes due to mitochondrial tRNALeU(UUR) gene mutation in Japanese patients

Y. Yazaki, H. Katagiri, Y. Oka, 1994, Diabetologia.

Auditory Findings in Patients with Maternally Inherited Diabetes and Deafness Harboring a Point Mutation in the Mitochondrial Transfer RNALeu (UUR) Gene

K. Kaga, Y. Oka, T. Yamasoba, 1996, The Laryngoscope.

Mitochondrial diabetes mellitus—glucose‐induced signaling defects and β‐cell loss

T. Kobayashi, T. Asano, H. Katagiri, 1995, Muscle & nerve. Supplement.

Endoplasmic reticulum stress and N-glycosylation modulate expression of WFS1 protein.

H. Katagiri, Y. Oka, H. Ishihara, 2004, Biochemical and biophysical research communications.

The role of N‐glycosylation in the targeting and stability of GLUT1 glucose transporter

Y. Yazaki, T. Asano, H. Katagiri, 1993, FEBS letters.

Mitochondrial gene mutation in islet-cell-antibody-positive patients who were initially non-insulin-dependent diabetics

Y. Yazaki, T. Kobayashi, H. Katagiri, 1993, The Lancet.

Endoplasmic reticulum stress induces Wfs1 gene expression in pancreatic beta-cells via transcriptional activation.

Y. Oka, M. Akiyama, K. Shinoda, 2005, European journal of endocrinology.

Distribution of two types of lymphocytes (intraepithelial and lamina-propria-associated) in the murine small intestine

Y. Oka, T. Toyota, T. Itoh, 2003, Cell and Tissue Research.

Neuronal pathway from the liver modulates energy expenditure and systemic insulin sensitivity

H. Katagiri, Y. Oka, Tetsuya Yamada, 2006, Autonomic Neuroscience.

Three mitogen-activated protein kinases inhibit insulin signaling by different mechanisms in 3T3-L1 adipocytes.

Y. Gotoh, T. Asano, H. Katagiri, 2003, Molecular endocrinology.

Long-Term Clinical Effects of Epalrestat, an Aldose Reductase Inhibitor, on Diabetic Peripheral Neuropathy

R. Kawamori, Y. Oka, Y. Akanuma, 2006, Diabetes Care.

Elevation of HDL-C in response to statin treatment is involved in the regression of carotid atherosclerosis.

S. Kono, H. Katagiri, Y. Oka, 2014, Journal of atherosclerosis and thrombosis.

International clinical harmonization of glycated hemoglobin in Japan: From Japan Diabetes Society to National Glycohemoglobin Standardization Program values

T. Sanke, M. Noda, T. Hanafusa, 2012, Diabetology International.

Maternal epidermal growth factor deficiency causes fetal hypoglycemia and intrauterine growth retardation in mice: possible involvement of placental glucose transporter GLUT3 expression.

Y. Taketani, Y. Oka, O. Tsutsumi, 1999, Endocrinology.

Maternal Epidermal Growth Factor Deficiency Causes Fetal Hypoglycemia and Intrauterine Growth Retardation in Mice: Possible Involvement of Placental Glucose Transporter GLUT3 Expression1.

Y. Taketani, Y. Oka, O. Tsutsumi, 1999, Endocrinology.

Cochlear histopathology associated with mitochondrial transfer RNALeu(UUR) gene mutation

T. Kobayashi, Y. Oka, T. Yamasoba, 1999, Neurology.

MKK6/3 and p38 MAPK Pathway Activation Is Not Necessary for Insulin-induced Glucose Uptake but Regulates Glucose Transporter Expression*

Y. Gotoh, T. Asano, M. Funaki, 2001, The Journal of Biological Chemistry.

4F2hc stabilizes GLUT1 protein and increases glucose transport activity.

T. Asano, H. Kurihara, H. Katagiri, 2011, American journal of physiology. Cell physiology.

WFS1 (Wolfram syndrome 1) gene product: predominant subcellular localization to endoplasmic reticulum in cultured cells and neuronal expression in rat brain.

Yoshifumi Watanabe, Y. Oka, Y. Watanabe, 2001, Human molecular genetics.

Pin1 Associates with and Induces Translocation of CRTC2 to the Cytosol, Thereby Suppressing cAMP-responsive Element Transcriptional Activity*

T. Uchida, N. Horike, T. Asano, 2010, The Journal of Biological Chemistry.

MEK Kinase 1 Interacts with Focal Adhesion Kinase and Regulates Insulin Receptor Substrate-1 Expression*

Y. Oka, T. Kitamura, Toru Takahashi, 2003, The Journal of Biological Chemistry.

Molecular mimicry by Helicobacter pylori CagA protein may be involved in the pathogenesis of H. pylori‐associated chronic idiopathic thrombocytopenic purpura

Y. Oka, K. Shinohara, M. Shirai, 2004, British journal of haematology.

β‐Cell Loss and Glucose Induced Signalling Defects in Diabetes Mellitus Caused by Mitochondrial tRNALeu(UUR) Gene Mutation

T. Kobayashi, T. Asano, H. Katagiri, 1996, Diabetic medicine : a journal of the British Diabetic Association.

Cold Exposure Suppresses Serum Adiponectin Levels through Sympathetic Nerve Activation in Mice

H. Sasano, H. Katagiri, Y. Oka, 2006, Obesity.

ATF4-mediated induction of 4E-BP1 contributes to pancreatic beta cell survival under endoplasmic reticulum stress.

H. Aburatani, J. Miyazaki, N. Sonenberg, 2008, Cell metabolism.

Metformin decreases blood pressure and obesity in OLETF rats via improvement of insulin resistance.

Y. Oka, K. Inukai, S. Katayama, 1996, Hypertension research : official journal of the Japanese Society of Hypertension.

Glucose intolerance in spontaneously hypertensive and Wistar-Kyoto rats: enhanced gene expression and synthesis of skeletal muscle glucose transporter 4.

Y. Oka, T. Awata, S. Katayama, 1997, Hypertension research : official journal of the Japanese Society of Hypertension.

Glucose transporter levels in a male spontaneous non-insulin-dependent diabetes mellitus rat of the Otsuka Long-Evans Tokushima Fatty strain.

