Marlies Sachs

发表

Ubiquitin C-terminal hydrolase L1 (UCH-L1) loss causes neurodegeneration by altering protein turnover in the first postnatal weeks

Markus M. Rinschen, P. Saftig, J. Reichelt, 2019, Proceedings of the National Academy of Sciences.

Glomerulopathy Induced by Immunization with a Peptide Derived from the Goodpasture Antigen α3IV-NC1

U. Panzer, H. Mittrücker, Marlies Sachs, 2015, The Journal of Immunology.

MicroRNA-193a Regulates the Transdifferentiation of Human Parietal Epithelial Cells toward a Podocyte Phenotype.

U. Panzer, M. Saleem, D. Kerjaschki, 2015, Journal of the American Society of Nephrology : JASN.

Deubiquitinating Enzyme UCH-L1 Promotes Dendritic Cell Antigen Cross-Presentation by Favoring Recycling of MHC Class I Molecules

J. Reichelt, H. Mittrücker, A. Reinicke, 2019, The Journal of Immunology.

The expression of podocyte-specific proteins in parietal epithelial cells is regulated by protein degradation.

Marlies Sachs, C. Meyer-Schwesinger, R. Stahl, 2013, Kidney international.

Distinct Modes of Balancing Glomerular Cell Proteostasis in Mucolipidosis Type II and III Prevent Proteinuria.

O. Kretz, E. Krüger, Marlies Sachs, 2020, Journal of the American Society of Nephrology : JASN.

Characterization of the renal CD4+ T-cell response in experimental autoimmune glomerulonephritis.

U. Panzer, H. Mittrücker, Marlies Sachs, 2012, Kidney international.

Rho-kinase inhibition prevents proteinuria in immune-complex-mediated antipodocyte nephritis.

S. Balabanov, F. Thaiss, Marlies Sachs, 2012, AJP - Renal Physiology.

Thrombospondin Type 1 Domain-Containing 7A Localizes to the Slit Diaphragm and Stabilizes Membrane Dynamics of Fully Differentiated Podocytes.

Marlies Sachs, Wiebke Sachs, C. Meyer-Schwesinger, 2019, Journal of the American Society of Nephrology : JASN.

ADAM10-Mediated Ectodomain Shedding Is an Essential Driver of Podocyte Damage

O. Kretz, R. Lüllmann-Rauch, P. Saftig, 2021, Journal of the American Society of Nephrology : JASN.

UCH-L1 induces podocyte hypertrophy in membranous nephropathy by protein accumulation.

S. Balabanov, C. Cohen, Marlies Sachs, 2014, Biochimica et biophysica acta.

Goodpasture with a Peptide Derived from the Glomerulopathy Induced by Immunization

U. Panzer, H. Mittrücker, Marlies Sachs, 2015 .

Ubiquitin C-Terminal Hydrolase L1 is required for regulated protein degradation through the ubiquitin proteasome system in kidney.

J. Reichelt, A. Reinicke, Marlies Sachs, 2018, Kidney international.

Tripartite Separation of Glomerular Cell Types and Proteomes from Reporter-Free Mice

Markus M. Rinschen, J. Reichelt, Marlies Sachs, 2021, Journal of the American Society of Nephrology : JASN.

Alterations in the ubiquitin proteasome system in persistent but not reversible proteinuric diseases.

H. Schlüter, C. Cohen, M. Kretzler, 2014, Journal of the American Society of Nephrology : JASN.

Tripartite separation of glomerular cell-types and proteomes from reporter-free mice

Markus M. Rinschen, J. Reichelt, Marlies Sachs, 2020, bioRxiv.

Ubiquitin C-terminal hydrolase-l1 activity induces polyubiquitin accumulation in podocytes and increases proteinuria in rat membranous nephropathy.

S. Balabanov, F. Thaiss, Marlies Sachs, 2011, The American journal of pathology.

Non-functional ubiquitin C-terminal hydrolase L1 drives podocyte injury through impairing proteasomes in autoimmune glomerulonephritis

E. Krüger, J. Reichelt, Marlies Sachs, 2023, Nature communications.

Alterations in the ubiquitin proteasome system in persistent but not reversible proteinuric diseases.

H. Schlüter, C. Cohen, M. Kretzler, 2014, Journal of the American Society of Nephrology : JASN.

The expression of podocyte-specific proteins in parietal epithelial cells is regulated by protein degradation.

Marlies Sachs, C. Meyer-Schwesinger, R. Stahl, 2013, Kidney international.

The proteasome modulates endocytosis specifically in glomerular cells to promote kidney filtration.

Markus M. Rinschen, Marlies Sachs, Wiebke Sachs, 2024, Nature communications.