C. Opazo

发表

A novel functional low-density lipoprotein receptor-related protein 6 gene alternative splice variant is associated with Alzheimer's disease

Eduardo Pérez-Palma, Randall T. Moon, R. Moon, 2013, Neurobiology of Aging.

Intracellular Ca2+ homeostasis in rat round spermatids.

D. Benos, C. Opazo, L. Mercado, 1998, Biology of the cell.

Intracellular Ca2+ homeostasis in rat round spermatids

D. Benos, C. Opazo, L. Mercado, 1998 .

On stage single cell identification of rat spermatogenic cells

D. Benos, C. Opazo, N. Osses, 1997, Biology of the cell.

Canonical Wnt3a Modulates Intracellular Calcium and Enhances Excitatory Neurotransmission in Hippocampal Neurons*

R. Moon, J. Parodi, Miguel E. Avila, 2010, The Journal of Biological Chemistry.

Cysteine 144 Is a Key Residue in the Copper Reduction by the β‐Amyloid Precursor Protein

N. Inestrosa, C. Opazo, F. Ruiz, 1999, Journal of neurochemistry.

Amyloid-beta-peptide reduces copper(II) to copper(I) independent of its aggregation state.

N. Inestrosa, C. Opazo, F. Ruiz, 2000, Biological research.

Metalloenzyme-like activity of Alzheimer's disease beta-amyloid. Cu-dependent catalytic conversion of dopamine, cholesterol, and biological reducing agents to neurotoxic H(2)O(2).

Xudong Huang, Colin L Masters, Roberto Cappai, 2002, The Journal of biological chemistry.

Nitro-Oxidative Stress after Neuronal Ischemia Induces Protein Nitrotyrosination and Cell Death

A. Planas, A. Galán, Carolina Caballo, 2013, Oxidative medicine and cellular longevity.

The role of oxidative stress in the toxicity induced by amyloid β-peptide in Alzheimer’s disease

N. Inestrosa, F. Leighton, C. Opazo, 2000, Progress in Neurobiology.

Biphasic effects of copper on neurotransmission in rat hippocampal neurons

C. Opazo, G. D. De Ferrari, L. Aguayo, 2011, Journal of neurochemistry.

Differential Membrane Toxicity of Amyloid-β Fragments by Pore Forming Mechanisms.

C. Opazo, L. Aguayo, C. Peters, 2016, Journal of Alzheimer's disease : JAD.

Alzheimer's Aβ interacts with cellular prion protein inducing neuronal membrane damage and synaptotoxicity

C. Opazo, L. Aguayo, C. Peters, 2015, Neurobiology of Aging.

Neurodegenerative processes in Alzheimer’s disease: role of A_–AChE complexes and Wnt signaling

N. Inestrosa, C. Opazo, G. Ferrari, 2004 .

Vitamin E But Not 17β-Estradiol Protects against Vascular Toxicity Induced by β-Amyloid Wild Type and the Dutch Amyloid Variant

N. Inestrosa, G. Gil-Gómez, M. Valverde, 2002, The Journal of Neuroscience.

PSD95 Suppresses Dendritic Arbor Development in Mature Hippocampal Neurons by Occluding the Clustering of NR2B-NMDA Receptors

N. Inestrosa, M. Montecino, C. Opazo, 2014, PloS one.

PSD 95 Suppresses Dendritic Arbor Development in Mature Hippocampal Neurons by Occluding the Clustering of NR 2 B-NMDA Receptors

N. Inestrosa, M. Montecino, C. Opazo, 2014 .

Acetylcholinesterase promotes the aggregation of amyloid-beta-peptide fragments by forming a complex with the growing fibrils.

N. Inestrosa, C. Opazo, N C Inestrosa, 1997, Journal of molecular biology.

Proteasome activity and proteasome subunit transcripts in human spermatozoa separated by a discontinuous Percoll gradient

E. S. Díaz, C. Opazo, P. Morales, 2011, Andrologia.

Metalloenzyme-like Activity of Alzheimer's Disease β-Amyloid

C. Masters, Xudong Huang, R. Tanzi, 2002, The Journal of Biological Chemistry.

The Aβcentric Pathway of Alzheimer’s Disease

C. Rowe, C. Masters, R. Cherny, 2007 .

Intracellular amyloid formation in muscle cells of Aβ-transgenic Caenorhabditis elegans: determinants and physiological role in copper detoxification

C. Masters, A. Bush, R. Cherny, 2009, Molecular Neurodegeneration.

Copper reduction by copper binding proteins and its relation to neurodegenerative diseases

N. Inestrosa, C. Opazo, Nibaldo C. Inestrosa, 2003, Biometals.

β-Amyloid Causes Depletion of Synaptic Vesicles Leading to Neurotransmission Failure*

N. Inestrosa, J. Parodi, C. Opazo, 2009, The Journal of Biological Chemistry.

The role of oxidative stress in the toxicity induced by amyloid beta-peptide in Alzheimer's disease.