Y. Oka, K. Shima, Y. Noma, 1997, Diabetes research and clinical practice.

Studies with antipeptide antibody suggest the presence of at least two types of glucose transporter in rat brain and adipocyte.

M. Kasuga, T. Asano, Y. Oka, 1988, The Journal of biological chemistry.

Different Subcellular Distribution and Regulation of Expression of Insulin Receptor Substrate (IRS)-3 from Those of IRS-1 and IRS-2*

Y. Yazaki, T. Asano, M. Funaki, 1998, The Journal of Biological Chemistry.

Peginterferon (PEG-IFN) plus ribavirin combination therapy, but neither interferon nor PGE-IFN alone, induced type 1 diabetes in a patient with chronic hepatitis C.

T. Shimosegawa, Y. Hinokio, H. Katagiri, 2009, Internal medicine.

Co-localisation of insulin-like growth factor II and the proliferation marker MIB1 in hepatocellular carcinoma cells.

T. Sohda, Y. Oka, M. Okumura, 1997, Journal of clinical pathology.

Status of Research Funded by the American Diabetes Association — Year 4 I n 1998

J. Olefsky, Y. Oka, T. Muramatsu, 2003 .

Dissipating excess energy stored in the liver is a potential treatment strategy for diabetes associated with obesity.

T. Asano, T. Shimosegawa, H. Katagiri, 2005, Diabetes.

ANGPTL3 is increased in both insulin-deficient and -resistant diabetic states.

H. Katagiri, Y. Oka, K. Inukai, 2004, Biochemical and biophysical research communications.

Glucose-Regulated Translational Control of Proinsulin Biosynthesis With That of the Proinsulin Endopeptidases PG2 and PC3 in the Insulin-Producing MIN6 Cell Line

Y. Oka, H. Ishihara, C. Rhodes, 1996, Diabetes.

Characteristics of the antibodies of two patients who developed daytime hyperglycemia and morning hypoglycemia because of insulin antibodies.

S. Ito, Takefumi Mori, Y. Oka, 2009, Diabetes research and clinical practice.

NIDDM--genetic marker; glucose transporter, glucokinase, and mitochondria gene.

Y. Oka, 1994, Diabetes research and clinical practice.

Prevalence of the Trp64Arg missense mutation of the beta3-adrenergic receptor gene in Japanese subjects.

T. Asano, Y. Oka, H. Inoue, 1997, Metabolism: clinical and experimental.

Ethanol feeding induces insulin resistance with enhanced PI 3-kinase activation.

T. Asano, H. Katagiri, Y. Oka, 2003, Biochemical and biophysical research communications.

Bach 1 deficiency protects pancreatic β-cells from oxidative stress 1 injury 2 3 Running title : Protective effects of Bach 1 deficiency in pancreatic 4 β-cells

H. Katagiri, Y. Oka, Tetsuya Yamada, 2013 .

Pancreatic beta cell line MIN6 exhibits characteristics of glucose metabolism and glucose-stimulated insulin secretion similar to those of normal islets

J. Miyazaki, Y. Yazaki, T. Asano, 1993, Diabetologia.

Acyl-coenzyme A dehydrogenases are localized on GLUT4-containing vesicles via association with insulin-regulated aminopeptidase in a manner dependent on its dileucine motif.

T. Kodama, T. Asano, H. Katagiri, 2002, Molecular endocrinology.

Enhanced insulin-stimulated activation of phosphatidylinositol 3-kinase in the liver of high-fat-fed rats.

Y. Yazaki, T. Asano, M. Funaki, 1999, Diabetes.

Cloning of a rabbit brain glucose transporter cDNA and alteration of glucose transporter mRNA during tissue development.

M. Kasuga, T. Asano, Y. Oka, 1988, Biochemical and biophysical research communications.

Rabbit brain glucose transporter responds to insulin when expressed in insulin-sensitive Chinese hamster ovary cells.

M. Kasuga, T. Asano, Y. Oka, 1989, The Journal of biological chemistry.

glucose uptake by skeletal muscle but not adipocytes Activation of AMPK is essential for AICAR-induced

T. Asano, H. Katagiri, Y. Oka, 2016 .

Activation of AMPK is essential for AICAR-induced glucose uptake by skeletal muscle but not adipocytes.

T. Asano, H. Katagiri, Y. Oka, 2002, American journal of physiology. Endocrinology and metabolism.

Regulation of insulin-stimulated glucose transport by chronic glucose exposure in 3T3-L1 adipocytes.

Y. Oka, T. Hosaka, K. Yaga, 1999, Endocrine journal.

Plasma concentrations of vascular endothelial growth factor are associated with peripheral oedema in patients treated with thiazolidinedione

Y. Oka, M. Emoto, A. Taguchi, 2006, Diabetologia.

Association between HLA and islet cell antibodies in diabetic patients with a mitochondrial DNA mutation at base pair 3243

Å. Lernmark, T. Kobayashi, H. Katagiri, 1996, Diabetologia.

Glucokinase-defective NIDDM

Y. Yazaki, T. Asano, H. Katagiri, 1993, The Lancet.

[Hypoglycemia associated with hyperinsulinemia in a subject with type 2 diabetes and liver cirrhosis].

H. Katagiri, Y. Oka, H. Ishihara, 2006, Nihon Naika Gakkai zasshi. The Journal of the Japanese Society of Internal Medicine.

Imidapril, an angiotensin-converting enzyme inhibitor, improves insulin sensitivity by enhancing signal transduction via insulin receptor substrate proteins and improving vascular resistance in the Zucker fatty rat.

Y. Yazaki, T. Asano, M. Funaki, 1999, Metabolism: clinical and experimental.

Photoaffinity labeling of insulin-sensitive hexose transporters in intact rat adipocytes. Direct evidence that latent transporters become exposed to the extracellular space in response to insulin.

M. Czech, Y. Oka, 1984, The Journal of biological chemistry.

Secondary sulfonylurea failure: comparison of period until insulin treatment between diabetic patients treated with gliclazide and glibenclamide.

