A. Buisson

发表

Neurotrophin-3-induced PI-3 kinase/Akt signaling rescues cortical neurons from apoptosis

E. Mackenzie, G. Liot, C. Gabriel, 2004, Experimental Neurology.

Matching Gene Expression with Hypometabolism after Cerebral Ischemia in the Nonhuman Primate

Charles Auffray, Alain Buisson, Denis Vivien, 2002, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

The proteolytic activity of tissue-plasminogen activator enhances NMDA receptor-mediated signaling

Alain Buisson, Fabian Docagne, Denis Vivien, 2001, Nature Medicine.

2,7-Bis-(4-Amidinobenzylidene)-Cycloheptan-1-One Dihydrochloride, tPA Stop, Prevents tPA-Enhanced Excitotoxicity Both In Vitro and In Vivo

Karim Benchenane, E. Mackenzie, G. Liot, 2004, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Iron overload accelerates neuronal amyloid-β production and cognitive impairment in transgenic mice model of Alzheimer's disease

Alain Buisson, A. Buisson, T. Freret, 2014, Neurobiology of Aging.

Transforming Growth Factor-β1 Potentiates Amyloid-β Generation in Astrocytes and in Transgenic Mice*

E. Mackenzie, A. Delacourte, L. Buée, 2003, The Journal of Biological Chemistry.

Transforming growth factor‐βl as a regulator of the serpins/t‐PA axis in cerebral ischemia

E. Mackenzie, A. Buisson, D. Vivien, 1999, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Up‐regulation of a serine protease inhibitor in astrocytes mediates the neuroprotective activity of transforming growth factor β1

E. Mackenzie, A. Buisson, D. Vivien, 1998, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Transforming growth factor α‐induced expression of type‐1 plasminogen activator inhibitor in astrocytes rescues neurons from excitotoxicity

E. Mackenzie, C. Gabriel, C. Ali, 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Striatal Protection Induced by Lesioning the Substantia Nigra of Rats Subjected to Focal Ischemia

A. Buisson, M. Plotkine, J. Callebert, 1992, Journal of neurochemistry.

Serine Protease Inhibitors: Novel Therapeutic Targets for Stroke?

A. Buisson, D. Vivien, D. Vivien, 2000, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Transforming Growth Factor-β and Ischemic Brain Injury

E. Mackenzie, C. Ali, A. Buisson, 2003, Cellular and Molecular Neurobiology.

NMDA receptor dysfunction contributes to impaired brain‐derived neurotrophic factor‐induced facilitation of hippocampal synaptic transmission in a Tau transgenic model

L. Buée, A. Buisson, N. Sergeant, 2013, Aging cell.

NMDA receptor activation inhibits alpha-secretase and promotes neuronal amyloid-beta production.

E. Mackenzie, C. Gabriel, C. Ali, 2005, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Amyloid Fibers Reveal Themselves With Near‐Infrared

A. Buisson, M. Decressac, Michael Decressac, 2019, Movement disorders : official journal of the Movement Disorder Society.

Improved optical slicing by stimulated emission depletion light sheet microscopy.

A. Buisson, J. Vial, I. Wang, 2020, Biomedical optics express.

Copper-catalyzed amination of (bromophenyl)ethanolamine for a concise synthesis of aniline-containing analogues of NMDA NR2B antagonist ifenprodil.

L. Barré, A. Buisson, P. Marchand, 2010, Organic & biomolecular chemistry.

Involvement of CRF2 signaling in enterocyte differentiation

B. Bonaz, A. Buisson, M. Jacquier-Sarlin, 2017, World journal of gastroenterology.

Reply to “Tissue plasminogen activator and NMDA receptor cleavage”

A. Buisson, D. Vivien, Mónica Fernández-Monreal, 2003, Nature Medicine.

Ischemia-Induced Interleukin-6 as a Potential Endogenous Neuroprotective Cytokine against NMDA Receptor-Mediated Excitoxicity in the Brain

E. Mackenzie, C. Ali, A. Buisson, 2000, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Transforming growth factor-beta and ischemic brain injury.

E. Mackenzie, C. Ali, A. Buisson, 2003, Cellular and molecular neurobiology.

A transforming growth factor-beta antagonist unmasks the neuroprotective role of this endogenous cytokine in excitotoxic and ischemic brain injury.

E. Mackenzie, C. Ali, A. Buisson, 1999, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Evidence of Type I and Type II Transforming Growth Factor‐β Receptors in Central Nervous Tissues: Changes Induced by Focal Cerebral Ischemia

E. Mackenzie, M. Bernaudin, A. Buisson, 1998, Journal of Neurochemistry.

GluN2B Subunit Labeling with Fluorescent Probes and High-Resolution Live Imaging.

A. Buisson, O. Nicole, C. Perrio, 2017, Methods in molecular biology.

Confocal microscopy imaging of NR2B-containing NMDA receptors based on fluorescent ifenprodil-like conjugates.

Laetitia Mony, P. Paoletti, L. Barré, 2012, Bioconjugate chemistry.

Is tissue-type plasminogen activator a neuromodulator?

