A. Mahboubi

发表

DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1.

D. Green, S. Kasibhatla, T. Brunner, 1998, Molecular cell.

Regulation of joint destruction and inflammation by p53 in collagen-induced arthritis.

D. Green, G. Firestein, M. Pinkoski, 2002, The American journal of pathology.

MYC, FAS, Apoptosis, and Immune Tolerance

D. Green, A. Mahboubi, Yufang Shi, 1994 .

Regulation of the Fas Apoptotic Cell Death Pathway by Abl (*)

Seamus J. Martin, D. Green, T. Cotter, 1995, The Journal of Biological Chemistry.

Promotion and Inhibition of Activation‐Induced Apoptosis in T‐Cell Hybridomas by Oncogenes and Related Signals

D. Green, Yili Yang, J. Ashwell, 1994, Immunological reviews.

HIV-1 Vpr suppresses immune activation and apoptosis through regulation of nuclear factor kappa B.

D. Green, D. Weiner, S. Kudchodkar, 1997, Nature medicine.

HIV-1 Vpr suppresses immune activation and apoptosis through regulation of nuclear factor κB

D. Green, D. Weiner, S. Kudchodkar, 1997, Nature Medicine.

Protein kinase ctheta cooperates with calcineurin to induce Fas ligand expression during activation-induced T cell death.

D. Green, A. Altman, S. Kasibhatla, 1999, Journal of immunology.

Expression of Fas Ligand in Activated T Cells Is Regulated by c-Myc*

D. Green, N. Yoo, S. Kasibhatla, 2000, The Journal of Biological Chemistry.

During Activation-Induced T Cell Death Calcineurin to Induce Fas Ligand Expression Cooperates with q Protein Kinase C

D. Green, A. Altman, S. Kasibhatla, 1999 .

Probing the opioid receptor complex with (+)-trans-SUPERFIT. II. Evidence that μ ligands are noncompetitive inhibitors of the δ cx opioid peptide binding site

A. Mahboubi, K. Rice, R. Rothman, 1992, Peptides.

Probing the opioid receptor complex with (+)-trans-superfit. I. Evidence that [D-Pen2,D-Pen5]enkephalin interacts with high affinity at the δ cx binding site

A. Mahboubi, K. Rice, R. Rothman, 1991, Peptides.

Heat-shock protein 70 inhibits apoptosis by preventing recruitment of procaspase-9 to the Apaf-1 apoptosome

Dick D. Mosser, Richard I. Morimoto, Pankaj Tailor, 2000, Nature Cell Biology.

The end of the (cell) line: methods for the study of apoptosis in vitro.

D. Green, S. Martin, A. Mahboubi, 1995, Methods in cell biology.

Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas

Seamus J. Martin, D. Green, C. Ware, 1995, Nature.

Apoptotic cell death induced by c-myc is inhibited by bcl-2

D. Green, F. Echeverri, A. Mahboubi, 1992, Nature.

Interaction of β‐funaltrexamine with [3H]cycloFOXY binding in rat brain: Further evidence that β‐FNA alkylates the opioid receptor complex

J. Holaday, J. Long, A. Mahboubi, 1991 .

Functional Myc-Max heterodimer is required for activation-induced apoptosis in T cell hybridomas

D. Green, A. Mahboubi, A. McGahon, 1994, The Journal of experimental medicine.

Proteolysis of Fodrin (Non-erythroid Spectrin) during Apoptosis (*)

Seamus J. Martin, D. Green, A. Mahboubi, 1995, The Journal of Biological Chemistry.

FAS-induced apoptosis is mediated via a ceramide-initiated RAS signaling pathway.

D. Green, F. Lang, R. Kolesnick, 1995, Immunity.

Labeling by [3H]1,3-di(2-tolyl)guanidine of two high affinity binding sites in guinea pig brain: evidence for allosteric regulation by calcium channel antagonists and pseudoallosteric modulation by sigma ligands.

A. Mahboubi, K. Rice, R. Rothman, 1991, Molecular pharmacology.

Interaction of beta-funaltrexamine with [3H]cycloFOXY binding in rat brain: further evidence that beta-FNA alkylates the opioid receptor complex.

J. Holaday, J. Long, A. Mahboubi, 1991, Synapse.

Probes for narcotic receptor mediated phenomena. 18. Epimeric 6 alpha- and 6 beta-iodo-3,14-dihydroxy-17-(cyclopropylmethyl)-4,5 alpha-epoxymorphinans as potential ligands for opioid receptor single photon emission computed tomography: synthesis, evaluation, and radiochemistry of [125I]-6 beta-iodo-

K. Berman, C. George, M. Iadarola, 1992, Journal of medicinal chemistry.