I. Tsuji, Y. Hinokio, J. Satoh, 2005, Diabetes research and clinical practice.

Association Studies of Variants in the Genes Involved in Pancreatic β-Cell Function in Type 2 Diabetes in Japanese Subjects

M. Kanamori, M. Kasuga, T. Sanke, 2006, Diabetes.

Cell type-specific activation of metabolism reveals that β-cell secretion suppresses glucagon release from α-cells in rat pancreatic islets

H. Katagiri, Y. Oka, H. Endou, 2006 .

A Decreased Response of Cyclic Adenosine Monophosphate Concentrations to Glucagon in Liver Slices from Streptozotocin-induced Diabetic Rats

E. Ogata, Y. Oka, K. Yamashita, 1980, Diabetes.

Human plasma epidermal growth factor/beta-urogastrone is associated with blood platelets.

Y. Oka, D. Orth, Y. Oka, 1983, The Journal of clinical investigation.

An insulin-like growth factor II-producing histiocytoma associated with hypoglycemia: analysis of the peptide, its gene expression, and glucose transporter isoforms.

T. Asano, Y. Oka, M. Yamamoto, 1992, Metabolism: clinical and experimental.

The nucleotide sequence of cDNA for a mouse liver-type glucose transporter protein.

T. Asano, Y. Oka, Y. Akanuma, 1989, Nucleic acids research.

Interleukin-6 enhances glucose-stimulated insulin secretion from pancreatic beta-cells: potential involvement of the PLC-IP3-dependent pathway.

H. Katagiri, Y. Oka, Tetsuya Yamada, 2011, Diabetes.

Interleukin-6 Enhances Glucose-Stimulated Insulin Secretion From Pancreatic β-Cells

H. Katagiri, Y. Oka, Tetsuya Yamada, 2011, Diabetes.

Inhibitory effect of hyperglycemia on insulin-induced Akt/protein kinase B activation in skeletal muscle.

T. Asano, M. Funaki, Y. Oka, 2001, American journal of physiology. Endocrinology and metabolism.

Replacement of intracellular C-terminal domain of GLUT1 glucose transporter with that of GLUT2 increases Vmax and Km of transport activity.

Y. Yazaki, T. Asano, H. Katagiri, 1992, The Journal of biological chemistry.

Roles of PI 3-kinase and Ras on insulin-stimulated glucose transport in 3T3-L1 adipocytes.

Y. Yazaki, T. Asano, J. Terasaki, 1997, The American journal of physiology.

p85/p110-type Phosphatidylinositol Kinase Phosphorylates Not Only the D-3, but Also the D-4 Position of the Inositol Ring*

Y. Yazaki, T. Asano, M. Funaki, 1999, The Journal of Biological Chemistry.

Expression of the GLUT 1 Glucose Transporter Increases Thymidine Uptake in Chinese Hamster Ovary Cells at Low Glucose Concentrations 1

Y. Yazaki, T. Asano, H. Katagiri, 2006 .

Peptide-Based Radioimmunoassay Specific for GLUT1 Glucose Transporter

T. Asano, H. Katagiri, Y. Oka, 1991, Diabetes.

Expression of the GLUT1 glucose transporter increases thymidine uptake in Chinese hamster ovary cells at low glucose concentrations.

Y. Yazaki, T. Asano, H. Katagiri, 1991, Cancer research.

Hyperglycemia impairs the insulin signaling step between PI 3-kinase and Akt/PKB activations in ZDF rat liver.

T. Asano, M. Funaki, Y. Oka, 1999, Biochemical and biophysical research communications.

Cloning and increased expression with fructose feeding of rat jejunal GLUT5.

Y. Yazaki, T. Asano, H. Katagiri, 1993, Endocrinology.

Deletion of C-terminal 12 amino acids of GLUT1 protein does not abolish the transport activity.

Y. Yazaki, T. Asano, H. Katagiri, 1992, Biochemical and biophysical research communications.

Glucose binding enhances the papain susceptibility of the intracellular loop of the GLUT1 glucose transporter

Y. Yazaki, T. Asano, H. Katagiri, 1992, FEBS letters.

Importance of endothelial NF-κB signalling in vascular remodelling and aortic aneurysm formation.

T. Asano, H. Katagiri, Y. Oka, 2013, Cardiovascular research.

Insulin Action on Membrane Components: The Glucose Transporter and the Type II Insulinlike Growth Factor Receptor

M. Czech, Y. Oka, Yoshitomo Oka, 1985 .

A Novel 55-kDa Regulatory Subunit for Phosphatidylinositol 3-Kinase Structurally Similar to p55PIK Is Generated by Alternative Splicing of the p85 Gene (*)

Y. Yazaki, T. Asano, M. Funaki, 1996, The Journal of Biological Chemistry.

p85alpha gene generates three isoforms of regulatory subunit for phosphatidylinositol 3-kinase (PI 3-Kinase), p50alpha, p55alpha, and p85alpha, with different PI 3-kinase activity elevating responses to insulin.

Y. Yazaki, T. Asano, M. Funaki, 1997, The Journal of biological chemistry.

DNA fragmentation and detachment of enterocytes induced by anti-CD3 mAb-activated intraepithelial lymphocytes

J. Satoh, Y. Oka, T. Toyota, 2003, Cell and Tissue Research.

Desensitization of canine histamine H2 receptor expressed in Chinese hamster ovary cells.

T. Kodama, Y. Yazaki, T. Asano, 1993, Biochemical and biophysical research communications.

Interaction between the two signal transduction systems of the histamine H2 receptor: desensitizing and sensitizing effects of histamine stimulation on histamine-dependent cAMP production in Chinese hamster ovary cells.

Y. Yazaki, T. Asano, M. Funaki, 1996, The Biochemical journal.

Cognitive decline in a patient with anti-glutamic acid decarboxylase autoimmunity; case report

H. Yamasaki, E. Mori, Y. Oka, 2011, BMC neurology.

Unique phosphorylation mechanism of Gab1 using PI 3-kinase as an adaptor protein.

M. Shibuya, T. Asano, M. Funaki, 2001, Biochemical and biophysical research communications.

Five isoforms of the phosphatidylinositol 3‐kinase regulatory subunit exhibit different associations with receptor tyrosine kinases and their tyrosine phosphorylations

T. Asano, M. Funaki, H. Katagiri, 2001, FEBS letters.

Direct Stimulation of Basal Insulin Secretion by Physiological Concentrations of Leptin in Pancreatic β Cells.

