S. Caporali

发表

Estrogens and Progesterone Promote Persistent CCND1 Gene Activation during G1 by Inducing Transcriptional Derepression via c-Jun/c-Fos/Estrogen Receptor (Progesterone Receptor) Complex Assembly to a Distal Regulatory Element and Recruitment of Cyclin D1 to Its Own Gene Promoter

Lucia Altucci, Luigi Cicatiello, Alessandro Weisz, 2004, Molecular and Cellular Biology.

Estrogens do not modify MAP kinase-dependent nuclear signaling during stimulation of early G(1) progression in human breast cancer cells.

L. Altucci, F. Bresciani, A. Weisz, 2001, Cancer research.

Joint action of miR‐126 and MAPK/PI3K inhibitors against metastatic melanoma

A. Giuliani, M. Biffoni, A. Carè, 2019, Molecular oncology.

DNA damage induced by temozolomide signals to both ATM and ATR: role of the mismatch repair system.

J. Jiricny, E. Bonmassar, S. Caporali, 2004, Molecular pharmacology.

The cyclin-dependent kinase inhibitor PHA-848125 suppresses the in vitro growth of human melanomas sensitive or resistant to temozolomide, and shows synergistic effects in combination with this triazene compound.

E. Bonmassar, L. Levati, M. Ciomei, 2010, Pharmacological research.

AKT Is Activated in an Ataxia-Telangiectasia and Rad3-Related-Dependent Manner in Response to Temozolomide and Confers Protection against Drug-Induced Cell Growth Inhibition

E. Bonmassar, L. Levati, S. Caporali, 2008, Molecular Pharmacology.

NF-κB is activated in response to temozolomide in an AKT-dependent manner and confers protection against the growth suppressive effect of the drug

P. Ascierto, E. Bonmassar, G. Palmieri, 2012, Journal of Translational Medicine.

Sox2 is not required for melanomagenesis, melanoma growth and melanoma metastasis in vivo

S. D. Agostino, P. Lacal, P. Rossi, 2017, Oncogene.

Advances in Brief Estrogens Do Not Modify MAP Kinase-dependent Nuclear Signaling during Stimulation of Early G 1 Progression in Human Breast Cancer Cells 1

L. Altucci, F. Bresciani, A. Weisz, 2001 .

Identification of genes involved in growth inhibition of breast cancer cells transduced with estrogen receptor

A. Weisz, S. Caporali, A. Lucas, 2003, FEBS letters.

Circulating miR-1246 and miR-485-3p as Promising Biomarkers of Clinical Response and Outcome in Melanoma Patients Treated with Targeted Therapy

M. Negrini, F. De Galitiis, P. Lacal, 2022, Cancers.

Influence of fatty acid synthase inhibitor orlistat on the DNA repair enzyme O6-methylguanine-DNA methyltransferase in human normal or malignant cells in vitro.

E. Bonmassar, A. Aquino, S. Caporali, 2015, International journal of oncology.

A single cycle of treatment with temozolomide, alone or combined with O(6)-benzylguanine, induces strong chemoresistance in melanoma cell clones in vitro: role of O(6)-methylguanine-DNA methyltransferase and the mismatch repair system.

J. Jiricny, P. Lacal, E. Bonmassar, 2006, International journal of oncology.

High expression of the mismatch repair protein MSH6 is associated with poor patient survival in melanoma.

P. Marchetti, J. Jiricny, S. Caporali, 2014, American journal of clinical pathology.

Down-regulation of the PTTG1 proto-oncogene contributes to the melanoma suppressive effects of the cyclin-dependent kinase inhibitor PHA-848125.

E. Bonmassar, L. Levati, D. Del Bufalo, 2012, Biochemical pharmacology.

Distinct signaling pathways mediate stimulation of cell cycle progression and prevention of apoptotic cell death by estrogen in rat pituitary tumor PR1 cells.

D. Sarkar, L. Altucci, F. Bresciani, 2003, Molecular biology of the cell.

Cells deficient in the base excision repair protein, DNA polymerase beta, are hypersensitive to oxaliplatin chemotherapy

P. Johnston, G. Dianov, J. Parsons, 2010, Oncogene.

Targeting the PI3K/AKT/mTOR pathway overcomes the stimulating effect of dabrafenib on the invasive behavior of melanoma cells with acquired resistance to the BRAF inhibitor.

