T. Fujita

Insulin-Induced c-Jun N-Terminal Kinase Activation Is Negatively Regulated by Protein Kinase C δ.

K. Morino, H. Maegawa, A. Kashiwagi, 2001, Endocrinology.

A new antidiabetic agent (JTT-501) rapidly stimulates glucose disposal rates by enhancing insulin signal transduction in skeletal muscle

K. Morino, H. Maegawa, M. Haneda, 1999, Diabetologia.

Expression of a Dominant Negative SHP-2 in Transgenic Mice Induces Insulin Resistance*

K. Morino, H. Maegawa, M. Haneda, 1999, The Journal of Biological Chemistry.

Insulin-induced c-Jun N-terminal kinase activation is negatively regulated by protein kinase C delta.

K. Morino, H. Maegawa, A. Kashiwagi, 2001, Endocrinology.

Opposite regulation of tyrosine-phosphorylation of p130(Cas) by insulin and insulin-like growth factor I.

H. Maegawa, A. Kashiwagi, R. Kikkawa, 1998, Journal of biochemistry.

SHPTP2 serves adapter protein linking between Janus kinase 2 and insulin receptor substrates.

H. Maegawa, A. Kashiwagi, T. Obata, 1996, Biochemical and Biophysical Research Communications - BBRC.