Jie Shen

发表

Differential modulation of short-term plasticity at hippocampal mossy fiber and Schaffer collateral synapses by mitochondrial Ca2+

M. Frotscher, S. H. Lee, D. Lutz, 2020, PloS one.

Nigrostriatal Dopaminergic Deficits and Hypokinesia Caused by Inactivation of the Familial Parkinsonism-Linked Gene DJ-1

P. Calabresi, G. Bernardi, A. Pisani, 2005, Neuron.

Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress

Jie Shen, T. Kitada, C. Gautier, 2008, Proceedings of the National Academy of Sciences.

Presenilins are Essential for Regulating Neurotransmitter Release

T. Südhof, Jie Shen, Dawei Zhang, 2009, Nature.

α-Synuclein is phosphorylated in synucleinopathy lesions

E. Masliah, T. Iwatsubo, M. Hasegawa, 2002, Nature Cell Biology.

PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin

Atsushi Tanaka, Mark R. Cookson, M. Cookson, 2010, PLoS biology.

PS Dependent APP Cleavage Regulates Glucosylceramide Synthase and is Affected in Alzheimer's Disease

H. Tanila, Jie Shen, T. Hartmann, 2014, Cellular Physiology and Biochemistry.

Loss of leucine-rich repeat kinase 2 causes impairment of protein degradation pathways, accumulation of α-synuclein, and apoptotic cell death in aged mice

R. J. Kelleher, Raphael Kopan, E. Giaime, 2010, Proceedings of the National Academy of Sciences.

Presenilins in synaptic function and disease.

Jie Shen, A. Ho, 2011, Trends in molecular medicine.

Levodopa-induced dyskinesia and striatal signaling pathways

A. Pisani, Jie Shen, 2009, Proceedings of the National Academy of Sciences.

Presenilin-dependent transcriptional control of the Abeta-degrading enzyme neprilysin by intracellular domains of betaAPP and APLP.

F. Checler, C. A. D. Alves da Costa, C. Sunyach, 2005, Neuron.

Characterization of the reconstituted gamma-secretase complex from Sf9 cells co-expressing presenilin 1, nicastrin [correction of nacastrin], aph-1a, and pen-2.

Jie Shen, E. Parker, Julie Lee, 2005, Biochemistry.

alpha-Synuclein is phosphorylated in synucleinopathy lesions.

E. Masliah, T. Iwatsubo, M. Hasegawa, 2002, Nature cell biology.

Allele‐specific genetic interactions between Mitf and Kit affect melanocyte development

J. Qu, Jie Shen, L. Hou, 2010, Pigment cell & melanoma research.

Loss of leucine-rich repeat kinase 2 causes age-dependent bi-phasic alterations of the autophagy pathway

H. Cai, E. Giaime, J. Bonventre, 2012, Molecular Neurodegeneration.

Genetic analysis of Parkinson's disease-linked leucine-rich repeat kinase 2.

Jie Shen, Youren Tong, 2012, Biochemical Society transactions.

gamma-secretase functions through Notch signaling to maintain skin appendages but is not required for their patterning or initial morphogenesis.

Raphael Kopan, Jie Shen, T. Gridley, 2004, Developmental cell.

Brain region specific mitophagy capacity could contribute to selective neuronal vulnerability in Parkinson's disease

Jie Shen, J. Klose, T. Kitada, 2011, Proteome Science.

DJ-1 associates with synaptic membranes

N. Hattori, T. Hatano, H. Ariga, 2011, Neurobiology of Disease.

Restricted growth and insulin-like growth factor-1 deficiency in mice lacking presenilin-1 in the neural crest cell lineage

Jie Shen, S. Okuyama, M. Nakajima, 2009, International Journal of Developmental Neuroscience.

Loss of DJ-1 Does Not Affect Mitochondrial Respiration but Increases ROS Production and Mitochondrial Permeability Transition Pore Opening

E. Giaime, Jie Shen, T. Kitada, 2012, PloS one.

Parkin Protects against Mitochondrial Toxins and β-Amyloid Accumulation in Skeletal Muscle Cells*

Kenneth M. Rosen, Matthew S. Goldberg, C. Moussa, 2006, Journal of Biological Chemistry.