P. Lacal, L. Levati, S. Caporali, 2016, International journal of oncology.

Targeting the PTTG1 oncogene impairs proliferation and invasiveness of melanoma cells sensitive or with acquired resistance to the BRAF inhibitor dabrafenib

P. Marchetti, P. Lacal, L. Levati, 2017, Oncotarget.

Fatty acid synthase inhibitor orlistat impairs cell growth and down-regulates PD-L1 expression of a human T-cell leukemia line

I. Faraoni, E. Bonmassar, A. Aquino, 2019, Journal of chemotherapy.

XPF protein levels determine sensitivity of malignant melanoma cells to oxaliplatin chemotherapy: Suitability as a biomarker for patient selection

P. McHugh, Ricky A. Sharma, S. Caporali, 2013, International journal of cancer.

NF-κB is activated in response to temozolomide in an AKT-dependent manner and confers protection against the growth suppressive effect of the drug

P. Ascierto, E. Bonmassar, G. Palmieri, 2012, Journal of Translational Medicine.

Combined effect of temozolomide and hyperthermia on human melanoma cell growth and O6-methylguanine-DNA methyltransferase activity.

E. Bonmassar, S. Caporali, E. Pagani, 2007, International journal of oncology.

miR-126-3p down-regulation contributes to dabrafenib acquired resistance in melanoma by up-regulating ADAM9 and VEGF-A

A. Carè, N. Felli, P. Lacal, 2019, Journal of experimental & clinical cancer research : CR.

Activation of IP3‐Protein Kinase C‐α Signal Transduction Pathway Precedes the Changes of Plasma Cholesterol, Hepatic Lipid Metabolism and Induction of Low‐Density Lipoprotein Receptor Expression in 17‐β‐Oestradiol‐Treated Rats

M. Marino, S. Caporali, G. Bruscalupi, 2001, Experimental physiology.

β‐estradiol stimulation of DNA synthesis requires different PKC isoforms in HepG2 and MCF7 cells

M. Marino, S. Caporali, V. Pallottini, 2001, Journal of cellular physiology.

Desacyl‐ghrelin and synthetic GH‐secretagogues modulate the production of inflammatory cytokines in mouse microglia cells stimulated by β‐amyloid fibrils

V. Locatelli, A. Torsello, E. Bresciani, 2009, Journal of neuroscience research.

DNA mismatch repair protein expression and microsatellite instability in primary mucosal melanomas of the head and neck

S. Caporali, S. Rahimi, E. Alvino, 2007, Histopathology.

TARGETING OF PDGF-C/NRP-1 AUTOCRINE LOOP AS A NEW STRATEGY FOR COUNTERACTING THE INVASIVENESS OF MELANOMA RESISTANT TO BRAF INHIBITORS.

P. Lacal, L. Levati, S. Caporali, 2023, Pharmacological research.

Platelet-derived growth factor-C promotes human melanoma aggressiveness through activation of neuropilin-1

P. Lacal, L. Levati, S. Caporali, 2017, Oncotarget.

Platelet-derived growth factor-C promotes human melanoma aggressiveness through activation of neuropilin-1

P. Lacal, L. Levati, S. Caporali, 2017, Oncotarget.

miR-126-3p down-regulation contributes to dabrafenib acquired resistance in melanoma by up-regulating ADAM9 and VEGF-A

A. Carè, N. Felli, P. Lacal, 2019, Journal of Experimental & Clinical Cancer Research.

Desacyl‐ghrelin and synthetic GH‐secretagogues modulate the production of inflammatory cytokines in mouse microglia cells stimulated by β‐amyloid fibrils

V. Locatelli, A. Torsello, E. Bresciani, 2009, Journal of neuroscience research.

Antisense Evidence for Nuclear Factor-κB–Dependent Embryopathies Initiated by Phenytoin-Enhanced Oxidative Stress

P. G. Wells, K. Ha, J. Im, 2004 .

DNA damage induced by temozolomide signals to both ATM and ATR: role of the mismatch repair system.

W. Catterall, T. Scheuer, S. Caporali, 2004 .

Circulating interleukin-8 and osteopontin are promising biomarkers of clinical outcomes in advanced melanoma patients treated with targeted therapy

P. Lacal, L. Levati, S. Caporali, 2024, Journal of experimental & clinical cancer research : CR.