W. Schmitz

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In Cardiomyocyte Hypoxia, Insulin-Like Growth Factor-I-Induced Antiapoptotic Signaling Requires Phosphatidylinositol-3-OH-Kinase-Dependent and Mitogen-Activated Protein Kinase-Dependent Activation of the Transcription Factor cAMP Response Element-Binding Protein

P. Li, R. Dietz, Peifeng Li, 2001, Circulation.

Transcriptional regulation by cAMP in the heart

F. Müller, W. Schmitz, J. Neumann, 2000, Molecular and Cellular Biochemistry.

Transcription factor AP-2α triggers apoptosis in cardiac myocytes

H. Scheld, I. H. Engels, K. Schulze-Osthoff, 2004, Cell Death and Differentiation.

Annexin A4 N‐terminal peptide inhibits adenylyl cyclase 5 and limits β‐adrenoceptor‐mediated prolongation of cardiac action potential

F. Müller, W. Schmitz, V. Gerke, 2020, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

A proteasome inhibitor confers cardioprotection.

W. Schmitz, J. Neumann, H. Lüss, 2002, Cardiovascular research.

Ryanodine Receptor–Mediated Calcium Leak Drives Progressive Development of an Atrial Fibrillation Substrate in a Transgenic Mouse Model

S. Nattel, X. Wehrens, D. Dobrev, 2014, Circulation.

Identification and characterization of diadenosine 5′,5‴‐P1,P2‐diphosphate and diadenosine 5′,5‴‐P1,P3‐triphosphate in human myocardial tissue

H. Schlüter, J. Jankowski, W. Schmitz, 1999, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Heart-directed Expression of a Human Cardiac Isoform of cAMP-Response Element Modulator in Transgenic Mice*

P. Kirchhof, F. Müller, W. Schmitz, 2005, Journal of Biological Chemistry.

Effects of caffeine, halothane, succinylcholine, phenylephrine and isoproterenol on myocardial force of contraction of malignant hyperthermia susceptible swine

J. Scholz, W. Schmitz, N. Roewer, 1991, Acta anaesthesiologica Scandinavica.

Impaired cardiac contraction and relaxation and decreased expression of sarcoplasmic Ca2+‐ATPase in mice lacking the CREM gene

F. Müller, W. Schmitz, G. Schütz, 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Cardiomyocyte‐specific inactivation of transcription factor CREB in mice

Michael D. Schneider, F. Müller, W. Schmitz, 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Evidence for a functional role of calsequestrin 2 in mouse atrium

W. Schmitz, U. Gergs, J. Neumann, 2017, Acta physiologica.

Altered sinus nodal and atrioventricular nodal function in freely moving mice overexpressing the A1 adenosine receptor.

G. Breithardt, P. Kirchhof, W. Schmitz, 2003, American journal of physiology. Heart and circulatory physiology.

Usefulness of increased myocardial cyclic adenosine 3',5'-monophosphate content as a sign of rejection after cardiac transplantation.

W. Schmitz, P. Lichtlen, H. E. von der Leyen, 1995, The American journal of cardiology.

Changes in cardiac beta 1- and beta 2-adrenoceptor densities after human cardiac transplantation: relation to transplant coronary vasculopathy and pretransplantation disease.

M. Steinfath, W. Schmitz, A. Haverich, 1992, American heart journal.

Increased myocardial cAMP in patients with transplant coronary vasculopathy

W. Schmitz, A. Haverich, P. Lichtlen, 1992, The Lancet.

Isoprenaline Stimulates Gene Transcription of the Inhibitory G Protein a-Subunit G . ia-2 in Rat Heart

K. Boheler, W. Schmitz, T. Eschenhagen, 2005 .

Isoprenaline-induced increase in the 40/41 kDa pertussis toxin substrates and functional consequences on contractile response in rat heart

M. Steinfath, J. Schulte am Esch, W. Schmitz, 2004, Naunyn-Schmiedeberg's Archives of Pharmacology.

Distinct down-regulation of cardiac β1- and β2-adrenoceptors in different human heart diseases

M. Steinfath, W. Schmitz, A. Haverich, 1991, Naunyn-Schmiedeberg's Archives of Pharmacology.

Isoprenaline-induced increase in mRNA levels of inhibitory G-protein α-subunits in rat heart

W. Schmitz, T. Eschenhagen, A. Warnholtz, 1991, Naunyn-Schmiedeberg's Archives of Pharmacology.

Effects of metoprolol on myocardial β-adrenoceptors and Giα-proteins in patients with congestive heart failure

M. Steinfath, W. Schmitz, P. Hanrath, 1996, European Journal of Clinical Pharmacology.

Isoprenaline stimulates gene transcription of the inhibitory G protein alpha-subunit Gi alpha-2 in rat heart.

K. Boheler, F. Müller, W. Schmitz, 1993, Circulation research.

Effect of captopril on myocardial β-adrenoceptor density and Giα-proteins in patients with mild to moderate heart failure due to dilated cardiomyopathy

M. Steinfath, J. Schulte am Esch, W. Schmitz, 2004, European Journal of Clinical Pharmacology.

Activation and inactivation of cAMP-response element-mediated gene transcription in cardiac myocytes.

F. Müller, W. Schmitz, J. Neumann, 2001, Cardiovascular research.

Quantification of the cAMP response element binding protein in ventricular nuclear protein from failing and nonfailing human hearts.

H. Scheld, M. Böhm, F. Müller, 1997, Biochemical and biophysical research communications.

The early response genes c-jun and HSP-70 are induced in regional cardiac stunning in conscious mammals.

F. Müller, W. Schmitz, N. Rolf, 2000, The Journal of thoracic and cardiovascular surgery.

Regulation of the human Giα-2 gene promotor activity in embryonic chicken cardiomyocytes

L. Weinstein, W. Schmitz, J. Weil, 2004, Basic Research in Cardiology.

Mechanical unloading of the rat heart involves marked changes in the protein kinase-phosphatase balance.

W. Schmitz, H. Ehmke, A. Schwoerer, 2008, Journal of molecular and cellular cardiology.

Reduced α1‐ and β2‐adrenoceptor‐mediated positive inotropic effects in human end‐stage heart failure

M. Steinfath, W. Schmitz, A. Haverich, 1992 .

Phosphodiesterase inhibition and positive inotropic effects.

W. Schmitz, H. E. von der Leyen, H. Scholz, 1989, Journal of cardiovascular pharmacology.

INCREASE IN MYOCARDIAL Gi-PROTEINS IN HEART FAILURE

W. Schmitz, P. Kalmar, H. Scholz, 1988, The Lancet.

Increased messenger RNA level of the inhibitory G protein alpha subunit Gi alpha-2 in human end-stage heart failure.