T. Asano, Y. Oka, H. Ishihara, 1997, Endocrinology.

The JNK pathway modulates expression and phosphorylation of 4E‐BP1 in MIN6 pancreatic β‐cells under oxidative stress conditions

H. Katagiri, Y. Oka, H. Ishihara, 2010, Cell biochemistry and function.

[Neuropathy is associated with depression independently of health-related quality of life in Japanese patients with diabetes].

S. Yoshida, S. Awata, Y. Oka, 2010, Seishin shinkeigaku zasshi = Psychiatria et neurologia Japonica.

Human GLUT-2 overexpression does not affect glucose-stimulated insulin secretion in MIN6 cells.

J. Miyazaki, Y. Yazaki, T. Asano, 1995, The American journal of physiology.

Overexpression of mitochondrial FAD-linked glycerol-3-phosphate dehydrogenase does not correct glucose-stimulated insulin secretion from diabetic GK rat pancreatic islets

Y. Oka, H. Ishihara, A. Matsutani, 1998, Diabetologia.

Overexpression of hexokinase I but not GLUT1 glucose transporter alters concentration dependence of glucose-stimulated insulin secretion in pancreatic beta-cell line MIN6.

J. Miyazaki, Y. Yazaki, T. Asano, 1994, The Journal of biological chemistry.

Constitutively active PDX1 induced efficient insulin production in adult murine liver.

H. Sasano, T. Asano, H. Katagiri, 2005, Biochemical and biophysical research communications.

Neural relay from the liver induces proliferation of pancreatic beta cells: a path to regenerative medicine using the self-renewal capabilities.

H. Katagiri, Y. Oka, Junta Imai, 2009, Communicative & integrative biology.

Co‐localization of glucokinase with actin filaments

T. Asano, H. Katagiri, Y. Oka, 1997, FEBS letters.

Repetitive mitochondrial Ca2+ signals synchronize with cytosolic Ca2+ oscillations in the pancreatic beta-cell line, MIN6

J. Miyazaki, T. Asano, Y. Oka, 1998, Diabetologia.

Overexpression of dominant negative mutant hepatocyte nuclear factor (HNF)-1α inhibits arginine-induced insulin secretion in MIN6 cells

Y. Oka, A. Matsutani, H. Inoue, 1999, Diabetologia.

Insulinotropin PACAP potentiates insulin-stimulated glucose uptake in 3T3 L1 cells☆

S. Shioda, M. Nakata, Y. Oka, 1999, Peptides.

Identification of Glypican3 as a novel GLUT4-binding protein.

H. Aburatani, Y. Oka, M. Emoto, 2008, Biochemical and biophysical research communications.

Identification of a novel mechanism regulating β-cell mass: Neuronal relay from the liver to pancreatic β-cells

H. Katagiri, Y. Oka, Junta Imai, 2009, Islets.

Overexpression of glucose transporter modulates insulin biosynthesis in insulin producing cell line

T. Asano, Y. Oka, Y. Akanuma, 1990, FEBS letters.

Promoter polymorphism of SLC11A1 (formerly NRAMP1) confers susceptibility to autoimmune type 1 diabetes mellitus in Japanese.

T. Shimosegawa, Y. Hinokio, J. Satoh, 2004, Tissue antigens.

Efficient and controlled gene expression in mouse pancreatic islets by arterial delivery of tetracycline-inducible adenoviral vectors.

H. Katagiri, Y. Oka, H. Ishihara, 2007, Journal of molecular endocrinology.

Two glucose transporter isoforms are sorted differentially and are expressed in distinct cellular compartments.

Y. Yazaki, T. Asano, H. Katagiri, 1992, The Biochemical journal.

The glucose transport activity of GLUT1 is markedly decreased by substitution of a single amino acid with a different charge at residue 415.

Y. Yazaki, T. Asano, H. Katagiri, 1991, Biochemical and biophysical research communications.

Substitution of leucine for tryptophan 412 does not abolish cytochalasin B labeling but markedly decreases the intrinsic activity of GLUT1 glucose transporter.

T. Asano, H. Katagiri, Y. Oka, 1991, The Journal of biological chemistry.

C-terminal truncated glucose transporter is locked into an inward-facing form without transport activity

T. Asano, H. Katagiri, Y. Oka, 1990, Nature.

Inhibition of pancreatic β‐cell glucokinase by antisense RNA expression in transgenic mice: mouse strain‐dependent alteration of glucose tolerance

J. Miyazaki, Y. Yazaki, F. Tashiro, 1995, FEBS letters.

DPP-4 Inhibition Ameliorates Pancreatic β-Cell Failure and Improves Glucose Tolerance in the Mouse Model of Wolfram Syndrome

H. Katagiri, Y. Oka, H. Ishihara, 2015 .

Detection of variants in the mitochondrial glycerophosphate dehydrogenase gene in Japanese NIDDM patients

Y. Oka, A. Matsutani, Y. Takeuchi, 1997, Diabetologia.

Wolfram syndrome 1 gene negatively regulates ER stress signaling in rodent and human cells.

M. Permutt, Kathryn L. Lipson, F. Urano, 2010, The Journal of clinical investigation.

A Novel Protein Kinase B (PKB)/AKT-binding Protein Enhances PKB Kinase Activity and Regulates DNA Synthesis*

Y. Uchijima, N. Horike, T. Asano, 2005, Journal of Biological Chemistry.

Behavioral and gene expression analyses of Wfs1 knockout mice as a possible animal model of mood disorder

Y. Oka, H. Ishihara, K. Iwamoto, 2008, Neuroscience Research.

The Insulinlike Growth Factor Receptors

M. Czech, Y. Oka, C. Mottola, 1986 .

WFS1-deficiency increases endoplasmic reticulum stress, impairs cell cycle progression and triggers the apoptotic pathway specifically in pancreatic beta-cells.

