J. Lawrence

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Sites That Govern Translational Repression Phosphorylation of Phas-i in Five (s/t)p Multiple Mechanisms Control

T. Haystead, I. Mothe-Satney, Daqing Yang, 2022 .

Control of Ser2448 Phosphorylation in the Mammalian Target of Rapamycin by Insulin and Skeletal Muscle Load*

S. Bodine, J. Lawrence, T. Reynolds, 2002, The Journal of Biological Chemistry.

Ser-64 and Ser-111 in PHAS-I Are Dispensable for Insulin-stimulated Dissociation from eIF4E*

I. Mothe-Satney, J. Lawrence, Gail E. Ferguson, 2003, Journal of Biological Chemistry.

Two Motifs in the Translational Repressor PHAS-I Required for Efficient Phosphorylation by Mammalian Target of Rapamycin and for Recognition by Raptor*

J. Lawrence, K. Choi, L. McMahon, 2003, Journal of Biological Chemistry.

Insulin signaling and the control of PHAS-I phosphorylation.

G. Brunn, J. Lawrence, 2001, Progress in molecular and subcellular biology.

Identification of an adipocyte protein that binds to calmodulin in the absence of Ca2+ and is phosphorylated in response to insulin and tumor-promoting phorbol esters.

J. Lawrence, J. McDonald, 1989, The Journal of biological chemistry.

Effects of Insulin and Transgenic Overexpression of UDP-glucose Pyrophosphorylase on UDP-glucose and Glycogen Accumulation in Skeletal Muscle Fibers*

E. Barrett, Y. Pak, T. Harris, 2005, Journal of Biological Chemistry.

Disruption of the Gene Encoding the Mitogen-regulated Translational Modulator PHAS-I in Mice*

D. Stumpo, P. Blackshear, J. Lawrence, 1997, The Journal of Biological Chemistry.

Glucose transporters in single skeletal muscle fibers. Relationship to hexokinase and regulation by contractile activity.

S. Salmons, J. Lawrence, J. Manchester, 1994, The Journal of biological chemistry.

Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo

G. Yancopoulos, S. Bodine, D. Glass, 2001, Nature Cell Biology.

The Mammalian Target of Rapamycin Phosphorylates Sites Having a (Ser/Thr)-Pro Motif and Is Activated by Antibodies to a Region near Its COOH Terminus

T. Haystead, G. Brunn, P. Fadden, 1997, The Journal of Biological Chemistry.

Glucose transport in L6 myoblasts overexpressing GLUT1 and GLUT4.

D. James, J. Lawrence, L. J. Robinson, 1993, The Journal of biological chemistry.

Regulation of Both Glycogen Synthase and PHAS-I by Insulin in Rat Skeletal Muscle Involves Mitogen-activated Protein Kinase-independent and Rapamycin-sensitive Pathways (*)

A. Depaoli-Roach, A. Saltiel, J. Lawrence, 1996, The Journal of Biological Chemistry.

mTOR‐dependent stimulation of the association of eIF4G and eIF3 by insulin

J. Shabanowitz, D. Hunt, R. Rhoads, 2006, The EMBO journal.

Glucose transport and phosphorylation in single cardiac myocytes: rate-limiting steps in glucose metabolism.

J. Nerbonne, O. H. Lowry, J. Lawrence, 1994, The American journal of physiology.

Alterations in Hepatic Metabolism in fld Mice Reveal a Role for Lipin 1 in Regulating VLDL-Triacylglyceride Secretion

T. Harris, J. Lawrence, B. Finck, 2008, Arteriosclerosis, thrombosis, and vascular biology.

of Inflammatory Factors Activity in Adipocytes To Inhibit Secretion

T. Harris, J. Lawrence, S. Keller, 2014 .

Lipin 1 Represses NFATc4 Transcriptional Activity in Adipocytes To Inhibit Secretion of Inflammatory Factors

T. Harris, J. Lawrence, S. Keller, 2010, Molecular and Cellular Biology.

Phosphorylation of the glucose transporter in rat adipocytes. Identification of the intracellular domain at the carboxyl terminus as a target for phosphorylation in intact-cells and in vitro.

D. James, J. Lawrence, J. Hiken, 1990, The Journal of biological chemistry.

Isoproterenol stimulates phosphorylation of the insulin-regulatable glucose transporter in rat adipocytes.

D. James, J. Lawrence, J. Hiken, 1989, Proceedings of the National Academy of Sciences of the United States of America.

GLUT4 phosphorylation and inhibition of glucose transport by dibutyryl cAMP.

