A. Domenighetti

发表

Characterization of mechanisms of myocardial remodeling in genetic models of cardiac hypertrophy

A. Domenighetti, 2005 .

S100A1 deficiency results in prolonged ventricular repolarization in response to sympathetic activation.

C. Heizmann, P. Erne, A. Domenighetti, 2008, General physiology and biophysics.

Vinculin directly binds zonula occludens-1 and is essential for stabilizing connexin-43-containing gap junctions in cardiac myocytes

A. Domenighetti, R. Ross, O. Platoshyn, 2014, Journal of Cell Science.

Cullin E3 Ligase Activity Is Required for Myoblast Differentiation.

A. Domenighetti, S. Lange, J. Blondelle, 2017, Journal of molecular biology.

An FHL1-containing complex within the cardiomyocyte sarcomere mediates hypertrophic biomechanical stress responses in mice.

A. McCulloch, Yusu Gu, N. Dalton, 2008, The Journal of clinical investigation.

Neuropeptide Y (NPY) potentiates phenylephrine-induced mitogen-activated protein kinase activation in primary cardiomyocytes via NPY Y5 receptors.

R. Palmiter, A. Domenighetti, T. Pedrazzini, 2000, Proceedings of the National Academy of Sciences of the United States of America.

An intercellular regenerative calcium wave in porcine coronary artery endothelial cells in primary culture

M. Frieden, A. Domenighetti, F. Chabaud, 1998, The Journal of physiology.

Elevated dietary sodium intake exacerbates myocardial hypertrophy associated with cardiac-specific overproduction of angiotensin II

L. Delbridge, A. Domenighetti, C. Huggins, 2004, Journal of the renin-angiotensin-aldosterone system : JRAAS.

Elevated intracardiac angiotensin II leads to cardiac hypertrophy and mechanical dysfunction in normotensive mice

L. Delbridge, A. Domenighetti, C. Huggins, 2003, Journal of the renin-angiotensin-aldosterone system : JRAAS.

Threshold effects of glucose transporter-4 (GLUT4) deficiency on cardiac glucose uptake and development of hypertrophy.

A. Domenighetti, J. Proietto, A. Dunn, 2003, Journal of molecular endocrinology.

TRANSITORY REDUCTION IN ANGIOTENSIN AT2 RECEPTOR EXPRESSION LEVELS IN POSTINFARCT REMODELLING IN RAT MYOCARDIUM

L. Delbridge, A. Domenighetti, C. Curl, 2004, Clinical and experimental pharmacology & physiology.

Building a Professional Identity and an Academic Career Track in Translational Medicine

N. Rosenblum, A. Domenighetti, V. Melotte, 2019, Front. Med..

Loss of myogenic potential and fusion capacity of muscle stem cells isolated from contractured muscle in children with cerebral palsy.

R. Lieber, A. Domenighetti, H. Chambers, 2018, American journal of physiology. Cell physiology.

Commentary: Muscle Microbiopsy to Delineate Stem Cell Involvement in Young Patients: A Novel Approach for Children With Cerebral Palsy

R. Lieber, A. Domenighetti, 2021, Frontiers in Physiology.

Targeted GLUT-4 deficiency in the heart induces cardiomyocyte hypertrophy and impaired contractility linked with Ca(2+) and proton flux dysregulation.

L. Delbridge, A. Domenighetti, C. Curl, 2010, Journal of molecular and cellular cardiology.

Neuropeptide Y ( NPY ) potentiates phenylephrine-induced mitogen-activated protein kinase activation in primary cardiomyocytes via NPY Y 5 receptors

R. Palmiter, A. Domenighetti, T. Pedrazzini, 2000 .

Conference report on contractures in musculoskeletal and neurological conditions

Sabrina S. M. Lee, W. Rymer, Y. Hathout, 2020, Muscle & nerve.

