D. Meechan

发表

Diminished dosage of 22q11 genes disrupts neurogenesis and cortical development in a mouse model of 22q11 deletion/DiGeorge syndrome

Anthony-Samuel LaMantia, A. LaMantia, D. Meechan, 2009, Proceedings of the National Academy of Sciences.

Modeling a model: Mouse genetics, 22q11.2 Deletion Syndrome, and disorders of cortical circuit development

A. LaMantia, D. Meechan, E. Tucker, 2015, Progress in Neurobiology.

No evidence for parental imprinting of mouse 22q11 gene orthologs

J. Lieberman, R. Hamer, A. LaMantia, 2006, Mammalian Genome.

When half is not enough: gene expression and dosage in the 22q11 deletion syndrome.

A. LaMantia, D. Meechan, T. Maynard, 2006, Gene expression.

Cognitive ability is associated with altered medial frontal cortical circuits in the LgDel mouse model of 22q11.2DS.

A. LaMantia, D. Meechan, T. Maynard, 2015, Cerebral cortex.

Mitochondrial localization and function of a subset of 22q11 deletion syndrome candidate genes

J. Lieberman, A. LaMantia, D. Meechan, 2008, Molecular and Cellular Neuroscience.

Specific mesenchymal/epithelial induction of olfactory receptor, vomeronasal, and gonadotropin‐releasing hormone (GnRH) neurons

A. LaMantia, C. Dulac, D. Meechan, 2010, Developmental dynamics : an official publication of the American Association of Anatomists.

Single nucleotide polymorphisms in the leptin receptor gene: studies in anorexia nervosa

A. Blakemore, D. Meechan, H. Eastwood, 2004, Psychiatric genetics.

Cortical Interneurons Require Jnk1 to Enter and Navigate the Developing Cerebral Cortex

D. Meechan, E. Tucker, Danielle R. Adney, 2014, The Journal of Neuroscience.

Complementary genetic and functional analyses of genes involved in adiposity

D. Meechan, 2003 .

22q11 Deletion Syndrome

A. LaMantia, D. Meechan, T. Maynard, 2015 .

Ranbp1, Deleted in DiGeorge/22q11.2 Deletion Syndrome, is a Microcephaly Gene That Selectively Disrupts Layer 2/3 Cortical Projection Neuron Generation.

A. LaMantia, D. Meechan, T. Maynard, 2015, Cerebral cortex.

Cxcr4 regulation of interneuron migration is disrupted in 22q11.2 deletion syndrome

A. LaMantia, D. Meechan, E. Tucker, 2012, Proceedings of the National Academy of Sciences.

22q11 Gene dosage establishes an adaptive range for sonic hedgehog and retinoic acid signaling during early development.

A. LaMantia, D. Meechan, T. Maynard, 2013, Human molecular genetics.

22 q 11 Gene dosage establishes an adaptive range for sonic hedgehog and retinoic acid signaling during early development

A. LaMantia, D. Meechan, T. Maynard, 2012 .

Mitochondrial Dysfunction Leads to Cortical Under-Connectivity and Cognitive Impairment

A. LaMantia, D. Meechan, T. Maynard, 2019, Neuron.

Limited influence of olanzapine on adult forebrain neural precursors in vitro

R. Hamer, J. Lieberman, A. LaMantia, 2006, Neuroscience.

Deleted in DiGeorge / 22 q 11 . 2 Deletion Syndrome , is a Microcephaly Gene That Selectively Disrupts Layer 2 / 3 Cortical Projection Neuron Generation

A. LaMantia, D. Meechan, T. Maynard, 2015 .

Three phases of DiGeorge/22q11 deletion syndrome pathogenesis during brain development: Patterning, proliferation, and mitochondrial functions of 22q11 genes

A. LaMantia, D. Meechan, E. Tucker, 2011, International Journal of Developmental Neuroscience.

Gene dosage in the developing and adult brain in a mouse model of 22q11 deletion syndrome

J. Lieberman, A. LaMantia, D. Meechan, 2006, Molecular and Cellular Neuroscience.

Out of Line or Altered States? Neural Progenitors as a Target in a Polygenic Neurodevelopmental Disorder

A. LaMantia, D. Meechan, T. Maynard, 2023, Developmental Neuroscience.

22q11 Gene dosage establishes an adaptive range for sonic hedgehog and retinoic acid signaling during early development.

A. LaMantia, D. Meechan, T. Maynard, 2013, Human molecular genetics.