J. Slot, D. James, J. Lawrence, 1993, The Journal of biological chemistry.

GLUT4 facilitates insulin stimulation and cAMP-mediated inhibition of glucose transport.

D. James, J. Lawrence, R. Piper, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Phosphorylation of PHAS-I by mitogen-activated protein (MAP) kinase. Identification of a site phosphorylated by MAP kinase in vitro and in response to insulin in rat adipocytes.

T. Haystead, J. Lawrence, C. Haystead, 1994, The Journal of biological chemistry.

Molecular cloning and tissue distribution of PHAS-I, an intracellular target for insulin and growth factors.

J. Lawrence, M. Velleca, S. Pang, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Insulin Receptor Substrate-2 Proteasomal Degradation Mediated by a Mammalian Target of Rapamycin (mTOR)-induced Negative Feedback Down-regulates Protein Kinase B-mediated Signaling Pathway in β-Cells*

J. Lawrence, C. Rhodes, L. M. Dickson, 2005, Journal of Biological Chemistry.

Amino acids and leucine allow insulin activation of the PKB/mTOR pathway in normal adipocytes treated with wortmannin and in adipocytes from db/db mice

E. Van Obberghen, J. Lawrence, N. Gautier, 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Control of glycogen synthase and phosphorylase by insulin in rat adipocytes which is unrelated to the concentration of cyclic AMP.

J. Larner, J. Lawrence, 1979, Biochimica et biophysica acta.

Hormonal effects on fat cell adenosine 3',5'-monophosphate dependent protein kinase.

J. Larner, J. Lawrence, J. Guinovart, 1978, Biochimica et biophysica acta.

In Rat Hepatocytes Glucagon Increases Mammalian Target of Rapamycin Phosphorylation on Serine 2448 but Antagonizes the Phosphorylation of Its Downstream Targets Induced by Insulin and Amino Acids*

E. Van Obberghen, J. Lawrence, N. Gautier, 2004, Journal of Biological Chemistry.

Insulin stimulates protein synthesis in skeletal muscle by enhancing the association of eIF-4E and eIF-4G.

S. Kimball, L. Jefferson, J. Lawrence, 1997, The American journal of physiology.

Insulin action in denervated rat hemidiaphragms. Decreased hormonal stimulation of glycogen synthesis involves both glycogen synthase and glucose transport.

J. Lawrence, R. Smith, 1984, The Journal of biological chemistry.

Modulation of the protein kinase activity of mTOR.

J. Lawrence, L. McMahon, K. M. Choi, 2004, Current topics in microbiology and immunology.

Branched-chain amino acids are essential in the regulation of PHAS-I and p70 S6 kinase by pancreatic beta-cells. A possible role in protein translation and mitogenic signaling.

M. Mcdaniel, G. Kwon, C. Marshall, 1998, The Journal of biological chemistry.

cAMP- and rapamycin-sensitive regulation of the association of eukaryotic initiation factor 4E and the translational regulator PHAS-I in aortic smooth muscle cells.

E. Krebs, L. Graves, K. Bornfeldt, 1995, Proceedings of the National Academy of Sciences of the United States of America.

Stimulation of glucose transport and glucose transporter phosphorylation by okadaic acid in rat adipocytes.

D. James, J. Lawrence, J. Hiken, 1990, The Journal of biological chemistry.

Effect of glycogen synthase overexpression on insulin-stimulated muscle glucose uptake and storage.

Z. Ding, J. Ivy, A. Scrimgeour, 2004, American journal of physiology. Endocrinology and metabolism.

Phosphorylase kinase isozymes and phosphorylase in denervated skeletal muscles

J. Lawrence, Roderic L. Smith, 1990, Muscle & nerve.

Rat skeletal muscle phosphorylase kinase: turnover and control of isozyme levels in culture.

J. Lawrence, W. Salsgiver, 1986, The American journal of physiology.

Evidence that levels of malate dehydrogenase and fumarase are increased by cAMP in rat myotubes.

J. Lawrence, W. Salsgiver, 1984, The American journal of physiology.

Levels of enzymes of energy metabolism are controlled by activity of cultured rat myotubes.

J. Lawrence, W. Salsgiver, 1983, The American journal of physiology.

Combining Microinjection with Microanalytical Methods to Investigate Regulation of Metabolic Pathways

J. Lawrence, J. Manchester, 1999 .

Autoantibodies to the insulin receptor activate glycogen synthase in rat adipocytes

C. Kahn, J. Larner, J. Lawrence, 1978, Molecular and Cellular Biochemistry.

Activation of ribosomal protein S6 kinases does not increase glycogen synthesis or glucose transport in rat adipocytes.