Loss of FHL1 induces an age-dependent skeletal muscle myopathy associated with myofibrillar and intermyofibrillar disorganization in mice.

J. Seidman, R. Lieber, C. Seidman, 2014, Human molecular genetics.

Differential DNA methylation and transcriptional signatures characterize impairment of muscle stem cells in pediatric human muscle contractures after brain injury

R. Lieber, A. Domenighetti, M. Schipma, 2021, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Management of Cardiac Involvement Associated With Neuromuscular Diseases: A Scientific Statement From the American Heart Association.

Paul J. Wang, D. Judge, A. Domenighetti, 2017, Circulation.

Control of the adaptive response of the heart to stress via the Notch1 receptor pathway

A. Domenighetti, M. Nemir, T. Pedrazzini, 2008, The Journal of experimental medicine.

Cardiac Sodium Channel Nav1.5 Is Regulated by a Multiprotein Complex Composed of Syntrophins and Dystrophin

Patrick Ruchat, H. Lehr, A. Domenighetti, 2006, Circulation research.

5'RNA-Seq identifies Fhl1 as a genetic modifier in cardiomyopathy.

J. Seidman, M. Bulyk, P. D. De Jager, 2014, The Journal of clinical investigation.

The titin N2B and N2A regions: biomechanical and metabolic signaling hubs in cross-striated muscles

C. Ottenheijm, E. Ehler, A. Domenighetti, 2021, Biophysical Reviews.

Tropomodulin 1 directly controls thin filament length in both wild-type and tropomodulin 4-deficient skeletal muscle

V. Fowler, A. Domenighetti, J. Ochala, 2015, Development.

Myocardial Gene Expression Associated with Genetic Cardiac Hypertrophy in the Absence of Hypertension

Matthew E. Ritchie, L. Delbridge, G. Smyth, 2008, Hypertension Research.

Skeletal muscle intermediate filaments form a stress-transmitting and stress-signaling network

Sameer B. Shah, R. Lieber, A. Ryan, 2015, Journal of Cell Science.

Potassium Supplementation Reduces Cardiac and Renal Hypertrophy Independent of Blood Pressure in DOCA/Salt Mice

M. Burnier, A. Domenighetti, T. Pedrazzini, 2005, Hypertension.

Impact of salt on cardiac differential gene expression and coronary lesion in normotensive mineralocorticoid-treated mice.

Ju Chen, Qing Wang, Alexandra Simon, 2012, American journal of physiology. Regulatory, integrative and comparative physiology.

Functional and metabolic remodelling in GLUT4-deficient hearts confers hyper-responsiveness to substrate intervention.

G K Smyth, L. Delbridge, G. Smyth, 2008, Journal of molecular and cellular cardiology.

Erratum: Angiotensin II-mediated adaptive and maladaptive remodeling of cardiomyocyte excitation-contraction coupling (Circulation Research (2009) 105 (42-50))

L. Delbridge, M. Egger, E. Niggli, 2009 .

Angiotensin II–Mediated Adaptive and Maladaptive Remodeling of Cardiomyocyte Excitation–Contraction Coupling

L. Delbridge, M. Egger, E. Niggli, 2009, Circulation research.

Angiotensin II–Mediated Phenotypic Cardiomyocyte Remodeling Leads to Age-Dependent Cardiac Dysfunction and Failure

Qing Wang, L. Delbridge, M. Egger, 2005, Hypertension.

Chronic angiotensin II stimulation in the heart produces an acquired long QT syndrome associated with IK1 potassium current downregulation.

A. Domenighetti, T. Pedrazzini, C. Boixel, 2007, Journal of molecular and cellular cardiology.

as a genetic modifier in cardiomyopathy

M. Bulyk, A. Aboukhalil, C. Seidman, 2014 .

Adaptive and maladaptive remodeling of cardiomyocyte excitation-contraction coupling by angiotensin II.

M. Egger, A. Domenighetti, 2010, Trends in cardiovascular medicine.