W. Schmitz, A. Haverich, T. Eschenhagen, 1992, Circulation research.

Long term beta-adrenoceptor-mediated up-regulation of Gi alpha and G(o) alpha mRNA levels and pertussis toxin-sensitive guanine nucleotide-binding proteins in rat heart.

J. Schulte am Esch, W. Schmitz, M. Diederich, 1992, Molecular pharmacology.

Changes in cardiac β-Adrenoceptors in human heart diseases: Relationship to the degree of heart failure and further evidence for etiology-related regulation of β1 and β2 subtypes☆

M. Steinfath, W. Schmitz, P. Kalmar, 1993 .

Endotoxin and cytokines induce direct cardiodepressive effects in mammalian cardiomyocytes via induction of nitric oxide synthase.

W. Schmitz, H. Scholz, M. Thoenes, 1996, Journal of molecular and cellular cardiology.

cAMP response element binding protein is expressed and phosphorylated in the human heart.

H. Scheld, M. Böhm, F. Müller, 1995, Circulation.

Genetic disruption of G proteins, Gi2α or Goα, does not abolish inotropic and chronotropic effects of stimulating muscarinic cholinoceptors in atrium

L. Birnbaumer, F. Müller, W. Schmitz, 2009, British journal of pharmacology.

Mechanism underlying the reduced positive inotropic effects of the phosphodiesterase III inhibitors pimobendan, adibendan and saterinone in failing as compared to nonfailing human cardiac muscle preparations

W. Schmitz, A. Haverich, H. Leyen, 1991, Naunyn-Schmiedeberg's Archives of Pharmacology.

Phosphodiesterase inhibition and positive inotropy in failing human myocardium.

F. Müller, W. Schmitz, T. Eschenhagen, 1992, Basic research in cardiology.

Bovine hereditary cardiomyopathy: an animal model of human dilated cardiomyopathy.

F. Müller, W. Schmitz, M. Diederich, 1995, Journal of molecular and cellular cardiology.

The stress-responsive MAP kinase p38 is activated by low-flow ischemia in the in situ porcine heart.

W. Schmitz, G. Heusch, R. Schulz, 2000, Journal of molecular and cellular cardiology.

Compartmentalization of NO signaling cascade in skeletal muscles.

M. Wellner, W. Schmitz, I. Buchwalow, 2005, Biochemical and biophysical research communications.

Evidence against a role of a pertussis toxin-sensitive guanine nucleotide-binding protein in the alpha1-adrenoceptor-mediated positive inotropic effect in the heart

M. Böhm, W. Schmitz, H. Scholz, 1987, Naunyn-Schmiedeberg's Archives of Pharmacology.

Evidence for the existence of inositol tetrakisphosphate in mammalian heart. Effect of alpha 1-adrenoceptor stimulation.

J. Scholz, W. Schmitz, H. Scholz, 1990, Circulation research.

Impaired relaxation in transgenic mice overexpressing junctin.

D. Bers, W. Schmitz, I. Buchwalow, 2003, Cardiovascular research.

Basal and isoprenaline-stimulated cAMP content in failing versus nonfailing human cardiac preparations.

W. Schmitz, P. Kalmar, H. Scholz, 1989, Journal of cardiovascular pharmacology.

Effects of metoprolol on myocardial beta-adrenoceptors and Gi alpha-proteins in patients with congestive heart failure.

M. Steinfath, W. Schmitz, P. Hanrath, 1996, European journal of clinical pharmacology.

Messenger RNA expression and immunological quantification of phospholamban and SR-Ca(2+)-ATPase in failing and nonfailing human hearts.

W. Schmitz, B. Linck, 2000, Cardiovascular research.

Postnatal changes in contractile time parameters, calcium regulatory proteins, and phosphatases.

F. Müller, W. Schmitz, G. Bodor, 1998, American journal of physiology. Heart and circulatory physiology.

Expression of calcium regulatory proteins in short-term hibernation and stunning in the in situ porcine heart.

F. Müller, W. Schmitz, G. Heusch, 1998, Cardiovascular research.

Regional distribution of β1- and β2-adrenoceptors in the failing and nonfailing human heart

M. Steinfath, W. Schmitz, P. Kalmar, 2004, European Journal of Clinical Pharmacology.

Design of new beta1-selective adrenoceptor ligands as potential radioligands for in vivo imaging.

O. Schober, S. Luthra, V. Pike, 2003, Bioorganic & medicinal chemistry.

Biochemical changes in malignant hyperthermia susceptible swine: cyclic AMP, inositol phosphates, α1, β1‐ and β2‐adrenoceptors in skeletal and cardiac muscle

J. Scholz, M. Steinfath, W. Schmitz, 1993 .

Decrease in β1- and increase in β2-adrenoceptors in long-term follow-up after orthotopic cardiac transplantation

M. Steinfath, W. Schmitz, A. Haverich, 1992 .

Cardiac α1‐adrenoceptor densities in different mammalian species

M. Steinfath, W. Schmitz, H. Scholz, 1992 .

The role of alpha 1-adrenergic and muscarinic receptors in cardiac function.

F. Müller, W. Schmitz, T. Eschenhagen, 1991, European heart journal.

Modulation of SR Ca2+ release by the triadin-to-calsequestrin ratio in ventricular myocytes.

F. Müller, W. Schmitz, H. Baba, 2012, American journal of physiology. Heart and circulatory physiology.

Transgenic triadin 1 overexpression alters SR Ca2+ handling and leads to a blunted contractile response to beta-adrenergic agonists.

M. Lohse, W. Schmitz, L. Hein, 2004, Cardiovascular research.

Cardiac overexpression of the human 5-HT4 receptor in mice.

P. Kirchhof, S. Hauptmann, A. Böckler, 2010, American journal of physiology. Heart and circulatory physiology.

Age-dependent biochemical and contractile properties in atrium of transgenic mice overexpressing junctin.

P. Kirchhof, W. Schmitz, H. Baba, 2004, American journal of physiology. Heart and circulatory physiology.

Increased Messenger RNA Level ofthe Inhibitory G Protein a Subunit Gia,2 inHumanEnd-Stage HeartFailure

W. Schmitz, U. Mende, V. Döring, 2000 .

Phosphodiesterase inhibition in ventricular cardiomyocytes from guinea‐pig hearts

W. Schmitz, H. Scholz, B. Stein, 1992, British journal of pharmacology.

Alpha-adrenoceptor-mediated positive inotropic effect of phenylephrine in isolated human ventricular myocardium.

W. Schmitz, A. Mügge, H. Scholz, 1984, European journal of pharmacology.

Reduced expression of Ca2+-regulating proteins in the upper gastrointestinal tract of patients with achalasia.