J. Lawrence, T. Lin, 1994, The Journal of biological chemistry.

Ras signaling in the activation of glucose transport by insulin.

O. H. Lowry, J. Lawrence, J. Manchester, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Mitogen-activated protein kinase activation is not sufficient for stimulation of glucose transport or glycogen synthase in 3T3-L1 adipocytes.

D. James, J. Lawrence, L. J. Robinson, 1993, The Journal of biological chemistry.

Activation of rat adipocyte glycogen synthase by insulins.

J. Larner, J. Lawrence, J. Guinovart, 1977, The Journal of biological chemistry.

Evidence for electron transfer reactions involved in the Cu2+ -dependent thiol activation of fat cell glucose utilization.

M. Czech, J. Lawrence, W. Lynn, 1974, The Journal of biological chemistry.

Evidence for the involvement of sulfhydryl oxidation in the regulation of fat cell hexose transport by insulin.

M. Czech, J. Lawrence, W. Lynn, 1974, Proceedings of the National Academy of Sciences of the United States of America.

Activation of hexose transport by concanavalin A in isolated brown fat cells. Effects of cell surface modification with neuraminidase and trypsin on lectin and insulin action.

M. Czech, J. Lawrence, W. Lynn, 1974, The Journal of biological chemistry.

Tetrodotoxin-insensitive sodium channels. Ion flux studies of neurotoxin action in a clonal rat muscle cell line.

W. Catterall, J. Lawrence, 1981, The Journal of biological chemistry.

Control of glycogen synthase and phosphorylase by amylin in rat skeletal muscle. Hormonal effects on the phosphorylation of phosphorylase and on the distribution of phosphate in the synthase subunit.

J. Lawrence, J. N. Zhang, 1994, The Journal of biological chemistry.

Branched-chain Amino Acids Are Essential in the Regulation of PHAS-I and p70 S6 Kinase by Pancreatic β-Cells

M. Mcdaniel, Guang Xu, G. Kwon, 1998, The Journal of Biological Chemistry.

Activation of Mammalian Target of Rapamycin (mTOR) by Insulin Is Associated with Stimulation of 4EBP1 Binding to Dimeric mTOR Complex 1*

J. Lawrence, C. Rhodes, Lifu Wang, 2006, Journal of Biological Chemistry.

Insulin resistance in denervated skeletal muscle. Inability of insulin to stimulate dephosphorylation of glycogen synthase in denervated rat epitrochlearis muscles.

P. Roach, J. Lawrence, R. Smith, 1988, The Journal of biological chemistry.

Insulin receptor substrate-2 proteasomal degradation mediated by a mammalian target of rapamycin (mTOR)-induced negative feedback down-regulates protein kinase B-mediated signaling pathway in beta-cells.

J. Lawrence, C. Rhodes, L. M. Dickson, 2005, The Journal of biological chemistry.

Glycogen synthase: activation by insulin and effect of transgenic overexpression in skeletal muscle.

P. Roach, J. Lawrence, J. Manchester, 1997, Biochemical Society transactions.

Insulin control of glycogen synthesis.

P. Roach, J. Larner, J. Lawrence, 1978, Advances in cyclic nucleotide research.

Insulin activates a PD 098059‐sensitive kinase that is involved in the regulation of p70S6K and PHAS‐I

J. Lawrence, P. Scott, 1997, FEBS letters.

Insulin and diabetes cause reciprocal changes in the association of eIF-4E and PHAS-I in rat skeletal muscle.

T. Haystead, S. Kimball, L. Jefferson, 1996, The American journal of physiology.

Rat skeletal muscle glycogen synthase: phosphorylation of the purified enzyme by cAMP-dependent and -independent protein kinases.

J. Lawrence, J. Hiken, 1985, Archives of biochemistry and biophysics.

Glycogen synthase in rat adipocytes and skeletal muscle is phosphorylated on both serine and threonine

J. Lawrence, J. Hiken, 1984, FEBS letters.

Mutational analysis of sites in the translational regulator, PHAS‐I, that are selectively phosphorylated by mTOR

G. Brunn, J. Lawrence, D. Yang, 1999, FEBS letters.

Phosphorylation of the translational repressor PHAS-I by the mammalian target of rapamycin.