H. Aburatani, J. Miyazaki, F. Tashiro, 2006, Human molecular genetics.

WFS 1 ( Wolfram syndrome 1 ) gene product : predominant subcellular localization to endoplasmic reticulum in cultured cells and neuronal expression in rat brain

Yoshifumi Watanabe, Y. Oka, H. Inoue, 2001 .

Unregulated elevation of glutamate dehydrogenase activity induces glutamine-stimulated insulin secretion: identification and characterization of a GLUD1 gene mutation and insulin secretion studies with MIN6 cells overexpressing the mutant glutamate dehydrogenase.

Y. Oka, H. Inoue, Y. Tanizawa, 2002, Diabetes.

beta-Adrenergic regulation of insulin and epidermal growth factor receptors in rat adipocytes.

M. Czech, Y. Oka, J. Pessin, 1983, The Journal of biological chemistry.

GAD antibody in mitochondrial diabetes associated with tRNA(UUR) mutation at position 3271.

Y. Oka, M. Taniyama, A. Shimada, 2002, Diabetes care.

Clinical relevance of heteroplasmic concentration of mitochondrial A 3243 G mutation in leucocytes

T. Asano, M. Funaki, H. Katagiri, 1999, Diabetologia.

Hepatic glucokinase modulates obesity predisposition by regulating BAT thermogenesis via neural signals.

H. Katagiri, Y. Oka, Tetsuya Yamada, 2012, Cell metabolism.

Cell Metabolism Liver GK Regulates BAT Thermogenesis

H. Katagiri, Y. Oka, Tetsuya Yamada, 2012 .

Association of TCF7L2 polymorphisms with susceptibility to type 2 diabetes in 4,087 Japanese subjects

M. Kasuga, K. Nanjo, K. Tokunaga, 2008, Journal of Human Genetics.

Pituitary adenylate cyclase‐activating polypeptide (PACAP) is an islet substance serving as an intra‐islet amplifier Of glucose‐induced insulin secretion in rats

S. Shioda, M. Nakata, Y. Oka, 1997, The Journal of physiology.

Induction of hypothalamic serum- and glucocorticoid-induced protein kinase-1 gene expression and its relation to plasma des-acyl ghrelin in energy homeostasis in mice.

Y. Oka, K. Nonogaki, Kana Ohashi-Nozue, 2006, Biochemical and biophysical research communications.

Hepatic peroxisome proliferator-activated receptor-γ-fat-specific protein 27 pathway contributes to obesity-related hypertension via afferent vagal signals.

H. Katagiri, Y. Oka, Tetsuya Yamada, 2012, European heart journal.

Increased hepatic expression of ganglioside-specific sialidase, NEU3, improves insulin sensitivity and glucose tolerance in mice.

H. Aburatani, K. Yamaguchi, Y. Hinokio, 2007, Metabolism: clinical and experimental.

Expression of glucose transporter isoforms with aging.

Y. Yazaki, T. Asano, H. Katagiri, 1992, Gerontology.

The WFS1 gene, responsible for low frequency sensorineural hearing loss and Wolfram syndrome, is expressed in a variety of inner ear cells

Y. Oka, J. Timmermans, K. Cryns, 2003, Histochemistry and Cell Biology.

MEK kinase 1 mediates the antiapoptotic effect of the Bcr-Abl oncogene through NF-κB activation

Y. Oka, Toru Takahashi, Y. Tanizawa, 2003, Oncogene.

Disruption of the WFS1 gene in mice causes progressive beta-cell loss and impaired stimulus-secretion coupling in insulin secretion.

H. Katagiri, Y. Oka, H. Ishihara, 2004, Human molecular genetics.

Neural relay from the liver induces proliferation of pancreatic β cells

H. Katagiri, Y. Oka, Junta Imai, 2009 .

Symposium on gene abnormalities in medical diseases. 2. Genetic defects and diabetes mellitus-defects of glucokinase gene and mitochondrial gene in diabetic patients.

Y. Oka, 1997, Internal medicine.

Isolation of full-length cDNA of mouse PAX4 gene and identification of its human homologue.

K. Nakai, H. Inoue, Y. Oka, 1998, Biochemical and biophysical research communications.

Direct demonstration of rapid insulin-like growth factor II Receptor internalization and recycling in rat adipocytes. Insulin stimulates 125I-insulin-like growth factor II degradation by modulating the IGF-II receptor recycling process.

M. Czech, Y. Oka, L. Rozek, 1985, The Journal of biological chemistry.

Mechanisms That Regulate Membrane Growth Factor Receptors

M. Czech, Y. Oka, J. Pessin, 1985 .

Resistin-like Molecule β Activates MAPKs, Suppresses Insulin Signaling in Hepatocytes, and Induces Diabetes, Hyperlipidemia, and Fatty Liver in Transgenic Mice on a High Fat Diet*

Y. Uchijima, N. Horike, Koichi Nishiyama, 2005, Journal of Biological Chemistry.

Serotonin systems upregulate the expression of hypothalamic NUCB2 via 5-HT2C receptors and induce anorexia via a leptin-independent pathway in mice.

Y. Oka, K. Nonogaki, Yukie Ohba, 2008, Biochemical and biophysical research communications.

Overexpression of constitutively activated glutamate dehydrogenase induces insulin secretion through enhanced glutamate oxidation.

H. Katagiri, Y. Oka, H. Mizuguchi, 2004, American journal of physiology. Endocrinology and metabolism.

Establishment of a pancreatic beta cell line that retains glucose-inducible insulin secretion: special reference to expression of glucose transporter isoforms.

J. Miyazaki, K. Yamamura, T. Asano, 1990, Endocrinology.

Wolfram syndrome 1 gene (WFS1) product localizes to secretory granules and determines granule acidification in pancreatic beta-cells.

Y. Oka, M. Hatanaka, M. Akiyama, 2011, Human molecular genetics.

of Rat GLUT5 and Functional Analysis of Chime& Proteins of GLUT1 Glucose Transporter and - GLUT5 Fructose Transporter

Y. Yazaki, T. Asano, H. Katagiri, 1995 .

WFS1 protein modulates the free Ca2+ concentration in the endoplasmic reticulum

H. Katagiri, Y. Oka, H. Ishihara, 2006, FEBS letters.

Olfactory receptors are expressed in pancreatic β-cells and promote glucose-stimulated insulin secretion

J. Miyazaki, M. Kanzaki, H. Katagiri, 2018, Scientific Reports.

Leptin induces proliferation of pancreatic beta cell line MIN6 through activation of mitogen-activated protein kinase.

