B. Hoang

发表

TARGETING TORC 2 IN MULTIPLE MYELOMA WITH A NEW mTOR KINASE INHIBITOR Running title : TORC 2 and multiple myeloma

J. Gera, A. Lichtenstein, T. Guglielmelli, 2010 .

Effect of autophagy on multiple myeloma cell viability

A. Lichtenstein, B. Hoang, P. Frost, 2009, Molecular Cancer Therapeutics.

Molecular basis for interaction between Icap1 alpha PTB domain and beta 1 integrin.

D. Chang, T. Springer, B. Hoang, 2002, The Journal of biological chemistry.

Molecular Basis for Interaction between Icap1α PTB Domain and β1 Integrin*

D. Chang, T. Springer, B. Hoang, 2002, Journal of Biological Chemistry.

IL-6-induced stimulation of c-myc translation in multiple myeloma cells is mediated by myc internal ribosome entry site function and the RNA-binding protein, hnRNP A1.

J. Gera, A. Lichtenstein, B. Hoang, 2008, Cancer research.

IL-6-induced Enhancement of c-Myc Translation in Multiple Myeloma Cells

J. Gera, A. Lichtenstein, B. Hoang, 2010, The Journal of Biological Chemistry.

AKT activity regulates the ability of mTOR inhibitors to prevent angiogenesis and VEGF expression in multiple myeloma cells

Y. Shi, A. Lichtenstein, B. Hoang, 2007, Oncogene.

Metabolomics Identifies Pyrimidine Starvation as the Mechanism of 5-Aminoimidazole-4-Carboxamide-1-β-Riboside-Induced Apoptosis in Multiple Myeloma Cells

A. Lichtenstein, B. Hoang, P. Frost, 2013, Molecular Cancer Therapeutics.

Metabolomics Identi fi es Pyrimidine Starvation as the Mechanism of 5-Aminoimidazole-4-Carboxamide-1-b-Riboside-Induced Apoptosis in Multiple Myeloma Cells

A. Lichtenstein, B. Hoang, P. Frost, 2013 .

Interleukin-6 activates phosphoinositol-3′ kinase in multiple myeloma tumor cells by signaling through RAS-dependent and, separately, through p85-dependent pathways

Huajun Yan, J. Gera, A. Lichtenstein, 2004, Oncogene.

SGK Kinase Activity in Multiple Myeloma Cells Protects against ER Stress Apoptosis via a SEK-Dependent Mechanism

A. Lichtenstein, B. Hoang, P. Frost, 2016, Molecular Cancer Research.

MNK kinases facilitate c-myc IRES activity in rapamycin-treated multiple myeloma cells

R. Fukunaga, Y. Shi, J. Gera, 2013, Oncogene.

Oncogenic RAS mutations in myeloma cells selectively induce cox-2 expression, which participates in enhanced adhesion to fibronectin and chemoresistance.

L. Goodglick, Huajun Yan, Li Zhu, 2006, Blood.

CRK SH3N Domain Diminishes Cell Invasiveness of Non-Small Cell Lung Cancer.

F. Moatamed, B. Hoang, G. Pezeshkpour, 2013, Genes & cancer.

chemoresistance expression which participates in enhanced adhesion to fibronectin and Oncogenic ras mutations in myeloma cells selectively induce COX-2

Huajun Yan, A. Lichtenstein, B. Hoang, 2013 .

Targeting TORC2 in multiple myeloma with a new mTOR kinase inhibitor.

