D. Bates

发表

Vinblastine sensitizes leukemia cells to cyclin-dependent kinase inhibitors, inducing acute cell cycle phase-independent apoptosis

A. Eastman, D. Bates, B. Salerni, 2011, Cancer biology & therapy.

Confounding off-target effects of BH3 mimetics at commonly used concentrations: MIM1, UMI-77, and A-1210477

A. Eastman, D. Bates, R. Soderquist, 2019, Cell Death & Disease.

Manipulating the apoptotic pathway: potential therapeutics for cancer patients.

D. Bates, L. Lewis, Darcy J. P. Bates, 2013, British journal of clinical pharmacology.

Multiple BH3 Mimetics Antagonize Antiapoptotic MCL1 Protein by Inducing the Endoplasmic Reticulum Stress Response and Up-regulating BH3-only Protein NOXA*

A. Eastman, D. Bates, B. Salerni, 2011, The Journal of Biological Chemistry.

Vinblastine Induces Acute, Cell Cycle Phase–Independent Apoptosis in Some Leukemias and Lymphomas and Can Induce Acute Apoptosis in Others when Mcl-1 Is Suppressed

A. Eastman, D. Bates, B. Salerni, 2010, Molecular Cancer Therapeutics.

Gossypol overcomes stroma-mediated resistance to the BCL2 inhibitor ABT-737 in chronic lymphocytic leukemia cells ex vivo

A. Eastman, D. Bates, A. Danilov, 2013, Leukemia.

Vinblastine Rapidly Induces NOXA and Acutely Sensitizes Primary Chronic Lymphocytic Leukemia Cells to ABT-737

A. Eastman, D. Bates, C. Lowrey, 2013, Molecular Cancer Therapeutics.

Vincristine activates c-Jun N-terminal kinase in chronic lymphocytic leukaemia in vivo.

A. Eastman, D. Bates, L. Lewis, 2015, British journal of clinical pharmacology.

Toward a cure for chronic lymphocytic leukemia: an attack on multiple fronts

A. Eastman, D. Bates, A. Danilov, 2013, Expert review of anticancer therapy.

Microtubule destabilising agents: far more than just antimitotic anticancer drugs

A. Eastman, D. Bates, Darcy J. P. Bates, 2017, British journal of clinical pharmacology.

Rapid induction of apoptosis in chronic lymphocytic leukemia cells by the microtubule disrupting agent BNC105

A. Eastman, D. Bates, A. Danilov, 2016, Cancer biology & therapy.

Strategically Timing Inhibition of Phosphatidylinositol 3-Kinase to Maximize Therapeutic Index in Estrogen Receptor Alpha–Positive, PIK3CA-Mutant Breast Cancer

A. Eastman, D. Bates, E. Demidenko, 2016, Clinical Cancer Research.

Therapeutic sensitivity to Rac GTPase inhibition requires consequential suppression of mTORC1, AKT, and MEK signaling in breast cancer

D. Bates, E. Demidenko, K. Shee, 2017, Oncotarget.

Nitroalkene fatty acids mediate activation of Nrf2/ARE-dependent and PPARγ-dependent transcription by distinct signaling pathways and with significantly different potencies.

A. Townsend, D. Bates, P. K. Smitherman, 2011, Biochemistry.

Noncatalytic interactions between glutathione S-transferases and nitroalkene fatty acids modulate nitroalkene-mediated activation of peroxisomal proliferator-activated receptor gamma.

A. Townsend, D. Bates, C. Morrow, 2009, Biochemistry.

Modulation of nitrated lipid signaling by multidrug resistance protein 1 (MRP1): glutathione conjugation and MRP1-mediated efflux inhibit nitrolinoleic acid-induced, PPARgamma-dependent transcription activation.

D. Bates, C. Morrow, M. Wright, 2006, Biochemistry.

Confounding off-target effects of BH3 mimetics at commonly used concentrations: MIM1, UMI-77, and A-1210477

A. Eastman, D. Bates, R. Soderquist, 2019, Cell Death & Disease.

Abstract 2993: Vinblastine induces very rapid apoptosis in chronic lymphocytic leukemia and mantle cell lymphoma

A. Eastman, D. Bates, C. Lowrey, 2010 .