E. Mackenzie, G. Bu, Mathias Cacquevel, 2004, Molecular and Cellular Neuroscience.

Complement anaphylatoxin C3a is selectively protective against NMDA-induced neuronal cell death

E. Mackenzie, C. Ali, A. Buisson, 2001, Neuroreport.

Tubulin tyrosination regulates synaptic function and is disrupted in Alzheimer’s disease

A. Buisson, P. Delagrange, Fabien Lanté, 2022, Brain : a journal of neurology.

Reduction of Ischemic Brain Damage by Nitrous Oxide and Xenon

E. Mackenzie, A. Buisson, J. Abraini, 2003, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Reciprocal disruption of neuronal signaling and Aβ production mediated by extrasynaptic NMDA receptors: a downward spiral

A. Buisson, T. Rush, 2014, Cell and Tissue Research.

Neuroprotection by nitrous oxide and xenon and its relation to minimum alveolar concentration.

E. Mackenzie, A. Buisson, J. Abraini, 2004, Anesthesiology.

Akt-dependent expression of NAIP-1 protects neurons against amyloid-{beta} toxicity.

E. Mackenzie, E. White, C. Gabriel, 2005, The Journal of biological chemistry.

Interaction Between αCaMKII and GluN2B Controls ERK-Dependent Plasticity

P. De Koninck, B. Bontempi, A. Buisson, 2012, The Journal of Neuroscience.

The neuroprotective effect of a nitric oxide inhibitor in a rat model of focal cerebral ischaemia

A. Buisson, M. Plotkine, R. Boulu, 1992, British journal of pharmacology.

Activation of Extrasynaptic, But Not Synaptic, NMDA Receptors Modifies Amyloid Precursor Protein Expression Pattern and Increases Amyloid-β Production

A. Buisson, O. Nicole, Javier Becerril-Ortega, 2010, The Journal of Neuroscience.

DCG‐IV Selectively Attenuates Rapidly Triggered NMDA‐induced Neurotoxicity in Cortical Neurons

D. Choi, A. Buisson, Shan Ping Yu, 1996, The European journal of neuroscience.

The inhibitory mGluR agonist, s-4-carboxy-3-hydroxy-phenylglycine selectively attenuates NMDA neurotoxicity and oxygen-glucose deprivation-induced neuronal death

D. Choi, A. Buisson, 1995, Neuropharmacology.

Synthesis and in Vitro Characterisation of Ifenprodil‐Based Fluorescein Conjugates as GluN1/GluN2B N‐Methyl‐D‐aspartate Receptor Antagonists

E. Mackenzie, L. Barré, A. Buisson, 2013, Chembiochem : a European journal of chemical biology.

Neuronal viability is controlled by a functional relation between synaptic and extrasynaptic NMDA receptors

A. Buisson, O. Nicole, F. Léveillé, 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Nitric oxide: an endogenous anticonvulsant substance

A. Buisson, M. Plotkine, R. Boulu, 1993, Neuroreport.

Transforming growth factor-beta 1 potentiates amyloid-beta generation in astrocytes and in transgenic mice.

E. Mackenzie, A. Delacourte, L. Buée, 2003, The Journal of biological chemistry.

Transforming growth factor-beta1-modulated cerebral gene expression.

C. Auffray, E. Mackenzie, G. Liot, 2002, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Impaired α-tubulin re-tyrosination leads to synaptic dysfunction and is a feature of Alzheimer’s disease

A. Buisson, P. Delagrange, Fabien Lanté, 2021, bioRxiv.

NMDA Receptor Activation Inhibits α-Secretase and Promotes Neuronal Amyloid-β Production

E. Mackenzie, C. Gabriel, C. Ali, 2005, The Journal of Neuroscience.

Autophagy Is Required for Memory Formation and Reverses Age-Related Memory Decline

P. Codogno, Allyson K. Friedman, S. Moriceau, 2019, Current Biology.

Astrocyte-neuron interplay is critical for Alzheimer's disease pathogenesis and is rescued by TRPA1 channel blockade.

A. Buisson, M. Albrieux, M. Jacquier-Sarlin, 2021, Brain : a journal of neurology.

Akt-dependent Expression of NAIP-1 Protects Neurons against Amyloid-β Toxicity*

E. Mackenzie, E. White, C. Gabriel, 2005, Journal of Biological Chemistry.

Neuroprotection Mediated by Glial Cell Line-Derived Neurotrophic Factor: Involvement of a Reduction of NMDA-Induced Calcium Influx by the Mitogen-Activated Protein Kinase Pathway

E. Mackenzie, C. Ali, A. Buisson, 2001, The Journal of Neuroscience.

A key function for microtubule-associated-protein 6 in activity-dependent stabilisation of actin filaments in dendritic spines

L. Blanchoin, C. Delphin, J. Deloulme, 2018, Nature Communications.

Synthesis, evaluation and metabolic studies of radiotracers containing a 4-(4-[18F]-fluorobenzyl)piperidin-1-yl moiety for the PET imaging of NR2B NMDA receptors.