W. Schmitz, J. Fischer, J. Konturek, 2006, World journal of gastroenterology.

On the Cardiac Contractile, Electrophysiological and Biochemical Effects of Endothall, a Protein Phosphatase Inhibitor

S. Huke, F. Müller, W. Schmitz, 2000, Pharmacology.

Inotropic effects of diadenosine tetraphosphate (AP4A) in human and animal cardiac preparations.

S. Huke, H. Scheld, H. Schlüter, 1999, The Journal of pharmacology and experimental therapeutics.

Human 5-HT4 receptor stimulation in atria of transgenic mice

S. Hauptmann, A. Böckler, W. Schmitz, 2013, Naunyn-Schmiedeberg's Archives of Pharmacology.

Enhanced cardiac function in mice overexpressing protein phosphatase Inhibitor-2.

A. Depaoli-Roach, W. Schmitz, H. Baba, 2005, Cardiovascular research.

Interleukin-6 correlates with hemodynamic impairment during dobutamine administration in chronic heart failure.

G. Breithardt, H. Scheld, G. Assmann, 1996, International journal of cardiology.

Effects of adenosine analogues on contractile response and CAMP content in guinea-pig isolated ventricular myocytes

W. Schmitz, H. Scholz, J. Neumann, 1989, Naunyn-Schmiedeberg's Archives of Pharmacology.

Characterization of biochemical effects of CGS 21680C, an A2-adenosine receptor agonist, in the mammalian ventricle.

W. Schmitz, H. Scholz, J. Neumann, 1997, Journal of cardiovascular pharmacology.

Effects of adenosine analogues on force and cAMP in the heart. Influence of adenosine deaminase.

W. Schmitz, H. Scholz, J. Neumann, 1989, European journal of pharmacology.

Mechanism of the "Antiadrenergic" Effects of Adenosine on Myocardial Force of Contraction

M. Böhm, W. Schmitz, H. Scholz, 1987 .

Functional studies in atrium overexpressing A1‐adenosine receptors

F. Müller, W. Schmitz, G. Matherne, 1999, British journal of pharmacology.

Identification and expression of a novel isoform of cAMP response element modulator in the human heart

H. Scheld, E. Oetjen, F. Müller, 1998, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Comparison of Contractile Responses in Isolated Mouse Aorta and Pulmonary Artery: Influence of Strain and Sex

W. Schmitz, F. Balzer, U. Gergs, 2006, Journal of cardiovascular pharmacology.

Evidence for Arrhythmogenic Effects of A2A-Adenosine Receptors

P. Kirchhof, F. Müller, W. Schmitz, 2019, Front. Pharmacol..

Regulation and possible functional implications of G-protein mRNA expression in nonfailing and failing ventricular myocardium.

J. Schulte am Esch, W. Schmitz, T. Eschenhagen, 1992, Basic research in cardiology.

Autonomic Dysfunction in Patients with Arrhythmogenic Right Ventricular Cardiomyopathy: Biochemical Evidence of Altered Signaling Pathways

G. Breithardt, J. Gerss, W. Schmitz, 2014, Pacing and clinical electrophysiology : PACE.

Inhibitor-2 prevents protein phosphatase 1-induced cardiac hypertrophy and mortality.

P. Kirchhof, A. Depaoli-Roach, W. Schmitz, 2008, American journal of physiology. Heart and circulatory physiology.

Sphingosine‐1‐Phosphate Receptor 1 Regulates Cardiac Function by Modulating Ca2+ Sensitivity and Na+/H+ Exchange and Mediates Protection by Ischemic Preconditioning

J. Stypmann, D. Herr, K. Hiller, 2016, Journal of the American Heart Association.

Isoproterenol infusion induces alterations in expression of hypertrophy-associated genes in rat heart.

E. Lakatta, M. Crow, M. Boluyt, 1995, The American journal of physiology.

Relation between contractile function and regulatory cardiac proteins in hypertrophied hearts.

E. Krause, W. Schmitz, H. Scholz, 1996, The American journal of physiology.

Progressive cardiac hypertrophy and dysfunction in atrial natriuretic peptide receptor (GC-A) deficient mice

E. Ehler, W. Schmitz, M. Kuhn, 2002, Heart.

Increased effects of C-type natriuretic peptide on cardiac ventricular contractility and relaxation in guanylyl cyclase A-deficient mice.

S. Lohmann, W. Schmitz, M. Kuhn, 2002, Cardiovascular research.

Effects of α- and β-adrenergic agonists, phosphodiesterase inhibitors and adenosine on isolated human heart muscle preparations

E. Erdmann, W. Schmitz, H. Scholz, 1987 .

Distinct down-regulation of cardiac beta 1- and beta 2-adrenoceptors in different human heart diseases.

M. Steinfath, W. Schmitz, A. Haverich, 1991, Naunyn-Schmiedeberg's archives of pharmacology.

Biochemical mechanism(s) of stunning in conscious dogs.

F. Müller, W. Schmitz, N. Rolf, 2000, American journal of physiology. Heart and circulatory physiology.

Pharmacological characterization of A2-adenosine receptors in guinea-pig ventricular cardiomyocytes.

W. Schmitz, H. Scholz, B. Stein, 1994, Journal of molecular and cellular cardiology.

On the contractile function of protein phosphatases in isolated human coronary arteries

S. Huke, H. Scheld, F. Müller, 1999, Naunyn-Schmiedeberg's Archives of Pharmacology.

Effect of 2,3-butanedione monoxime on force of contraction and protein phosphorylation in bovine smooth muscle

W. Schmitz, H. Aken, H. Van Aken, 1999, Naunyn-Schmiedeberg's Archives of Pharmacology.

The protein phosphatase inhibitor cantharidin alters vascular endothelial cell permeability.

S. Huke, F. Müller, W. Schmitz, 1999, The Journal of pharmacology and experimental therapeutics.

Contractility and inhibition of protein phosphatases by cantharidin.

S. Huke, F. Müller, W. Schmitz, 1998, General pharmacology.

The mechanism of action of cantharidin in smooth muscle

S. Huke, F. Müller, W. Schmitz, 1998, British journal of pharmacology.

Reduced a1- and fl2-adrenoceptor-mediated positive inotropic effects in human end-stage heart failure

M. Steinfath, W. Schmitz, H. Scholz, 2006 .

Cardiac Dysfunction

M. Lohse, F. Müller, W. Schmitz, 2008 .

Role of protein phosphatases in regulation of cardiac inotropy and relaxation.

S. Huke, F. Müller, W. Schmitz, 2001, American journal of physiology. Heart and circulatory physiology.

Purified cardiac cell membranes with high (Na+ + K+) ATPase activity contain significant NADH-vanadate reductase activity

F. L. Crane, E. Erdmann, W. Schmitz, 1979, Nature.

Gene construction, expression and functional testing of an inotropic peptide from the venom of the black scorpion Hottentotta judaicus.