Christine C. Hudson, P. Houghton, R. Abraham, 1997, Science.

Direct inhibition of the signaling functions of the mammalian target of rapamycin by the phosphoinositide 3‐kinase inhibitors, wortmannin and LY294002.

R. Abraham, C. Sabers, G. Brunn, 1996, The EMBO journal.

Insulin stimulates dephosphorylation of phosphorylase in rat epitrochlearis muscles.

J. Lawrence, A. Davis, J. Hiken, 1989, The Journal of biological chemistry.

Control of PHAS-I by Insulin in 3T3-L1 Adipocytes

P. Blackshear, A. Saltiel, J. Lawrence, 1995, The Journal of Biological Chemistry.

Multiple phosphorylation of rabbit skeletal muscle glycogen synthase. Evidence for interactions among phosphorylation sites and the resolution of electrophoretically distinct forms of the subunit.

A. Depaoli-Roach, P. Roach, J. Lawrence, 1983, The Journal of biological chemistry.

Tetrodotoxin-insensitive sodium channels. Binding of polypeptide neurotoxins in primary cultures of rat muscle cells.

W. Catterall, J. Lawrence, 1981, The Journal of biological chemistry.

Tetrodotoxin-sensitive sodium channels in rat muscle cells developing in vitro.

S. Sherman, W. Catterall, J. Lawrence, 1983, The Journal of biological chemistry.

Evidence for alpha adrenergic activation of phosphorylase and inactivation of glycogen synthase in rat adipocytes. Effects of alpha and beta adrenergic agonists and antagonists on glycogen synthase and phosphorylase.

J. Larner, J. Lawrence, J. Larner,, 1977, Molecular pharmacology.

PHAS/4E-BPs as regulators of mRNA translation and cell proliferation.

R. Abraham, J. Lawrence, 1997, Trends in biochemical sciences.

Brain extract induces synaptic characteristics in the basal lamina of cultured myotubes

J. Sanes, J. Lawrence, D. Feldman, 1984, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Activation of glycogen synthase in rat adipocytes by insulin and glucose involves increased glucose transport and phosphorylation.

J. Larner, J. Lawrence, J. Larner,, 1978, The Journal of biological chemistry.

Farnesylthiosalicylic acid inhibits mammalian target of rapamycin (mTOR) activity both in cells and in vitro by promoting dissociation of the mTOR-raptor complex.

R. Santen, W. Yue, J. Lawrence, 2005, Molecular endocrinology.

Hormonal control of glycogen synthase in rat hemidiaphragms. Effects of insulin and epinephrine on the distribution of phosphate between two cyanogen bromide fragments.

A. Depaoli-Roach, P. Roach, J. Lawrence, 1983, The Journal of biological chemistry.

Phosphorylation of phosphoprotein phosphatase inhibitor-2 (I-2) in rat fat cells.

A. Depaoli-Roach, J. Lawrence, J. Hiken, 1988, Biochemical and biophysical research communications.

Insulin action in denervated skeletal muscle. Evidence that the reduced stimulation of glycogen synthesis does not involve decreased insulin binding.

J. Lawrence, R. Smith, 1985, The Journal of biological chemistry.

Regulation of Proline-rich Akt Substrate of 40 kDa (PRAS40) Function by Mammalian Target of Rapamycin Complex 1 (mTORC1)-mediated Phosphorylation*

T. Harris, J. Lawrence, Lifu Wang, 2008, Journal of Biological Chemistry.

Evidence of insulin-stimulated phosphorylation and activation of the mammalian target of rapamycin mediated by a protein kinase B signaling pathway.

R. Roth, A. D. Kohn, G. Brunn, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Fat Cell–Specific Ablation of Rictor in Mice Impairs Insulin-Regulated Fat Cell and Whole-Body Glucose and Lipid Metabolism

M. Magnuson, T. Harris, J. Lawrence, 2010, Diabetes.

PRAS40 Regulates mTORC1 Kinase Activity by Functioning as a Direct Inhibitor of Substrate Binding*

R. Roth, T. Harris, J. Lawrence, 2007, Journal of Biological Chemistry.

Insulin-dependent stimulation of protein synthesis by phosphorylation of a regulator of 5'-cap function

A. Gingras, N. Sonenberg, A. Pause, 1994, Nature.

Identification of Phosphorylation Sites in the Translational Regulator, PHAS-I, That Are Controlled by Insulin and Rapamycin in Rat Adipocytes*

T. Haystead, P. Fadden, J. Lawrence, 1997, The Journal of Biological Chemistry.

Activation of glycogen synthase by insulin in rat adipocytes. Evidence of hormonal stimulation of multisite dephosphorylation by glucose transport-dependent and -independent pathways.

