Lizhi He

发表

Inhibition of ERK activation enhances the repair of double-stranded breaks via non-homologous end joining by increasing DNA-PKcs activation.

L. Tao, F. Wei, Judy Yan, 2013, Biochimica et biophysica acta.

ERK1 and ERK2 kinases activate hydroxyurea-induced S-phase checkpoint in MCF7 cells by mediating ATR activation.

L. Tao, F. Wei, D. Tang, 2011, Cellular signalling.

Specific Reduction of Fas-Associated Protein with Death Domain (FADD) in Clear Cell Renal Cell Carcinoma

D. Tang, Lizhi He, Hui Xu, 2009, Cancer investigation.

Co-existence of high levels of the PTEN protein with enhanced Akt activation in renal cell carcinoma.

D. Tang, Lizhi He, Xin-chan Feng, 2007, Biochimica et biophysica acta.

PTEN inhibits BMI1 function independently of its phosphatase activity

D. Tang, Lizhi He, A. Kapoor, 2009, Molecular Cancer.

Bmi1 promotes prostate tumorigenesis via inhibiting p16(INK4A) and p14(ARF) expression.

D. Tang, Lizhi He, A. Kapoor, 2008, Biochimica et biophysica acta.

SIPL1-facilitated PTEN ubiquitination contributes to its association with PTEN.

F. Wei, D. Tang, Xiaozeng Lin, 2014, Cellular signalling.

Age- and gene-dosage-dependent cre-induced abnormalities in the retinal pigment epithelium.

Lizhi He, A. Marneros, J. Dunaief, 2014, The American journal of pathology.

Prognostic and Therapeutic Potential of the OIP5 Network in Papillary Renal Cell Carcinoma

D. Tang, Xiaozeng Lin, Lizhi He, 2021, Cancers.

Identification of a novel Wee1 isoform.

Jerry H. Wang, D. Tang, Lizhi He, 2005, Biochimica et biophysica acta.

Interaction of p14ARF with Brca1 in cancer cell lines and primary breast cancer

D. Tang, Lizhi He, Xin-chan Feng, 2008, Cell biology international.

ATM activity contributes to the tumor-suppressing functions of p14ARF

Dahai Zhu, D. Tang, Lizhi He, 2004, Oncogene.

Shank-interacting protein-like 1 promotes tumorigenesis via PTEN inhibition in human tumor cells.

D. Tang, Lizhi He, A. Ingram, 2010, The Journal of clinical investigation.

SIPL1 enhances the proliferation, attachment, and migration of CHO cells by inhibiting PTEN function.

Judy Yan, D. Tang, Lizhi He, 2014, International journal of molecular medicine.

Macrophages are essential for the early wound healing response and the formation of a fibrovascular scar.

Lizhi He, A. Marneros, Alexander G. Marneros, 2013, The American journal of pathology.

Properties and Simulating Research of Epoxy Resin/Micron-SiC/Nano-SiO2 Composite

Lizhi He, Junguo Gao, N. Guo, 2022, Energies.

Genome-scale identification of transcription factors that mediate an inflammatory network during breast cellular transformation

Kevin Struhl, Aviv Regev, Asaf Rotem, 2018, Nature Communications.

Doxycycline Inhibits Polarization of Macrophages to the Proangiogenic M2-type and Subsequent Neovascularization*

Lizhi He, A. Marneros, 2014, The Journal of Biological Chemistry.

Doxycycline in macrophage polarization 1 Doxycycline inhibits polarization of macrophages to the proangiogenic M 2-type and subsequent neovascularization *

Lizhi He, A. Marneros, 2014 .

CYB5D2 enhances HeLa cells survival of etoposide-induced cytotoxicity.

L. Tao, F. Wei, D. Tang, 2011, Biochemistry and cell biology = Biochimie et biologie cellulaire.

NFAT restricts osteochondroma formation from entheseal progenitors.

Bin Zhou, Bin Zhou, Lizhi He, 2016, JCI insight.

Bmi1 promotes prostate tumorigenesis via inhibiting p16 and p14 expression

D. Tang, Lizhi He, A. Kapoor, 2020 .

Bmi 1 promotes prostate tumorigenesis via inhibiting p 16 INK 4 A and p 14 ARF expression

D. Tang, Lizhi He, A. Kapoor, 2008 .

LINC00520 is induced by Src, STAT3, and PI3K and plays a functional role in breast cancer

Andrew H. Beck, David G Hendrickson, J. Rinn, 2016, Oncotarget.

Construction of a set of novel and robust gene expression signatures predicting prostate cancer recurrence

F. Wei, D. Tang, Xiaozeng Lin, 2018, Molecular oncology.

ATM activation is accompanied with earlier stages of prostate tumorigenesis.

D. Tang, Lizhi He, Xin-chan Feng, 2006, Biochimica et biophysica acta.

α-Mannosidase 2C1 attenuates PTEN function in prostate cancer cells.

D. Tang, Lizhi He, J. Scholey, 2011, Nature communications.

Global characterization of macrophage polarization mechanisms and identification of M2-type polarization inhibitors

Kai-Yao Huang, Tzong-Yi Lee, N. Slavov, 2021, Cell reports.

FAM84B promotes prostate tumorigenesis through a network alteration

F. Wei, D. Tang, Xiaozeng Lin, 2019, Therapeutic advances in medical oncology.

Common reduction of the Raf kinase inhibitory protein in clear cell renal cell carcinoma

Jason De Melo, Judy Yan, D. Tang, 2014, Oncotarget.

Inflammatory regulatory network mediated by the joint action of NF-kB, STAT3, and AP-1 factors is involved in many human cancers

A. Regev, K. Struhl, Lizhi He, 2019, Proceedings of the National Academy of Sciences.

Regulatory network controlling tumor-promoting inflammation in human cancers

A. Regev, K. Struhl, Lizhi He, 2018, bioRxiv.

The ESCRT protein CHMP5 restrains skeletal progenitor cell senescence by preserving endo-lysosomal-mitochondrial network

Cole M. Haynes, Lizhi He, Haibo Liu, 2020, bioRxiv.

YAP and TAZ are transcriptional co-activators of AP-1 proteins and STAT3 during breast cellular transformation

Z. Weng, K. Struhl, F. Wei, 2021, bioRxiv.

MafB, WDR77, and ß-catenin interact with each other and have similar genome association profiles

Z. Weng, K. Struhl, Lizhi He, 2022, bioRxiv.

IQGAP2, A candidate tumour suppressor of prostate tumorigenesis.

L. Tao, F. Wei, Judy Yan, 2012, Biochimica et biophysica acta.

p14ARF inhibits the growth of p53 deficient cells in a cell-specific manner.

D. Tang, Lizhi He, Yanxia Li, 2006, Biochimica et biophysica acta.

Inflammatory regulatory network mediated by the joint action of NF-kB, STAT3, and AP-1 factors is involved in many human cancers

A. Regev, K. Struhl, Lizhi He, 2019, Proceedings of the National Academy of Sciences.

HSD17B6 delays type 2 diabetes development via inhibiting SREBP activation.

F. Wei, D. Tang, Xiaozeng Lin, 2023, Metabolism: clinical and experimental.