P. Kirchhof, F. Müller, W. Schmitz, 2012, Toxicon : official journal of the International Society on Toxinology.

Role of guanine nucleotide-binding protein in the regulation by adenosine of cardiac potassium conductance and force of contraction. Evaluation with pertussis toxin

M. Böhm, W. Schmitz, H. Scholz, 1986, Naunyn-Schmiedeberg's Archives of Pharmacology.

Pertussis toxin prevents adenosine receptor- and m-cholinoceptor-mediated sinus rate slowing and AV conduction block in the guinea-pig heart

M. Böhm, W. Schmitz, H. Scholz, 2004, Naunyn-Schmiedeberg's Archives of Pharmacology.

Positive inotropic effect of bay K 8644: cAMP-independence and lack of inhibitory effect of adenosine

M. Böhm, W. Schmitz, H. Scholz, 1985, Naunyn-Schmiedeberg's Archives of Pharmacology.

Functional evidence against the existence of β2-adrenoceptors mediating positive inotropic effects in guinea-pig right ventricular myocardium

W. Schmitz, A. Mügge, H. Scholz, 1996, Naunyn-Schmiedeberg's Archives of Pharmacology.

Regional expression of protein phosphatase type 1 and 2A catalytic subunit isoforms in the human heart.

H. Scheld, F. Müller, W. Schmitz, 2000, Journal of molecular and cellular cardiology.

Cantharidin enhances norepinephrine-induced vasoconstriction in an endothelium-dependent fashion.

F. Müller, W. Schmitz, J. Neumann, 2000, The Journal of pharmacology and experimental therapeutics.

Profiling of histone H3 lysine 9 trimethylation levels predicts transcription factor activity and survival in acute myeloid leukemia.

Yipeng Wang, H. Klein, S. Koschmieder, 2010, Blood.

Beta-adrenoceptor stimulation-induced increase in cardiac Gi-protein expression and in carbachol sensitivity.

J. Schulte am Esch, W. Schmitz, T. Eschenhagen, 1991, European heart journal.

Effects of cantharidin on force of contraction and phosphatase activity in nonfailing and failing human hearts

F. Müller, W. Schmitz, J. Neumann, 1996, British journal of pharmacology.

Evidence for isoproterenol-induced phosphorylation of phosphatase inhibitor-1 in the intact heart.

A. Nairn, W. Schmitz, R. Gupta, 1991, Circulation research.

Inositol-1,4,5-trisphosphate and malignant hyperthermia

J. Scholz, W. Schmitz, M. Patten, 1991, The Lancet.

Functional evidence for the existence of adenosine receptors in the human heart.

M. Böhm, W. Schmitz, A. Mügge, 1985, European journal of pharmacology.

Differential regulation of mRNA specific for beta 1- and beta 2-adrenergic receptors in human failing hearts. Evaluation of the absolute cardiac mRNA levels by two independent methods.

W. Schmitz, W. Kübler, T. Eschenhagen, 1996, Journal of molecular and cellular cardiology.

Effects of the beta2-adrenoceptor agonists fenoterol and salbutamol on force of contraction in isolated human ventricular myocardium

W. Schmitz, A. Mügge, H. Scholz, 1985, Klinische Wochenschrift.

Effects on transmembrane action potential, slow inward current and force of contraction in ventricular cardiac muscle of BRL 31660, a new antiarrhythmic drug with class I and class IV activity

W. Schmitz, H. Scholz, R. Brückner, 1985, Naunyn-Schmiedeberg's Archives of Pharmacology.

Urethral relaxation after electrostimulation in the guinea pig is independent of nitric oxide.

L. Hertle, W. Schmitz, B. von Heyden, 1997, The Journal of urology.

Heat shock protein 70 (HSP70) in gastric adaptation to aspirin in Helicobacter pylori infection.

E. Hahn, W. Schmitz, J. Konturek, 2001, Journal of physiology and pharmacology : an official journal of the Polish Physiological Society.

Expression of cardiac calcium regulatory proteins in atrium v ventricle in different species.

F. Müller, W. Schmitz, L. Jones, 1999, Journal of molecular and cellular cardiology.

Enhanced protein phosphorylation in hypertensive hypertrophy.

W. Schmitz, O. Brodde, I. Heinroth-Hoffmann, 2001, Cardiovascular research.

Successful overexpression of wild-type inhibitor-2 of PP1 in cardiovascular cells

F. Müller, W. Schmitz, F. Balzer, 2018, Naunyn-Schmiedeberg's Archives of Pharmacology.

Effects of (−)-Phenylephrine on force of contraction in the presence of cocaine and hydrocortisone in cat papillary muscle

W. Schmitz, A. Mügge, H. Scholz, 1984, Naunyn-Schmiedeberg's Archives of Pharmacology.

Reduced serine–16 and threonine–17 phospholamban phosphorylation in stunning of conscious dogs

W. Schmitz, H. Aken, J. Neumann, 2006, Basic Research in Cardiology.

Interaction of beta-adrenoceptor and adenosine receptor agonists on phosphorylation. Identification of target proteins in mammalian ventricles.

E. Krause, W. Schmitz, R. Gupta, 1995, Journal of molecular and cellular cardiology.

Stimulatory effect of vanadate on cyclic AMP levels in cat papillary muscle.

E. Erdmann, W. Schmitz, H. Scholz, 1980, Biochemical pharmacology.

In vivo isoproterenol treatment leads to downregulation of the mRNA encoding the cAMP response element binding protein in the rat heart.

F. Müller, W. Schmitz, P. Bokník, 1995, Biochemical and biophysical research communications.

Comparison of the Stereoselective Effects of a Thiadiazinone Derivative on Contractile Parameters and Protein Phosphorylation in the Mammalian Ventricle

W. Schmitz, H. Scholz, J. Neumann, 1995, Journal of cardiovascular pharmacology.

Increased sensitivity to α-adrenoceptor stimulation but intact purinergic and muscarinergic effects in prehypertensive cardiac hypertrophy of spontaneously hypertensive rats

M. Böhm, W. Schmitz, H. Scholz, 1986, Naunyn-Schmiedeberg's Archives of Pharmacology.

Minimally invasive closed-chest ultrasound-guided substance delivery into the pericardial space in mice

P. Kirchhof, W. Schmitz, I. Piccini, 2012, Naunyn-Schmiedeberg's Archives of Pharmacology.

Calcium-independent activation of the contractile apparatus in smooth muscle of mouse aorta by protein phosphatase inhibition

W. Schmitz, F. Balzer, J. Neumann, 2002, Naunyn-Schmiedeberg's Archives of Pharmacology.

Effects of the phosphatase inhibitor calyculin A on the phosphorylation of C-protein in mammalian ventricular cardiomyocytes.