J. Lawrence, C. James, 1984, The Journal of biological chemistry.

Insulin Mediates Glucose-stimulated Phosphorylation of PHAS-I by Pancreatic Beta Cells

M. Mcdaniel, Guang Xu, R. Munivenkatappa, 1998, The Journal of Biological Chemistry.

Construction and Characterization of a Conditionally Active Version of the Serine/Threonine Kinase Akt*

M. Birnbaum, A. Barthel, R. Roth, 1998, The Journal of Biological Chemistry.

Attenuation of Mammalian Target of Rapamycin Activity by Increased cAMP in 3T3-L1 Adipocytes*

J. Lawrence, P. Scott, 1998, The Journal of Biological Chemistry.

PHAS-I as a link between mitogen-activated protein kinase and translation initiation.

N. Sonenberg, T. Haystead, A. Pause, 1994, Science.

Mammalian Target of Rapamycin-dependent Phosphorylation of PHAS-I in Four (S/T)P Sites Detected by Phospho-specific Antibodies*

R. Abraham, G. Brunn, J. Lawrence, 2000, The Journal of Biological Chemistry.

Control of the Translational Regulators PHAS-I and PHAS-II by Insulin and cAMP in 3T3-L1 Adipocytes*

J. Lawrence, T. Lin, 1996, The Journal of Biological Chemistry.

An acetylcholine receptor precursor alpha subunit that binds alpha-bungarotoxin but not d-tubocurare.

J. Merlie, J. Lawrence, J. Lindstrom, 1986, Proceedings of the National Academy of Sciences of the United States of America.

Muscle-Specific Deletion of Rictor Impairs Insulin-Stimulated Glucose Transport and Enhances Basal Glycogen Synthase Activity

John C. Lawrence, Susanna R. Keller, Mark A. Magnuson, 2007, Molecular and Cellular Biology.

Effects of insulin, methoxamine, and calcium on glycogen synthase in rat adipocytes.

J. Lawrence, J. Larner,, 1978, Molecular pharmacology.

Lipin 1 is an inducible amplifier of the hepatic PGC-1α/PPARα regulatory pathway

T. Harris, J. Lawrence, D. Kelly, 2006 .

Regulation of phosphorylation of nicotinic acetylcholine receptors in mouse BC3H1 myocytes.

J. Merlie, J. Lawrence, M. Smith, 1987, Proceedings of the National Academy of Sciences of the United States of America.

Insulin Controls Subcellular Localization and Multisite Phosphorylation of the Phosphatidic Acid Phosphatase, Lipin 1*

J. Shabanowitz, D. Hunt, T. Harris, 2007, Journal of Biological Chemistry.

Insulin-stimulated phosphorylation of lipin mediated by the mammalian target of rapamycin

J. Lawrence, I. Mothe‐Satney, Todd A Huffman, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Control of Protein Synthesis by Insulin

J. Lawrence, J. F. Christian, 2007 .

TOR Signaling

T. Harris, J. Lawrence, 2003, Science's STKE.

Increasing cAMP attenuates activation of mitogen-activated protein kinase.

J. Lawrence, X. Kong, B. Sevetson, 1993, Proceedings of the National Academy of Sciences of the United States of America.

Phosphorylation of the translational regulator, PHAS‐I, by protein kinase CK2

T. Haystead, P. Fadden, J. Lawrence, 1998, FEBS letters.

A conserved phosphatase cascade that regulates nuclear membrane biogenesis

J. Dixon, Matthew S. Gentry, S. Wiley, 2007, Proceedings of the National Academy of Sciences.

Mitogen-activated Protein Kinase-independent Pathways Mediate the Effects of Nerve Growth Factor and cAMP on Neuronal Survival*

J. Lawrence, D. Creedon, E. Johnson, 1996, The Journal of Biological Chemistry.

Activity-dependent accumulation of basal lamina by cultured rat myotubes.

J. Sanes, J. Lawrence, John C. Lawrence, 1983, Developmental biology.

Mammalian Target of Rapamycin Complex 1 (mTORC1) Activity Is Associated with Phosphorylation of Raptor by mTOR*

T. Sturgill, T. Harris, J. Lawrence, 2009, Journal of Biological Chemistry.

PHAS proteins as mediators of the actions of insulin, growth factors and cAMP on protein synthesis and cell proliferation.