N. Inestrosa, F. Leighton, C. Opazo, 2000, Progress in neurobiology.

Crosslinking of amyloid-beta peptide to brain acetylcholinesterase.

N. Inestrosa, C. Opazo, N C Inestrosa, 1998, Molecular and chemical neuropathology.

Pore- Forming Neurotoxin-Like Mechanism for A b Oligomer-Induced Synaptic Failure

J. Parodi, C. Opazo, L. Aguayo, 2009 .

Stable Complexes Involving Acetylcholinesterase and Amyloid-β Peptide Change the Biochemical Properties of the Enzyme and Increase the Neurotoxicity of Alzheimer’s Fibrils

B. P. Doctor, N. Inestrosa, C. Opazo, 1998, The Journal of Neuroscience.

Inhibition of amyloid beta-induced synaptotoxicity by a pentapeptide derived from the glycine zipper region of the neurotoxic peptide

C. Opazo, V. Streltsov, L. Aguayo, 2013, Neurobiology of Aging.

Radioiodinated clioquinol as a biomarker for β‐amyloid: Zn2+ complexes in Alzheimer's disease

C. Rowe, C. Masters, A. Bush, 2006, Aging cell.

The N‐terminal copper‐binding domain of the amyloid precursor protein protects against Cu2+ neurotoxicity in vivo

A. Bush, N. Inestrosa, Mauricio González, 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Fibrinogen nitrotyrosination after ischemic stroke impairs thrombolysis and promotes neuronal death.

A. Planas, G. Gil-Gómez, I. López-Vílchez, 2015, Biochimica et biophysica acta.

Nature of the neurotoxic membrane actions of amyloid-β on hippocampal neurons in Alzheimer's disease

Robert W. Peoples, C. Opazo, L. Aguayo, 2014, Neurobiology of Aging.

Synaptotoxicity of Alzheimer Beta Amyloid Can Be Explained by Its Membrane Perforating Property

J. Parodi, C. Opazo, L. Aguayo, 2010, PloS one.

Alzheimer β-amyloid blocks epileptiform activity in hippocampal neurons

C. Opazo, L. Aguayo, F. Sepúlveda, 2009, Molecular and Cellular Neuroscience.

Soluble Aβ(1-40) peptide increases excitatory neurotransmission and induces epileptiform activity in hippocampal neurons.

C. Opazo, L. Aguayo, H. Haensgen, 2011, Journal of Alzheimer's disease : JAD.

Ibuprofen inhibits the synaptic failure induced by the amyloid-β peptide in hippocampal neurons.

C. Opazo, J. Fuentealba, L. Aguayo, 2013, Journal of Alzheimer's disease : JAD.

Vitamin E but not 17beta-estradiol protects against vascular toxicity induced by beta-amyloid wild type and the Dutch amyloid variant.

N. Inestrosa, G. Gil-Gómez, M. Valverde, 2002, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Inhibition of amyloid beta-induced synaptotoxicity by a pentapeptide derived from the glycine zipper region of the neurotoxic peptide

C. Opazo, V. Streltsov, L. Aguayo, 2013, Neurobiology of Aging.

Ibuprofen inhibits the synaptic failure induced by the amyloid-β peptide in hippocampal neurons.

C. Opazo, J. Fuentealba, L. Aguayo, 2013, Journal of Alzheimer's disease : JAD.

Transition metals and Alzheimer's disease

C. Opazo, 2005 .

Soluble Aβ(1-40) peptide increases excitatory neurotransmission and induces epileptiform activity in hippocampal neurons.

C. Opazo, L. Aguayo, H. Haensgen, 2011, Journal of Alzheimer's disease : JAD.

An extracellular mechanism that can explain the neurotoxic effects of α-synuclein aggregates in the brain

C. Opazo, L. Aguayo, C. Opazo, 2012, Front. Physio..