E. Krause, W. Schmitz, H. Scholz, 1995, Biochemical pharmacology.

Transcription factor cAMP response element modulator (Crem) restrains Pdgf-dependent proliferation of vascular smooth muscle cells in mice

J. Fischer, F. Müller, W. Schmitz, 2014, Pflügers Archiv - European Journal of Physiology.

Increased responsiveness to stimulation of alpha- but not beta-adrenoceptors in the hereditary cardiomyopathy of the Syrian hamster. Intact adenosine- and cholinoceptor-mediated isoprenaline antagonistic effect.

M. Böhm, W. Schmitz, H. Scholz, 1986, European journal of pharmacology.

Alpha-1 adrenoceptor-mediated positive inotropic effect and inositol trisphosphate increase in mammalian heart.

J. Scholz, M. Steinfath, M. Lohse, 1988, The Journal of pharmacology and experimental therapeutics.

Adenosine inhibits the positive inotropic effect of 3‐isobutyl‐1‐methylxanthine in papillary muscles without effect on cyclic AMP or cyclic GMP

M. Böhm, W. Schmitz, H. Scholz, 1988, British journal of pharmacology.

Isolated hearts of Delta KPQ SCN5A mutated (Long-QT3) mice exhibit bradycardia, early afterdepolarizations, and torsade de pointes arrhythmias which are suppressed by pacing or mexiletine

G. Breithardt, P. Kirchhof, P. Carmeliet, 2002 .

Negative inotropic effects of aldosterone antagonists in isolated human and guinea-pig ventricular heart muscle

W. Schmitz, A. Mügge, H. Scholz, 1984, Klinische Wochenschrift.

Activity of cAMP-dependent protein kinase and Ca2+/calmodulin-dependent protein kinase in failing and nonfailing human hearts.

W. Schmitz, H. Scholz, J. Neumann, 1999, Cardiovascular research.

Endogenous Diadenosine Tetraphosphate, Diadenosine Pentaphosphate, and Diadenosine Hexaphosphate in Human Myocardial Tissue

H. Schlüter, J. Jankowski, W. Schmitz, 2004, Hypertension.

Negative chronotropic and inotropic effects exerted by diadenosine hexaphosphate (AP6A) via A1‐adenosine receptors

H. Scheld, H. Schlüter, F. Müller, 1996, British journal of pharmacology.

Biochemical and electrophysiological mechanisms of the positive inotropic effect of calyculin A, a protein phosphatase inhibitor.

W. Schmitz, K. Wiechen, H. Scholz, 1994, The Journal of pharmacology and experimental therapeutics.

Vanadate and its significance in biochemistry and pharmacology.

E. Erdmann, K. Werdan, W. Schmitz, 1984, Biochemical pharmacology.

Quantitation of clobazam in human plasma using high-performance liquid chromatography.

F. Müller, W. Schmitz, J. Neumann, 1999, Journal of chromatographic science.

A positive inotropic effect of adenosine in cardiac preparations of right atria from diseased human hearts

W. Schmitz, R. Silber, A. Simm, 2009, Naunyn-Schmiedeberg's Archives of Pharmacology.

Effect of vanadium in the +5, +4 and +3 oxidation states on cardiac force of contraction, adenylate cyclase and (Na+ + K+)-ATPase activity.

E. Erdmann, K. Werdan, W. Schmitz, 1982, Biochemical pharmacology.

A positive inotropic effect of ATP in the human cardiac atrium.

W. Schmitz, R. Silber, A. Simm, 2008, American journal of physiology. Heart and circulatory physiology.

Cardiac Hypertrophy and Impaired Relaxation in Transgenic Mice Overexpressing Triadin 1*

W. Schmitz, H. Baba, Yvonne M. Kobayashi, 2001, The Journal of Biological Chemistry.

Pertussis toxin unmasks stimulatory myocardial A2-adenosine receptors on ventricular cardiomyocytes.

W. Schmitz, H. Scholz, J. Neumann, 1993, Journal of molecular and cellular cardiology.

Cardiac effects of adenosine and adenosine analogs in guinea-pig atrial and ventricular preparations: evidence against a role of cyclic AMP and cyclic GMP.

W. Schmitz, H. Scholz, W. Meyer, 1985, The Journal of pharmacology and experimental therapeutics.

The Inhibitory Effect of Bupivacaine on Prostaglandin E2 (EP1) Receptor Functioning: Mechanism of Action

W. Schmitz, H. Van Aken, K. Hahnenkamp, 2001, Anesthesia and analgesia.

F. Müller, W. Schmitz, M. Hasselblatt, 2007, Acta histochemica.

Inotropic effects of diadenosine tetraphosphate in isolated canine cardiac preparations.

H. Schlüter, F. Müller, W. Schmitz, 1999, Journal of cardiovascular pharmacology.

Enhanced Cardiomyocyte NLRP3 Inflammasome Signaling Promotes Atrial Fibrillation

Corey L. Reynolds, S. Lemaire, S. Nattel, 2018, Circulation.

Calmodulin kinase II-mediated sarcoplasmic reticulum Ca2+ leak promotes atrial fibrillation in mice.

U. Schotten, X. Wehrens, D. Dobrev, 2009, The Journal of clinical investigation.

receptors(RyR2s)ishypothesizedtocontributetoarrhythmogenesisinAF,butthemolecularmechanisms areincompletelyunderstood.Here,wehaveshownthatmicewithageneticgain-of-functiondefectinRyr2 (whichwetermedRyr2 R176Q/+ mice)didnotexhibitspontaneousAFbutthatrapidatrialpacingunmasked anincreasedvulnerabilitytoA

U. Schotten, X. Wehrens, D. Dobrev, 2009 .

Distinct contractile and molecular differences between two goat models of atrial dysfunction: AV block-induced atrial dilatation and atrial fibrillation.

M. Allessie, U. Schotten, S. Nattel, 2009, Journal of molecular and cellular cardiology.

Atrial Fibrillation Is Associated With Increased Activity of

U Ravens, E Wettwer, D. Dobrev, 2022 .

Overexpression of cAMP-response element modulator causes abnormal growth and development of the atrial myocardium resulting in a substrate for sustained atrial fibrillation in mice.

P. Kirchhof, U. Schotten, É. Marijon, 2013, International journal of cardiology.

Genetic inhibition of Nuclear Factor of Activated T-cell c2 (NFATc2) prevents atrial fibrillation in CREM transgenic mice.

X. Wehrens, D. Dobrev, F. Müller, 2021, Cardiovascular research.

Inhibition of protein phosphatase 1 by inhibitor-2 exacerbates progression of cardiac failure in a model with pressure overload.

F. Müller, W. Schmitz, H. Baba, 2008, Cardiovascular research.

Postnatal changes in contractile time parameters, calcium regulatory proteins, and phosphatases.