L. Barré, A. Buisson, R. Labas, 2011, European journal of medicinal chemistry.

TRPA1 channels promote astrocytic Ca2+ hyperactivity and synaptic dysfunction mediated by oligomeric forms of amyloid-β peptide

A. Buisson, M. Albrieux, M. Jacquier-Sarlin, 2017, Molecular Neurodegeneration.

Astrocyte-neuron interplay is critical for Alzheimer’s disease pathogenesis and is rescued by TRPA1 channel blockade

A. Buisson, M. Albrieux, S. Boisseau, 2021, bioRxiv.

Selective impairment of some forms of synaptic plasticity by oligomeric amyloid-β peptide in the mouse hippocampus: implication of extrasynaptic NMDA receptors.

P. Dutar, A. Buisson, Vincent Villette, 2012, Journal of Alzheimer's disease : JAD.

Reverse glial glutamate uptake triggers neuronal cell death through extrasynaptic NMDA receptor activation

C. Melon, A. Buisson, O. Nicole, 2009, Molecular and Cellular Neuroscience.

Synapses, NMDA receptor activity and neuronal Aβ production in Alzheimer’s disease

A. Buisson, Javier Becerril-Ortega, K. Bordji, 2011, Reviews in the neurosciences.

Synaptotoxicity in Alzheimer's Disease Involved a Dysregulation of Actin Cytoskeleton Dynamics through Cofilin 1 Phosphorylation

A. Buisson, M. Jacquier-Sarlin, E. Borel, 2018, The Journal of Neuroscience.

VEGF counteracts amyloid-β-induced synaptic dysfunction.

J. Honnorat, A. Buisson, N. Chounlamountri, 2021, Cell reports.

Membrane‐delimited modulation of NMDA currents by metabotropic glutamate receptor subtypes 1/5 in cultured mouse cortical neurons.

S P Yu, S L Sensi, L M Canzoniero, 1997, The Journal of physiology.

Effect of Aβ Oligomers on Neuronal APP Triggers a Vicious Cycle Leading to the Propagation of Synaptic Plasticity Alterations to Healthy Neurons

A. Buisson, Fabien Lanté, M. Rolland, 2020, The Journal of Neuroscience.

The amyloid-β oligomer Aβ*56 induces specific alterations in neuronal signaling that lead to tau phosphorylation and aggregation

A. Buisson, S. Lesné, J. Götz, 2017, Science Signaling.

Density of GABAB Receptors Is Reduced in Granule Cells of the Hippocampus in a Mouse Model of Alzheimer’s Disease

R. Shigemoto, R. Luján, Y. Fukazawa, 2020, International journal of molecular sciences.

Reduction in the neuronal surface of post and presynaptic GABAB receptors in the hippocampus in a mouse model of Alzheimer's disease

R. Shigemoto, R. Luján, Y. Fukazawa, 2019, Brain pathology.

Assembly of The Mitochondrial Complex I Assembly Complex Suggests a Regulatory Role for Deflavination

M. Soler‐Lopez, P. Mas, D. Hart, 2020, Angewandte Chemie.

A key function for microtubule-associated-protein 6 in activity-dependent stabilisation of actin filaments in dendritic spines

L. Blanchoin, C. Delphin, J. Deloulme, 2018, Nature Communications.

Oxygen glucose deprivation-induced astrocyte dysfunction provokes neuronal death through oxidative stress.

A. Nieoullon, A. Buisson, L. Kerkerian-Le Goff, 2014, Pharmacological research.

Increased Expression of Transforming Growth Factor-β after Cerebral Ischemia in the Baboon: An Endogenous Marker of Neuronal Stress?

E. Mackenzie, J. Toutain, C. Ali, 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Activity-Dependent Tau Protein Translocation to Excitatory Synapse Is Disrupted by Exposure to Amyloid-Beta Oligomers

A. Buisson, Fabien Lanté, I. Arnal, 2014, The Journal of Neuroscience.

Treating early postnatal circuit defect delays Huntington’s disease onset and pathology in mice

E. Barbier, S. Humbert, J. Deloulme, 2022, Science.

Disruption of dopaminergic transmission remodels tripartite synapse morphology and astrocytic calcium activity within substantia nigra pars reticulata

M. Savasta, A. Buisson, M. Albrieux, 2015, Glia.

Pyr1-Mediated Pharmacological Inhibition of LIM Kinase Restores Synaptic Plasticity and Normal Behavior in a Mouse Model of Schizophrenia

C. Nguyen, A. Buisson, Fabien Lanté, 2021, Frontiers in Pharmacology.

Comparison of the pharmacological properties of GK11 and MK801, two NMDA receptor antagonists: towards an explanation for the lack of intrinsic neurotoxicity of GK11

A. Privat, A. Buisson, G. Desmadryl, 2007, Journal of neurochemistry.

A Transforming Growth Factor-β Antagonist Unmasks the Neuroprotective Role of This Endogenous Cytokine in Excitotoxic and Ischemic Brain Injury

E. Mackenzie, C. Ali, A. Buisson, 1999 .