Y. Oka, A. Matsutani, Y. Tanizawa, 1997, Biochemical and biophysical research communications.

Regulation of PPARγ transcriptional activity in 3T3-L1 adipocytes

H. Katagiri, Y. Oka, K. Inukai, 2003 .

Role of JTT-501, a new insulin sensitiser, in restoring impaired GLUT4 translocation in adipocytes of rats fed a high fat diet

Y. Yazaki, T. Asano, J. Terasaki, 1998, Diabetologia.

A human pancreatic islet inwardly rectifying potassium channel: cDNA cloning, determination of the genomic structure and genetic variations in Japanese NIDDM patients

M. Permutt, A. Kuwano, H. Katagiri, 1996, Diabetologia.

p85α Gene Generates Three Isoforms of Regulatory Subunit for Phosphatidylinositol 3-Kinase (PI 3-Kinase), p50α, p55α, and p85α, with Different PI 3-Kinase Activity Elevating Responses to Insulin*

Y. Yazaki, T. Asano, M. Funaki, 1997, The Journal of Biological Chemistry.

DOC2B: A Novel Syntaxin-4 Binding Protein Mediating Insulin-Regulated GLUT4 Vesicle Fusion in Adipocytes

Y. Oka, M. Emoto, A. Taguchi, 2009, Diabetes.

Simultaneous Copy Number Losses within Multiple Subtelomeric Regions in Early-Onset Type2 Diabetes Mellitus

H. Katagiri, Y. Oka, Tetsuya Yamada, 2014, PloS one.

Frequent Loss of Genome Gap Region in 4p16.3 Subtelomere in Early-Onset Type 2 Diabetes Mellitus

M. Emi, Y. Hinokio, H. Katagiri, 2011, Experimental diabetes research.

WFS1 Is a Novel Component of the Unfolded Protein Response and Maintains Homeostasis of the Endoplasmic Reticulum in Pancreatic β-Cells*

Kathryn L. Lipson, F. Urano, Y. Oka, 2005, Journal of Biological Chemistry.

A case showing an association between type 1 diabetes mellitus and Kabuki syndrome.

T. Asano, Y. Oka, T. Ogihara, 2003, Diabetes research and clinical practice.

Characterization of rat GLUT5 and functional analysis of chimeric proteins of GLUT1 glucose transporter and GLUT5 fructose transporter.

Y. Yazaki, T. Asano, H. Katagiri, 1995, Endocrinology.

Resistin is regulated by C/EBPs, PPARs, and signal-transducing molecules.

T. Asano, H. Katagiri, Y. Oka, 2002, Biochemical and biophysical research communications.

Blockade of the Nuclear Factor-&kgr;B Pathway in the Endothelium Prevents Insulin Resistance and Prolongs Life Spans

T. Asano, H. Katagiri, Y. Oka, 2012, Circulation.

Expression and possible function of glucose transporter protein GLUT1 during preimplantation mouse development from oocytes to blastocysts.

Y. Taketani, Y. Oka, O. Tsutsumi, 1992, Biochemical and biophysical research communications.

Wolfram syndrome 1 (Wfs1) mRNA expression in the normal mouse brain during postnatal development

K. Yamamoto‐Hanada, Y. Oka, K. Shinoda, 2009, Neuroscience Research.

A gene encoding a transmembrane protein is mutated in patients with diabetes mellitus and optic atrophy (Wolfram syndrome)

P. Behn, M. Permutt, G. Sobue, 1998, Nature Genetics.

Colocalization of vascular endothelial growth factor (vascular permeability factor) and insulin in pancreatic islet cells.

S. Mori, Y. Oka, M. Kuroda, 1995, The Journal of clinical endocrinology and metabolism.

Baseline and 1-year interim follow-up assessment of Japanese patients initiating insulin therapy who were enrolled in the cardiovascular risk evaluation in people with type 2 diabetes on insulin therapy study: an international, multicenter, observational study

K. Node, T. Hanafusa, R. Kawamori, 2013, Cardiovascular Diabetology.

Troglitazone treatment increases plasma vascular endothelial growth factor in diabetic patients and its mRNA in 3T3-L1 adipocytes.

Y. Oka, M. Emoto, A. Matsutani, 2001, Diabetes.

PKC mediates cyclic stretch‐induced cardiac hypertrophy through Rho family GTPases and mitogen‐activated protein kinases in cardiomyocytes

Y. Oka, Jing-kun Pan, K. Baker, 2005, Journal of cellular physiology.

Possible Relevance of HLA-DRB1*0403 Haplotype in Insulin Autoimmune Syndrome Induced by α-Lipoic Acid, Used as a Dietary Supplement

Y. Hinokio, H. Katagiri, Y. Oka, 2007, Diabetes Care.

Genetic analysis of Japanese patients with persistent hyperinsulinemic hypoglycemia of infancy: nucleotide-binding fold-2 mutation impairs cooperative binding of adenine nucleotides to sulfonylurea receptor 1.

K. Tachibana, Y. Oka, T. Amachi, 2000, Diabetes.

Clinical features of diabetes mellitus with the mitochondrial DNA 3243 (A-G) mutation in Japanese: maternal inheritance and mitochondria-related complications.

Yutaka Seino, Takashi Kadowaki, Masashi Kobayashi, 2003, Diabetes research and clinical practice.

Bach1 deficiency protects pancreatic β-cells from oxidative stress injury.

H. Katagiri, Y. Oka, Tetsuya Yamada, 2013, American journal of physiology. Endocrinology and metabolism.

Construction of a prediction model for type 2 diabetes mellitus in the Japanese population based on 11 genes with strong evidence of the association

J. Chan, W. So, R. Ma, 2009, Journal of Human Genetics.

Differential targeting of facilitative glucose transporters in polarized epithelial cells.