W. Schmitz, T. Muller, G. Bodor, 1998, The American journal of physiology.

Radioimmunochemical quantification of Gi alpha in right and left ventricles from patients with ischaemic and dilated cardiomyopathy and predominant left ventricular failure.

M. Steinfath, M. Böhm, P. Schnabel, 1994, Journal of molecular and cellular cardiology.

Nitric oxide synthase in muscular dystrophies: a re-evaluation

M. Wellner, F. Müller, W. Schmitz, 2006, Acta Neuropathologica.

An in situ evidence for autocrine function of NO in the vasculature.

M. Wellner, H. Haller, W. Schmitz, 2004, Nitric oxide : biology and chemistry.

Expression of constitutive nitric oxide synthase in rat and human gastrointestinal tract.

J. Stachura, W. Schmitz, J. Konturek, 1999, Biochimica et biophysica acta.

Protein phosphatase 2A affects myofilament contractility in non-failing but not in failing human myocardium

C. D. dos Remedios, F. Müller, W. Schmitz, 2011, Journal of Muscle Research and Cell Motility.

Enrichment of G protein α-subunit mRNAs in the AV-conducting system of the mammalian heart

U. Laufs, W. Schmitz, J. Weil, 1995 .

Reduced myocardial sarcoplasmic reticulum Ca(2+)-ATPase protein expression in compensated primary and secondary human cardiac hypertrophy.

U. Schotten, W. Schmitz, P. Hanrath, 1999, Journal of molecular and cellular cardiology.

Ischemic Preconditioning by Unstable Angina Reduces the Release of CK‐MB Following CABG and Stimulates Left Ventricular HSP‐72 Protein Expression

G. Breithardt, H. Scheld, W. Schmitz, 2005, Journal of cardiac surgery.

Adrenergic and muscarinic receptor regulation and therapeutic implications in heart failure.

F. Müller, W. Schmitz, P. Bokník, 1996 .

Inhibition of the effects of adenosine on force of contraction and the slow calcium inward current by pertussis toxin is associated with myocardial lesions.

M. Böhm, W. Schmitz, H. Scholz, 1988, Cardiovascular research.

Pertussis toxin sensitive and insensitive effects of adenosine and carbachol in murine atria overexpressing A1‐adenosine receptors

W. Schmitz, G. Matherne, J. Neumann, 2003, British journal of pharmacology.

Inotropic actions of adenosine derivatives in the mammalian heart

W. Schmitz, H. Scholz, J. Neumann, 1993 .

Functional properties of the rat phosphatase 1α promoter

F. Müller, W. Schmitz, K. Kikuchi, 2001, Molecular and Cellular Biochemistry.

Adrenergic and muscarinic receptor regulation and therapeutic implications in heart failure

F. Müller, W. Schmitz, P. Bokník, 2004, Molecular and Cellular Biochemistry.

Protein phosphatase activity is increased in a rat model of long-term β-adrenergic stimulation

F. Müller, W. Schmitz, S. Herzig, 2000, Naunyn-Schmiedeberg's Archives of Pharmacology.

Dissociation of the effects of forskolin and dibutyryl cAMP on force of contraction and phospholamban phosphorylation in human heart failure.

P. Karczewski, E. Krause, W. Schmitz, 1999, Journal of cardiovascular pharmacology.

Molecular aspects of adrenergic signal transduction in cardiac failure

M. Böhm, R. Hajjar, P. Schnabel, 1998, Journal of Molecular Medicine.

Differential antagonism by 1,3-dipropylxanthine-8-cyclopentylxanthine and 9-chloro-2-(2-furanyl)-5,6-dihydro-1,2,4-triazolo(1,5-c)quinazolin-5-im ine of the effects of adenosine derivatives in the presence of isoprenaline on contractile response and cyclic AMP content in cardiomyocytes. Evidence for

W. Schmitz, H. Scholz, J. Neumann, 1990, Journal of Pharmacology and Experimental Therapeutics.

Positive inotropic effect of carbachol and inositol phosphate levels in mammalian atria after pretreatment with pertussis toxin.

W. Schmitz, H. Scholz, C. Kohl, 1990, The Journal of pharmacology and experimental therapeutics.

Human 5-HT4 receptor stimulation in atria of transgenic mice

S. Hauptmann, A. Böckler, W. Schmitz, 2013, Naunyn-Schmiedeberg's Archives of Pharmacology.

Gene dose-dependent atrial arrhythmias, heart block, and brady-cardiomyopathy in mice overexpressing A(3) adenosine receptors.

G. Breithardt, P. Kirchhof, W. Schmitz, 2004, Cardiovascular research.

Regional expression of phospholamban in the human heart.

C. Unkel, F. Müller, W. Schmitz, 1999, Cardiovascular research.

Effects of adenosine receptor and muscarinic cholinergic receptor agonists on cardiac protein phosphorylation. Influence of pertussis toxin.

W. Schmitz, L. Jones, G. Bodor, 1994, Journal of Pharmacology and Experimental Therapeutics.

Adenosine inhibition of catecholamine-induced increase in force of contraction in guinea-pig atrial and ventricular heart preparations. Evidence against a cyclic AMP- and cyclic GMP-dependent effect.

B. Haubitz, M. Böhm, W. Schmitz, 1984, The Journal of pharmacology and experimental therapeutics.

Evidence for adenosine receptor-mediated isoprenaline-antagonistic effects of the adenosine analogs PIA and NECA on force of contraction in guinea-pig atrial and ventricular cardiac preparations

M. Böhm, W. Schmitz, H. Scholz, 1985, Naunyn-Schmiedeberg's Archives of Pharmacology.

Effects of PIA and NECA on contractile behaviour and cAMP content in isolated cardiomyocytes in the presence of the A1-adenosine receptor antagonist DPCPX☆

W. Schmitz, H. Scholz, J. Neumann, 1989 .

Phenotyping of Mice with Heart Specific Overexpression of A2A-Adenosine Receptors: Evidence for Cardioprotective Effects of A2A-Adenosine Receptors

P. Kirchhof, F. Müller, W. Schmitz, 2018, Front. Pharmacol..

Evidence for physiological functions of protein phosphatases in the heart: evaluation with okadaic acid.

W. Schmitz, R. Gupta, H. Scholz, 1993, The American journal of physiology.

The effect of the protein phosphatases inhibitor cantharidin on β‐adrenoceptor‐mediated vasorelaxation

F. Müller, W. Schmitz, P. Neumann, 1997, British journal of pharmacology.

Increased expression of cardiac phosphatases in patients with end-stage heart failure.