J. Gera, A. Lichtenstein, T. Guglielmelli, 2010, Blood.

Therapeutic potential of targeting IRES-dependent c-myc translation in multiple myeloma cells during ER stress

Y. Shi, B. Holmes, J. Gera, 2016, Oncogene.

MNK1-Induced eIF-4E Phosphorylation in Myeloma Cells: A Pathway Mediating IL-6-Induced Expansion and Expression of Genes Involved in Metabolic and Proteotoxic Responses

J. Gera, A. Lichtenstein, B. Hoang, 2014, PloS one.

Regulation of D-cyclin translation inhibition in myeloma cells treated with mammalian target of rapamycin inhibitors: rationale for combined treatment with extracellular signal–regulated kinase inhibitors and rapamycin

J. Gera, A. Lichtenstein, B. Hoang, 2009, Molecular Cancer Therapeutics.

Mechanism by which mammalian target of rapamycin inhibitors sensitize multiple myeloma cells to dexamethasone-induced apoptosis.

Huajun Yan, J. Gera, A. Lichtenstein, 2006, Cancer research.

The PP242 Mammalian Target of Rapamycin (mTOR) Inhibitor Activates Extracellular Signal-regulated Kinase (ERK) in Multiple Myeloma Cells via a Target of Rapamycin Complex 1 (TORC1)/ Eukaryotic Translation Initiation Factor 4E (eIF-4E)/RAF Pathway and Activation Is a Mechanism of Resistance*

J. Gera, A. Lichtenstein, B. Hoang, 2012, The Journal of Biological Chemistry.

Mammalian Target of Rapamycin Inhibitors Induce Tumor Cell Apoptosis In Vivo Primarily by Inhibiting VEGF Expression and Angiogenesis

J. Gera, A. Lichtenstein, B. Hoang, 2013, Journal of oncology.

multiple myeloma cells in a xenograft model In vivo anti-tumor effects of the mTOR inhibitor, CCI-779, against human

F. Moatamed, Huajun Yan, P. Frost, 2013 .

In vivo antitumor effects of the mTOR inhibitor CCI-779 against human multiple myeloma cells in a xenograft model.

F. Moatamed, Huajun Yan, P. Frost, 2004, Blood.

Running Head: In vivo efficacy of CCI-779 in myeloma

Huajun Yan, P. Frost, J. Gera, 2004 .

Delayed mammary gland involution in mice with mutation of the insulin-like growth factor binding protein 5 gene.

Bao Hoang, T. Wood, J. Pintar, 2007, Endocrinology.

Mechanism by Which mTOR Inhibitors Sesitize Multiple Myeloma Cells to Dexamethasone-Induced Apoptosis.

Huajun Yan, J. Gera, A. Lichtenstein, 2005 .

DEPTOR is linked to a TORC1-p21 survival proliferation pathway in multiple myeloma cells

R. Finn, J. Gera, A. Lichtenstein, 2014, Genes & cancer.

Anti-CD138-targeted interferon is a potent therapeutic against multiple myeloma.

S. Morrison, Juan Zhang, A. Lichtenstein, 2015, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.

ROCK-II-induced membrane blebbing and chromatin condensation require actin cytoskeleton.

D. Chang, B. Hoang, Yuhong Song, 2002, Experimental cell research.

Genetic Analysis on the Role of Integrin during Axon Guidance in Drosophila

B. Hoang, A. Chiba, 1998, The Journal of Neuroscience.

Targeting TORC2 in multiple myeloma with a new mTOR kinase inhibitor.

J. Gera, A. Lichtenstein, T. Guglielmelli, 2010, Blood.

The PP242 Mammalian Target of Rapamycin (mTOR) Inhibitor Activates Extracellular Signal-regulated Kinase (ERK) in Multiple Myeloma Cells via a Target of Rapamycin Complex 1 (TORC1)/ Eukaryotic Translation Initiation Factor 4E (eIF-4E)/RAF Pathway and Activation Is a Mechanism of Resistance*

J. Gera, A. Lichtenstein, B. Hoang, 2012, The Journal of Biological Chemistry.

Metabolomics Identifies Pyrimidine Starvation as the Mechanism of 5- Aminoimidazole-4-carboxamide-1-β-riboside (aicar) Induced Apoptosis in Multiple Myeloma Cells

B. Hoang, P. Frost, Yijiang Shi, 2022 .