T. Haystead, P. Fadden, J. Lawrence, 1997, Advances in enzyme regulation.

Control of glycogen synthesis is shared between glucose transport and glycogen synthase in skeletal muscle fibers.

P. Roach, J. Lawrence, J. Manchester, 2000, American journal of physiology. Endocrinology and metabolism.

The Rapamycin-Binding Domain Governs Substrate Selectivity by the Mammalian Target of Rapamycin

R. Abraham, J. Lawrence, K. Choi, 2002, Molecular and Cellular Biology.

Studies on the mechanism of resistance to rapamycin in human cancer cells.

P. Houghton, R. Abraham, M. Danks, 1998, Molecular pharmacology.

Protection against oxidative stress-induced insulin resistance in rat L6 muscle cells by mircomolar concentrations of alpha-lipoic acid.

J. Lawrence, I. Goldfine, B. Maddux, 2001, Diabetes.

New Insights Into the Role and Mechanism of Glycogen Synthase Activation by Insulin

P. Roach, J. Lawrence, 1997, Diabetes.

Lipin 1 is an inducible amplifier of the hepatic PGC-1alpha/PPARalpha regulatory pathway.

T. Harris, J. Lawrence, D. Kelly, 2006, Cell metabolism.

Phosphorylase kinase isozymes in normal and electrically stimulated skeletal muscles.

S. Salmons, J. Lawrence, J. Hiken, 1986, The American journal of physiology.

Inhibition of acetylcholine receptor assembly by activity in primary cultures of embryonic rat muscle cells.

J. Merlie, J. Lawrence, J. Lindstrom, 1986, The Journal of biological chemistry.

Signal transduction and protein phosphorylation in the regulation of cellular metabolism by insulin.

J. Lawrence, Jr J C Lawrence, 1992, Annual review of physiology.

Insulin Control of Glycogen Metabolism in Knockout Mice Lacking the Muscle-Specific Protein Phosphatase PP1G/RGL

A. Depaoli-Roach, A. Scrimgeour, J. Lawrence, 2001, Molecular and Cellular Biology.

Hexose transport in isolated brown fat cells. A model system for investigating insulin action on membrane transport.

M. Czech, J. Lawrence, W. Lynn, 1974, The Journal of biological chemistry.

Inhibitor-1 Is Not Required for the Activation of Glycogen Synthase by Insulin in Skeletal Muscle*

P. Greengard, A. Fienberg, P. Allen, 1999, The Journal of Biological Chemistry.

Insulin, growth factors, and cAMP Antagonism in the signal transduction pathways

L. Graves, J. Lawrence, 1996, Trends in Endocrinology & Metabolism.

Control of enzyme activities in individual myotubes cultured without nerve.

J. Lawrence, P. Nemeth, L. Solanki, 1985, The American journal of physiology.

Control of phosphorylase in cultured rat skeletal muscle cells. Changes in synthesis and degradation resulting from differentiation and muscle activity.

J. Lawrence, W. Salsgiver, C. James, 1984, The Journal of biological chemistry.

mTOR-dependent control of skeletal muscle protein synthesis.

J. Lawrence, 2001, International journal of sport nutrition and exercise metabolism.

Insulin stimulates the generation of an adipocyte phosphoprotein that is isolated with a monoclonal antibody against the regulatory subunit of bovine heart cAMP-dependent protein kinase.

C. W. Scott, M. Mumby, J. Lawrence, 1986, Proceedings of the National Academy of Sciences of the United States of America.

Increased glycogen accumulation in transgenic mice overexpressing glycogen synthase in skeletal muscle.

P. Roach, J. Lawrence, J. Manchester, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Phosphorylation of nerve growth factor receptor proteins in sympathetic neurons and PC12 cells. In vitro phosphorylation by the cAMP-independent protein kinase FA/GSK-3.

P. Roach, J. Lawrence, E. Johnson, 1986, The Journal of biological chemistry.

Surprises of genetic engineering

S. Dimauro, P. Roach, N. Raben, 2001, Neurology.

Effects of aging and exercise on insulin action in rat adipocytes are correlated with changes in fat cell volume.

J. Lawrence, J. Holloszy, G. Cartee, 1989, Journal of Gerontology.

Multiple Mechanisms Control Phosphorylation of PHAS-I in Five (S/T)P Sites That Govern Translational Repression

T. Haystead, Daqing Yang, P. Fadden, 2000, Molecular and Cellular Biology.