W. Schmitz, T. Eschenhagen, L. Jones, 1997, Journal of molecular and cellular cardiology.

In Vivo β-Adrenergic Stimulation Leads to Biphasic Regulation of Giα-2 Gene Transcriptional Activity in Rat Heart

F. Müller, W. Schmitz, T. Eschenhagen, 1994 .

Effects of carbachol and (−)‐N6‐phenylisopropyladenosine on myocardial inositol phosphate content and force of contraction

J. Scholz, W. Schmitz, H. Scholz, 1990, British journal of pharmacology.

Effects ofcantharidin on force ofcontraction andphosphatase activity innonfailing andfailing humanhearts

W. Schmitz, J. Neumann, P. Bokník, 1996 .

Overexpression of the Catalytic Subunit of Protein Phosphatase 2A Impairs Cardiac Function*

Ulrich Gergs, Larissa Fabritz, W. Schmitz, 2004, Journal of Biological Chemistry.

Modulation of cardiac contractility by serine/threonine protein phosphatase type 5.

W. Schmitz, D. Kučerová, I. Buchwalow, 2012, International journal of cardiology.

Positive inotropic effects of the calcium sensitizer CGP 48506 in guinea pig myocardium.

W. Schmitz, E. Gams, H. Scholz, 1996, The Journal of pharmacology and experimental therapeutics.

Positive inotropic effects of the calcium sensitizer CGP 48506 in failing human myocardium.

I. Grupp, W. Schmitz, A. Haverich, 1996, The Journal of pharmacology and experimental therapeutics.

Proarrhythmia in a non-failing murine model of cardiac-specific Na+/Ca2+ exchanger overexpression: whole heart and cellular mechanisms

G. Breithardt, P. Kirchhof, P. Milberg, 2012, Basic Research in Cardiology.

Stress and high heart rate provoke ventricular tachycardia in mice expressing triadin.

G. Breithardt, P. Kirchhof, M. Schäfers, 2007, Journal of molecular and cellular cardiology.

Autonomic Dysfunction in Patients with Brugada Syndrome: Further Biochemical Evidence of Altered Signaling Pathways

Ulrich Gergs, Günter Breithardt, Matthias Paul, 2011, Pacing and clinical electrophysiology : PACE.

Altered signal transduction in cardiac ventricle overexpressing A(1)-adenosine receptors.

W. Schmitz, G. Matherne, J. Neumann, 2003, Cardiovascular research.

Mechanisms of the contractile effects of levosimendan in the mammalian heart.

W. Schmitz, H. Scholz, J. Neumann, 1997, The Journal of pharmacology and experimental therapeutics.

University of Birmingham Phenotyping of mice with heart specific overexpression of A2A-adenosine receptors

P. Kirchhof, F. Müller, W. Schmitz, 2018 .

Isoprenaline-like effects of the phosphodiesterase inhibitor 3-isobutyl-1-methylxanthine on mechanical, biochemical and electrophysiological parameters in the mammalian heart

W. Schmitz, H. Scholz, M. Nose, 1985, Experientia.

Effects of 1,3-dipropyl-8-cyclopentylxanthine (DPCPX), a highly selective adenosine receptor antagonist, on force of contraction in guinea-pig atrial and ventricular cardiac preparations

J. Scholz, M. Lohse, W. Schmitz, 1989, Naunyn-Schmiedeberg's Archives of Pharmacology.

Adenosine and adenosine analogs inhibit phosphodiesterase activity in the heart

W. Schmitz, H. Scholz, M. Nose, 1984, Naunyn-Schmiedeberg's Archives of Pharmacology.

Increase in IP3 precedes alpha-adrenoceptor-induced increase in force of contraction in cardiac muscle.

J. Scholz, M. Steinfath, W. Schmitz, 1987, European journal of pharmacology.

ADRENALINE, ADENOSINE, AND FEEDBACK

M. Böhm, W. Schmitz, H. Scholz, 1985, The Lancet.

Biochemical mechanisms of hibernation and stunning in the human heart.

G. Breithardt, H. Scheld, O. Schober, 2002, Cardiovascular research.

The human CASQ2 mutation K206N is associated with hyperglycosylation and altered cellular calcium handling.

E. Schulze-Bahr, A. Wilde, F. Müller, 2010, Journal of molecular and cellular cardiology.

Cardiac expression of the CREM repressor isoform CREM-IbΔC-X in mice leads to arrhythmogenic alterations in ventricular cardiomyocytes

X. Wehrens, F. Müller, W. Schmitz, 2016, Basic Research in Cardiology.

Critical Role of Transcription Factor Cyclic AMP Response Element Modulator in &bgr;1-Adrenoceptor–Mediated Cardiac Dysfunction

M. Lohse, F. Müller, W. Schmitz, 2009, Circulation.

Existence and alpha 1-adrenergic stimulation of inositol polyphosphates in mammalian heart.

J. Scholz, J. Schulte am Esch, W. Schmitz, 1992, Molecular pharmacology.

Ca(++)-dependent constitutive nitric oxide synthase is not involved in the cyclic GMP-increasing effects of carbachol in ventricular cardiomyocytes.

W. Schmitz, A. Mülsch, H. Scholz, 1993, The Journal of pharmacology and experimental therapeutics.

A novel intronic promoter of the Crem gene induces small ICER (smICER) isoforms

K. Schulze-Osthoff, F. Müller, W. Schmitz, 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Protein phosphorylation in isolated trabeculae from nonfailing and failing human hearts

P. Karczewski, E. Krause, W. Schmitz, 2004, Molecular and Cellular Biochemistry.

Stimulatory effect of vanadate on (Na+ + K+)-ATPase activity and on 3H-ouabain-binding in a cat heart cell membrane preparation

E. Erdmann, W. Schmitz, H. Scholz, 1979, Nature.

Positive inotropism of vanadate in cat papillary muscle

E. Erdmann, W. Schmitz, H. Scholz, 1978, Nature.

Long-term beta adrenoceptor-mediated alteration in contractility and expression of phospholamban and sarcoplasmic reticulum Ca(++)-ATPase in mammalian ventricle.

F. Müller, W. Schmitz, H. Baba, 1998, The Journal of pharmacology and experimental therapeutics.

Overexpression of junctin causes adaptive changes in cardiac myocyte Ca(2+) signaling.

Peter Lipp, Lars Kaestner, P. Lipp, 2006, Cell calcium.

High non-specific binding of the β1-selective radioligand 2-125I-ICI-H

O. Schober, S. Luthra, V. Pike, 2003, Nuklearmedizin.

Pertussis toxin does not inhibit the alpha 1-adrenoceptor-mediated effect on inositol phosphate production in the heart.

J. Scholz, M. Steinfath, M. Lohse, 1987, European journal of pharmacology.

Myocardial Phosphodiesterase Activity in Different Mammalian Species

W. Schmitz, E. Kruse, 1978 .

Does an impaired adenosine mediated feedback control play a role in the development of hereditary dystrophic cardiomyopathy?

M. Böhm, W. Schmitz, H. Scholz, 1986, Cardiovascular research.

Functional properties of transgenic mouse hearts overexpressing both calsequestrin and the Na(+)-Ca(2+) exchanger.

