B. Butts

发表

Elevated basal reactive oxygen species and phospho‐Akt in murine keratinocytes resistant to ultraviolet B–induced apoptosis

B. Butts, K. Kwei, G. T. Bowden, 2003, Molecular carcinogenesis.

Attenuation of catalase activity in the malignant phenotype plays a functional role in an in vitro model for tumor progression.

B. Butts, K. Kwei, M. Briehl, 2001, Cancer letters.

Generation and characterization of antibodies specific for caspase-cleaved neo-epitopes: a novel approach

K. Vora, H. Mehmet, B. Butts, 2011, Cell Death and Disease.

Increased tumor necrosis factor-alpha sensitivity of MCF-7 cells transfected with NAD(P)H:quinone reductase.

B. Butts, M. Briehl, L. Siemankowski, 2000, Cancer research.

The genome of epsilon15, a serotype-converting, Group E1 Salmonella enterica-specific bacteriophage.

S. J. Billington, B. Butts, A. Kropinski, 2007, Virology.

Caspases and Cell Death

H. Mehmet, B. Butts, L. Hernandez, 2009 .

Inhibition of O-GlcNAcase leads to elevation of O-GlcNAc tau and reduction of tauopathy and cerebrospinal fluid tau in rTg4510 mice

N. Hastings, B. Butts, R. Hargreaves, 2017, Molecular Neurodegeneration.

The genome of ε15, a serotype-converting, Group E1 Salmonella enterica-specific bacteriophage

S. J. Billington, Andrew M. Kropinski, B. Butts, 2007 .

Two molecular mechanisms of apoptosis resistance

B. Butts, 2002 .

The permeability transition pore triggers Bax translocation to mitochondria during neuronal apoptosis

T. Laessig, K. Heidenreich, B. Butts, 2005, Cell Death and Differentiation.

Targeted Quantitative Mass Spectrometry Analysis of Protein Biomarkers From Previously Stained Single Formalin-Fixed Paraffin-Embedded Tissue Sections.

M. Westfall, B. Butts, S. Hill, 2023, Laboratory investigation; a journal of technical methods and pathology.

Glycogen Synthase Kinase-3β Phosphorylates Bax and Promotes Its Mitochondrial Localization during Neuronal Apoptosis

T. Laessig, K. Heidenreich, B. Butts, 2004, The Journal of Neuroscience.

Maturation-dependent sensitivity of oligodendrocyte lineage cells to apoptosis: implications for normal development and disease

H. Mehmet, B. Butts, C. Houde, 2008, Cell Death and Differentiation.

Distinct mechanisms of neuronal apoptosis are triggered by antagonism of Bcl‐2/Bcl‐x(L) versus induction of the BH3‐only protein Bim

K. Heidenreich, B. Butts, D. Linseman, 2005, Journal of neurochemistry.

Proteasome inhibition elicits a biphasic effect on neuronal apoptosis via differential regulation of pro-survival and pro-apoptotic transcription factors

K. Heidenreich, B. Butts, D. Linseman, 2005, Molecular and Cellular Neuroscience.

Inhibition of O-GlcNAcase leads to elevation of O-GlcNAc tau and reduction of tauopathy and cerebrospinal fluid tau in rTg4510 mice

Lynn A. Hyde, N. Hastings, B. Butts, 2017, Molecular Neurodegeneration.

Identification of a functional peroxisome proliferator activated receptor response element in the 3' untranslated region of the human bcl-2 gene.

B. Butts, M. Briehl, N. Tran, 2004, International journal of oncology.

Insulin-like Growth Factor-I Suppresses Degradation of the Pro-survival Transcription Factor Myocyte Enhancer Factor 2D (MEF2D) During Neuronal Apoptosis

T. Laessig, K. Heidenreich, B. Butts, 2003, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.