J. Cabungcal

发表

Accurate performance of a rat model of schizophrenia in the water maze depends on visual cue availability and stability: A distortion in cognitive mapping abilities?

F. Schenk, D. Preissmann, L. Bertholet, 2011, Behavioural Brain Research.

Geometrical properties of three-dimensional binocular eye position in light sleep

H. Misslisch, B. Hess, K. Hepp, 2002, Vision Research.

Perineuronal nets protect fast-spiking interneurons against oxidative stress

T. Hensch, H. Morishita, R. Kraftsik, 2013, Proceedings of the National Academy of Sciences.

Early-Life Insults Impair Parvalbumin Interneurons via Oxidative Stress: Reversal by N-Acetylcysteine

R. Kraftsik, M. Cuénod, K. Do, 2013, Biological Psychiatry.

Cortical fast-spiking parvalbumin interneurons enwrapped in the perineuronal net express the metallopeptidases Adamts8, Adamts15 and Neprilysin

M. Hawrylycz, Amy Bernard, E. Lein, 2014, Molecular Psychiatry.

Mitochondrial, exosomal miR137-COX6A2 and gamma synchrony as biomarkers of parvalbumin interneurons, psychopathology, and neurocognition in schizophrenia

M. Murray, K. Spencer, M. Cuénod, 2021, Molecular Psychiatry.

Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: A “central hub” in schizophrenia pathophysiology?

M. Cuénod, K. Do, J. Cabungcal, 2016, Schizophrenia Research.

Monkeyed Pontine Burst Neurons in the Ocular Counterroll Modulates the Preferred Direction

E. M. Klier, D. Angelaki, K. Hepp, 2015 .

Caught in vicious circles: a perspective on dynamic feed-forward loops driving oxidative stress in schizophrenia

M. Cuénod, J. Cabungcal, P. Conus, 2021, Molecular Psychiatry.

Glutathione deficit impairs myelin maturation in experimental models : relevance for white matter integrity in schizophrenia patients

J. Thiran, A. Griffa, R. Gruetter, 2022 .

Developmental oxidative stress leads to T-type Ca2+ channel hypofunction in thalamic reticular nucleus of mouse models pertinent to schizophrenia

K. Do, J. Cabungcal, Z. Rovó, 2022, Molecular Psychiatry.

Caught in vicious circles: a perspective on dynamic feed-forward loops driving oxidative stress in schizophrenia; Response to “Adaptive changes to oxidative stress in schizophrenia by Lena Palaniyappan”

M. Cuénod, J. Cabungcal, P. Conus, 2022, Molecular Psychiatry.

Ocular counterroll modulates the preferred direction of saccade-related pontine burst neurons in the monkey.

K Hepp, V Henn, H Scherberger, 2001, Journal of neurophysiology.

Glutathione deficit impairs myelin maturation: relevance for white matter integrity in schizophrenia patients

R Gruetter, P Hagmann, A. Griffa, 2014, Molecular Psychiatry.

Nutritional Intervention for Developmental Brain Damage: Effects of Lactoferrin Supplementation in Hypocaloric Induced Intrauterine Growth Restriction Rat Pups

S. Sizonenko, K. Do, J. Cabungcal, 2019, Front. Endocrinol..

Redox Dysregulation Affects the Ventral But Not Dorsal Hippocampus: Impairment of Parvalbumin Neurons, Gamma Oscillations, and Related Behaviors

R. Kraftsik, M. Cuénod, K. Do, 2010, The Journal of Neuroscience.

Redox dysregulation in the pathophysiology of schizophrenia and bipolar disorder: insights from animal models.

Thomas Werge, A. Ingason, T. Werge, 2013, Antioxidants & redox signaling.

