J. Lindner

发表

Cytosolic phosphoenolpyruvate carboxykinase does not solely control the rate of hepatic gluconeogenesis in the intact mouse liver.

M. Magnuson, S. Burgess, T. He, 2007, Cell metabolism.

Mode of suppression of pituitary and gonadal function after acute or prolonged administration of a luteinizing hormone-releasing hormone antagonist in normal men.

J. Lindner, W. Vale, J. Rivier, 1989, The Journal of clinical endocrinology and metabolism.

Characterization of human anti-luteinizing hormone-releasing hormone (LRH) antibodies in the serum of a patient with isolated gonadotropin deficiency treated with synthetic LRH.

S. Marney, J. Lindner, W. Vale, 1981, The Journal of clinical endocrinology and metabolism.

Human islets expressing HNF1A variant have defective &bgr; cell transcriptional regulatory networks

Diane C. Saunders, D. Harlan, S. Levy, 2018, The Journal of clinical investigation.

Energy Homeostasis and Gastrointestinal Endocrine Differentiation Do Not Require the Anorectic Hormone Peptide YY

D. Drucker, Susan E. Schonhoff, A. Leiter, 2005, Molecular and Cellular Biology.

Sulfonylurea Receptor Type 1 Knock-out Mice Have Intact Feeding-stimulated Insulin Secretion despite Marked Impairment in Their Response to Glucose*

J. Gromada, O. Larsson, P. Berggren, 2002, The Journal of Biological Chemistry.

Glucokinase Thermolability and Hepatic Regulatory Protein Binding Are Essential Factors for Predicting the Blood Glucose Phenotype of Missense Mutations*

M. Magnuson, H. Collins, F. Matschinsky, 2007, Journal of Biological Chemistry.

Rictor/mTORC2 Is Essential for Maintaining a Balance Between β-Cell Proliferation and Cell Size

Weiping Yuan, M. Magnuson, J. Lindner, 2011, Diabetes.

Combined administration of a gonadotropin-releasing hormone antagonist and testosterone in men induces reversible azoospermia without loss of libido.

J. Lindner, W. Vale, P. Conn, 1991, The Journal of clinical endocrinology and metabolism.

Efficient DNA cassette exchange in mouse embryonic stem cells by staggered positive‐negative selection

M. Magnuson, J. Lindner, Julie R. Jones, 2004, Genesis.

Epidermal growth factor stimulates secretion of human chorionic gonadotropin by cultured human choriocarcinoma cells.

G. Carpenter, S. Cohen, J. Lindner, 1978, The Journal of clinical endocrinology and metabolism.

Combined administration of a gonadotropin-releasing hormone antagonist and testosterone in men induces reversible azoospermia without loss of libido.

J. Lindner, W. Vale, P. Conn, 1991, Journal of Clinical Endocrinology and Metabolism.

MafA and MafB Regulate Genes Critical to β-Cells in a Unique Temporal Manner

I. Artner, R. Stein, Y. Hang, 2010, Diabetes.

Human Chorionic Gonadotropin α-Subunit from Cultured Choriocarcinoma (JEG) Cells: Comparison of the Subunit Secreted Free with That Prepared from Secreted Human Chorionic Gonadotropin

J. Lindner, D. Puett, D. Rabin, 1979 .

A luteinizing hormone-releasing hormone agonist decreases biological activity and modifies chromatographic behavior of luteinizing hormone in man.

J. Lindner, R. Evans, D. Rabin, 1984, The Journal of clinical investigation.

Regulation of pituitary glycoprotein alpha-subunit secretion after administration of a luteinizing hormone-releasing hormone antagonist in normal men.

J. Lindner, W. Vale, J. Rivier, 1990, The Journal of clinical endocrinology and metabolism.

Persistence of concordant luteinizing hormone (LH), testosterone, and alpha-subunit pulses after LH-releasing hormone antagonist administration in normal men.

J D Veldhuis, J. Lindner, W. Vale, 1990, The Journal of clinical endocrinology and metabolism.

Maintenance of the ratio of bioactive to immunoreactive follicle-stimulating hormone in normal men during chronic luteinizing hormone-releasing hormone agonist administration.

J. Lindner, W. Vale, J. Rivier, 1988, The Journal of clinical endocrinology and metabolism.

Mind bomb 1 is required for pancreatic β-cell formation

M. C. Jørgensen, J. Ahnfelt-Rønne, P. Serup, 2012, Proceedings of the National Academy of Sciences.

α Cell Function and Gene Expression Are Compromised in Type 1 Diabetes

Diane C. Saunders, D. Harlan, S. Levy, 2018, Cell reports.

Dual Roles for Glucokinase in Glucose Homeostasis as Determined by Liver and Pancreatic β Cell-specific Gene Knock-outs Using Cre Recombinase*

M. Magnuson, A. Cherrington, J. Lindner, 1999, The Journal of Biological Chemistry.

alpha Cell Function and Gene Expression Are Compromised in Type 1 Diabetes

Diane C. Saunders, D. Harlan, S. Levy, 2019 .

Targeted oncogenesis of hormone-negative pancreatic islet progenitor cells.

C. Wright, M. Magnuson, J. Lindner, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Deletion of PPARγ in adipose tissues of mice protects against high fat diet-induced obesity and insulin resistance

R. Kesterson, M. Magnuson, B. Kahn, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Multiallelic disruption of the rictor gene in mice reveals that mTOR complex 2 is essential for fetal growth and viability.

J. Woo, M. Magnuson, J. Lindner, 2006, Developmental cell.

Impaired Tricarboxylic Acid Cycle Activity in Mouse Livers Lacking Cytosolic Phosphoenolpyruvate Carboxykinase*

Craig R Malloy, A Dean Sherry, M. Magnuson, 2004, Journal of Biological Chemistry.

Deletion of PPARgamma in adipose tissues of mice protects against high fat diet-induced obesity and insulin resistance.

R. Kesterson, M. Magnuson, B. Kahn, 2005, Proceedings of the National Academy of Sciences of the United States of America.

, Subir the Anorectic Hormone Peptide YY Endocrine Differentiation Do Not Require Energy Homeostasis and Gastrointestinal

D. Drucker, Susan E. Schonhoff, A. Leiter, 2005 .

3-6-2018 α Cell Function and Gene Expression Are Compromised in Type 1 Diabetes

Rita, Diane C. Saunders, D. Harlan, 2018 .

Dissociation of LH and FSH Responses to LHRH during Estrogen Therapy of Patients with Ovarian Failure

J. Lindner, D. Rabin, R. Linde, 1982, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.