J. Slot, D. James, Y. Oka, 1996, The American journal of physiology.

Circulating oxidized LDL: a biomarker and a pathogenic factor

H. Katagiri, Y. Oka, Y. Ishigaki, 2009, Current opinion in lipidology.

A hepatic amino acid/mTOR/S6K-dependent signalling pathway modulates systemic lipid metabolism via neuronal signals

T. Asano, H. Katagiri, Y. Oka, 2015, Nature Communications.

Adiposity and Cardiovascular Disorders: Disturbance of the Regulatory System Consisting of Humoral and Neuronal Signals

Hideki Katagiri, H. Katagiri, Y. Oka, 2007, Circulation research.

Involvement of Apolipoprotein E in Excess Fat Accumulation and Insulin Resistance

M. Kanzaki, H. Katagiri, Y. Oka, 2007, Diabetes.

WRN gene 1367 Arg allele protects against development of type 2 diabetes mellitus.

Y. Hinokio, J. Satoh, Y. Oka, 2005, Diabetes research and clinical practice.

Humoral regulation of resistin expression in 3T3-L1 and mouse adipose cells.

T. Asano, H. Katagiri, Y. Oka, 2002, Diabetes.

Involvement of Endoplasmic Stress Protein C/EBP Homologous Protein in Arteriosclerosis Acceleration With Augmented Biological Stress Responses

H. Katagiri, Y. Oka, Tetsuya Yamada, 2011, Circulation.

Increased hypothalamic 5-HT2A receptor gene expression and effects of pharmacologic 5-HT2A receptor inactivation in obese Ay mice.

Y. Oka, K. Nonogaki, K. Nozue, 2006, Biochemical and biophysical research communications.

Obesity alters circadian expressions of molecular clock genes in the brainstem

H. Katagiri, Y. Oka, Tetsuya Yamada, 2009, Brain Research.

Peptidyl-prolyl Cis/Trans Isomerase NIMA-interacting 1 Associates with Insulin Receptor Substrate-1 and Enhances Insulin Actions and Adipogenesis

T. Uchida, N. Horike, T. Asano, 2011, The Journal of Biological Chemistry.

Carboxy terminus of glucose transporter 3 contains an apical membrane targeting domain.

D. James, Y. Oka, K. Inukai, 2004, Molecular endocrinology.

Increased insulin demand promotes while pioglitazone prevents pancreatic beta cell apoptosis in Wfs1 knockout mice

M. Miyazaki, Y. Oka, M. Hatanaka, 2009, Diabetologia.

Wolfram Syndrome in the Japanese Population; Molecular Analysis of WFS1 Gene and Characterization of Clinical Features

Yuichiro Yamada, Y. Oka, M. Akiyama, 2014, PloS one.

Signals from intra-abdominal fat modulate insulin and leptin sensitivity through different mechanisms: neuronal involvement in food-intake regulation.

H. Aburatani, T. Asano, H. Katagiri, 2006, Cell metabolism.

Altered Expression Levels and Impaired Steps in the Pathway to Phosphatidylinositol 3-Kinase Activation via Insulin Receptor Substrates 1 and 2 in Zucker Fatty Rats

Y. Yazaki, T. Asano, J. Terasaki, 1998, Diabetes.

Social isolation affects the development of obesity and type 2 diabetes in mice.

Y. Oka, K. Nonogaki, K. Nozue, 2007, Endocrinology.

Inter-organ metabolic communication involved in energy homeostasis: potential therapeutic targets for obesity and metabolic syndrome.

H. Katagiri, Y. Oka, Tetsuya Yamada, 2008, Pharmacology & therapeutics.

Epidermal growth factor in human milk and the effects of systemic EGF injection on intestinal calcium transport in suckling rats.

H. Greene, Y. Oka, F. Ghishan, 1986, Endocrinologia experimentalis.

Rat epidermal growth factors: purification and tissue content.

W. Suzuki, Y. Oka, M. Aburada, 1989, Endocrinologia japonica.

Expression of GLUT-4 glucose transporter in unweighted soleus muscle of normal and STZ-induced diabetic rats.

Y. Yazaki, T. Asano, H. Katagiri, 1993, The American journal of physiology.

A negative feedback system between brain serotonin systems and plasma active ghrelin levels in mice.

Y. Oka, K. Nonogaki, Kana Ohashi-Nozue, 2006, Biochemical and biophysical research communications.

Characterization of a novel murine preadipocyte line, AP‐18, isolated from subcutaneous tissue: Analysis of adipocyte‐related gene expressions

J. Satoh, M. Fukumoto, H. Katagiri, 2010, Cell biology international.

Altered expression of glucose transporter isoforms with aging in rats — selective decrease in GluT4 in the fat tissue and skeletal muscle

T. Asano, H. Katagiri, Y. Oka, 1991, Diabetologia.

Hyperphagia alters expression of hypothalamic 5-HT2C and 5-HT1B receptor genes and plasma des-acyl ghrelin levels in Ay mice.

Y. Oka, K. Nonogaki, K. Nozue, 2006, Endocrinology.

Association of upregulated activity of KATP channels with impaired insulin secretion in UCP1‐expressing insulinoma cells

C. Tei, Y. Oka, H. Ishihara, 2002, The Journal of physiology.

A case of slowly progressive type 1 diabetes with insulin independence maintained for 10 years with α-glucosidase inhibitor monotherapy.

H. Katagiri, Y. Oka, Tetsuya Yamada, 2012, Internal medicine.

Atf6α-null mice are glucose intolerant due to pancreatic β-cell failure on a high-fat diet but partially resistant to diet-induced insulin resistance.

M. Fukumoto, H. Katagiri, Y. Oka, 2012, Metabolism: clinical and experimental.

Increase in human placental glucose transporter-1 during pregnancy

H. Kurachi, M. Sakata, Y. Oka, 1995 .

Regulation of Pancreatic β Cell Mass by Neuronal Signals from the Liver

T. Asano, H. Katagiri, Y. Oka, 2008, Science.

Structural and functional analysis of the canine histamine H2 receptor by site-directed mutagenesis: N-glycosylation is not vital for its action.

Y. Yazaki, T. Asano, H. Katagiri, 1995, The Biochemical journal.

The type II insulin-like growth factor receptor is internalized and recycles in the absence of ligand.