Inactivation of Pink1 Gene in Vivo Sensitizes Dopamine-producing Neurons to 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) and Can Be Rescued by Autosomal Recessive Parkinson Disease Genes, Parkin or DJ-1

David S. Park, H. Anisman, R. Slack, 2012, The Journal of Biological Chemistry.

Age-Dependent Dopaminergic Neurodegeneration and Impairment of the Autophagy-Lysosomal Pathway in LRRK-Deficient Mice

Guodong Huang, E. Giaime, Jie Shen, 2017, Neuron.

The Roles of Kinases in Familial Parkinson's Disease

M. Cookson, T. Dawson, Jie Shen, 2007, The Journal of Neuroscience.

The protein kinase TOUSLED is required for maintenance of transcriptional gene silencing in Arabidopsis

Jian-Kang Zhu, Jie Shen, Xuhui Hong, 2007, EMBO reports.

PINK 1 Is Selectively Stabilized on Impaired Mitochondria to Activate

M. Cookson, R. Youle, Jie Shen, 2010 .

Reduced beta-amyloid production and increased inflammatory responses in presenilin conditional knock-out mice.

Richard D. Kim, C. Lemere, Jie Shen, 2004, The Journal of biological chemistry.

Hydrocephalus and abnormal subcommissural organ in mice lacking presenilin-1 in Wnt1 cell lineages

K. Matsuda, Jie Shen, S. Okuyama, 2011, Brain Research.

A nasal proteosome adjuvant activates microglia and prevents amyloid deposition

H. Weiner, D. Frenkel, Adi Dembinsky-Vaknin, 2008, Annals of neurology.

A chemical genetic approach to probe the function of PINK1 in regulating mitochondrial dynamics

Xuedong Liu, Jie Shen, E. Giaime, 2014, Cell Research.

Role of presenilin-1 in cortical lamination and survival of Cajal-Retzius neurons.

Jie Shen, Mary Wines-Samuelson, M. Handler, 2005, Developmental biology.

Characterization of Two Distinct Lymphoproliferative Diseases Caused by Ectopic Expression of the Notch Ligand DLL4 on T Cells

Jie Shen, C. Tadokoro, J. Lafaille, 2013, PloS one.

Mitochondrial membrane potential decrease caused by loss of PINK1 is not due to proton leak, but to respiratory chain defects

Alexander M. Wolf, N. Hattori, M. Toyomizu, 2011, Neurobiology of Disease.

Notch1 and 2 cooperate in limb ectoderm to receive an early Jagged2 signal regulating interdigital apoptosis.

Zhenyi Liu, Raphael Kopan, Jie Shen, 2005, Developmental biology.

Presenilins are required for maintenance of neural stem cells in the developing brain

Jie Shen, W. Kim, 2008, Molecular Neurodegeneration.

Dominant negative mechanism of Presenilin-1 mutations in FAD

Jie Shen, Hirotaka Watanabe, 2017, Proceedings of the National Academy of Sciences.

Effect of presenilins in the apoptosis of thymocytes and homeostasis of CD8+ T cells.

M. Serrano, M. Badura, Jie Shen, 2007, Blood.

Protocols for assessing neurodegenerative phenotypes in Alzheimer’s mouse models

Jie Shen, Jongkyun Kang, Hirotaka Watanabe, 2021, STAR protocols.

Partial Loss of Presenilin Impairs Age-Dependent Neuronal Survival in the Cerebral Cortex

Jie Shen, Mary Wines-Samuelson, Hirotaka Watanabe, 2014, The Journal of Neuroscience.

Function and Dysfunction of Presenilin

Jie Shen, 2013, Neurodegenerative Diseases.

Mitochondrial Dysfunction and Oxidative Damage in parkin-deficient Mice*

Joachim Klose, Stefan Krauss, Matthew S. Goldberg, 2004, Journal of Biological Chemistry.

Potentiation in Hippocampal CA1 Pyramidal Neurons Caffeine-Mediated Presynaptic Long-Term

Eduardo D. Martín, M. White, D. Burks, 2015 .

Presenilins regulate synaptic plasticity in the perforant pathways of the hippocampus

V. Bolshakov, S. H. Lee, Jie Shen, 2023, Molecular Brain.

Inactivation of Presenilin in inhibitory neurons results in decreased GABAergic responses and enhanced synaptic plasticity

V. Bolshakov, S. H. Lee, Jie Shen, 2021, Molecular brain.

Cell-autonomous role of Presenilin in age-dependent survival of cortical interneurons

Jie Shen, Jongkyun Kang, 2020, Molecular neurodegeneration.

