G. Henkel

发表

Ras-mediated phosphorylation of a conserved threonine residue enhances the transactivation activities of c-Ets1 and c-Ets2

Michael C. Ostrowski, D. Hume, C. Hauser, 1996, Molecular and cellular biology.

Differentiation of the mononuclear phagocyte system during mouse embryogenesis: the role of transcription factor PU.1.

Michael C. Ostrowski, D. Hume, S. Mckercher, 1999, Blood.

Commitment to the monocytic lineage occurs in the absence of the transcription factor PU.1.

S. Mckercher, R. Maki, G. Henkel, 1999, Blood.

VX-322: a novel dual receptor tyrosine kinase inhibitor for the treatment of acute myelogenous leukemia.

Jiehua Zhou, G. Henkel, Yung-mae M. Yao, 2012, Journal of medicinal chemistry.

Design, synthesis, and evaluation of a novel dual FMS-like tyrosine kinase 3/stem cell factor receptor (FLT3/c-KIT) inhibitor for the treatment of acute myelogenous leukemia.

V. Galullo, G. Henkel, J. Ma, 2011, Journal of medicinal chemistry.

Discovery of a novel highly potent broad-spectrum heterocyclic chemical series of arenavirus cell entry inhibitors.

N. V. Sokolova, G. Henkel, Alexander N. Freiberg, 2021, Bioorganic & medicinal chemistry letters.

SAR studies of 4-acyl-1,6-dialkylpiperazin-2-one arenavirus cell entry inhibitors.

N. V. Sokolova, Landon R. Whitby, D. Boger, 2019, Bioorganic & medicinal chemistry letters.

IL-Converting Enzyme/Caspase-1 Inhibitor VX-765 Blocks the Hypersensitive Response to an Inflammatory Stimulus in Monocytes from Familial Cold Autoinflammatory Syndrome Patients

K. Beaumont, P. Larsen, G. Henkel, 2005, The Journal of Immunology.

Syndrome Patients from Familial Cold Autoinflammatory to an Inflammatory Stimulus in Monocytes VX-765 Blocks the Hypersensitive Response IL-Converting Enzyme / Caspase-1 Inhibitor

K. Beaumont, P. Larsen, G. Henkel, 2005 .

An Intron Transcriptional Enhancer Element Regulates IL-4 Gene Locus Accessibility in Mast Cells1

G. Henkel, J. Hural, Melissa A Brown, 2000, The Journal of Immunology.

Nuclear factor of activated T cells is associated with a mast cell interleukin 4 transcription complex

G. Henkel, M. Brown, L. Timmerman, 1996, Molecular and cellular biology.

PU.1 and GATA: components of a mast cell-specific interleukin 4 intronic enhancer.

G. Henkel, M. Brown, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Targeted disruption of the PU.1 gene results in multiple hematopoietic abnormalities.

A. Feeney, M. Klemsz, S. Mckercher, 1996, The EMBO journal.

Identification of three genes up-regulated in PU.1 rescued monocytic precursor cells.

S. Mckercher, R. Maki, G. Henkel, 2002, International immunology.

PU.1 but not ets-2 is essential for macrophage development from embryonic stem cells.

S. Mckercher, R. Maki, G. Henkel, 1996, Blood.

Defective trophoblast function in mice with a targeted mutation of Ets2.

S. Mckercher, R. Maki, M. Flannery, 1998, Genes & development.

Discovery of Adamantane Carboxamides as Ebola Virus Cell Entry and Glycoprotein Inhibitors.

N. V. Sokolova, G. Henkel, D. Lorimer, 2020, ACS medicinal chemistry letters.

Severe mammarenaviral disease in Guinea pigs effectively treated by an orally bioavailable fusion inhibitor, alone or in combination with favipiravir.

G. Henkel, B. Gowen, Brady T. Hickerson, 2022, Antiviral research.

A DNase I-hypersensitive site in the second intron of the murine IL-4 gene defines a mast cell-specific enhancer.

G. Henkel, M. Brown, D. Weiss, 1992, Journal of immunology.

Regulates IL-4 Gene Locus Accessibility in An Intron Transcriptional Enhancer Element

Melissa A. Brown, G. Henkel, J. Hural, 2000 .

The transcription factor PU.1 does not regulate lineage commitment but has lineage‐specific effects

S. Mckercher, R. Maki, G. Henkel, 1999, Journal of leukocyte biology.