M. Czech, Y. Oka, 1986, The Journal of biological chemistry.

Differing Roles of Akt and Serum- and Glucocorticoid-regulated Kinase in Glucose Metabolism, DNA Synthesis, and Oncogenic Activity*

Y. Gotoh, T. Asano, H. Katagiri, 2003, Journal of Biological Chemistry.

Leptin increases the viability of isolated rat pancreatic islets by suppressing apoptosis.

Y. Oka, Y. Tanizawa, S. Okuya, 2001, Endocrinology.

Mammalian Sugar Transporters:Their Localization and Link to Cellular Functions.

Y. Oka, K. Takata, H. Hirano, 1993 .

Neuropathy is associated with depression independently of health‐related quality of life in Japanese patients with diabetes

S. Yoshida, S. Awata, Y. Oka, 2009, Psychiatry and clinical neurosciences.

Eradication of insulin resistance

Y. Hinokio, H. Katagiri, Y. Oka, 2009, The Lancet.

Optimal cutoff point of waist circumference and use of home blood pressure as a definition of metabolic syndrome: the Ohasama study.

K. Asayama, M. Kikuya, T. Ohkubo, 2008, American journal of hypertension.

Reliability and validity of the Japanese version of the World Health Organization‐Five Well‐Being Index in the context of detecting depression in diabetic patients

P. Bech, Y. Hinokio, S. Yoshida, 2007, Psychiatry and clinical neurosciences.

Milnacipran, a serotonin and norepinephrine reuptake inhibitor, induces appetite-suppressing effects without inducing hypothalamic stress responses in mice.

Y. Oka, T. Kuboki, K. Nonogaki, 2007, American journal of physiology. Regulatory, integrative and comparative physiology.

Ishihara, H. et al. Type I phosphatidylinositol-4-phosphate 5-kinases. Cloning of the third isoform and deletion/substitution analysis of members of this novel lipid kinase family. J. Biol. Chem. 273, 8741-8748

Y. Yazaki, T. Asano, Y. Oka, 1998 .

Glucose transporter GLUT1 mRNA expression in the ontogeny of glucose incorporation in mouse preimplantation embryos.

Y. Taketani, Y. Oka, O. Tsutsumi, 1994, Biochemical and biophysical research communications.

Multiple cranial mononeuropathies with acetylcholine receptor antibody in mitochondrial diabetes.

Y. Oka, T. Muramatsu, M. Taniyama, 2003, Diabetes Care.

Enhanced phosphoinositide hydrolysis via overexpression of phospholipase C beta1 or delta1 inhibits stimulus-induced insulin release in insulinoma MIN6 cells.

Y. Yazaki, T. Asano, Y. Oka, 1999, Biochemical and biophysical research communications.

Superoxide dismutase in developing mouse retina.

T. Asano, H. Katagiri, Y. Oka, 1994, Japanese journal of ophthalmology.

The N-terminal 34 residues of the 55 kDa regulatory subunits of phosphoinositide 3-kinase interact with tubulin.

T. Asano, M. Funaki, H. Katagiri, 2000, The Biochemical journal.

Effect of mouse epidermal growth factor/urogastrone on the functional maturation of rat intestine.

H. Greene, Y. Oka, F. Ghishan, 1983, Endocrinology.

Replication of genome-wide association studies of type 2 diabetes susceptibility in Japan.

M. Kasuga, K. Tokunaga, J. Takeda, 2008, The Journal of clinical endocrinology and metabolism.

Characterization of GLUT3 protein expressed in Chinese hamster ovary cells.

Y. Yazaki, T. Asano, H. Katagiri, 1992, The Biochemical journal.

Carboxy-terminal modulator protein induces Akt phosphorylation and activation, thereby enhancing antiapoptotic, glycogen synthetic, and glucose uptake pathways.

Y. Uchijima, N. Horike, T. Asano, 2007, American journal of physiology. Cell physiology.

Multiple tumors in mitochondrial diabetes associated with tRNALeu(UUR) mutation at position 3264.

Y. Oka, T. Muramatsu, M. Taniyama, 2003, Diabetes care.

The relationship of microvesicles to the plasmalemma of rat adipocytes.

M. Czech, Y. Oka, J. Pessin, 1983, European journal of cell biology.

Enhanced expression of glucose transporter GLUT3 in tumorigenic HeLa cell hybrids associated with tumor suppressor dysfunction.

T. Suzuki, E. Stanbridge, J. Redpath, 1999, European journal of biochemistry.

Insulin activates the appearance of insulin-like growth factor II receptors on the adipocyte cell surface.

M. Czech, Y. Oka, C. Mottola, 1984, Proceedings of the National Academy of Sciences of the United States of America.

Regulation of PPAR gamma transcriptional activity in 3T3-L1 adipocytes.

H. Katagiri, Y. Oka, K. Inukai, 2003, Biochemical and biophysical research communications.

Mammalian Sugar Transporters:Their Localization and Link to Cellular Functions.

Y. Oka, K. Takata, H. Hirano, 1993 .

Expression and localization of glucose transporter 1 (GLUT1) in the rat oviduct: a possible supplier of glucose to embryo during early embryonic development.

H. Kurachi, M. Sakata, Y. Oka, 1995, Biochemical and biophysical research communications.

[Positional cloning of the gene(WFS1) for Wolfram syndrome].

Y. Oka, H. Inoue, Y. Tanizawa, 2000, Rinsho byori. The Japanese journal of clinical pathology.

Relationship of dysregulation of glucose metabolism with white-coat hypertension: the Ohasama study

K. Asayama, M. Kikuya, T. Ohkubo, 2010, Hypertension Research.

Increased Liver Glucose-Transporter Protein and mRNA in Streptozocin-Induced Diabetic Rats

T. Asano, Y. Oka, Y. Akanuma, 1990, Diabetes.

Targeting of GLUT1-GLUT5 chimeric proteins in the polarized cell line Caco-2.

Y. Yazaki, T. Asano, H. Katagiri, 1997, Molecular endocrinology.

Regulation of glucose transporter 1 (GLUT1) gene expression by epidermal growth factor in bovine corneal endothelial cells.

H. Katagiri, Y. Oka, K. Ishida, 1995, Japanese Journal of Ophthalmology.