Presenilins regulate synaptic plasticity and mitochondrial calcium homeostasis in the hippocampal mossy fiber pathway

M. Frotscher, V. Bolshakov, Mohanad Mossalam, 2017, Molecular Neurodegeneration.

Presenilin-1 Knockin Mice Reveal Loss-of-Function Mechanism for Familial Alzheimer’s Disease

R. J. Kelleher, E. Tsvetkov, V. Bolshakov, 2015, Neuron.

Presenilins regulate calcium homeostasis and presynaptic function via ryanodine receptors in hippocampal neurons

Jie Shen, F. A. Lai, Bei Wu, 2013, Proceedings of the National Academy of Sciences.

Inactivation of presenilins causes pre-synaptic impairment prior to post-synaptic dysfunction

T. Südhof, A. Kirkwood, Jie Shen, 2010, Journal of neurochemistry.

Characterization of Age-Dependent and Progressive Cortical Neuronal Degeneration in Presenilin Conditional Mutant Mice

Xinran Liu, R. J. Kelleher, C. Aoki, 2010, PloS one.

Indirect Regulation of Presenilins in CREB-mediated Transcription*

R. J. Kelleher, Jie Shen, V. Beglopoulos, 2009, Journal of Biological Chemistry.

Loss of Presenilin Function Causes Impairments of Memory and Synaptic Plasticity Followed by Age-Dependent Neurodegeneration

E. Kandel, A. Kirkwood, R. J. Kelleher, 2004, Neuron.

APP Processing and Synaptic Plasticity in Presenilin-1 Conditional Knockout Mice

E. Kandel, M. Wilson, Linus D. Sun, 2001, Neuron.

Skeletal and CNS Defects in Presenilin-1-Deficient Mice

S. Tonegawa, D. Selkoe, R. Bronson, 1997, Cell.

Synaptic function of nicastrin in hippocampal neurons

T. Südhof, S. H. Lee, Jie Shen, 2014, Proceedings of the National Academy of Sciences.

PTEN-inducible kinase 1 (PINK1)/Park6 is indispensable for normal heart function

T. Mak, V. Rao, Ludger Hauck, 2011, Proceedings of the National Academy of Sciences.

Interaction between Reelin and Notch Signaling Regulates Neuronal Migration in the Cerebral Cortex

P. Rakic, N. Šestan, Jie Shen, 2008, Neuron.

Impaired dopamine release and synaptic plasticity in the striatum of PINK1-deficient mice

Douglas R. Porter, A. Pisani, P. Bonsi, 2007, Proceedings of the National Academy of Sciences.

Transcriptional repression of p53 by parkin and impairment by mutations associated with autosomal recessive juvenile Parkinson's disease

Patrick M. Abou-Sleiman, Nicholas W. Wood, Andrew West, 2009, Nature Cell Biology.

Notch activation induces apoptosis in neural progenitor cells through a p53-dependent pathway.

R. Klein, Raphael Kopan, Jie Shen, 2004, Developmental biology.

Conditional Deletion of Notch1 and Notch2 Genes in Excitatory Neurons of Postnatal Forebrain Does Not Cause Neurodegeneration or Reduction of Notch mRNAs and Proteins*

Raphael Kopan, Jie Shen, Mary Wines-Samuelson, 2012, The Journal of Biological Chemistry.

Notch1 Signaling Influences V2 Interneuron and Motor Neuron Development in the Spinal Cord

Raphael Kopan, M. Tansey, Jie Shen, 2006, Developmental Neuroscience.

Presenilins in the Developing, Adult, and Aging Cerebral Cortex

Jie Shen, Mary Wines-Samuelson, Jie Shen, 2005, The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry.

Role of Presenilin‐1 in Murine Neural Development

Jie Shen, M. Handler, Xudong Yang, 2000, Annals of the New York Academy of Sciences.

A regulatory network involving Foxn4, Mash1 and delta-like 4/Notch1 generates V2a and V2b spinal interneurons from a common progenitor pool

D. Rowitch, W. Richardson, Jie Shen, 2007, Development.

Altered Ca2+ homeostasis in the skeletal muscle of DJ−1 null mice

Jie Shen, José R Lopez, A. Shtifman, 2011, Neurobiology of Aging.

