A. Ghosh

发表

Protection of apoptotic cell death by protein A

T. Das, G. Sa, P. Ray, 2000, Apoptosis.

Adipose tissue senescence and inflammation in aging is reversed by the young milieu.

Nathan R. Qi, A. Ghosh, M. O’Brien, 2018, The journals of gerontology. Series A, Biological sciences and medical sciences.

The Cbl family and other ubiquitin ligases: destructive forces in control of antigen receptor signaling.

Lei Duan, M. Dimri, H. Band, 2004, Immunity.

Candidate exome capture identifies mutation of SDCCAG8 as the cause of a retinal-renal ciliopathy

S. Levy, C. Dang, R. Lewis, 2010, Nature Genetics.

An essential role of ubiquitination in Cbl-mediated negative regulation of the Src-family kinase Fyn.

H. Band, A. Ghosh, C. Andoniou, 2002, Signal transduction.

The non‐receptor tyrosine kinase Syk is a target of Cbl‐mediated ubiquitylation upon B‐cell receptor stimulation

H. Band, Jiong Wu, A. Ghosh, 2001, The EMBO journal.

D-SPHEROID DEFECTS IN NPHP KNOCKDOWN CELLS ARE RESCUED BY THE 2 SOMATOSTATIN AGONIST OCTREOTIDE

T. Hurd, F. Hildebrandt, A. Ghosh, 2012 .

3D spheroid defects in NPHP knockdown cells are rescued by the somatostatin receptor agonist octreotide.

T. Hurd, F. Hildebrandt, A. Ghosh, 2012, American journal of physiology. Renal physiology.

Lifespan extension drug interventions affect adipose tissue inflammation in aging.

A. Ghosh, Richard A. Miller, M. O’Brien, 2019, The journals of gerontology. Series A, Biological sciences and medical sciences.

Elevated Endoplasmic Reticulum Stress Response Contributes to Adipose Tissue Inflammation in Aging.

A. Ghosh, Jianhua Liu, S. Garg, 2015, The journals of gerontology. Series A, Biological sciences and medical sciences.

Toll-like receptor 4 (TLR4) deficient mice are protected from adipose tissue inflammation in aging

A. Ghosh, M. O’Brien, T. Mau, 2017, Aging.

Aging is associated with increased regulatory T-cell function

R. Banerjee, A. Ghosh, S. Garg, 2014, Aging cell.

Insilco molecular docking study of Dihydroisoquinolinium Derivatives as DPP IV Inhibitors in Type II Diabetes Mellitus

A. Ghosh, S. Samajdar, S. Palit, 2017 .

Negative regulation of Lck by Cbl ubiquitin ligase

H. Band, A. Ghosh, S. Miyake, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Exome Capture Reveals ZNF423 and CEP164 Mutations, Linking Renal Ciliopathies to DNA Damage Response Signaling

Corinne Stoetzel, Richard A. Lewis, Colin A. Johnson, 2012, Cell.

Tumor macrophages as a target for Capsaicin mediated immunotherapy.

A. Ghosh, S. Basu, Sreyashi Basu, 2012, Cancer letters.

Impaired autophagy activity is linked to elevated ER-stress and inflammation in aging adipose tissue

A. Ghosh, S. Garg, M. O’Brien, 2016, Aging.

FAN1 mutations cause karyomegalic interstitial nephritis, linking chronic kidney failure to defective DNA damage repair

H. Moch, S. Levy, T. Hurd, 2012, Nature Genetics.

Nephronophthisis-Associated CEP164 Regulates Cell Cycle Progression, Apoptosis and Epithelial-to-Mesenchymal Transition

R. Goldschmeding, F. Hildebrandt, A. Ghosh, 2014, PLoS genetics.

Nephronophthisis-Associated CEP 164 Regulates Cell Cycle Progression , Apoptosis and Epithelial-to-Mesenchymal Transition

R. Goldschmeding, F. Hildebrandt, G. Van, 2014 .

Mutation analysis of 18 nephronophthisis associated ciliopathy disease genes using a DNA pooling and next generation sequencing strategy

Colin A. Johnson, T. Hurd, D. Milford, 2010, Journal of Medical Genetics.

Renal-retinal ciliopathy gene Sdccag8 regulates DNA damage response signaling.

S. Bekker-Jensen, T. Hurd, A. Kispert, 2014, Journal of the American Society of Nephrology : JASN.

Explorer FAN 1 mutations cause karyomegalic interstitial nephritis , linking chronic kidney failure to defective DNA damage repair

H. Moch, S. Levy, T. Hurd, 2017 .

FAN 1 mutations cause karyomegalic interstitial nephritis , linking chronic kidney failure to defective DNA damage repair

Soliman, Agata, Kerstin, 2013 .

Human retinopathy-associated ciliary protein retinitis pigmentosa GTPase regulator mediates cilia-dependent vertebrate development.

A. Swaroop, H. Khanna, A. Ghosh, 2010, Human molecular genetics.

Biochemical Basis for the Requirement of Kinase Activity for Cbl-dependent Ubiquitinylation and Degradation of a Target Tyrosine Kinase*

Lei Duan, V. Band, H. Band, 2004, Journal of Biological Chemistry.

Ablation of the X-linked retinitis pigmentosa 2 (Rp2) gene in mice results in opsin mislocalization and photoreceptor degeneration.

A. Swaroop, H. Khanna, T. Hurd, 2013, Investigative ophthalmology & visual science.

Immunotherapy of Tumors with Neuroimmune Ligand Capsaicin

A. Ghosh, S. Basu, J. Beltran, 2007, The Journal of Immunology.

Neuroimmune Ligand Capsaicin Immunotherapy of Tumors with

A. Ghosh, S. Basu, J. Beltran, 2007 .

Cbl-mediated Ubiquitinylation Is Required for Lysosomal Sorting of Epidermal Growth Factor Receptor but Is Dispensable for Endocytosis*

Rick A. Rogers, Pengcheng Zhou, D. Bowtell, 2003, Journal of Biological Chemistry.

Accumulation of the Raf-1 Kinase Inhibitory Protein (Rkip) Is Associated with Cep290-mediated Photoreceptor Degeneration in Ciliopathies*

H. Khanna, T. Hurd, S. Patil, 2011, The Journal of Biological Chemistry.

Fas-associated factor 1 is a negative regulator in capsaicin induced cancer cell apoptosis.

A. Ghosh, S. Basu, Sreyashi Basu, 2010, Cancer letters.

Novel role of autophagy-associated Pik3c3 gene in gonadal white adipose tissue browning in aged C57/Bl6 male mice

A. Ghosh, M. O’Brien, T. Mau, 2018, Aging.

Functional Analysis of Retinitis Pigmentosa 2 (RP2) Protein Reveals Variable Pathogenic Potential of Disease-Associated Missense Variants

H. Khanna, T. Hurd, A. Ghosh, 2011, PloS one.

Exome Capture Reveals ZNF423 and CEP164 Mutations, Linking Renal Ciliopathies to DNA Damage Response Signaling

Colin A. Johnson, S. Levy, Shaohui Wang, 2012, Cell.

Mutation analysis of 18 nephronophthisis associated ciliopathy disease genes using a DNA pooling and next generation sequencing strategy

Colin A. Johnson, T. Hurd, D. Milford, 2010, Journal of Medical Genetics.