S. Huke, F. Müller, W. Schmitz, 2000, The Journal of pharmacology and experimental therapeutics.

Cardiac ai-adrenoceptor densities in different mammalian species

W. Schmitz, H. Scholz, M. Nose, 2006 .

Effects of Isomazole on Force of Contraction and Phosphodiesterase Isoenzymes I‐IV in Nonfailing and Failing Human Hearts

W. Schmitz, A. Haverich, H. E. von der Leyen, 1991, Journal of cardiovascular pharmacology.

Cardiac alpha 1-adrenoceptor densities in different mammalian species.

Y. Y. Chen, M. Steinfath, W. Schmitz, 1992, British Journal of Pharmacology.

Stimulatory (insulin-mimetic) and inhibitory (ouabain-like) action of vanadate on potassium uptake and cellular sodium and potassium in heart cells in culture.

E. Erdmann, K. Werdan, W. Schmitz, 1982, Biochimica et biophysica acta.

Chronic treatment with carbachol sensitizes the myocardium to cAMP-induced arrhythmia.

M. Steinfath, M. Böhm, W. Schmitz, 1996, Circulation.

Annexin A4 is a novel direct regulator of adenylyl cyclase type 5

F. Müller, W. Schmitz, V. Gerke, 2015, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Negative inotropic effect of vanadate in ventricular myocardium in the presence of 3-isobutyl-1-methylxanthine or isoprenaline

E. Erdmann, K. Werdan, W. Schmitz, 2004, Naunyn-Schmiedeberg's Archives of Pharmacology.

On the cardiac contractile, biochemical and electrophysiological effects of cantharidin, a phosphatase inhibitor.

W. Schmitz, M. Apel, M. Tepel, 1995, The Journal of pharmacology and experimental therapeutics.

Inositol-1,4,5-trisphosphate increase by diadenosine tetraphosphate in preparations from failing human myocardium

H. Schlüter, F. Müller, W. Schmitz, 1999, Naunyn-Schmiedeberg's Archives of Pharmacology.

Reduced alpha 1- and beta 2-adrenoceptor-mediated positive inotropic effects in human end-stage heart failure.

M. Steinfath, W. Schmitz, H. E. von der Leyen, 1992, British journal of pharmacology.

Evidence against a role of (Na++K+-ATPase in the alpha-adrenoceptor mediated positive inotropic effect of phenylephrine

E. Erdmann, K. Werdan, W. Schmitz, 1980, Experientia.

On the role of junctin in cardiac Ca2+ handling, contractility, and heart failure.

F. Müller, W. Schmitz, K. Schlüter, 2007, American journal of physiology. Heart and circulatory physiology.

Messenger RNA expression and immunological quantification of phospholamban and SR-Ca(2+)-ATPase in failing and nonfailing human hearts.

F. Müller, W. Schmitz, T. Eschenhagen, 1996, Cardiovascular research.

Triadin is a critical determinant of cellular Ca cycling and contractility in the heart.

F. Müller, W. Schmitz, H. Baba, 2007, American journal of physiology. Heart and circulatory physiology.

Altered function in atrium of transgenic mice overexpressing triadin 1.

W. Schmitz, H. Baba, Yvonne M. Kobayashi, 2002, American journal of physiology. Heart and circulatory physiology.

Junctional sarcoplasmic reticulum transmembrane proteins in the heart

F. Müller, W. Schmitz, J. Neumann, 2002, Basic Research in Cardiology.

Importance of cardiac cell membranes in vanadate-induced NADH oxidation

E. Erdmann, W. Schmitz, H. Scholz, 1981, Nature.

Regulation of the human Gi alpha-2 gene promotor activity in embryonic chicken cardiomyocytes.

L. Weinstein, W. Schmitz, J. Weil, 1996, Basic Research in Cardiology.

On the mechanism of positive inotropic effects of alpha-adrenoceptor agonists

J. Scholz, W. Schmitz, H. Scholz, 2007, Basic Research in Cardiology.

Cardiac alpha-receptors and cardiac hypertrophy in genetic predisposition to hypertension.

M. Böhm, W. Schmitz, H. Scholz, 1986, American heart journal.

Increased sensitivity to alpha-adrenoceptor stimulation but intact purinergic and muscarinergic effects in prehypertensive cardiac hypertrophy of spontaneously hypertensive rats.

M. Böhm, W. Schmitz, H. Scholz, 1986, Naunyn-Schmiedeberg's archives of pharmacology.

High Selectivity for Inhibition of Phosphodiesterase III and Positive Inotropic Effects of MCI‐154 in Guinea Pig Myocardium

W. Schmitz, H. Scholz, B. Armah, 1993, Journal of cardiovascular pharmacology.

Possible involvement of inositol-lipid metabolism in malignant hyperthermia.

J. Scholz, J. Schulte am Esch, W. Schmitz, 1991, British Journal of Anaesthesia.

Effects of Beta‐ and Alpha‐adrenergic Agonists, Adenosine, and Carbachol in Heart Muscle Isolated from Malignant Hyperthermia Susceptible Swine

M. Böhm, W. Schmitz, N. Roewer, 1988, Anesthesiology.

New positive inotropic agents acting by phosphodiesterase inhibition or alpha 1-adrenergic stimulation.

J. Scholz, W. Schmitz, H. E. von der Leyen, 1989, Pharmacological research.

Knockout of ATF1 leads to enhanced cardiac contractility and output

F. Müller, W. Schmitz, G. Schütz, 2008 .

First Report on an Inotropic Peptide Activating Tetrodotoxin-Sensitive, “Neuronal” Sodium Currents in the Heart

P. Kirchhof, E. Schulze-Bahr, T. Budde, 2015, Circulation. Heart failure.

Sarcoplasmic reticulum Ca2+ release in neonatal rat cardiac myocytes.

W. Schmitz, I. Buchwalow, L. Jones, 2011, Journal of molecular and cellular cardiology.

Successful overexpression of wild-type inhibitor-2 of PP1 in cardiovascular cells

F. Müller, W. Schmitz, F. Balzer, 2018, Naunyn-Schmiedeberg's Archives of Pharmacology.

Evidence for alpha 1-adrenoceptor-mediated increase of inositol trisphosphate in the human heart.

J. Scholz, W. Schmitz, P. Kalmar, 1989, Journal of cardiovascular pharmacology.

Inotropic response in CHF: myocarditis vs dilated cardiomyopathy.

W. Schmitz, J. Scholz, P. Kalmar, 1988, American Heart Journal.

Enrichment of G protein alpha-subunit mRNAS in the AV-conducting system of the mammalian heart.

U. Laufs, W. Schmitz, J. Weil, 1995, Journal of molecular and cellular cardiology.