Familial Frontotemporal Dementia-Associated Presenilin-1 c.548G>T Mutation Causes Decreased mRNA Expression and Reduced Presenilin Function in Knock-In Mice

R. J. Kelleher, D. Xia, Jie Shen, 2012, The Journal of Neuroscience.

Impaired Neurotransmitter Release in Alzheimer’s and Parkinson’s Diseases

Jie Shen, Jie Shen, 2010, Neurodegenerative Diseases.

Defective signal transduction in B lymphocytes lacking presenilin proteins

R. Xavier, A. Shaw, S. Mohanty, 2008, Proceedings of the National Academy of Sciences.

Regulation of CRE-dependent transcription by presenilins: prospects for therapy of Alzheimer's disease.

Jie Shen, V. Beglopoulos, 2006, Trends in pharmacological sciences.

Conditional Inactivation of Presenilin 1 Prevents Amyloid Accumulation and Temporarily Rescues Contextual and Spatial Working Memory Impairments in Amyloid Precursor Protein Transgenic Mice

R. Morris, A. Kirkwood, Jie Shen, 2005, The Journal of Neuroscience.

Adult hippocampal neurogenesis occurs in the absence of Presenilin 1 and Presenilin 2

Jie Shen, J. Béïque, Yingben Xue, 2018, Scientific Reports.

An Evolutionarily Conserved Role of Presenilin in Neuronal Protection in the Aging Drosophila Brain

N. Perrimon, Jie Shen, Jongkyun Kang, 2017, Genetics.

Loss of Aβ43 Production Caused by Presenilin-1 Mutations in the Knockin Mouse Brain

R. J. Kelleher, D. Xia, Jie Shen, 2016, Neuron.

Presenilins are Essential for Regulating Neurotransmitter Release

T. Südhof, Jie Shen, Dawei Zhang, 2009, Nature.

Conditional Forebrain Inactivation of Nicastrin Causes Progressive Memory Impairment and Age-Related Neurodegeneration

T. Südhof, K. Tabuchi, Jie Shen, 2009, The Journal of Neuroscience.

The presenilin hypothesis of Alzheimer's disease: Evidence for a loss-of-function pathogenic mechanism

R. J. Kelleher, Jie Shen, 2007, Proceedings of the National Academy of Sciences.

Gene-targeting technologies for the study of neurological disorders

Jie Shen, V. Beglopoulos, Vassilios Beglopoulos, 2007, NeuroMolecular Medicine.

Hippocampal Spatial Memory Impairments Caused by the Familial Alzheimer’s Disease-Linked Presenilin 1 M146V Mutation

M. Mattson, Jie Shen, V. Beglopoulos, 2005, Neurodegenerative Diseases.

Presenilin-Dependent Transcriptional Control of the Aβ-Degrading Enzyme Neprilysin by Intracellular Domains of βAPP and APLP

F. Checler, C. Sunyach, C. A. D. Costa, 2005, Neuron.

Reduced β-Amyloid Production and Increased Inflammatory Responses in Presenilin Conditional Knock-out Mice*

Richard D. Kim, C. Lemere, Jie Shen, 2004, Journal of Biological Chemistry.

Presenilin-1 Function in the Adult Brain

Jie Shen, 2002 .

Presenilin-Mediated Modulation of Capacitative Calcium Entry

M. Frosch, R. Tanzi, A. Yoo, 2000, Neuron.

Heterogeneous populations of ES cells in the generation of a floxed Presenilin‐1 allele

Jie Shen, Huakui Yu, J. Kessler, 2000, Genesis.

Analysis of Local Chromatin States Reveals Gene Transcription Potential during Mouse Neural Progenitor Cell Differentiation.

Haihe Ruan, Guohong Li, Jie Shen, 2020, Cell reports.

Analysis of Local Chromatin States Reveals Gene Transcription Potential during Mouse Neural Progenitor Cell Differentiation.

Haihe Ruan, Guohong Li, Jie Shen, 2020, Cell reports.

Notch controls embryonic Schwann cell differentiation, postnatal myelination and adult plasticity

F. Guillemot, R. Mirsky, L. Wrabetz, 2009, Nature Neuroscience.

Parkin Regulates Mitosis and Genomic Stability through Cdc20/Cdh1.

Liewei Wang, J. Jen, Y. Pang, 2015, Molecular cell.

