I. Uphues

发表

Association of impaired phosphatidylinositol 3-kinase activity in GLUT1-containing vesicles with malinsertion of glucose transporters into the plasma membrane of fibroblasts from a patient with severe insulin resistance and clinical features of Werner syndrome.

A. Niendorf, H. Joost, H. Häring, 2000, The Journal of clinical endocrinology and metabolism.

LKB1 and AMPK differentially regulate pancreatic β-cell identity

G. Rutter, Y. Dor, F. Reimann, 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Cardiac insulin resistance is associated with an impaired recruitment of phosphatidylinositol 3-kinase to GLUT4 vesicles

I. Uphues, J. Eckel, M. Till, 2000, International Journal of Obesity.

LKB1 and AMPK differentially regulate pancreatic β-cell identity

G. Rutter, Y. Dor, F. Reimann, 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Diversification of cardiac insulin signaling involves the p85 alpha/beta subunits of phosphatidylinositol 3-kinase.

A. Kessler, I. Uphues, D. Ouwens, 2001, American journal of physiology. Endocrinology and metabolism.

Diversification of cardiac insulin signaling involves the p85α/β subunits of phosphatidylinositol 3-kinase

A. Kessler, I. Uphues, D. Ouwens, 2001 .

Contraction-induced translocation of the glucose transporter Glut4 in isolated ventricular cardiomyocytes.

I. Uphues, J. Eckel, H. Reinauer, 1992, Biochemical and biophysical research communications.

Insulin‐dependent translocation of the small GTP‐binding protein rab3C in cardiac muscle: studies on insulin‐resistant Zucker rats

Y. Chern, I. Uphues, J. Eckel, 1995, FEBS letters.

Molecular analysis of insulin resistance in isolated ventricular cardiomyocytes of obese Zucker rats.

I. Uphues, J. Eckel, J. Eckel, 1997, The American journal of physiology.

Alterations in β-Cell Calcium Dynamics and Efficacy Outweigh Islet Mass Adaptation in Compensation of Insulin Resistance and Prediabetes Onset

C. Cohrs, I. Uphues, Chunguang Chen, 2016, Diabetes.

Insulin-induced translocation of the glucose transporter GLUT4 in cardiac muscle: studies on the role of small-molecular-mass GTP-binding proteins.

B. Goud, I. Uphues, J. Eckel, 1994, The Biochemical journal.

Diversiﬁcation of cardiac insulin signaling involves the p85 a / b subunits of phosphatidylinositol 3-kinase

A. Kessler, I. Uphues, J. Eckel, 2000 .

Photoaffinity labelling of cardiac membrane GTP-binding proteins in response to insulin.

I. Uphues, J. Eckel, T. Kolter, 1994, European journal of biochemistry.

BI 456906: Discovery and preclinical pharmacology of a novel GCGR/GLP-1R dual agonist with robust anti-obesity efficacy

D. Drucker, I. Uphues, H. Neubauer, 2022, Molecular metabolism.

Regulation of subcellular distribution of GLUT4 in cardiomyocytes: Rab4A reduces basal glucose transport and augments insulin responsiveness.

A. Schürmann, H. Joost, I. Uphues, 2012, Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association.

Plasma Dihydroceramides Are Diabetes Susceptibility Biomarker Candidates in Mice and Humans.

Ioannis Xenarios, Frédéric Fumeron, Robin Liechti, 2017, Cell reports.

Molecular phenotyping of multiple mouse strains under metabolic challenge uncovers a role for Elovl2 in glucose-induced insulin secretion

I. Xenarios, R. Scharfmann, G. Rutter, 2017, Molecular metabolism.

Hes3 Is Expressed in the Adult Pancreatic Islet and Regulates Gene Expression, Cell Growth, and Insulin Release*

S. Bornstein, S. Poser, I. Uphues, 2014, The Journal of Biological Chemistry.

Failure of insulin-regulated recruitment of the glucose transporter GLUT4 in cardiac muscle of obese Zucker rats is associated with alterations of small-molecular-mass GTP-binding proteins.

B. Goud, I. Uphues, J. Eckel, 1995, The Biochemical journal.