G. Burgess

发表

Transformation of interleukin-3-dependent cells without participation of Stat5/bcl-xL: cooperation of akt with raf/erk leads to p65 nuclear factor kappaB-mediated antiapoptosis involving c-IAP2.

H. Nakshatri, A. King, V. Gelfanov, 2001, Blood.

Dissociation of nuclear events on p21 RAS transformation of FDC-P1 myeloid cells: c-jun/AP-1 expression versus c-myc transcription.

A. Miller, G. Burgess, S. Litz-Jackson, 1992, Blood.

Forced over-expression of the myeloid zinc finger gene MZF-1 inhibits apoptosis and promotes oncogenesis in interleukin-3-dependent FDCP.1 cells.

K. Cornetta, R. Hromas, G. Burgess, 1996, Leukemia.

Role of p21 RAS in p210 bcr-abl transformation of murine myeloid cells.

G. Burgess, H. Boswell, T. Tauchi, 1993, Blood.

A novel mast cell growth factor (MCGF-3) produced by marrow-adherent cells that synergizes with interleukin 3 and interleukin 4.

D. Williams, G. Burgess, H. Boswell, 1990, Experimental hematology.

Role for E2F1 in p210 BCR-ABL downstream regulation of c-myc transcription initiation. Studies in murine myeloid cells.

E. Williamson, G. Burgess, S. Litz-Jackson, 1995, Leukemia.

Cyclic AMP negatively controls c-myc transcription and G1 cell cycle progression in p210 BCR-ABL transformed cells: inhibitory activity exerted through cyclin D1 and cdk4

G. Burgess, S. Litz-Jackson, H. Boswell, 1997, Leukemia.

Mutant H-ras over-expression inhibits a random apoptotic nuclease in myeloid leukemia cells.

R. Hromas, R. Hoffman, G. Burgess, 1993, Leukemia research.

Regulation of the c-jun Gene in p210 BCR-ABL Transformed Cells Corresponds With Activity of JNK, the c-jun N-Terminal Kinase

H. Nakshatri, A. Kraft, E. Williamson, 1998 .

Regulation of the c-jun gene in p210 BCR-ABL transformed cells corresponds with activity of JNK, the c-jun N-terminal kinase.

H. Nakshatri, A. Kraft, E. Williamson, 1998, Blood.

Role for c-jun N-terminal kinase in treatment-refractory acute myeloid leukemia (AML): signaling to multidrug-efflux and hyperproliferation

D. Mercola, G. Burgess, H. Boswell, 2002, Leukemia.

Role for cyclic AMP in the postreceptor control of cytokine-stimulated stromal cell growth factor production.

D. Williams, G. Burgess, H. Boswell, 1990, Leukemia.

A mutant RAS gene acts through protein kinase C to augment interleukin-3 dependent proliferation in a fastidious immortal myeloid cell line.

G. Burgess, H. Boswell, D. English, 1989, Leukemia.

p210 bcr-abl confers overexpression of inosine monophosphate dehydrogenase: an intrinsic pathway to drug resistance mediated by oncogene.

F. Collart, G. Burgess, S. Litz-Jackson, 1994, Leukemia.

A RAS oncogene imparts growth factor independence to myeloid cells that abnormally regulate protein kinase C: a nonautocrine transformation pathway.

G. Burgess, H. Boswell, E. Srour, 1990, Experimental hematology.