Parkin Regulates Mitosis a nd Genomic Stability through Cdc 20 / Cdh 1 Graphical Abstract Highlights

Boqin, Liewei Wang, J. Jen, 2015 .

LRRK2 regulates synaptic vesicle endocytosis.

N. Hattori, T. Hatano, W. Seol, 2008, Experimental cell research.

Mitochondria and Dopamine New Insights into Recessive Parkinsonism

M. Cookson, Jie Shen, 2004, Neuron.

APP Family Regulates Neuronal Excitability and Synaptic Plasticity but Not Neuronal Survival

V. Bolshakov, S. H. Lee, Jie Shen, 2020, Neuron.

PINK1 heterozygous mutations induce subtle alterations in dopamine‐dependent synaptic plasticity

A. Pisani, N. Mercuri, P. Bonsi, 2014, Movement disorders : official journal of the Movement Disorder Society.

Therapeutic effect of γ-secretase inhibition in KrasG12V-driven non-small cell lung carcinoma by derepression of DUSP1 and inhibition of ERK.

Manuel Serrano, Teresa Palomero, A. Ferrando, 2012, Cancer cell.

Transcriptional Activation of Low-Density Lipoprotein Receptor Gene by DJ-1 and Effect of DJ-1 on Cholesterol Homeostasis

T. Niki, K. Takahashi-Niki, S. Iguchi-Ariga, 2012, PloS one.

CREB Binding Protein Is Required for Both Short-Term and Long-Term Memory Formation

J. V. van Deursen, Jie Shen, Hirotaka Watanabe, 2010, The Journal of Neuroscience.

R1441C mutation in LRRK2 impairs dopaminergic neurotransmission in mice

A. Pisani, E. Pothos, Jie Shen, 2009, Proceedings of the National Academy of Sciences.

PINK1 Triggers Autocatalytic Activation of Parkin to Specify Cell Fate Decisions

Xuedong Liu, Xuedong Liu, E. Giaime, 2014, Current Biology.

ROS-dependent regulation of Parkin and DJ-1 localization during oxidative stress in neurons.

David S. Park, T. Mak, R. Slack, 2012, Human molecular genetics.

14-3-3 binding to LRRK2 is disrupted by multiple Parkinson's disease-associated mutations and regulates cytoplasmic localization

A. Prescott, R. Nichols, D. Alessi, 2010, The Biochemical journal.

Postnatal Deletion of Numb/Numblike Reveals Repair and Remodeling Capacity in the Subventricular Neurogenic Niche

Y. Jan, L. Jan, J. García-Verdugo, 2006, Cell.

Antisilencing role of the RNA-directed DNA methylation pathway and a histone acetyltransferase in Arabidopsis

Jie Shen, Weiqiang Qian, Z. Gong, 2012, Proceedings of the National Academy of Sciences.

Amyloid-Independent Mechanisms in Alzheimer's Disease Pathogenesis

Li-Huei Tsai, Ralph A. Nixon, L. Tsai, 2010, The Journal of Neuroscience.

Regulation of gene expression by the APP family in the adult cerebral cortex

H. Cha, Jie Shen, Jongkyun Kang, 2022, Scientific reports.

Presenilin-1 mutations and Alzheimer’s disease

R. J. Kelleher, Jie Shen, 2017, Proceedings of the National Academy of Sciences.

Regulation of cholesterol and sphingomyelin metabolism by amyloid-β and presenilin

B. Strooper, M. Duering, Jie Shen, 2005, Nature Cell Biology.

Trans-Dominant Negative Effects of Pathogenic PSEN1 Mutations on γ-Secretase Activity and Aβ Production

R. J. Kelleher, Jie Shen, T. Tai, 2013, The Journal of Neuroscience.

Parkin-deficient Mice Exhibit Nigrostriatal Deficits but Not Loss of Dopaminergic Neurons*

Bryan L Roth, Matthew S Goldberg, Carlos Cepeda, 2003, Journal of Biological Chemistry.

Effect of retinoid X receptor-α nuclear export inhibition on apoptosis of neurons in vivo and in vitro.

YingChun Liu, Jie Shen, Min Wang, 2017, Molecular medicine reports.