Redox dysregulation, neurodevelopment, and schizophrenia

M. Cuénod, K. Do, J. Cabungcal, 2009, Current Opinion in Neurobiology.

Erratum: Nrf2-dependent persistent oxidative stress results in stress-induced vulnerability to depression

K. Do, J. Cabungcal, J. Molet, 2016, Molecular Psychiatry.

Low protein-induced intrauterine growth restriction as a risk factor for schizophrenia phenotype in a rat model: assessing the role of oxidative stress and neuroinflammation interaction

K. Do, J. Cabungcal, C. Yzydorczyk, 2023, Translational Psychiatry.

The Thalamic Reticular Nucleus in Schizophrenia and Bipolar Disorder: Role of Parvalbumin-Expressing Neuron Networks and Oxidative Stress

T. Salt, S. Berretta, M. Cuénod, 2017, Molecular Psychiatry.

Juvenile Antioxidant Treatment Prevents Adult Deficits in a Developmental Model of Schizophrenia

M. Cuénod, P. O’Donnell, L. Hong, 2014, Neuron.

A developmental redox dysregulation leads to spatio-temporal deficit of parvalbumin neuron circuitry in a schizophrenia mouse model

R. Kraftsik, M. Cuénod, K. Do, 2019, Schizophrenia Research.

MMP9/RAGE pathway overactivation mediates redox dysregulation and neuroinflammation, leading to inhibitory/excitatory imbalance: a reverse translation study in schizophrenia patients

M. Cuénod, L. Tenenbaum, K. Do, 2019, Molecular Psychiatry.

Oxidative stress-driven parvalbumin interneuron impairment as a common mechanism in models of schizophrenia

A. Grace, J. Coyle, T. Hensch, 2017, Molecular Psychiatry.

Glutathione deficit during development induces anomalies in the rat anterior cingulate GABAergic neurons: Relevance to schizophrenia

R. Kraftsik, M. Cuénod, J. Hornung, 2006, Neurobiology of Disease.

Effect of light sleep on three-dimensional eye position in static roll and pitch

H. Scherberger, K. Hepp, H. Misslisch, 2001, Vision Research.

Nrf2-dependent persistent oxidative stress results in stress-induced vulnerability to depression

K. Do, J. Cabungcal, J. Molet, 2017, Molecular Psychiatry.

Timely N-Acetyl-Cysteine and Environmental Enrichment Rescue Oxidative Stress-Induced Parvalbumin Interneuron Impairments via MMP9/RAGE Pathway: A Translational Approach for Early Intervention in Psychosis

M. Cuénod, L. Xin, J. Cabungcal, 2021, Schizophrenia bulletin.

Cellular Neuroscience

M. Cuénod, K. Do, J. Cabungcal, 2022 .

Prolonged Period of Cortical Plasticity upon Redox Dysregulation in Fast-Spiking Interneurons

T. Hensch, H. Morishita, K. Do, 2015, Biological Psychiatry.

Transitory glutathione deficit during brain development induces cognitive impairment in juvenile and adult rats: Relevance to schizophrenia

F. Schenk, M. Cuénod, K. Do, 2007, Neurobiology of Disease.

Glutathione Deficit and Redox Dysregulation in Animal Models of Schizophrenia

F. Schenk, M. Cuénod, K. Do, 2011 .

Thalamic reticular nucleus impairments and abnormal prefrontal control of dopamine system in a developmental model of schizophrenia: prevention by N-acetylcysteine

A. Grace, M. Cuénod, K. Do, 2021, Molecular Psychiatry.

M. Cuénod, J. Cabungcal, P. Conus, 2022, Molecular Psychiatry.

A lack of GluN2A-containing NMDA receptors confers a vulnerability to redox dysregulation: Consequences on parvalbumin interneurons, and their perineuronal nets

K. Do, J. Cabungcal, Romain Cardis, 2018, Neurobiology of Disease.

Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: A “central hub” in schizophrenia pathophysiology?

M. Cuénod, K. Do, J. Cabungcal, 2016, Schizophrenia Research.

Glutathione deficit during development induces anomalies in the rat anterior cingulate GABAergic neurons: Relevance to schizophrenia

R. Kraftsik, M. Cuénod, J. Hornung, 2006, Neurobiology of Disease.

Oxidative Stress Affects Prefrontal-Basal Ganglia-Thalamo-Cortical Circuits Involved in Selected Attention

K. Do, J. Cabungcal, P. Baumann, 2020, Biological Psychiatry.