A Cdc20-APC Ubiquitin Signaling Pathway Regulates Presynaptic Differentiation

A. Bonni, A. Kim, Jie Shen, 2009, Science.

Reply to Gaiano et al.: Expression of Notch Proteins in Pyramidal Neurons in Vivo

Raphael Kopan, Jie Shen, Mary Wines-Samuelson, 2012, The Journal of Biological Chemistry.

A Presenilin-1 Mutation Identified in Familial Alzheimer Disease with Cotton Wool Plaques Causes a Nearly Complete Loss of γ-Secretase Activity*

R. J. Kelleher, Jie Shen, W. Xia, 2010, The Journal of Biological Chemistry.

PINK1 and Parkin complementarily protect dopaminergic neurons in vertebrates.

R. Takahashi, Y. Taniguchi, Jie Shen, 2013, Human molecular genetics.

Potential therapeutic role of fibroblast growth factor 21 in neurodegeneration: Evidence for ameliorating parkinsonism via silent information regulator 2 homolog 1 and implication for gene therapy

Jie Shen, Xiangdong Gao, W. Yao, 2020, Neuropharmacology.

[Ginsenoside Rbl attenuates beta-amyloid peptide25-35 -induced tau hyperphosphorylation in cortical neurons].

Jie Shen, Tianwen Huang, Li-min Chen, 2005, Yao xue xue bao = Acta pharmaceutica Sinica.

Regulation of mitochondrial permeability transition pore by PINK1

E. Giaime, Jie Shen, D. Chan, 2012, Molecular Neurodegeneration.

Impaired dopamine release and synaptic plasticity in the striatum of Parkin−/− mice

A. Pisani, E. Pothos, P. Platania, 2009, Journal of neurochemistry.

α-Synuclein occurs in lipid-rich high molecular weight complexes, binds fatty acids, and shows homology to the fatty acid-binding proteins

Rebecca A. Betensky, Matthew S. Goldberg, D. Selkoe, 2001, Proceedings of the National Academy of Sciences of the United States of America.

Absence of nigral degeneration in aged parkin/DJ‐1/PINK1 triple knockout mice

Jie Shen, T. Kitada, Youren Tong, 2009, Journal of neurochemistry.

Molecular Neurodegeneration Increased Dj-1 Expression under Oxidative Stress and in Alzheimer's Disease Brains

C. Lemere, M. Goldberg, Jie Shen, 2022 .

Histological analysis of neurodegeneration in the mouse brain.

Jie Shen, H. Yamaguchi, 2013, Methods in molecular biology.

CRTC1 mediates preferential transcription at neuronal activity-regulated CRE/TATA promoters

Jie Shen, A. Parra-Damas, C. Saura, 2017, Scientific Reports.

Olfactory ensheathing cells and neuropathic pain

Jie Shen, Fei-long Zou, Wen-Jun Zhang, 2023, Frontiers in Cell and Developmental Biology.

α-Synuclein and LRRK2: Partners in Crime

Jie Shen, Youren Tong, 2009, Neuron.

Absence of dopaminergic neuronal degeneration and oxidative damage in aged DJ-1-deficient mice

Jie Shen, H. Yamaguchi, 2007, Molecular Neurodegeneration.

Increased levels of NMDA receptor NR2A subunits at pre‐ and postsynaptic sites of the hippocampal CA1: An early response to conditional double knockout of presenilin 1 and 2

C. Aoki, Hermina Nedelescu, Jie Shen, 2009, The Journal of comparative neurology.

Adult hippocampal neurogenesis occurs in the absence of Presenilin 1 and Presenilin 2

Jie Shen, J. Béïque, Yingben Xue, 2018, Scientific Reports.

Protein Kinases Linked to the Pathogenesis of Parkinson's Disease

Jie Shen, 2004, Neuron.

Increased DJ-1 expression under oxidative stress and in Alzheimer's disease brains

C. Lemere, Jie Shen, W. Xia, 2009, Molecular Neurodegeneration.

Enhanced sensitivity to group II mGlu receptor activation at corticostriatal synapses in mice lacking the familial parkinsonism-linked genes PINK1 or Parkin

A. Pisani, P. Bonsi, P. Platania, 2009, Experimental Neurology.

[JNK/p38 MAPK involves in ginsenoside Rb1 attenuating beta-amyloid peptide (25-35) -induced tau protein hyperphosphorylation in embryo rat cortical neurons].

Jie Shen, Jing Zhang, Tianwen Huang, 2008, Yao xue xue bao = Acta pharmaceutica Sinica.